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Community‐level interventions for improving access to food in low‐ and middle‐income countries

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Background

After decades of decline since 2005, the global prevalence of undernourishment reverted and since 2015 has increased to levels seen in 2010 to 2011. The prevalence is highest in low‐ and middle‐income countries (LMICs), especially Africa and Asia. Food insecurity and associated undernutrition detrimentally affect health and socioeconomic development in the short and long term, for individuals, including children, and societies. Physical and economic access to food is crucial to ensure food security. Community‐level interventions could be important to increase access to food in LMICs.

Objectives

To determine the effects of community‐level interventions that aim to improve access to nutritious food in LMICs, for both the whole community and for disadvantaged or at‐risk individuals or groups within a community, such as infants, children and women; elderly, poor or unemployed people; or minority groups.

Search methods

We searched for relevant studies in 16 electronic databases, including trial registries, from 1980 to September 2019, and updated the searches in six key databases in February 2020. We applied no language or publication status limits.

Selection criteria

We included randomised controlled trials (RCTs), cluster randomised controlled trials (cRCTs) and prospective controlled studies (PCS). All population groups, adults and children, living in communities in LMICs exposed to community‐level interventions aiming to improve food access were eligible for inclusion. We excluded studies that only included participants with specific diseases or conditions (e.g. severely malnourished children).

Eligible interventions were broadly categorised into those that improved buying power (e.g. create income‐generation opportunities, cash transfer schemes); addressed food prices (e.g. vouchers and subsidies); addressed infrastructure and transport that affected physical access to food outlets; addressed the social environment and provided social support (e.g. social support from family, neighbours or government).

Data collection and analysis

Two authors independently screened titles and abstracts, and full texts of potentially eligible records, against the inclusion criteria. Disagreements were resolved through discussion or arbitration by a third author, if necessary.

For each included study, two authors independently extracted data and a third author arbitrated disagreements. However, the outcome data were extracted by one author and checked by a biostatistician.

We assessed risk of bias for all studies using the Effective Practice and Organization of Care (EPOC) risk of bias tool for studies with a separate control group.

We conducted meta‐analyses if there was a minimum of two studies for interventions within the same category, reporting the same outcome measure and these were sufficiently homogeneous. Where we were able to meta‐analyse, we used the random‐effects model to incorporate any existing heterogeneity. Where we were unable to conduct meta‐analyses, we synthesised using vote counting based on effect direction.

Main results

We included 59 studies, including 214 to 169,485 participants, and 300 to 124, 644 households, mostly from Africa and Latin America, addressing the following six intervention types (three studies assessed two different types of interventions).

Interventions that improved buying power:

Unconditional cash transfers (UCTs) (16 cRCTs, two RCTs, three PCSs): we found high‐certainty evidence that UCTs improve food security and make little or no difference to cognitive function and development and low‐certainty evidence that UCTs may increase dietary diversity and may reduce stunting. The evidence was very uncertain about the effects of UCTs on the proportion of household expenditure on food, and on wasting. Regarding adverse outcomes, evidence from one trial indicates that UCTs reduce the proportion of infants who are overweight.

Conditional cash transfers (CCTs) (nine cRCTs, five PCSs): we found high‐certainty evidence that CCTs result in little to no difference in the proportion of household expenditure on food and that they slightly improve cognitive function in children; moderate‐certainty evidence that CCTs probably slightly improve dietary diversity and low‐certainty evidence that they may make little to no difference to stunting or wasting. Evidence on adverse outcomes (two PCSs) shows that CCTs make no difference to the proportion of overweight children.

Income generation interventions (six cRCTs, 11 PCSs): we found moderate‐certainty evidence that income generation interventions probably make little or no difference to stunting or wasting; and low‐certainty evidence that they may result in little to no difference to food security or that they may improve dietary diversity in children, but not for households.

Interventions that addressed food prices:

Food vouchers (three cRCTs, one RCT): we found moderate‐certainty evidence that food vouchers probably reduce stunting; and low‐certainty evidence that that they may improve dietary diversity slightly, and may result in little to no difference in wasting.

Food and nutrition subsidies (one cRCT, three PCSs): we found low‐certainty evidence that food and nutrition subsidies may improve dietary diversity among school children. The evidence is very uncertain about the effects on household expenditure on healthy foods as a proportion of total expenditure on food (very low‐certainty evidence).

Interventions that addressed the social environment:

Social support interventions (one cRCT, one PCS): we found moderate‐certainty evidence that community grants probably make little or no difference to wasting; low‐certainty evidence that they may make little or no difference to stunting. The evidence is very uncertain about the effects of village savings and loans on food security and dietary diversity.

None of the included studies addressed the intervention category of infrastructure changes. In addition, none of the studies reported on one of the primary outcomes of this review, namely prevalence of undernourishment.

Authors' conclusions

The body of evidence indicates that UCTs can improve food security. Income generation interventions do not seem to make a difference for food security, but the evidence is unclear for the other interventions. CCTs, UCTs, interventions that help generate income, interventions that help minimise impact of food prices through food vouchers and subsidies can potentially improve dietary diversity. UCTs and food vouchers may have a potential impact on reducing stunting, but CCTs, income generation interventions or social environment interventions do not seem to make a difference on wasting or stunting. CCTs seem to positively impact cognitive function and development, but not UCTs, which may be due to school attendance, healthcare visits and other conditionalities associated with CCTs.

PICOs

Population
Intervention
Comparison
Outcome

The PICO model is widely used and taught in evidence-based health care as a strategy for formulating questions and search strategies and for characterizing clinical studies or meta-analyses. PICO stands for four different potential components of a clinical question: Patient, Population or Problem; Intervention; Comparison; Outcome.

See more on using PICO in the Cochrane Handbook.

Improving access to food in low‐ and middle‐income countries

Review question

We looked at the effect of community‐level interventions to improve access to nutritious food in low‐ and middle‐income countries (LMICs) on people, households and communities.

Background

Food security exists when people have physical, social and economic access to sufficient, safe, nutritious foods to be healthy. The number of people who do not have enough to eat in the world has started increasing since 2015. Most of these people live in LMICs, especially in Asia and Africa. Not being able to access nutritious food, either because of not having enough money or because of not having somewhere to shop or find food near where people live, affects the health and socioeconomic situation of people and societies, both in the short and long term. Strategies focusing on communities may be important for increasing access to food in LMICs.

Study characteristics

We found 59 studies assessing different interventions in LMICs, including 214 to 169,485 participants and 300 to 124,644 households, mainly in Africa and Latin America. Many studies assessed cash transfers, which are welfare programmes where money is provided to households. Of these, 21 studies evaluated unconditional cash transfers, where there are no conditions for receiving the money, and 14 studies assessed conditional cash transfers, where there are specific conditions required to meet in order to receive the money. Seventeen studies looked at income generation interventions (for example, livestock management or self‐help groups), four studies at food vouchers, four studies at providing food and nutrition subsidies, and two studies looked at social support interventions such as village savings and loans and community grant programmes.

Search date

The evidence is current to February 2020.

Key results

Interventions that improved buying power:

Unconditional cash transfers improve food security and make little or no difference to cognitive function (thoughts and understanding) and development (high‐quality evidence), may increase dietary diversity (variety of the foods that people or households eat from different food groups) and reduce stunting (poor growth) (low‐quality evidence). It is very uncertain whether UCTs reduce the proportion of household expenditure on food and reduce wasting. Regarding adverse outcomes, evidence from one trial indicates that UCTs reduce the proportion of infants who are overweight.

Conditional cash transfers make little to no difference in the proportion of household expenditure on food and slightly improve cognitive function in children (high‐quality evidence), probably slightly improve dietary diversity (moderate‐quality evidence), and may make little to no difference to stunting or wasting (low bodyweight) (low‐quality evidence). Evidence on adverse outcomes (two studies) shows that CCTs make no difference to the proportion of overweight children.

Income generation strategies make little or no difference to stunting or wasting (moderate‐quality evidence), may result in little to no difference to food security and may improve dietary diversity in children but not for households (low‐quality evidence).

Interventions that addressed food prices:

Food vouchers probably reduce stunting (moderate‐quality evidence), may slightly improve dietary diversity and may result in little to no difference in wasting (low‐quality evidence).

Food and nutrition subsidies may improve dietary diversity among school children (low‐quality evidence). We are very uncertain about the effects on household expenditure on healthy foods as a proportion of total expenditure on food (very low‐quality evidence).

Interventions that addressed the social environment:

Social support interventions such as community grants probably make little to no difference to wasting (moderate‐quality evidence) and may make little or no difference to stunting (low‐quality evidence). We are very uncertain about the effects of village savings and loans on food security or dietary diversity (very low‐quality evidence).

None of the included studies addressed the intervention category of infrastructure changes and none of the included studies reported on one of the primary outcomes: prevalence of undernourishment.

Some limitations of the review include not having all necessary information about what was measured (outcomes), judgements that had to be made regarding which outcome measures to report and inability to pool the results of all studies reporting on the same outcome. Another limitation was that we were unable to find out what specific intervention features enable or impede the effective implementation of the intervention.

Authors' conclusions

Implications for practice

This review provides policy makers with a comprehensive evidence base, ranging from randomised controlled trials (RCTs) to prospective controlled studies, evaluating the effects of a wide range of community‐level interventions to address access to food in low‐ and middle‐income countries (LMICs).

The body of evidence indicates that unconditional cash transfers (UCTs) can improve food security, income‐generation interventions do not seem to make a difference for food security, but the evidence is unclear for the other interventions. Conditional cash transfers (CCTs), UCTs, those that help generate income, and those that help minimise impact of food prices through food vouchers and subsidies can potentially improve dietary diversity. UCTs and food vouchers may have a potential impact on reducing stunting, but CCTs, income‐generation interventions or social environment interventions do not seem to make a difference on wasting or stunting. CCTs seem to positively impact cognitive function and development but not UCTs. This may be due to the fact that in CCTs, beneficiaries are required to meet specific conditionalities such as attending school, visiting the health clinic regularly for growth monitoring or supplementation. None of the included studies reported on the primary outcome prevalence of undernourishment; in retrospect this was expected given that this is mostly used as a national‐level indicator of food security.

We found no studies reporting specific adverse outcomes. Three studies, one assessing UCTs and two assessing CCTs, reported on increased risk of overweight and obesity, our predefined adverse outcome. The effects on this outcome are unclear and we should thus not discard overweight and obesity as potential harms of these interventions. This is particularly problematic because in LMIC populations, where these interventions are implemented, overweight and obesity are often already a problem. Hawkes 2020 reported that this was the case in cash or food transfers and voucher interventions in Mexico, Egypt and the US where the unintended negative outcomes of these programmes include poorer diet quality and obesity and diabetes‐related NCDs, due to increased intake of foods high in energy, sugar, fat and salt, resulting from the programme itself, or from the income from the programme that enabled people to purchase these types of foods. In some cases, the targeted populations experienced both undernutrition and overnutrition, such as the existence of undernourished children and obese mothers in the same household or community. Thus it is possible that these programmes may exacerbate existing problems. Potential solutions to prevent this could be related to specifying which types of foods can be purchased with interventions such as vouchers, and accompanying health education with transfers.

As these interventions are often implemented at national level, direct implications for practitioners and the community are less clear. Organisations involved in the development and implementation of interventions to improve access to food may be able to better focus their time and resources by optimally designing or choosing programmes which maximise the intended outcome.

Implications for research

Here, we draw on the EPICOT framework – which stands for Evidence, Population, Intervention, Comparison, Outcomes, and Time stamp (Brown 2006) – to suggest gaps in the evidence base that future research could address. There is enough evidence from RCTs for CCTs and UCTs but not for the other intervention types assessed in this review (i.e. those addressing food prices (e.g. policies, discounts, vouchers and subsidies); addressing infrastructure and transport that affect physical access to food outlets; and those addressing the social environment and providing social support (e.g. social support from family, neighbours or government)).

In general, higher‐quality RCTs and prospective controlled studies are required, particularly concerning methods to minimise the issues with selection and attrition bias.

The multiplicity of outcome measures made analysis for this review challenging. It is thus important for future studies to have a similar set of outcomes that we can usefully compare across studies, and that are most relevant for assessing food and nutrition security at the community or household level. If primary studies measured similar outcomes, it would have likely been possible to pool the majority of results across included studies for this review, resulting in clearer review findings.

None of the included studies clearly and specifically reported on adverse events. Although these types of interventions do not tend to have the same extent of harms as a clinical intervention, for example, it is still important to consider what the potential harms may be. Although overweight or obesity is a potential harm, the studies did not report on this as such.

Most studies reported results from one to two years of the intervention. Longer‐term studies of interventions aimed to improve food access in vulnerable communities or households are required.

Summary of findings

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Summary of findings 1. Unconditional cash transfers compared to no intervention for food security

Unconditional cash transfers compared to no intervention for food security

Patient or population: children, adults, households
Setting: poor rural and urban households in LMICs
Intervention: UCTs
Comparison: no intervention

Outcomes

Impact

№ of participants
(studies)

Certainty of the evidence
(GRADE)

Comments

Prevalence of undernourishment

0 included studies measured this outcome.

Proportion of household expenditure on food
follow‐up: range 1–2 years

1 study showed a clear effect favouring UCTs, 2 studies showed unclear effect potentially favouring UCTs and 2 studies showed clear effect favouring the control. Data not pooled.

11271 households
(5 RCTs)

⊕⊝⊝⊝
Very lowa,b,c

Evidence is very uncertain about the effects of UCTs on the proportion of household expenditure on food.

Food security
assessed with: proportion of households consuming > 1 meal per day; modified HFIAS; FSI
follow‐up: range 1–2 years

6 studies showed a clear effect favouring UCTs.

A meta‐analysis of 3 of these studies showed a small improvement in food security scores (SMD 0.18, 95% CI 0.13 to 0.23; 6209 households)

10,251 households, 7604 children (6 RCTs)

⊕⊕⊕⊕
High

UCTs improve food security.

Dietary diversity
assessed with: dietary diversity scores (i.e. number of food groups consumed); proportion with minimum dietary diversity
follow‐up: range 1–2 years

5 studies showed a clear effect favouring UCTs and 5 studies show an unclear effect potentially favouring UCTs.

Data not pooled.

12,631 households, 890 children (10 RCTs)

⊕⊕⊝⊝
Lowa,b

UCTs may increase dietary diversity.

Stunting
assessed with: HAZ < –2SD
follow‐up: 2 years

1 study showed a clear effect favouring UCTs, 2 studies showed an unclear effect favouring UCTs and 1 study showed an unclear effect favouring the control.

A meta‐analysis of 2 of these studies showed a reduction in stunting with UCTs (OR 0.62, 95% CI 0.46 to 0.84; 2914 children)

4713 children
(4 RCTs)

⊕⊕⊝⊝
Lowa,b

UCTs may reduce stunting.

Wasting
assessed with: WHZ < –2SD
follow‐up: range 2 years

1 study showed an unclear effect potentially favouring UCTs and 3 studies showed an unclear effect potentially favouring the control. Data not pooled.

6396 children
(4 RCTs)

⊕⊝⊝⊝
Very lowa,b,c

We are uncertain whether UCTs reduce wasting.

Cognitive function and development
assessed with: cognitive test scores, language scores
follow‐up: 2 years

3 studies reported unclear effect potentially favouring intervention.

10,813 children

(3 RCTs)

⊕⊕⊕⊕
High

UCTs make little or no difference on cognitive function and development.

*No meta‐analyses carried out.

CI: confidence interval; FSI: Food Security Index; HAZ: height‐for‐age z‐score; HFIAS: Household Food Insecurity Access Scale; LMIC: low‐ and middle‐income country; OR: odds ratio; RCT: randomised controlled trial; SD: standard deviation; SMD: standardised mean difference; UCT: unconditional cash transfer; WHZ: weight‐for‐height z‐score.

GRADE Working Group grades of evidence
High certainty: we are very confident that the true effect lies close to that of the estimate of the effect.
Moderate certainty: we are moderately confident in the effect estimate: the true effect is likely to be close to the estimate of the effect, but there is a possibility that it is substantially different.
Low certainty: our confidence in the effect estimate is limited: the true effect may be substantially different from the estimate of the effect.
Very low certainty: we have very little confidence in the effect estimate: the true effect is likely to be substantially different from the estimate of effect.

aDowngraded one level for risk of bias: at least one study was at high overall risk of bias due to selection or attrition bias, or both.
bDowngraded one level for inconsistency: there was wide variance of point estimates.
cDowngraded one level for imprecision: wide confidence intervals.

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Summary of findings 2. Conditional cash transfers compared to no intervention for food security

Conditional cash transfers compared to no intervention for food security

Patient or population: children, adults, households
Setting: poor urban and rural communities in LMICs
Intervention: CCTs
Comparison: no intervention

Outcomes

Impact

№ of participants
(studies)

Certainty of the evidence
(GRADE)

Comments

Prevalence of undernourishment

0 included studies measured this outcome.

Proportion of household expenditure spent on food
follow‐up: 9 months to 2 years

1 study showed a clear effect potentially favouring the control and 1 study showed an unclear effect favouring the control. Data not pooled.

4760 households
(2 RCTs)

⊕⊕⊕⊕
High

CCTs result in little to no difference in the proportion of household expenditure on food.

Food security

0 included studies measured this outcome.

Dietary diversity
assessed with: Food Consumption Score
follow‐up: 7 months to 2.5 years

Meta‐analysis of 2 studies showed a clear effect favouring CCTs (MD 0.45, 95% CI 0.25 to 0.65)

3937 households
(2 RCTs)

⊕⊕⊕⊝
Moderatea

CCTs probably slightly improve dietary diversity

Stunting
assessed with: HAZ < –2SD
follow‐up: range 20 months to 3 years

3 studies showed an unclear effect potentially favouring CCTs and 1 study showed an unclear effect potentially favouring the control.

A meta‐analysis of 3 of these studies showed an unclear effect favouring CCTs (MD –2.51, 95% CI –7.78, 2.75)

3529 children
(4 RCTs)

⊕⊕⊝⊝
Lowa,b

CCTs may make little or no difference to the proportion of stunted children.

Wasting
assessed with: WHZ < –2SD
follow‐up: 2 years

A meta‐analysis of 2 studies showed an unclear effect favouring CCTs (MD –2.50 95% CI –8.04 to 3.04)

2116 children
(2 RCTs)

⊕⊕⊝⊝
Lowb,c

CCTs may make little or no difference in wasting.

Cognitive function and development
assessed with: cognitive test scores; cognitive and socioemotional outcomes scores
follow‐up: range 9 months to 2 years

A meta‐analysis of 2 studies showed a slight improvement with CCTs (SMD 0.13, 95% CI 0.09 to 0.18)

5383 children
(2 RCTs)

⊕⊕⊕⊕
High

CCTs slightly improve cognitive function in children.

*No meta‐analyses carried out.

CCT: conditional cash transfer; CI: confidence interval; HAZ: height‐for‐age z‐score; MD: mean difference; RCT: randomised controlled trial; SD: standard deviation; SMD: standardised mean difference; WHZ: weight‐for‐height z‐score.

GRADE Working Group grades of evidence
High certainty: we are very confident that the true effect lies close to that of the estimate of the effect.
Moderate certainty: we are moderately confident in the effect estimate: the true effect is likely to be close to the estimate of the effect, but there is a possibility that it is substantially different.
Low certainty: our confidence in the effect estimate is limited: the true effect may be substantially different from the estimate of the effect.
Very low certainty: we have very little confidence in the effect estimate: the true effect is likely to be substantially different from the estimate of effect.

aDowngraded one level for risk of bias: at least one study was at high overall risk of bias due to selection or attrition bias, or both.
bDowngraded one level imprecision: wide confidence intervals.
cDowngraded one level for inconsistency: wide variation in point estimates.

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Summary of findings 3. Income‐generation interventions compared to no intervention for food security

Income‐generation interventions compared to no intervention for food security

Patient or population: children, adults, households
Setting: poor rural communities in LMICs
Intervention: income‐generation interventions (e.g. livestock transfers, community development programmes)
Comparison: no intervention

Outcomes

Impact

№ of participants
(studies)

Certainty of the evidence
(GRADE)

Comments

Prevalence of undernourishment

0 included studies reported this outcome.

Proportion of household expenditure on food
follow‐up: range 1–2 years

2 studies reported this outcome but did not provide relevant numerical data or indicated clearly the direction of effect.

434 households (2 prospective controlled studies)

Food security
assessed with: proportion experiencing food security; Household food security score
follow‐up: 3–4 months

1 trial reported no effect measure and 1 trial showed an unclear effect potentially favouring the control.

2193 households (1 trial)

⊕⊕⊝⊝
Lowa,b

Income‐generation interventions may result in little to no difference in food security.

Dietary diversity
assessed with: DDS, HDDS, MDD
follow‐up: 2 years

2 trials showed a clear effect favouring income‐generation interventions, 1 trial showed an unclear effect favouring the intervention and 1 trial showed an unclear effect favouring control.

A meta‐analysis of 3 of these studies showed that the intervention improved the proportion of children achieving MDD (OR 1.28, 95% CI 1.11 to 1.47)

3677 households and 3790 children (4 RCTs)

⊕⊕⊝⊝
Lowa,c

Income‐generation interventions may improve dietary diversity in children and may result in little or no difference to household dietary diversity.

Stunting
assessed with: HAZ
follow‐up: 12 months

Meta‐analysis of 2 studies showed no difference to stunting (OR 1.00, 95% CI 0.84 to 1.19)

3466 children (2 RCTs)

⊕⊕⊕⊝
Moderated

Income‐generation interventions probably make little or no difference to stunting.

Wasting
assessed with: WHZ
follow‐up: 2 years

Meta‐analysis of 2 studies showed unclear effect favouring the intervention (OR 1.13, 95% CI 0.92 to 1.40)

3500 children (2 trials)

⊕⊕⊕⊝
Moderated

Income‐generation interventions probably make little or no difference to wasting.

Cognitive function and development

0 included studies reported this outcome.

CI: confidence interval; DDS: Dietary Diversity Score; HAZ: height‐for‐age z‐score; HDDS: Household Dietary Diversity Score; MDD: minimum dietary diversity; OR: odds ratio; RCT: randomised controlled trial; WHZ: weight‐for‐height z‐score.

GRADE Working Group grades of evidence
High certainty: we are very confident that the true effect lies close to that of the estimate of the effect.
Moderate certainty: we are moderately confident in the effect estimate: the true effect is likely to be close to the estimate of the effect, but there is a possibility that it is substantially different.
Low certainty: our confidence in the effect estimate is limited: the true effect may be substantially different from the estimate of the effect.
Very low certainty: we have very little confidence in the effect estimate: the true effect is likely to be substantially different from the estimate of effect.

aDowngraded one level for risk of bias: at least one study was at high overall risk of bias due to selection or attrition bias, or both.
bDowngraded one level for indirectness: results are from a single study which assessed a public works programme and the effects may be different from other types of income generation interventions. Additionally public works programmes are often implemented in different ways in different settings.
cDowngraded one level for inconsistency: wide variation in point estimates.
dDowngraded one level for imprecision: wide confidence intervals.

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Summary of findings 4. Food vouchers compared to no intervention for food security

Food vouchers compared to no intervention for food security

Patient or population: poor households
Setting: urban and agrarian communities in LMICs
Intervention: food vouchers
Comparison: no intervention

Outcomes

Impact

№ of participants
(studies)

Certainty of the evidence
(GRADE)

Comments

Prevalence of undernourishment

0 included studies reported this outcome.

Proportion of household expenditure on food

0 included studies reported this outcome.

Food security

0 included studies reported this outcome.

Dietary diversity
assessed with: FCS
follow‐up: 7 months to 1 year

2 studies reported improved dietary diversity (not pooled).

2459 households (2 RCT)

⊕⊕⊝⊝
Lowa,b

Food vouchers may improved dietary diversity slightly.

Stunting (HAZ < –2SD)

follow‐up: 12 months

1 study reported reduced stunting (OR 0.48, 95% CI 0.31 to 0.73)

1633 children (1 RCT)

⊕⊕⊕⊝

Moderatec

Food vouchers probably reduce stunting.

Wasting (WHZ < –2SD)

follow‐up: 12 months

1 study reports an unclear effect potentially favouring the control (OR 1.17, 95% CI 0.75, 1.82)

1633 children (1 RCT)

⊕⊕⊝⊝

Lowc,d

Food vouchers may result in little to no difference in wasting

Cognitive function and development

0 included studies reported this outcome.

CI: confidence interval; FCS: Food Consumption Score; HAZ: height‐for‐age z‐score; OR: odds ratio; RCT: randomised controlled trial; SD: standard deviation; WHZ: weight‐for‐height z‐score.

GRADE Working Group grades of evidence
High certainty: we are very confident that the true effect lies close to that of the estimate of the effect.
Moderate certainty: we are moderately confident in the effect estimate: the true effect is likely to be close to the estimate of the effect, but there is a possibility that it is substantially different.
Low certainty: our confidence in the effect estimate is limited: the true effect may be substantially different from the estimate of the effect.
Very low certainty: we have very little confidence in the effect estimate: the true effect is likely to be substantially different from the estimate of effect.

aDowngraded one level for overall risk of bias: two studies at high risk of selection and attrition bias.
bDowngraded one level for inconsistency: confidence intervals had minimal overlap.
cDowngraded one level for indirectness: findings are from one single study that assessed a programme of fresh food vouchers redeemed at designated vendors. Food vouchers may be implemented in different ways across different settings, e.g. for staple foods alone, or with, no vendor‐ restrictions.
dDowngraded one level for imprecision: findings ranged from an important harm to important benefit.

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Summary of findings 5. Food and nutrition subsidies compared to no intervention for food security

Food and nutrition subsidies compared to no intervention for food security

Patient or population: primary schools and households and members of healthcare plan
Setting: urban and rural settings in LMICs
Intervention: food and nutrition subsidies
Comparison: no intervention

Outcomes

Impact

№ of participants
(studies)

Certainty of the evidence
(GRADE)

Comments

Prevalence of undernourishment

0 included studies reported this outcome.

Proportion of household expenditure on food
assessed with: ratio of healthy to total food expenditure
follow‐up: 28 months

1 study reported that food rebates of 10% improved the ratio of healthy, to total food expenditure

169,485 households (1 prospective controlled study)

⊕⊝⊝⊝
Very lowa,b

The evidence is very uncertain about the effects of food rebates on household expenditure on healthy foods.

Food security

0 included studies reported this outcome.

Dietary diversity

1 study reported a clear effect favouring nutrition subsidies.

656 children (1 RCT)

⊕⊕⊝⊝

Lowc ,d

Nutrition subsidies may improve dietary diversity among school children

Stunting

0 included studies reported this outcome.

Wasting

0 included studies reported this outcome.

Cognitive function and development

0 included studies reported this outcome.

LMIC: low‐ and middle‐income country; RCT: randomised controlled trial.

GRADE Working Group grades of evidence
High certainty: we are very confident that the true effect lies close to that of the estimate of the effect.
Moderate certainty: we are moderately confident in the effect estimate: the true effect is likely to be close to the estimate of the effect, but there is a possibility that it is substantially different.
Low certainty: our confidence in the effect estimate is limited: the true effect may be substantially different from the estimate of the effect.
Very low certainty: we have very little confidence in the effect estimate: the true effect is likely to be substantially different from the estimate of effect.

aDowngraded one level for risk of bias: high risk of selection bias due to disparate baseline expenditure on healthy food as a ratio of total expenditure between households in the intervention and control group.
bDowngraded one level for indirectness: results are from a single study that assessed food rebates at a supermarket in South Africa. The population was restricted to members of the health insurance company's program, who are usually healthier and wealthier in general. Effects in other populations may differ.

cDowngraded one level for indirectness: results are from a single study that assessed the effects of providing nutrition subsidies to schools. Subsidies to individuals or households may have different effects.
dDowngraded one level for risk of bias: study was at high overall risk of bias due to attrition bias.

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Summary of findings 6. Social support compared to no intervention for food security

Social support compared to no intervention for food security

Patient or population: households at risk of food insecurity
Setting: poor communities in LMICs
Intervention: village savings and loans groups and community cash transfers
Comparison: no intervention

Outcomes

Impact

№ of participants
(studies)

Certainty of the evidence
(GRADE)

Comments

Prevalence of undernourishment

0 included studies reported this outcome.

Proportion of household expenditure on food

0 included studies reported this outcome.

Food security
assessed with: self‐reported months of food sufficiency
follow‐up: 3 years

1 study reported an unclear effect favouring village savings and loans

1687 households (1 prospective controlled study)

⊕⊝⊝⊝
Very lowa

The evidence is very uncertain about the effects of village savings and loan on food security.

Dietary diversity
assessed with: HDDS
follow‐up: 3 years

1 study showed an unclear effect favouring the control.

1615 households (1 prospective controlled study)

⊕⊝⊝⊝
Very lowa

The evidence is very uncertain about the effects of village savings and loan on dietary diversity.

Stunting

assessed with: HAZ < –2SD

follow‐up: 2 years

1 study showed an unclear effect favouring the control.

1481 children (1 RCT)

⊕⊕⊝⊝

Lowb ,c

Community grants may make little or no difference to stunting.

Wasting

assessed with: WHZ < –2SD

follow‐up: 2 years

1 study showed an unclear effect favouring a community grant programme.

1481 children (1 RCT)

⊕⊕⊕⊝

Moderateb

Community grants probably make little or no difference to wasting.

Cognitive function and development

0 included studies reported this outcome.

*No meta‐analyses carried out.
CI: confidence interval; HAZ: height‐for‐age z‐score; HDDS: Household Dietary Diversity Score; LMIC: low‐ and middle‐income country; RCT: randomised controlled trial; SD: standard deviation; WHZ: weight‐for‐height z‐score.

GRADE Working Group grades of evidence
High certainty: we are very confident that the true effect lies close to that of the estimate of the effect.
Moderate certainty: we are moderately confident in the effect estimate: the true effect is likely to be close to the estimate of the effect, but there is a possibility that it is substantially different.
Low certainty: our confidence in the effect estimate is limited: the true effect may be substantially different from the estimate of the effect.
Very low certainty: we have very little confidence in the effect estimate: the true effect is likely to be substantially different from the estimate of effect.

aDowngraded one level for indirectness: results from a single study which assessed the effects of microfinance program to villages in Mozambique. Effects of other types of social support interventions may be different. As this was a prospective controlled study the certainty of evidence started at low.
bDowngraded one level for indirectness: results are from a single study which assessed the effects of a community cash transfer programme implemented in rural villages in Indonesia. Village management teams allocated funds to different types of social support interventions, Effects in urban populations and with different intervention implementation may differ.

cDowngraded one level for imprecision: wide confidence interval.

Background

Description of the condition

Food security "exists when all people, at all times, have physical, social and economic access to sufficient, safe and nutritious food that meets their dietary needs and food preferences for an active and healthy life" (FAO 2019). When these conditions are not met, the population and people within it are said to be food insecure.

Food insecurity and associated undernutrition affect health and socioeconomic development on different levels (Black 2013; Ecker 2012; Victora 2008). For adults, it has been associated with an increased risk of disability, morbidity and mortality, and with a decrease in income‐generation potential (Black 2008; Black 2013; Victora 2008). Food insecurity is also associated with mental health problems such as depression and anxiety, both in high‐income as well as low‐ and middle‐income settings (Arenas 2019; Carter 2011; Cole 2011; Hadley 2006; Hadley 2008; Maynard 2018). Children who are affected may experience impaired physical and cognitive development, and decreased school performance (Black 2008; Black 2013; Liu 2012; Victora 2008). At the macro‐level, undernutrition is associated with direct and indirect costs. Direct costs are due to increased healthcare costs for preventing and treating affected individuals (Black 2013; Victora 2008). Indirect costs are due to poor productivity and losses of human resources due to mental and physical under‐performance and death (Victora 2008). Given these far‐reaching consequences, and considering that food security is defined as a human right by the United Nations (FAO 2003), it is important to address food insecurity.

Building on the first 2010 to 2015 Millennium Development Goal (MDG), which was to eradicate extreme poverty and hunger, the second 2015 Sustainable Development Goal (SDG) aimed to end hunger by 2030 and sought to "achieve food security and improve nutrition and promote sustainable agriculture" (UN 2015). Progress towards this goal has been insufficient. Following decades of decline, the global prevalence of undernourishment, has, since 2015, increased to levels seen in 2010–2011, approximately 11% (FAO 2019). Although this prevalence is highest in Asia, it has been sharply increasing in Africa which is now home to 30% of the world's undernourished population (FAO 2019). The global number of undernourished people, estimated at 820 million in 2018, has been steadily rising particularly in Africa, Latin America and Western Asia. Globally, the prevalence and number of stunted children under five years of age has decreased since 2012, although this is uneven as Africa and Asia account for more than 90% of stunted and wasted children globally (FAO 2019; SOWC 2019). Factors that have delayed improvements in rates of chronic hunger include the food price crisis of 2008, brought about by trade restrictions of major food exporters, biofuels policies and increased commodity speculation, among others (Ecker 2012). The higher demand for food due to changing dietary patterns and growing population, and food price increases and volatility due to climate change are other factors that will contribute to food insecurity in the long term (Ecker 2012).

Food security is a complex concept that encompasses several different dimensions (Ecker 2012; FAO 2013; FAO 2019; Gross 2000), where 1. food availability refers to the quantity of food that is physically available in the relevant vicinity of a population during a given period (ACF‐IN 2008); 2. food access is a measure of the capacity of a household to acquire sufficient and appropriate foods to ensure a diet that is diverse, nutrient‐rich and safe, and that satisfies the nutrient needs of its members during a given period, which is often influenced by the proximity and price of food (ACF‐IN 2008; WHO 2013); 3. food utilisation refers to the intake of food by the people within a household and how the body assimilates the nutrients physiologically; and 4. food stability introduces the condition of time to the food security concept, that is it refers to chronic or transient food insecurity (FAO 2003). Chronic food insecurity refers to long‐term, persistent lack of food and results from continued problems with structural poverty, relating to the inability of the labour market to produce enough jobs to keep people out of poverty, low incomes and with lack of sufficient social safety nets to assist the poor (Ecker 2012; FAO 2003; Rank 2003). In contrast, transient food insecurity refers to food and nutrient shortages during certain periods of food crises due to natural disasters, economic collapse or conflict (Ecker 2012; FAO 2003). In addition, the nutrition dimension was added to the food security concept at the 2009 World Food Summit (Ecker 2012) as food insecurity is associated with nutrient deficiencies and poor nutritional outcomes. Furthermore, food and nutrient intake interact in a bidirectional manner with health status (Ecker 2012). This means that nutritional status is the primary measure of food security.

The four dimensions of food security operate at different levels of influence, although these are often inter‐related (Ecker 2012; Gross 2000). At the macro‐level (national, regional, global) and meso‐level (community), food security issues are mainly related to food availability and stability, whereas at the micro‐level they are mainly related to food access and utilisation by households and individuals (Ecker 2012; Gross 2000; Pinstrup‐Andersen 2009). Food security in one level does not ensure food security at another level (Gross 2000). For example, food might be available at the national level but not accessible for certain disadvantaged communities or districts, or among lower income or otherwise marginalised population groups. In Ghana, despite improvements in reducing poverty and increasing food production, there has been less progress in reducing undernutrition and disparities remain (FAO 2013; Hjelm 2013). There, poorer households and those headed by women tend to be more food insecure due to their low‐diversity diets compared with the wealthier or male‐headed households (FAO 2013; Hjelm 2013). In Nepal, there is still widespread undernutrition despite the country producing sufficient food, and those living in rural areas are at a higher risk of food insecurity and have a higher prevalence of undernutrition and stunting in children as poor infrastructures and poverty limit their physical and economic access to food (FAO 2013; MOHP 2012). Furthermore, households might have access to food, but this does not guarantee that all individuals in the household are able to access and utilise sufficient amounts of good quality and safe food. This is because the distribution of food within the household may be influenced by cultural beliefs, practices, attitudes, gender and age‐specific roles and responsibilities, as well as decision‐making hierarchies (Gittelsohn 2003; Pinstrup‐Andersen 2009; Renzaho 2010).

In addition to the burden of undernutrition, low‐ and middle‐income countries (LMICs) also have high rates of overweight and obesity that are on the rise (Hossain 2007; Popkin 2012; Subramanian 2011). In an analysis of data from 54 LMICs, 27% of women were overweight (Subramanian 2011). The prevalence of overweight in 2008 ranged from approximately 18% in low‐income countries to 59% in upper middle‐income countries, with a mean prevalence of 28% in the African region (WHO 2010). Among children under five years of age, the prevalence of overweight and obesity is also increasing, with 12.9% of boys and 13.4% of girls overweight in LMICs in 2013 (Ng 2014). Most of this burden is concentrated in Africa and Asia, regions that accounted for almost three‐quarters of the global share of overweight children in 2018 (FAO 2019; SOWC 2019). The increased rates of overweight and obesity are associated with the nutrition transition and poorer‐quality diets increasingly consisting of more affordable processed foods, high intake of refined sugars and fats, and increased intake of food away from home, further exacerbated by decreased levels of physical activity (Popkin 2012; SOWC 2019). In LMICs, the consumption of processed or junk foods and sugar‐sweetened beverages has increased, with 54% of the global consumption of soft drinks 1997 and 2010 occurring in LMICs (Basu 2013). These dietary patterns are partly the result of high food prices, which cause consumers, particularly those in poorer households, to buy less‐expensive foods. These are often energy dense (higher in calories) and less nutritious (containing fewer nutrients per serving size). Consumption of these foods is, therefore, associated with increased risk of overweight, obesity and micronutrient deficiencies. In this context, it is important to consider the quantity and quality of the food intake in any intervention.

Description of the intervention

Scoping review: preparation for this systematic review

The complexity of food security allows for a wide range of interventions addressing its different dimensions at varying levels of influence. In order to better conceptualise the framework for our review with regards to the type(s) of intervention(s) to assess, the eligibility criteria for study selection and the outcomes to be assessed, we conducted a scoping review of existing systematic reviews of interventions addressing food security in LMICs (more information about the methods is available on request).

We included 29 systematic reviews in the scoping review (references available on request). Fourteen reviews addressed food availability, mainly assessing food production interventions and food utilisation (13 reviews, including five which also addressed availability), specifically around issues of nutrition education for people to improve their dietary intake. Seven reviews addressed food access. The scoping review also revealed that the included reviews were unclear regarding the description of participants and settings, types of interventions and comparisons, or the outcomes they would assess (Table 1). The quality of reviews varied considerably, some with very low‐quality scores using the AMSTAR tool (Shea 2009).

Open in table viewer
Table 1. Summary of PICOS and of AMSTAR scores of included systematic reviews, and how existing reviews informed the PICOS of a new Cochrane Review

Domain

Finding

How it informed our review question or methods

Setting

  • 12 reviews did not specify the setting

  • 11 reviews stated the community as the setting

  • 3 reviews stated the setting was LMICs

  • 3 reviews specified a school as the setting

We chose the community as the setting, defined as a group of people with diverse characteristics who were linked by social ties, share common perspectives and engage in joint action in geographical locations or settings (MacQueen 2001).

Participants

  • 5 reviews did not specify the types of participants for inclusion

  • 11 reviews included infants and children (up to school‐aged children)

  • 1 review included adults and adolescents

  • 6 reviews included pregnant women or mothers in the immediate postpartum period. 1 of these also targeted other adults who could be linked to women who may have breastfed. Many of these were assessing interventions on breastfeeding or complementary feeding.

  • 1 review included only parents of children aged 2–5 years, as it assessed influence of parenting practices on children's dietary habits

  • 2 reviews included all people living in a community

  • 3 reviews included only poor people who were recipients of some service, e.g. recipients of a government conditional cash‐transfer programme

As existing reviews specifically addressed specific high‐risk groups, we did not focus on these. Instead, we included all individuals across all ages that belonged to the community where relevant interventions had been implemented.

Intervention (including its duration)

  • 14 reviews addressed interventions related to the availability of food, 5 of which also assessed interventions influencing utilisation of food, such as nutrition education

  • 13 reviews assessed interventions addressing food utilisation

  • 7 reviews assessed interventions addressing access to food (2 of which had a low AMSTAR score of 4)

  • 28 reviews did not specify the duration of the intervention, and only 1 included interventions with a minimum duration of 3 months. As a result, the duration and the follow‐up times of the interventions varied considerably within and across reviews

Of the 14 reviews that addressed food availability, 5 also assessed food utilisation (e.g. combination of community gardens and nutrition education). As fewer reviews addressed food access, we included interventions that had addressed this dimension of food security.

We included interventions with any duration but extracted outcomes that were measured ≥ 3 months after implementation.

Control

  • 18 reviews did not specify a control group

  • 6 reviews compared the intervention with either no intervention, an alternative intervention or placebo

  • 3 reviews did not have any control group

  • 2 reviews stated that included studies needed to have a control group, but did not specify further

We included studies in which these interventions, individually or in combination, were compared to no intervention or to other eligible intervention.

Outcomes assessed

The specific outcomes assessed across the included reviews varied considerably and often they were not clearly specified at the outset.

The most common and important outcomes reported in these reviews were related to dietary intake, anthropometric measurements, and biochemical and clinical indicators, to describe the impact of the intervention on nutritional status. Other outcomes measured included food purchase or expenditure, food production, morbidity and mortality, and breastfeeding initiation rates or duration.

Often, reviews measured the same outcome in different ways. For example, anthropometric indicators assessed differed, as did their classifications, across the included reviews. This makes it difficult to compare results across reviews and to reach a conclusion about the effectiveness of a specific intervention.

The most commonly specified outcomes measured food and nutrition security, and nutritional status. We also focused on these outcomes. Examples included: diet diversity scores and hunger measures; and anthropometric, biochemical and dietary intake indicators. We clearly defined, a priori, the specific outcome measures and metrics that we included in our review.

Study designs

  • 11 reviews did not specify which study designs they would include

  • 3 reviews included only RCTs

  • 1 review included only CCTs

  • 1 review included only impact evaluations

  • 13 reviews included a variety of study designs, which included ≥ 2 of: RCTs, BAS, quasi‐RCTs, analytical cohort studies, ITS, CCTs, randomised field trials and CSS

However, the definitions of the study design labels used were not always clear and varied across the included reviews.

The study design labels used varied across included reviews and were not always clearly defined.

We included both randomised and non‐randomised studies, as we expect that existing RCTs in the area of food security would be scarce. We wanted to include the best available evidence for our review question. We clearly defined the type of study designs included in our review.

Search strategies

Most reviews ran comprehensive searches. They used a comprehensive set of keywords and searched a variety of relevant databases. Only 5 reviews did not indicate search terms either in the text or in an appendix.

  • 2 reviews conducted searches until 2012

  • 11 reviews searched until 2010–2011

  • 9 reviews searched before 2010

  • 7 reviews did not specify the date of the last search

Our review included updated searches across a variety of relevant databases and websites. We drew on common keywords used across these included reviews.

Reporting

The methods sections of most reviews were often not reported clearly. The reporting of results in these reviews, in terms of characteristics of included studies, was also poor.

Poor reporting of the characteristics of included studies makes it difficult to assess the context in which these results were obtained. Thus, it is difficult to generalise the results.

We clearly reported on the characteristics of included studies, so that the context in which the interventions were implemented was clearly understood.

AMSTAR scores

  • 9 reviews were of low quality (AMSTAR score: 0–4)

  • 11 reviews were of moderate quality (AMSTAR score: 5–8)

  • 8 reviews were of high quality (AMSTAR score: 9–11)

  • 1 review did not have a score as it did not include any studies

Of the 8 high‐quality reviews, 5 assessed interventions that aimed to improve food availability or utilisation (or both), and 3 assessed interventions addressing food access. The other 2 included reviews that addressed food access were of low quality (AMSTAR = 4).

We contributed to the evidence base on interventions addressing food access by producing a high‐quality systematic review that assessed the effectiveness of the interventions on relevant outcomes, such as nutritional status.

BAS: before‐and‐after study; CCT: controlled clinical trial; CSS: cross‐sectional study; ITS: interrupted time series; LMIC: low‐ and middle‐income country; RCT: randomised controlled trial.

Interventions selected based on scoping review results

Based on the findings of the scoping review, we decided to focus this Cochrane Review on community‐level interventions that aim to improve access to nutritious food in LMICs; as we found that there are fewer reviews addressing food access compared to food availability or utilisation. Furthermore, we know that in many areas of LMICs, nutritious food is available at a national level, but physical distance and financial constraints prevent thousands of people from accessing the food (FAO 2013). As explained above, increased intake of ultraprocessed food products and sugar‐sweetened beverages has contributed to the rise in overweight and obesity in LMICs and poor diet quality is also responsible for micronutrient deficiencies. Thus, interventions should aim to improve access to nutritious food. Nutritious foods can be defined as those that are nutrient dense, that is providing substantial amounts of vitamins and minerals (Pennington 2007). This includes fresh or minimally processed foods from the different food groups, such as whole grains, lean meats, dairy products, legumes, vegetables and fruits, and excludes ultraprocessed products and sugar‐sweetened beverages that provide empty calories (Drewnowski 2005; Ministry of Health of Brazil 2014).

The interventions addressing food access include those aimed at infrastructure and transport, food prices, the social environment, coping strategies and buying power. In our scoping review, we found no systematic reviews addressing infrastructure and transport or coping strategies. We did, however, find reviews focusing on food prices, social environment and buying power, but these did not assess all relevant outcomes and not all were of good quality. Therefore, we included all these interventions addressing food access in this review.

We chose to assess community‐level interventions because every community member residing in the setting where they are implemented can potentially benefit from them (McLeroy 2003). These types of interventions have been shown to be effective (Bhandari 2003; Mohammadifard 2009), and include interventions that take place across cities or within community institutions, such as schools, neighbourhoods, churches or work sites. The intervention may involve individuals, families, organisations or public policy.

This review focused on LMICs as they experience the greatest burden from food insecurity and malnutrition and because another Cochrane Review will address food security in developed countries (Burns 2010).

How the intervention might work

Based on the literature cited in the above sections, and on guidance on how to use logic models in systematic reviews (Rohwer 2016), we developed a logic model that illustrates how interventions addressing food insecurity might work in improving the nutritional status of individuals (Figure 1). In this model, we present interventions that address food availability, access and utilisation. The interventions may operate at different levels of influence, including the macro (national, regional, global), meso (community) and micro (household and individual) levels. As mentioned above, food security at one level does not ensure food security at another level (Gross 2000). As our review focused on chronic food insecurity, the logic model does not include interventions that address transient food insecurity.


Food security logic model: how interventions influence food and nutritional security.

Food security logic model: how interventions influence food and nutritional security.

Although this logic model encompasses three dimensions of food security – availability, access and utilisation – we only explored how interventions addressing access to food may lead to food and nutrition security. As mentioned above, access to food concerns the ability of households (and communities) to acquire sufficient and appropriate foods to ensure a diet that is diverse, nutrient dense and safe, and that satisfies the nutrient needs of its members (ACF‐IN 2008; WHO 2013). This logic model provides examples of interventions that address the determinants of food access. These include income‐ or employment‐generating opportunities, coping strategies (e.g. borrowing money from a community fund, childcare), social grants, food price policies and regulations, rural infrastructure development, and food or cash vouchers. The direct effects of these interventions include increased financial resources in the household, reduced food prices, increased social support and assistance (e.g. from family, neighbours or the government), having adequate facilities to store food, and ensuring that there is affordable transport to food outlets as well as existence of food outlets closer to where people live (Cotta 2013; Ecker 2012; FAO 2012). Many of these factors interact with each other. For example, having more money may enable the household to buy a fridge to store fresh food, being able to borrow money increases the money available to buy food or the existence of adequate road infrastructure may lead to decreased food prices. These direct effects all lead to a common intermediate effect, which is better ability of households to acquire healthy and nutritious food. The acquisition of healthy food is dependent on there being food available. Being able to acquire healthy food makes it easier for households to make healthy food choices, which in turn influences their intake of healthy and safe food. This represents the interaction across the different dimensions of food security. When the intermediate effects across all dimensions of food security are in place – that is, when nutritious food is commonly available in sufficient quantities at fair prices – households are able to acquire healthy food, all individuals within the household can eat healthy food that meets their nutritional requirements as well as their preferences, and long‐term outcomes of food and nutrition security, and thus of improved nutritional status of everyone in the household and in the community, are achievable.

One potentially harmful unintended consequence of interventions that improve access to food is the increased risk of overweight or obesity (Cotta 2013; Ruel 2013), particularly if there is increased intake of energy‐dense ultraprocessed products and sugar‐sweetened beverages (Lignani 2011). People may choose to acquire these foods because of lower cost; lack of knowledge about healthy diets; or other social, cultural or individual preferences (Ruel 2013).

Although we are assessing interventions addressing access to food, it is important to note that, in order to have long‐term food and nutrition security, all three dimensions need to be in place: food needs to be available; people need to be able to access it; and they also need to know how to choose the food, prepare and store it appropriately (Pinstrup‐Andersen 2009; WHO 2013).

Why it is important to do this review

Although many interventions are being implemented to address food insecurity globally, the lack of sufficient improvements in levels of undernutrition over time, particularly in LMICs, highlights the need for the effectiveness of these interventions to be assessed. Furthermore, our scoping review highlighted that existing reviews addressing access to food in LMICs were not of high methodological quality. Therefore, we aimed to apply rigorous Cochrane Review methods to produce a high‐quality review to identify effective interventions addressing food access. This evidence would then inform relevant stakeholders' decisions about which interventions to implement in order to achieve desirable results and ensure that scarce resources are utilised efficiently. Furthermore, improving access to food would help improve overall food security and the health and nutritional status of populations, which are requisites for the socioeconomic development of individuals and societies (FAO 2003).

Objectives

Primary objective

To determine the effects of community‐level interventions that aim to improve access to nutritious food in LMICs, for both the whole community and for disadvantaged or at‐risk individuals or groups within a community, such as infants, children and women; elderly, poor or unemployed people; or minority groups.

Secondary objectives

To determine the features of community‐level interventions that enable or impair the effective implementation of these interventions to improve access to food.

To identify unintended consequences of interventions to improve access to food.

Methods

Criteria for considering studies for this review

Types of studies

We included randomised controlled trials (RCTs) and cluster randomised controlled trials (cRCTs). We also included non‐randomised studies because: 1. we did not expect to find many RCTs that would answer our question; and 2. to increase the external validity of the review findings. In these studies, observations are made before and after an intervention has been implemented or an exposure has occurred, both in an intervention and a control group. These types of studies are sometimes referred to as prospective analytical cohort studies or controlled before‐after studies. We collectively termed them prospective controlled studies (PCS). We planned to include interrupted time series (ITS), but found none. ITS studies observe the effects of an intervention at multiple time points before and after an intervention. ITS studies needed to have at least three time points both before and after the intervention in order to be included.

Types of participants

We included all population groups living in communities in LMICs exposed to community‐level interventions aiming to improve food access. For the purpose of this review, we defined a community as a group of people with diverse characteristics who are linked by social ties, share common perspectives and engage in joint action in geographical locations or settings (MacQueen 2001). We included both adults and children living in those communities, as well as disadvantaged groups within those communities. LMICs were defined according to the World Bank 2020.

Most interventions addressing food insecurity are usually implemented in areas and among populations at high risk for food insecurity, such as low‐income areas, unemployed people, women and children. We did not restrict studies on the basis of social and demographic characteristics, and reported these characteristics in the review.

We excluded studies which only included participants with specific diseases or conditions (e.g. severely malnourished children) as these types of participants require specialised approaches to address malnutrition caused by these diseases or conditions.

Types of interventions

We included community‐level interventions that aim to improve access to food, as detailed in our logic model (Figure 1). Community‐level interventions were defined as those in which the community was the setting where the intervention was implemented, with every member of that community potentially benefiting from it (McLeroy 2003). This includes interventions that are district‐, city‐ or village‐wide or interventions that take place within community institutions such as schools, neighbourhoods, churches or work sites. The intervention may involve individuals, households, organisations or public policy. Based on the literature in this field, and on the findings of our scoping review, we decided to include the following interventions that address access to food (Table 2):

Open in table viewer
Table 2. Definition of interventions included in the review

Category of intervention

Definition

Types of interventions

Improve buying power

Interventions that generate/increase/maintain income to ensure economic access to food and other basic needs.

  • Cash transfers (conditional or unconditional)

  • Other income generation interventions, e.g.

    • Cash‐for‐work programmes

    • Microcredit/microenterprise development – facilitation of small business development through credit‐provision and training in specific business skills

    • Employment generating activities, that will generate/increase income

    • Agriculture‐related interventions – training /cash cropping/livestock ownership/other. These interventions are only included if they aim to increase income of households. Agricultural interventions only aiming to increase/ensure enough food for consumption are excluded.

Food prices

Interventions that reduce price of food and thus increase economic access to food.

  • Food stamps or vouchers (distribution of coupons or stamps that can be used to purchase foods in local markets, etc.)

  • Food subsidies/discounts

  • Policies/regulations that reduce/regulate food prices

Infrastructure/transport

Interventions that ensure people/communities have physical access to food/food outlets.

  • Rural infrastructure development; e.g. roads that enable access to shops/ markets

  • Interventions that ensure affordable transportation to markets/food outlets

  • Adequate food storage facilities

Social environment/support

Interventions that ensure people have social support/support network they can resort to for money/food in times of need, or access to adequate storage facilities (e.g. shared fridge) or services (e.g. transport/childcare) – leading to increased economic or physical access to food

Social support can be instrumental, emotional, informational, or companionship. We were interested in instrumental social support, i.e. practical help that can be accessed in times of need.

  • Childcare so parents can go to work

  • Borrowing money/food from neighbours/relatives

  • Community fund/village savings loans

  • Shared fridge/storage facilities

  • Shared transport

  • interventions that improve buying power (e.g. income‐generation opportunities, cash transfer schemes);

  • interventions addressing food prices (e.g. policies, discounts, vouchers and subsidies);

  • interventions addressing infrastructure and transport that affect physical access to food outlets;

  • interventions addressing the social environment and social support (e.g. social support from family, neighbours or government).

We included studies that compared these interventions, individually or in combination, to no intervention or to other eligible interventions, including treatment as usual.

We chose this broad approach because we did not expect to find many eligible studies to include for each of the intervention types.

As we anticipated variability in the duration of included interventions, we included interventions of any duration.

Although we were interested in interventions that have measured access to nutritious food, we did not apply this as an inclusion criterion. Instead, we captured this information when extracting the details of included interventions, if this was available.

We excluded interventions that addressed transient food insecurity (e.g. food aid during natural disasters and wars) and that provided short‐term relief from food insecurity (e.g. one‐off food voucher, food banks or soup kitchens). We also excluded interventions that provided food in the form of food baskets or in‐kind transfers of food. These types of interventions, according to our logic model, fall under the groups of interventions addressing availability and were, therefore, excluded. Other types of in‐kind transfers (e.g. livestock, food vouchers, etc), that were not directly providing food to participants but contributed to their economic access to food, were eligible for inclusion. Interventions that involved agricultural production also typically fall under 'food availability', however, if their aim was specifically to generate income, they were included.

Types of outcome measures

Given the complex nature of food security, we assessed outcomes at different levels, namely at the community, household and individual levels.

The findings of our scoping review showed that the types of outcomes measured across food security interventions vary considerably. For this reason, we took a broad approach regarding the outcomes to include.

Given that our main interest was in determining whether these interventions improve access to food and, consequently, food security and nutritional status, we included only interventions that had measured outcomes related to food access or nutritional status, or that used a food security measurement tool. We included any study that had at least one of the outcomes listed below.

Primary outcomes

Our primary outcomes included those that measure access to food at the household and community level. Following from our logic model, these were the following changes in the (FAO 2013; Smith 2006):

  • prevalence of undernourishment (i.e. proportion of people with insufficient intake to meet minimum dietary energy requirements (MDER) (inddex.nutrition.tufts.edu/data4diets/indicator/prevalence-undernourishment?back=/data4diets/indicators);

  • proportion of household expenditure on food (as proportion of household income or of total household expenditure);

  • proportion of households who were food secure (e.g. according to various measures or indices of food security and dietary diversity at an individual or household level), as measured in the included study.

Secondary outcomes

Secondary outcomes were those that reflect access to food and food availability and utilisation. Thus, they reflect nutritional status, which is the ultimate goal of food security interventions at the individual level. Following from our logic model, the secondary outcomes at the individual level were:

  • change in adequacy of dietary intake (e.g. food or energy intake and whether it meets energy and nutrient requirements; if intake was not assessed for adequacy, i.e. only calories reported, this was not reported in the review);

  • change in anthropometric indicators (e.g. stunting, wasting and underweight in children, according to height, weight, height‐for‐age z‐scores (HAZ), weight‐for‐height z‐scores (WHZ), and weight‐for‐age z‐scores (WAZ); underweight and overweight in adults according to body mass index (BMI) classifications);

  • change in biochemical indicators (e.g. micronutrient levels in the blood);

  • cognitive function and development during the intervention period (e.g. Denver Developmental Screening Test, Bayley Scales of Infant Development);

  • change in proportion of anxiety or depression (as described by the included study's authors);

  • morbidity (as described by the review authors);

  • adverse outcomes (e.g. proportion overweight or obese as a potentially harmful consequence of these type of interventions).

We only included outcomes that were measured at least three months after the intervention was implemented as outcomes measured earlier are not likely to reflect sustainable changes.

Search methods for identification of studies

Electronic searches

We searched electronic databases from 1980 onwards for relevant studies. We applied no language or publication status limits. We chose the year 1980 as the starting point because it was around this time that the term 'food security', encompassing access to food, started being used (Masset 2011). The initial searches were conducted in September 2016 in the following databases:

  • Ovid MEDLINE(R) Epub Ahead of Print 11 July 2016, Ovid MEDLINE 1946 to June week 5 2016, Ovid MEDLINE(R) In‐Process & Other Non‐Indexed Citations 11 July 2016, Ovid MEDLINE Daily Update 11 July 2016;

  • Cochrane Central Register of Controlled Trials (CENTRAL): Issue 6, 2016 (the Cochrane Library/Wiley);

  • Embase (Elsevier);

  • GreenFILE (EBSCO);

  • AfricaBib (africabib.org);

  • AGRIS;

  • AGRICOLA;

  • AFRICAN HEALTHLINE, African Journals Online (via Africa‐Wide Information, EBSCO);

  • Trials Register of Promoting Health Interventions (TRoPHI);

  • WHO Global Index Medicus;

  • Web of Science (Conference Proceedings Citation Index, Science Citation Index Expanded, Social Science Citation Index);

  • Sociological Abstracts (ProQuest);

  • International Bibliography of the Social Sciences (IBSS) (ProQuest);

  • Global Health (EBSCO);

  • ClinicalTrials.gov;

  • WHO International Clinical Trials Registry Platform.

A combination of text words and controlled vocabulary terms related to the interventions and possible outcome measures were used to develop a sensitive search strategy. The search strategies for the different databases are available in Appendix 1, which is an adaptation of the search strategy for the Cochrane Review assessing interventions to improve food security in developed countries (Burns 2010). We applied a study design filter to the search that has been developed by Joy Oliver, the information specialist at Cochrane South Africa. The original search strategy for MEDLINE published with the protocol of this review had to be revised by a librarian and adapted for Ovid MEDLINE, as it retrieved an excessive number of results. The Ovid MEDLINE search strategy was then modified to be adapted for the other databases and reported as appendices in our full review. We recruited the Cochrane Public Health Group's information specialist to advise on and implement the search strategy.

The search was updated in April 2019 and February 2020. For the updates, we followed the recommendations of Garner 2016. The Cochrane Public Health Group's information specialist defined a minimum set of databases that would have identified the original included studies and optimised the remaining database searches to improve the balance of sensitivity and precision of the search strategies. We searched the following databases from 1980 onwards:

  • Ovid MEDLINE(R) and Epub Ahead of Print, In‐Process & Other Non‐Indexed Citations, Daily and Versions;

  • CENTRAL (the Cochrane Library/Wiley);

  • Web of Science (Conference Proceedings Citation Index, Science Citation Index Expanded, Social Science Citation Index);

  • Global Health (EBSCO);

  • Sociological Abstracts (ProQuest);

  • International Clinical Trials Registry Platform.

The search strategies for each database included in the latest search, which is the same as for the April 2019 search, are available in Appendix 1. Appendix 2 presents the search strategies of the original search strategy in September 2016.

Some of the electronic databases specified above index a combination of published and unpublished studies, such as doctoral thesis and conference abstracts. Therefore, the electronic searches captured some of the unpublished studies. For further searching for unpublished studies, see Searching other resources.

Searching other resources

We identified additional studies through searching reference lists of similar reviews or contacting authors of included studies. However, much of the additional searches we were planning to undertake at the protocol stage were not carried out. We provide reasons in the Differences between protocol and review section.

Data collection and analysis

Selection of studies

Two author pairs (SD, AS, MV, AB, JO, VR, BS) independently screened all titles and abstracts retrieved to determine eligibility against the inclusion criteria. Full‐text copies of eligible titles and of those for which eligibility was unclear were retrieved for closer examination. Any disagreements regarding eligibility were resolved through discussion or through arbitration by a third author, if necessary. We recorded the reasons for excluding studies at the full‐text screening stage in the Characteristics of excluded studies table. We completed a PRISMA flow chart of study selection.

The initial title and abstract screening, from the first search, was carried out using Word documents. The full‐text screening and all subsequent screening was carried out using the Covidence platform.

Where we found relevant studies in a language other than English, Portuguese or Spanish, we planned to contact Cochrane Public Health for options for translations. We found studies in French, which a colleague reviewed against the eligibility criteria. As these were not eligible for inclusion, no translations were required. We found no studies in any other language that required translation.

We used EndNote X8 to manage the retrieved records and to remove duplicate reports of the same study. The study was considered the unit and all references related to the same study were grouped under the same identifier.

Data extraction and management

For each included study, author pairs (SD, AS, MV, AB, JO, VR, BS) extracted data independently and resolved disagreements through discussion or arbitration by a third author. We collected all data except those concerning outcomes using Covidence, using a standardised data collection form, which was piloted on two studies. One author extracted outcomes data using a standardised and piloted form in Microsoft Excel 2007 and a second author (a biostatistician; YB) checked all the data extracted. We based our data extraction form on the forms from Cochrane Public Health and Cochrane Effective Practice and Organisation of Care (EPOC), modified to suit our review. We extracted the following data.

  • Study design and methods (recruitment of participants, representativeness of sample, number of intervention groups, randomisation procedure, statistical methods).

  • Details about the participants, including PROGRESS‐Plus characteristics and number in each group at baseline and at the endpoint. PROGRESS‐Plus characteristics refer to characteristics of participants that can be used to identify disadvantaged groups and that allow us to differentiate the effects of the intervention across social categories (Tugwell 2010). These characteristics include: place of residence, race or ethnicity, occupation, gender, religion, education, socioeconomic status and social capital; and Plus characteristics include age). We extracted details about withdrawals and dropouts, if these were available.

  • Details about the intervention, including process measures (e.g. aims; social and cultural context; comparison interventions; length of the intervention; duration of follow‐up; implementation factors such as amount of conditional cash transfers, number of times transport is given or total amount of food vouchers given to each individual), and whether the intervention was universal or targeted. This information aimed to provide insight on the factors that may impair or facilitate implementation of the intervention, which addresses the second objective of this review. We also extracted information on whether the intervention aimed to improve access to nutritious food, how nutritious food was defined, if specific nutritious foods were targeted for increased access and what types of food were accessed by participants.

  • Description of outcomes used to measure effectiveness and how they were measured.

  • Primary outcomes at the household and community level.

  • Secondary outcomes at the individual level.

  • Other process measures including intervention cost and sustainability.

  • Source of study funding and sponsorship of the interventions.

We incorporated the Cochrane‐Campbell Methods Group Equity Checklist into our data extraction form (methods.cochrane.org/sites/methods.cochrane.org.equity/files/public/uploads/EquityChecklist2012.pdf); however, the included studies reported very little of this information.

We extracted information on potential confounders or moderators of the study outcomes. These included sociodemographic variables such as gender, ethnicity or race, and place of residence, and other PROGRESS‐Plus characteristics based on the details available in the studies.

When necessary, we contacted the authors of primary studies to for clarification or to seek missing information.

We used Review Manager 2014 for data management and analysis.

Assessment of risk of bias in included studies

Author pairs (SD, AS, MV, AB, JO, VR, BS) conducted the risk of bias assessment and resolved disagreements through discussion or arbitration by a third author. Risk of bias assessments were also carried out in Covidence.

We assessed the risk of bias for all RCTs and PCS using the EPOC risk of bias tool for studies with a separate control group (EPOC 2017). This tool assesses the same risk of bias domains as the Cochrane 'Risk of bias' tool for RCTs (Higgins 2011), namely sequence generation, allocation concealment, blinding of participants and personnel, blinding of outcome assessors, incomplete outcome data (including loss of clusters in the case of cRCTs), selective outcome reporting and other bias. It also includes additional domains to assess the risk of bias from inappropriate methods regarding: similarity of baseline outcome measurements, similarity of baseline characteristics and whether the study was protected against contamination. For other bias, we specifically assessed misclassification bias of the exposure, measurement bias and two domains related to cRCTs: incorrect analysis (i.e. whether the study adjusted for clustering) and recruitment bias. We assessed the risk of bias from lack of blinding of participants and personnel and of outcome assessors separately. We had planned to assess the risk of bias from lack of blinding separately for objective and subjective outcomes and to assess the risk of bias from incomplete outcome data separately for different outcomes. However, given the disparity and multiplicity of outcomes reported, we decided not to do this. However, we did consider whether the outcome was objective or subjective when assessing the risk of bias from lack of blinding.

We had planned to use the EPOC risk of bias tool for ITS study designs; however, we included no ITS studies.

For each item, we made a judgement of 'High risk', 'Unclear risk', or 'Low risk', with supportive information to justify these judgements provided in the Characteristics of included studies table. We incorporated the risk of bias assessment in the interpretation of our review findings, and we did not restrict analysis by degree of risk of bias. We presented a 'Risk of bias' graph and a summary figure.

To assess overall risk of bias at study level, we considered its risk of bias for two key domains: selection and attrition bias. For example, if a study was classified at high risk of either selection or attrition bias, it was classified as having overall high risk of bias.

Measures of treatment effect

Where data allowed, we conducted meta‐analysis using Review Manager 5 (Review Manager 2014). However, due to sparsely reported data, we were unable to conduct a meta‐analysis in many instances.

For binary outcomes, we planned to report the risk ratio (RR) of outcomes in the intervention group compared to the control group. Only one included study reported the RR as an overall effect measure for the intervention. The other 28 included studies that reported dichotomous outcomes reported the change in proportion using percentage points (pp) (68%) or using reported odds ratios (OR) (32%).

For continuous outcomes, and where baseline data were available, we reported the mean difference (MD) between the change in the intervention and control groups if the outcomes had been measured in the same way by all studies. If the continuous outcomes were measured in distinct ways in different studies in a comparison, we used the standardised mean difference (SMD) between the intervention and control groups. Where the change per group was not available, we used end values where randomisation was successful. If there was a reasonable risk of selection bias, and the change per group was not available, the study was not included in a meta‐analysis.

The included studies reported mostly estimates from regression or from difference‐in‐difference analyses, which were interpretable as an MD and thus were reported as such. None of the studies reported the effects per group, and in most cases the data were only available either for baseline or endline, and if it was available, often there was no measure of variance reported. Thus, we described the effect measures as reported in the included studies.

We reported 95% confidence intervals (CIs) alongside all effect estimates, when these were available or when it was possible to calculate them. Calculations of 95% CI were done in Review Manager (using the inverse variance option; Review Manager 2014) or using a Microsoft Excel 2007 spreadsheet with the formula to calculate the 95% CI from the regression estimate and standard error (SE) value. We report P values only where no 95% CI was reported or could be calculated to illustrate the strength of evidence for the effect size.

Unit of analysis issues

cRCTs that randomise groups rather than individuals to intervention groups and that report analyses at the individual level needed to also report the method used to account for clustering. A biostatistician (YB) checked all studies to ensure that the clustering effect had been accounted for correctly. If they had not taken the clustering effect into account in their analyses, we would have requested individual participant data, calculated an intracluster correlation coefficient (ICC), and re‐analysed the data appropriately. If we had been unable to obtain primary data, we would have attempted to find an appropriate ICC from the literature and adjust the sample size accordingly. We had planned to meta‐analyse the effect estimates and SEs from cRCTs using generic inverse‐variance methods in Review Manager 2014. If we had re‐analysed the data, we would have clearly marked the results as re‐analysed and we would have stated where re‐analysis had not been possible. However, we did not have to re‐analyse the data for included cRCTs as they all correctly accounted for clustering.

In cases where the outcomes were measured at multiple time points, we had planned to group the outcomes measured at similar time points where this was possible. For any particular outcome, if most studies were reporting a specific time point and only one study reported multiple time points, we reported the most commonly reported time point. In most cases, the time points were similar and, for the few instances where this was not the case, we extracted measures from all time points but reported the latest time point. Taking into account that the minimum duration after implementation at which we extracted outcomes was three months, the short‐term time point was three to six months.

We only considered outcomes reported immediately at the end of the intervention, not postintervention follow‐up.

In many cases, studies reported multiple outcome measures for the same outcome domains. To prioritise the outcomes, we selected the measure that provided the largest scale measure of the domain (i.e. the most comprehensive outcome). For example, in cases where individual and composite measures for the same outcome domain were reported for the same study, we preferentially reported composite measures as these are probably more useful to decision‐makers. For anthropometry, we did not report effects on weight and height units, but rather reported z‐scores for weight‐for‐age and height‐for‐age, in which weight and height are assessed against a reference standard. All outcomes reported in a study are presented in the Characteristics of included studies table, as well as an indication of which were selected for synthesis.

For interventions with multiple comparison groups, all groups that met this review's inclusion criteria were included. If there were more than two relevant comparison groups for the same intervention, we attempted to combine the relevant experimental and control groups to make a single pairwise comparison. This was the case in three studies, for which two interventions groups were combined. If this was not possible, we made multiple pairwise comparisons between the relevant groups and divided the sample size of the shared intervention group evenly across the comparisons to avoid double counting of participants in a meta‐analysis. If a meta‐analysis was not possible and we could not combine the results of different groups, we presented the results of all relevant groups.

Dealing with missing data

If there were unclear or missing data related to study methodology, participants lost to follow‐up, outcome data or statistics, we contacted the study's primary author via email. We recorded all communications with authors in Appendix 3.

We recorded all missing outcome data in the data extraction form and in the Characteristics of included studies table. If it was not possible to obtain missing outcome information after attempting to do so, we would have excluded these studies from the meta‐analysis. We did not exclude any studies due to missing outcome data.

Five included studies did not report the number analysed for at least one outcome (for which the number (n) is stated as not reported (NR)) (Ahmed 2019a; Ahmed 2019b; Andaleeb 2016; Ferre 2014; Hoddinott 2013). This lack of reporting reduces our confidence in the estimated treatment effect as we are unable to assess if the study was powered to detect an effect, analysed accounting for any clustering or if attrition bias was likely. In addition, it has been shown that trial sample sizes can influence treatment effect, with smaller studies reporting larger effect estimates (Dechartres 2013). With no sample sizes reported, it is difficult to ascertain whether this bias exists.

Assessment of heterogeneity

Where we were able to meta‐analyse, we assessed heterogeneity, or the variability among the studies included in a meta‐analysis, by visual inspection of overlap of CIs, and by assessing statistical heterogeneity with the Chi2 statistic (P < 0.1) (Deeks 2019). We calculated the I2 statistic to quantify heterogeneity; with an I2 statistic of 75% and above indicating substantial heterogeneity. We also calculated Tau2, which reflects the extent of variation among intervention effects in different studies, to assess heterogeneity (Deeks 2019). However, in most cases, we were unable to carry out meta‐analyses or create forest plots due to heterogeneity. Instead, we assessed clinical, methodological and conceptual heterogeneity, through tabulation of characteristics of studies included in the same synthesis. For specific comparisons and outcomes, we assessed clinical or conceptual heterogeneity by considering variability in the participants and interventions, or co‐interventions, including study duration, intervention dosing and outcome assessment. We assessed methodological heterogeneity by considering the variability in study design and risk of bias (Deeks 2019; Singh 2017).

Assessment of reporting biases

We had planned to assess the likelihood of reporting bias through funnel plots for each outcome with 10 or more included studies in a meta‐analysis (Sterne 2019). We would have assessed the funnel plots visually for sources of asymmetry, such as small‐study effects, publication bias or other. If it was likely that asymmetry was caused by small‐study effects, we would have conducted sensitivity analysis to explore how this affected the results and conclusions of the meta‐analysis. However, we were unable to do this as none of the compared outcomes were assessed by at least 10 studies.

Data synthesis

In most cases, we were unable to include all studies reporting a specific outcome domain in a meta‐analysis. This was due to studies reporting multiple measures for the same domain that could not be combined, either because there was missing information regarding variance measures, or because the effect measures reported could not be converted to a standardised metric. Therefore, we synthesised the data from all studies reporting on the same outcome domain using vote counting based on the effect direction method (McKenzie 2019). The results of individual studies were presented in one of four categories: 1. 'favours control' if the point estimate favoured the control and the 95% CI did not cross the null; 2. 'unclear effect; potentially favouring the control' if the point estimate favoured the control but the 95% CI crossed the null; 3. 'unclear effect; potentially favouring the intervention' if the point estimate favoured the intervention and the 95% CI crossed the null; and 4. 'favours intervention' if the point estimate favoured the intervention and the 95% CI did not cross the null. Where no CI was provided or could be calculated, we decided whether the effects were 'clear' or 'unclear' based on provided P values. However, P values did not inform the effect direction reported. Although this is a useful method to synthesise data when meta‐analysis is not possible, there are some limitations associated with this method, for example, it does not provide information on the magnitude of effects, does not account for differences in the relative sizes of the studies and is a less powerful method than that used to combine P values (McKenzie 2019).

Regarding the effect direction synthesis, we reported the number of studies with results in the different effect categories for each outcome domain, and the probability of observing this based on the multinomial distribution (for outcome domains with two or more studies and assuming the true proportion is 0.25 for all categories). This information was reported in the first paragraph reporting results for a specific outcome in the effects of interventions section. If a meta‐analysis of all studies in the outcome domain was possible, the multinomial distribution P value was not reported. To visually display the results for key outcomes included in the 'Summary of findings' tables, we created harvest plots for each comparison. These harvest plots depict data both from RCTs and from PCS for key summary of findings outcomes. We presented the results of the effect direction synthesis separately for RCTs and for PCS, in the Effects of interventions section.

In some instances, we were able to carry out a meta‐analysis, either for all studies reporting on the same outcome measure (n = 35), or for a subset of studies that could be combined in a meta‐analysis (n = 8). We conducted meta‐analyses in Review Manager 2014 if the included studies were sufficiently homogeneous (I2 < 75%) and if there was a minimum of two studies for any type of intervention being compared reporting the same outcome measure. Not pooling results in cases of high heterogeneity is an accepted approach in the Cochrane Handbook for Systematic Reviews of Interventions (Deeks 2019). We also explored heterogeneity and reported this when there was high heterogeneity. We did not have sufficient data to carry out any subgroup analysis or meta‐regression to more formally explore heterogeneity. We carried out meta‐analyses separately for each outcome and type of study design, and we used the random‐effects model for all analyses to account for any existing heterogeneity. We generated forest plots for each comparison and outcome where meta‐analyses could be carried out (see Data and analyses).

In preparation for synthesis, we first grouped all studies assessing the same intervention categories to identify which studies could be grouped under each preplanned comparison, and no changes to prespecified grouping were required. The comparison groups were based on the pre‐specified types of interventions listed in table 2: cash transfers (unconditional; conditional), income generation interventions, food vouchers, food subsidies, and social support interventions. Under each comparison, we tabulated the available data and time frames reported for each outcome, which helped identify what data were available, and thus where meta‐analysis was possible and where we had to synthesise using effect direction. A meta‐analysis was possible if the effect and variance estimates were available for all studies or could be calculated from the available data, and if all effect estimates were of the same type (e.g. OR or MD) or could be converted for the comparison (e.g. OR to SMD). Where necessary, we converted OR to SMD, MD to SMD, or MD to SMD to OR.

We prepared two additional types of tables to aid visualisation of available data. One was the 'Overview of included studies' table, summarising main characteristics and reported outcomes of included studies for each comparison (McKenzie 2019). In this table, we organised studies first by study design, with RCTs first followed by PCS, and second according to their overall risk of bias (low, unclear and then high risk of bias). The second type were tables with the results for individual studies, for each comparison, for scrutiny by the reader. The studies in these tables were also ordered according to their overall risk of bias.

We had planned to assess and discuss the implementation factors common to effective interventions, if this information was reported in included studies or in published process evaluations that are mentioned in the study report. However, there was insufficient information from included studies on this.

Subgroup analysis and investigation of heterogeneity

We did not have enough data per outcome and comparison to carry out subgroup analyses. If data allowed, we would have conducted subgroup analysis to assess effectiveness for people at different levels of disadvantage. In updates of this review, we will include the following subgroups.

  • Geographic location (e.g. urban versus rural, country or region).

  • Sex (male versus female).

  • Age (e.g. elderly people, adults, children, infants).

  • Baseline nutritional status (e.g. underweight, overweight, micronutrient deficiencies).

We would also have assessed important implementation factors through subgroups analyses, including the following.

  • Intensity of intervention (high intensity versus low intensity, e.g. in relation to amount of food vouchers or of conditional cash transfers).

  • Length of study and follow‐up (e.g. three to six months, more than six months to less than two years, and two years and beyond).

  • Whether the intervention specifically aimed to improve access to nutritious food.

These analyses would have allowed further exploration of heterogeneity. In order to compare the different subgroups with each other, we would have conducted a standard heterogeneity test in Review Manager 2014 across the subgroup results, by calculating the I2 statistic. We would have made sure that the subgroup data being compared were independent.

Sensitivity analysis

We conducted a sensitivity analysis to assess risk of bias for outcomes with five or more studies. Studies with overall low risk of bias were included in the sensitivity analysis. We reported the results of study designs separately.

Summary of findings and assessment of the certainty of the evidence

The 'Summary of findings' tables include information regarding the number of participants and studies for key outcomes, a summary of the intervention effect and a measure of the certainty of evidence for each outcome according to GRADE considerations. GRADE is a system of rating certainty of evidence in systematic reviews (Guyatt 2010). We rated the overall certainty of evidence for a particular outcome on‐line with GRADEpro as high, moderate, low or very low. All RCTs started at high‐certainty evidence and the following factors were considered to downgrade the certainty: overall risk of bias, consistency of effect, imprecision, indirectness and publication bias. All PCS started at low‐certainty evidence and the following factors would have been considered to upgrade the certainty: large magnitude of effect, dose‐response gradient and effect of plausible residual confounding. We did not upgrade the certainty of evidence for PCS as there were existing reasons for downgrading (Schünemann 2019). As most of the evidence was not from pooled data, we used the 'Summary of findings' table format for narrative synthesis.

We had planned to include a 'Summary of findings' table for the primary outcomes of this review. However, we decided to also include some of the secondary outcomes. The choice of outcome categories and specific outcome measures to report in the 'Summary of findings' table were decided by the review author team through in‐depth discussion until consensus was reached, taking into consideration which outcomes would be useful to decision‐makers. The 'Summary of findings' tables included the following outcomes.

  • Prevalence of undernourishment.

  • Proportion of household expenditure on food.

  • Proportion of households who were food secure.

  • Dietary diversity.

  • Stunting.

  • Wasting.

  • Cognitive function and development.

Three authors met to rate the evidence per outcome for each of the 'Summary of findings' tables. Decisions about whether to downgrade or not were made through discussion and reaching consensus. Evidence on different outcomes was available from different study designs. Where there was evidence for a particular outcome from both RCTs and PCS, we reported the data from the RCTs in the 'Summary of findings' table. When there were no data from RCTs for a particular outcome, we reported data from PCS that reported that outcome. We reported the certainty of the evidence in the Effects of interventions for outcomes which assessed with GRADE. For other outcomes, we report the risk of bias, as an indicator of the certainty of the evidence to consider when interpreting the findings.

Results

Description of studies

Results of the search

We screened titles and abstracts of 15,477 deduplicated records identified through searching electronic databases and reference searching of eligible studies. Of these, we assessed the full texts of 463 records against the eligibility criteria. After assessing available full‐texts, we included 59 studies reported in 116 records in this review. Some interventions were reported in more than one study, and two records each reported on two different studies. Of the remaining records: we placed 39 studies under awaiting classification as we could not access them or they were conference abstracts, 11 studies are still ongoing and we excluded 297 records with reasons. In the Characteristics of excluded studies table, we report a subset of key excluded studies. The study selection process is described in Figure 2.


Study flow diagram.

Study flow diagram.

Included studies

We included 59 studies in this review (Table 3). In this section we provide a summary overview of included studies. More details are provided in the results of interventions section, for studies included in each comparison, and in the Characteristics of included studies table.

Open in table viewer
Table 3. Summary of included studies

Intervention category

Intervention type

Studies and study designs

Improve buying power

Unconditional cash transfers

18 RCTs: Ahmed 2019a; Ahmed 2019b; Asfaw 2014; Baird 2013a; Brugh 2018; Daidone 2014; Fenn 2015; Fernald 2011; Gangopadhyay 2015; Haushofer 2013; Hjelm 2017; Hoddinott 2013; Merttens 2013; Miller 2011; Pellerano 2014; Schwab 2013; Skoufias 2013; Tonguet Papucci 2015

3 prospective controlled studies: Aguero 2006; Breisinger 2018; Renzaho 2017

Conditional cash transfers

9 RCTs: Baird 2013a; Evans 2014; Gertler 2000 (PROGRESA); Hidrobo 2014c; Kandpal 2016; Kurdi 2019; Kusuma 2017a; Macours 2012; Maluccio 2005

5 prospective controlled studies: Andersen 2015; Ferre 2014; Huerta 2006 (PROGRESA); Leroy 2008 (PROGRESA); Lopez Arana 2016

Income generationd

6 RCTs: Beegle 2017; Darrouzet Nardi 2016; Marquis 2018; Olney 2016; Osei 2017; Verbowski 2018

11 prospective controlled studies: Alaofe 2016; Alaofe 2019; Asadullah 2015; Doocy 2017; Jodlowski 2016; Kangmennaang 2017; Katz 2001; Kennedy 1989; Murshed E Jahan 2011; Porter 2016e; Weinhardt 2017

Food prices

Food vouchers

4 RCTs: Fenn 2015b; Hidrobo 2014c; Jensen 2011; Ponce 2017

0 prospective controlled studies

Food rebates/subsidies

1 RCT: Chen 2019

3 prospective controlled study: Andaleeb 2016; Chakrabarti 2018; Sturm 2013

Infrastructure changes

0 identified

Social environment

Village savings and loans

1 RCT: Kusuma 2017b

1 prospective controlled study: Brunie 2014

aBaird 2013 assesses both conditional and unconditional cash transfers.
bFenn 2015 assesses both unconditional cash transfers and food vouchers.
cHidrobo 2014 assesses both conditional cash transfers and food vouchers.
dThis includes different interventions that aimed to generate income of participants (e.g. integrated agricultural programmes, community development programmes).
ePorter 2016 assessed a public works (80%) (cash/food‐for‐work) or unconditional cash transfer government programme (20%). Results were reported for the entire population, not disaggregated according to intervention received.

RCT: randomised controlled trial.

Study design, sample size and follow‐up

Of the 59 studies included:

It is important to note that three studies, of different design, evaluated the same programme: PROGRESA (Gertler 2000 (PROGRESA); Huerta 2006 (PROGRESA); Leroy 2008 (PROGRESA)) (Table 4). In addition, two studies assessed the effects of a programme in Malawi in different study settings (Miller 2011; Brugh 2018), whereas two studies reported the effects of an income‐generation intervention during different periods (Alaofe 2016; Alaofe 2019).

Open in table viewer
Table 4. Description of included studies assessing the effects of Mexico's PROGRESA/Oportunidades conditional cash transfer programme

Study ID

Linked references

Study design and duration

Description of intervention

Sampling

Outcomes reported

Gertler 2000 (PROGRESA)

Gertler 2004; Hoddinott 2000; Hoddinott 2003a; Hoddinott 2004(?); Skoufias 2001; Skoufias 2007; Fernald 2008; Fernald 2009

Cluster‐RCT conducted between 1998 and 2000, where communities were randomly allocated to either receive the intervention immediately (intervention group) or to receive the intervention 2 years later (control group). In reality, control communities started receiving the intervention in late 1999, about 1.5 years after the intervention communities.

Timepoints of data collection (through household surveys – ENCEL):

  • March 1998 (pre‐intervention)

  • October/November 1998

  • May/June 1999

  • October/November 1999

  • September/December 2003 (follow‐up)

  • September/December 2007 (follow‐up)

'Oportunidades' (previously called Progresa) is a conditional cash transfer programme implemented by the Mexican government since April 1998.

Women in eligible households receive cash transfers every 2 months (a food and an education transfer) if they adhered to specific conditionalities: all family members attend preventive health services regularly; children aged 0–5 years and lactating mothers attended nutrition monitoring clinics for growth monitoring, immunisation, to obtain nutrition supplements, and for nutrition and hygiene education; pregnant women attend antenatal care, receive nutritional supplements and health education.

The education transfers included scholarships for school attendance and school supplies, and was dependent on children's school attendance.

The value of the transfers was about 20–30% to the household consumption expenditure preintervention.

506/50,000 eligible rural villages were randomly selected based on the index level of community poverty. Of these, 320 communities were allocated to the intervention group and 186 to the control group. Within each community, households were selected by proxy means testing and selection validated in a community assembly.

Some studies assessed outcomes in a subsample of the study population.

Fernald 2008 followed up on a sample of children in 2003: children aged 24–72 months in the 'Early intervention' group (from 144 communities), and children aged 2–5 years in the 'Late intervention' group (from 108 communities).

Fernald 2009, followed up a sample of children in 2007: 1093 children aged 8–10 years in the 'Early intervention' group, and 700 children aged 9–10 years in the 'Late intervention' group.

  • Household food consumption (Hoddinott 2000)

  • Dietary diversity (Hoddinott 2000)

  • Total caloric availability (Hoddinott 2000; 2003a)

  • Morbidity (children aged 0–5 years) (Gertler 2004)

  • Fernald 2008 and Fernald 2009 only assessed data that included the period when both the control and intervention groups were receiving the intervention (i.e. early vs late intervention). These data were not extracted for the review but were mentioned in the Discussion.

Huerta 2006 (PROGRESA)

Rivera 2004; Gertler 2004; Behrman 2001

Nested cohort study conducted on a subset of the larger cRCT sample (described above), including a random selection of 205 of original intervention communities and 142 of original 186 control communities. Additional household surveys conducted on health and nutrition indicators.

Time points of data collection:

  • August/September 1998 (i.e. no true baseline data available as by this time all intervention households were already receiving transfers);

  • September/December 1999;

  • November/December 2000 (both groups exposed to the programme for approximately 1 year)

As above

Subsample of children selected.

Behrman 2005 (?)

Rivera 2004: children aged < 12 months (461 children from 175 communities in the intervention and 334 children from 107 communities in the control).

Gertler 2004 and Huerta 2006: sample sizes not reported

  • Height (Behrman 2005; Gertler 2004)

  • Stunting (Gertler 2004)

  • Anaemia (Gertler 2004)

No outcome data reported for exposed vs non‐exposed groups after 1 year of follow‐up (Rivera 2004; Huerta 2006) (?).

Leroy 2008 (PROGRESA)

N/A

CBA: urban communities randomly selected for expansion of Oportunidades into 149 urban areas. The control group comprised eligible households that did not enrol in the programme.

Time points of data collection through household surveys:

  • September/December 2002 (preintervention)

  • July/November 2004

As above

Children aged < 24 months in 2002: 574 in intervention and 159 in control

  • Height

  • HAZ

  • Weight

  • WHZ

CBA: controlled before‐after study; cRCT: cluster randomised controlled trial; HAZ: height‐for‐age z‐score; N/A: not applicable/available; RCT: randomised controlled trial; WHZ: weight‐for‐height z‐score.

All studies had a control group. Most studies compared the intervention with a control group where there was no intervention implemented, six studies compared the intervention with another food security‐related intervention (Andaleeb 2016; Chakrabarti 2018; Gangopadhyay 2015; Hoddinott 2013; Renzaho 2017; Schwab 2013), and one study compared the intervention with another intervention unrelated to food security (Weinhardt 2017). Four studies had three intervention groups (Chen 2019;Gangopadhyay 2015;Ponce 2017; Verbowski 2018 ), four studies had four intervention groups (Brunie 2014; Fenn 2015; Hidrobo 2014; Skoufias 2013), and two studies had five intervention arms (Ahmed 2019a; Ahmed 2019b). In all but three cases the study groups were either not relevant for the review or they pertained to different comparisons, therefore, there were no issues with overlapping control groups. For three studies with two relevant intervention arms, these were combined. In some studies, the control group received the intervention at a later stage; this review only captured data for the period during which the control group was not yet receiving the intervention.

The unit of allocation or exposure to the intervention was at group level in 41 studies (including communities, electoral divisions, municipalities, parishes, districts, villages, other), at household level in 14 studies (Alaofe 2016; Andersen 2015; Asadullah 2015; Gangopadhyay 2015; Haushofer 2013; Huerta 2006 (PROGRESA); Jensen 2011; Jodlowski 2016; Kennedy 1989; Kurdi 2019; Leroy 2008 (PROGRESA); Macours 2012; Porter 2016; Sturm 2013), and individual level in four studies (including individuals, women, and children) (Aguero 2006; Baird 2013; Katz 2001; Murshed E Jahan 2011).

The sample size in included studies ranged from 214 to 169,485 participants and 300 to 124,644 households. All studies collected data on individual participants except for Sturm 2013, which collected supermarket sales data.

Maximum follow‐up was three months in one study (Hoddinott 2013), greater than three months to 12 months in 10 studies (Alaofe 2016; Alaofe 2019; Chen 2019; Fenn 2015; Gangopadhyay 2015; Hidrobo 2014; Jensen 2011; Miller 2011; Ponce 2017; Schwab 2013), greater than 12 months to two years in 25 studies, greater than two years to five years in 17 studies (Asfaw 2014; Baird 2013; Beegle 2017; Breisinger 2018; Brunie 2014; Chakrabarti 2018; Doocy 2017; Evans 2014;Hjelm 2017; Kurdi 2019; Lopez Arana 2016; Macours 2012; Murshed E Jahan 2011; Osei 2017; Sturm 2013; Tonguet Papucci 2015; Weinhardt 2017), and greater than five years to nine years in six studies (Aguero 2006; Andaleeb 2016; Andersen 2015; Asadullah 2015; Porter 2016; Renzaho 2017).

Interventions

Included interventions were classified according to the categories of interventions in our logic model (Figure 1; Table 2). See Table 3 for a summary of categories and types of interventions included in this review.

Fifty‐two studies assessed interventions aimed at increasing buying power, including:

Open in table viewer
Table 5. Income‐generation interventions – overview of included studies

Study (country of conduct)

Study design

Overall risk of biasa

Other key details of intervention

Population (sample size at baseline: Intervention/ Control)

Outcome domains and measures with available data

Time point of measurement

Darrouzet Nardi 2016 (Nepal)

cRCT

Unclear

Programme name: Heifer training curriculum

Programme description and frequency: participation in programme that focused on training regarding poverty alleviation, citizen empowerment, community development and optimisation of livestock management as means to generate income.

Provider: NGO (Heifer International)

Delivery: women's self‐help groups which met with a trained facilitator, supplemented by specific interactive instruction, workshops, guidance, and training. Biweekly meetings

Co‐interventions: none reported

Rural farming communities; HHs: 201/214; children (aged 6–60 months): 283/324

Dietary diversity:

  • Household dietary diversity index

  • Child minimum dietary diversity

Anthropometry

  • HAZ;

  • WAZ

1 and 2 years

Doocy 2017 (Democratic Republic of the Congo)

Prospective controlled study

High

Programme name: Intervention implemented as part of the Jenga Jamaa II project

Programme description and frequency: WEGs met weekly and meetings served as a delivery mechanism for a variety of interventions including literacy and numeracy, business and marketing training, and income‐generation activities. Savings and credit groups were started in each WEG. Beneficiaries were provided with a starter kit of basic materials for their income‐generation activity. Many WEG participants also received goats and energy‐efficient stoves. The FFS intervention provided farmers with experience‐based education on farming practices and postharvest handling as well as business and natural resource management skills. Each FFS group received semi‐monthly training sessions for 2 years. Each FFS group had a community demonstration plot, and group members also received starter packages of seeds and tools for use on individual farms. The FFS programmes focused on a variety of common crops in the region. The first year of training focused on knowledge of production systems and technologies; adoption of techniques and technologies and behaviour change were the focus in the second year

Provider: ADRA

Delivery: FFS – training sessions on agriculture techniques and other content by ADRA field agents.

Co‐interventions: after they finished the FFS intervention (2 years) some transitioned to farmer business associations, which were intended to improve access to credit and marketing opportunities.

Farming villages; HHs (WEG: 390/324; FFS: 338/324)

Food security:

  • HFIAS

  • Proportion of HHs improving a HFIAS category

Dietary diversity:

  • HDDS

  • Achieving target dietary diversity (based on HDDS)

3.5 years

Weinhardt 2017 (Malawi)

Prospective controlled study (non‐equivalent control group)

Unclear

Programme name: support to able‐bodied vulnerable groups to achieve food security (SAFE) programme

Programme description and frequency: programme comprised 4 components

  • Improving farming practices and sustainable agriculture through Farmer Field Schools

  • Increasing access to savings and investment through Village Savings and Loans Groups

  • Building capacity of local governance structures

  • Integrating HIV education and gender empowerment into programmes through training and education

Provider: NGO (CARE Malawi)

Delivery: community‐based programme

Co‐interventions: agricultural education programme for a few intervention and control participants

Rural HHs (598/301)

Food security:

  • Mean number of months with less food than necessary to meet needs

Anthropometry:

  • WAZ

  • HAZ

  • Moderate and severe underweight (< –2SD WAZ)

  • Child BMI

18 and 36 months

Jodlowski 2016 (Zambia)

Prospective controlled study

Low

Programme name: Copperbelt Rural Livelihoods Enhancement Support Project (CRLESP)

Programme description and frequency: ongoing training and one‐off transfer of livestock contingent on training participation. 1 female livestock offspring per transferred female had to be donated to a Pass‐on‐the‐Gift HH.

Provider: NGO (Heifer International)

Delivery: NR

Co‐interventions: none reported

Rural households (105/178)

Dietary diversity:

  • Household Dietary Diversity Index

  • Probability weighted dietary diversity score

6, 12 and 18 months

Asadullah 2015 (Bangladesh)

Prospective controlled study

High

Programme name: challenging the frontiers of poverty reduction – targeting the ultra‐poor (CFPR‐TUP)

Programme description and frequency: multicomponent intervention including orientation training, selection of income‐generation microenterprise by female participants with one‐off transfer of productive assets worth BDT 10,000 to support it (90% of households chose livestock combination), community savings, monthly health worker visits, weekly follow‐up for technical advice, building social capital (village support networks and sponsorship of community leaders), and weekly stipends (BDT 70).

Provider: NGO (Bangladesh Rural Advancement Committee (BRAC))

Delivery: NGO staff deliver training and assets

Co‐interventions: none reported

Ultra‐poor households (2633/2993)

Food security

  • Proportion experiencing food deficit always

Morbidity:

  • Perceived health status

  • Perceived health improvement

3, 6 and 9 years

Marquis 2018 (Ghana)

cRCT

Low

Programme name: Nutrition Links (NL)

Programme description and frequency: 12‐month intervention was an integrated package of agricultural inputs and training as well as education in nutrition, health care and child stimulation for participants. The intervention had 4 main components

  • Poultry for egg production

  • Home gardens

  • Weekly group education sessions throughout the year

  • Community‐wide education

Provider: "Heifer's Passing on the Gift (POG) community development programme, project staff, district agricultural extension officers, district government staff, University of Ghana's Nutrition Research and Training Centre

Delivery:

  • 4‐day training received chickens and initial feed for 1 month and vaccinations, and weekly technical assistance by the project staff

  • Training, received planting materials, and weekly technical assistance

  • Weekly group education sessions

  • Training that was accessible to all residents

Co‐interventions: none reported

Mother–infant pairs in rural communities (287/213).

Dietary diversity

  • Minimal diet diversity

Anthropometry:

  • WAZ;

  • LAZ/HAZ;

  • WLZ/WHZ

1 year

Olney 2016 (Burkina Faso)

cRCT

Unclear

Programme name: enhanced‐homestead food production (EHFP)

Programme description and frequency: integrated agriculture and nutrition programme. Agriculture interventions included provision of land with inputs (crops, animals and implements) and training. Nutrition intervention included behaviour change communication strategy for health and nutrition behaviours, delivered through visits by community volunteers twice per month.

Provider: NGO (Helen Keller International – HKI)

Delivery: agriculture interventions rolled out first to female village farm leaders, who then trained other mothers. Nutrition education carried out by older women leaders or health committee members.

Co‐interventions: none reported

Villages with agricultural homesteads (30/25). HHs: 514 (health committee); 512 (older women leaders); 741 (control)

Dietary diversity:

  • Household Dietary Diversity Index

  • Proportion of mothers consuming individual food groups in past 7 days

Anthropometry:

  • BMI (adult)

  • Underweight (adults) (BMI < 18.5 kg/m2)

2 years

Osei 2017 (Nepal)

cRCT

Unclear

Programme name: Enhanced Homestead Food Production (EHFP) programme

Programme description and frequency: training in improved gardening and poultry‐rearing practices; hosting of a village model farm, which served as a site for purchasing inputs and ongoing training for all the beneficiary women. For every season (rainy and winter) of the first year, each woman was given a one‐off free supply of seeds, saplings and locally bred chicks to establish their home gardens and poultry production. Throughout the period of the intervention, the women met monthly at the farm to refresh lessons on agriculture techniques and nutrition through social and behaviour change communications. During monthly home visits, the project staff and the female community health volunteers also reinforced the educational messages on breastfeeding and complementary feeding to all mothers.

Provider: NGO (Helen Keller International – HKI)

Delivery: 1 woman per group of intervention villages (5 or 6) was selected and trained by HKI and this woman then trained 20 other beneficiary women; meetings at farm; home visits by trained project staff, female community health volunteers and agriculture extension officers.

Co‐interventions: none reported.

Homesteads: mothers (1055/1051), children (1055/1051)

Food security

  • Prevalence of HH food insecurity

Anthropometry:

  • HAZ

  • Stunting (HAZ < –2SD)WAZ

  • Underweight (child) (WAZ < –2SD) and mother (BMI < 18.5 kg/m2)

  • WHZ

  • Wasting (WHZ < –2SD)

  • BMI (mother)

Biochemical indicators:

  • Mean haemoglobin concentration (child and mother)

Morbidity:

  • Prevalence of anaemia (child and mother)

2.5 years

Verbowski 2018 (Cambodia)

cRCT

Unclear

Programme name: Fish on Farms (FoF) project using the Enhanced Homestead Food Production (EHFP) programme

Programme description and frequency: basic agricultural inputs and training, and nutrition and hygiene education. The education focused on optimal nutrition for women and infants and young child practices, and the use of nutrient‐dense produce grown by farmers were demonstrated. The purpose of EHFP was to increase production and intakes of various types of vegetables, herbs and tree fruit. The aquaculture intervention was designed to increase the production of 3 types of small fish, which typically were consumed whole, as well as 3 types of large fish (typically sold for income or fillets consumed).

Provider: NGO (Helen Keller International – HKI, local)

Delivery: trained village health volunteers provided education sessions, through small group and 1‐to‐1 counselling. Cooking demonstrations were also conducted. Support was provided through village model farms (1 in each village).

Co‐interventions: none reported.

Rural HHs: EHFP + aquaculture (100), EHFP (100) and control (100)

Anthropometry:

  • Underweight (women) (BMI <18.5 kg/m2) and children (WAZ < –2SD);

  • Stunting (HAZ < –2SD);

  • Wasting (WHZ < –2SD)

Biochemical indicators:

  • Haemoglobin (non‐pregnant women)

  • Haemoglobin (children)

Morbidity:

  • Anaemia (non‐pregnant women)

  • Anaemia (children)

22 months

Murshed E Jahan 2011 (Bangladesh)

Prospective controlled study

Unclear

Programme name: Development of Sustainable Aquaculture Project (DSAP)

Programme description and frequency: farmers received support to efficiently implement integrated aquaculture‐agriculture (IAA) approaches under 2 models – 1 with a one‐off provision of a small grant for purchasing inputs (value not reported) and 1 without, with training provided (3 sessions in the first year, 2 in the second year and 1 in the third year).

Provider: NGO; WorldFish Center

Delivery: farmers trained in recording required information which was collected bi‐monthly by research assistants.

Co‐interventions: none reported

Small‐scale farmers (260/126).

Within intervention farmers: 127 grant farmers, 133 non‐grant farmers

Proportion of HH expenditure on food

3 years

Kennedy 1989 (Kenya)

Prospective controlled study

Unclear

Programme name: South Nyanza Sugar Factory (Sony) smallholder sugarcane outgrowers' scheme

Programme description and frequency: farmers were enrolled into the scheme to provide sugarcane to a new factory, with payments to farmers after every harvest (24 months after planting)

Provider: Kenyan government

Delivery: contract agreement between farmers and factory.

Co‐interventions: none reported

Smallholder farm HHs (181/231).

Within intervention: 139 sugar farmers and 42 new entrant

  • Proportion of HH expenditure on food

Adequacy of dietary intake

  • Percentage of HHs with caloric deficiency

  • Caloric adequacy of preschool children

Anthropometry

  • WAZ

  • Underweight (< 80% of standard for WAZ)

  • HAZ

  • Stunted (< 90% of standard for HAZ)

  • WHZ

  • Wasting (< 90% of standard for WHZ)

  • BMI (adult)

Morbidity:

  • Illness of women and children (all‐cause and diarrhoea)

2 years

Alaofe 2016 (Benin)

Prospective controlled study

Unclear

Programme name: Solar Market Gardens (SMG)

Programme description and frequency: drip irrigation powered by solar water pump, using a perennial stream or borehole, with continued maintenance and training to farmers provided.

Provider: NGO (Solar Electric Light Fund – SELF)

Delivery: installation of system and training of local technicians carried.

Co‐interventions: women's agriculture group activities.

Rural HHs (116/98)

In both intervention and control groups, HHs included women who participated in women's agriculture groups (59/38) or not (60/60)

Proportion of HH expenditure on food

1 year

Alaofe 2019 (Benin)

Prospective controlled study

Unclear

Programme name: Solar Market Garden (SMG)

Programme description and frequency: Installation of a low‐pressure drip irrigation system, combined with a solar‐powered water pump in each intervention village. Each SMG was used jointly by 30–35 women belonging to the local women's agriculture group (each woman farmed her own land of 120 m2).

Provider: NGO (Solar Electric Light Fund – SELF)

Delivery: expanded installation of SMG systems (from programme reported in Alaofe 2016).

Co‐interventions: women's agriculture group activities.

Women in rural HHs (415/359).

In both intervention and control groups, HHs included women who participated in women's agriculture groups (184/126) or not (228/233)

Dietary diversity:

  • HDDS

  • Women's Dietary Diversity Score

Anthropometry

  • BMI (adult);

  • Underweight (adult) (BMI <18.5 kg/m2)

Biochemical indictors:

  • Iron deficiency

  • Vitamin A deficiency

Morbidity:

  • Anaemia

  • Iron‐deficiency anaemia

1 year

Kangmennaang 2017 (Malawi)

Prospective controlled study

High

Programme name: the Malawi Farmer to Farmer Agroecology project (MAFFA).

Programme description and frequency: farmers do their own experimentation with agroecological methods. Farmers are also encouraged to share knowledge gained with other farmers. MAFFA encourages farmers to adopt a suit of innovations rather than just a single innovation and to encourage farmer‐led learning. In addition to crop diversification, many farmers increased or began to apply compost and manure to their rain‐fed fields. Some farmers also experimented with botanical pesticides. Also, MAFFA goes beyond agroecological training to focus on knowledge sharing, leadership support, nutrition and attention to social inequalities.

Provider: Soils, Food and Healthy Communities organisation of Ekwendeni Hospital, Chancellor College, University of Malawi as well as Malawian and Canadian scientists.

Delivery: training, educational activities, campaigns, provision of seeds. Farmers shared knowledge with other farmers.

Co‐interventions: none reported.

Smallholder farm HHs (793/408)

Food security:

  • HFIAS score

About 2 years

Beegle 2017 (Malawi)

cRCT

High

Programme name: Malawi Social Action Fund's Public Works Programme (MASAF PWP).

Programme description and frequency: the MASAF PWP aims to provide short‐term labour‐intensive activities. The programme was designed to be interlinked with Malawi's large‐scale fertiliser input subsidy programme through the implementation of the PWP in the planting months of the main agricultural season when the fertiliser distribution also occurs. Projects were mostly road rehabilitation or construction, with some afforestation and irrigation projects. The wage rate was USD 0.92/day for a total payment of USD 11.01 for a 12‐day wave, total of 4 waves.

Provider: Malawi government

Delivery: payments in the study districts were facilitated by the research team for the purposes of the evaluation, with physical delivery of the cash in conjunction with the district officials.

Co‐interventions: the national fertiliser subsidy programme provided fertiliser coupons that allow two bags of fertiliser to be purchased for MK 500 each. These coupons are more likely to be available to treated HHs.

10 poor and able‐bodied HHs per community were offered the programme; communities (144/38)

Food security:

  • Food Security Score

Dietary diversity:

  • Food Consumption Score

  • Number of food groups consumed

  • Food Security Score

3/4 months

Porter 2016 (Ethiopia)

Prospective controlled study

High

Programme name: Productive Safety Net Program (PSNP)

Programme description and frequency: 80% public works programme (food/cash‐for‐work; USD 0.56/day in 2008) and 20% unconditional transfers to those unable to work (value NR). Programme operated seasonally but predictably, i.e. not emergency.

Provider: Ethiopian government, with donor funding

Delivery: centrally co‐ordinated by Government

Co‐interventions: none reported

Poor and food insecure rural HHs (682/924)

Anthropometry (results presented for all programme participants; not disaggregated according to type of intervention received)

  • HAZ

  • WAZ

5 and 7 years

Katz 2001 (Nepal)

Prospective controlled study

High

Programme name: N/A

Programme description and frequency: part‐time (5 hours/week) employment for women; distributing weekly supplements to and recording data on married women of child‐bearing age in own or neighbouring communities. Monthly income valued at USD 15

Provider: Joint undertaking by USAID, academic institutions (Johns Hopkins University), NGOs (National Society for the Prevention of Blindness, Kedia Seva Mandir) and the Nepalese government

Delivery: NR

Co‐interventions: approximately 31% of women employed by the project reported having additional cash employment, but amounts are unknown

Women living in rural areas (350/520)

Anthropometry:

  • MUAC

2 years

aOverall risk of bias based on risk for selection and attrition bias

ADRA: Adventist Development and Relief Agency; BDT: Bangladeshi taka; BMI: body mass index; FFS: Farmer Field School; HAZ: height‐for‐age z‐score; HDDS: Household Dietary Diversity Score; HFIAS: Household Food Insecurity Access Scale; HH: household; LAZ: length‐for‐age z‐score; MUAC: mid‐upper arm circumference; NGO: non‐governmental organisation; NR: not reported; RCT: randomised controlled trial; SD: standard deviation; WLZ: weight‐for‐length z‐score; WAZ: weight‐for‐age z‐score; WEG: Women Empowerment Group.

Eight studies assessed interventions addressing food prices: four RCTs evaluated the effects of food vouchers (Fenn 2015; Hidrobo 2014; Jensen 2011; Ponce 2017); one cRCT (Chen 2019) and three PCS (Sturm 2013; Andaleeb 2016; Chakrabarti 2018) evaluated the effects of food and nutrition subsidies.

Two studies assessed social environment interventions: one cRCT (Kusuma 2017b) and one PCS (Brunie 2014) evaluated an intervention addressing the social environment, namely the effects of village savings and loans (VSL) and community grants.

Some studies assessed more than one type or category of intervention. Hidrobo 2014 and Fenn 2015 included a group for a CCT and another for food vouchers. Baird 2013 included two groups for conditional and unconditional cash transfers. Porter 2016 assessed a public works intervention providing either cash or food for work, or an UCT.

Participants

Twenty‐five studies included children or households in which children lived. Of these, five studies included households with children under 18 months of age (Andersen 2015; Fernald 2011; Marquis 2018; Olney 2016; Tonguet Papucci 2015); 10 studies included households with children under six years of age (Ahmed 2019a; Ahmed 2019b; Alaofe 2019; Daidone 2014; Fenn 2015; Kennedy 1989; Kurdi 2019; Osei 2017; Renzaho 2017; Verbowski 2018), and six with children under 18 years of age (Chen 2019; Kandpal 2016; Kusuma 2017b; Kusuma 2017a; Lopez Arana 2016; Pellerano 2014); three included households with children, without specifying their age (Aguero 2006; Asfaw 2014; Huerta 2006 (PROGRESA)). Baird 2013 included girls 13 to 22 years of age who had never married.

Five studies included adults; in one study, these were members of a healthcare plan (Sturm 2013), one included farmers (Murshed E Jahan 2011), one include men and women (Doocy 2017), and three studies included only women and their respective households (Alaofe 2016; Alaofe 2019; Katz 2001).

Twenty‐nine studies included households without specifying the inclusion of children (Andaleeb 2016; Asadullah 2015; Beegle 2017; Breisinger 2018; Brugh 2018; Brunie 2014; Chakrabarti 2018; Darrouzet Nardi 2016; Evans 2014; Ferre 2014; Gangopadhyay 2015; Gertler 2000 (PROGRESA); Haushofer 2013; Hidrobo 2014; Hjelm 2017; Hoddinott 2013; Jensen 2011; Jodlowski 2016; Kangmennaang 2017; Leroy 2008 (PROGRESA); Macours 2012; Maluccio 2005; Merttens 2013; Miller 2011; Ponce 2017; Porter 2016; Schwab 2013; Skoufias 2013; Weinhardt 2017).

We extracted information from included studies on the following PROGRESS‐Plus characteristics: age, place of residence, sex, ethnicity and language, occupation, education, socioeconomic status and social capital, where this was available. There was considerable variation in the reporting of these characteristics. Most studies (48/59) reported on an aspect of socioeconomic status, with 38 studies on age, 37 on sex, 34 on place of residence, 32 on education, 17 on ethnicity and language, 16 studies on occupation and 13 studies on social capital.

Setting and context

Most included studies were conducted in Africa (27): one each in Egypt (Breisinger 2018), the Democratic Republic of the Congo (Doocy 2017), Ghana (Marquis 2018), Ethiopia (Porter 2016), Lesotho (Pellerano 2014), Mozambique (Brunie 2014), Niger (Hoddinott 2013), and Tanzania (Evans 2014); two each in Benin (Alaofe 2016; Alaofe 2019), Burkina Faso (Olney 2016; Tonguet Papucci 2015), and South Africa (Aguero 2006; Sturm 2013); three in Zambia (Daidone 2014; Jodlowski 2016; Hjelm 2017); four in Kenya (Asfaw 2014; Haushofer 2013; Kennedy 1989; Merttens 2013); six in Malawi (Baird 2013; Beegle 2017; Brugh 2018; Kangmennaang 2017; Miller 2011; Weinhardt 2017).

Nineteen included studies were conducted in Asia: five in Bangladesh (Ahmed 2019a; Ahmed 2019b; Asadullah 2015; Ferre 2014; Murshed E Jahan 2011); four in Nepal (Darrouzet Nardi 2016; Katz 2001; Osei 2017; Renzaho 2017); three in India (Andaleeb 2016; Chakrabarti 2018; Gangopadhyay 2015) and two in China (Jensen 2011; Chen 2019), Indonesia (Kusuma 2017b; Kusuma 2017a); and one each in Cambodia (Verbowski 2018), Pakistan (Fenn 2015), and Philippines (Kandpal 2016).

Five included studies were conducted in South America: one each in Colombia (Lopez Arana 2016) and Peru (Andersen 2015), and three in Ecuador (Fernald 2011; Hidrobo 2014; Ponce 2017).

Two studies were conducted in Nicaragua, Central America (Macours 2012; Maluccio 2005), and four studies took place in Mexico, North America (Gertler 2000 (PROGRESA); Huerta 2006 (PROGRESA); Leroy 2008 (PROGRESA); Skoufias 2013). Two studies were conducted in Yemen, Middle East (Kurdi 2019; Schwab 2013).

All studies specifically targeted poor communities or households except two; one that included data from supermarkets in urban areas in South Africa (Sturm 2013), and one that targeted children enrolled in elementary schools in rural China (Chen 2019). Of those targeting communities, 24 studies did not specify the type of communities, 29 included rural communities, including farming communities, and four included urban communities.

Outcome measures

No included study assessed the primary outcome, namely the prevalence of undernourishment (i.e. people with insufficient food intake to meet their dietary requirements).

Eleven studies reported household expenditure on food (Alaofe 2016; Asfaw 2014; Brugh 2018; Ferre 2014; Hjelm 2017; Kennedy 1989; Macours 2012; Maluccio 2005; Merttens 2013; Miller 2011; Sturm 2013). Household expenditure was reported using different units, for example, household food expenditure per day, week or month; or as a proportion of total weekly or monthly household expenditure. One of these studies reported sales data, including the ratio of expenditure on healthy foods, on fruits and vegetables and on less desirable foods, compared to the total food expenditure (Sturm 2013).

Food security was reported as food security indices and dietary diversity measures. Thirteen studies reported food security outcomes using measures such as the proportion of participants experiencing food security or food deficit always, of households consuming more than one meal per day, Household Food Insecurity Access Scale (HFIAS) and Food Security Index (FSI) (Asadullah 2015; Beegle 2017; Brugh 2018; Brunie 2014; Daidone 2014; Doocy 2017; Haushofer 2013; Hjelm 2017; Kangmennaang 2017; Miller 2011; Osei 2017; Pellerano 2014; Weinhardt 2017). Twenty‐four studies reported on dietary diversity using a variety of measures including individual and Household Dietary Diversity Scores (HDDS), Food Consumption Scores (FCS), minimum dietary diversity (MDD) or minimum acceptable food consumption (Ahmed 2019a; Ahmed 2019b; Alaofe 2019; Asfaw 2014; Beegle 2017; Breisinger 2018; Brugh 2018; Brunie 2014; Chen 2019; Daidone 2014; Darrouzet Nardi 2016; Doocy 2017; Ferre 2014; Hidrobo 2014; Jodlowski 2016; Kurdi 2019; Marquis 2018; Merttens 2013; Miller 2011; Olney 2016; Pellerano 2014; Ponce 2017; Skoufias 2013; Tonguet Papucci 2015). Definitions for the food security and dietary diversity measures reported in included studies are provided in Table 6.

Open in table viewer
Table 6. Food security and dietary diversity indices reported by included studies

Index/scale (study ID of studies reporting this measure)

Definition

Interpretation

Reference cited

Household food security indices

Household Food Insecurity Access Scale (HFIAS)

(Daidone 2014; Hjelm 2017; Kangmennaang 2017)

or

Household Food Insecurity Access Prevalence (HFIAP)

(Doocy 2017; Osei 2017; Weinhardt 2017)

HFIAS: sum of responses to 9 questions related to 4 domains of food security of a HH during the past 4 weeks.

HFIAP: categorises HHs into 4 levels of HH food insecurity, based on the frequency and severity of food insecurity experienced by HHs.

HFIAS: score ranges from 0 to 27. The higher the score the more food insecure the HH.

HFIAP: categorised as: food secure, and mild, moderately and severely food insecure.

Coates J, Swindale A, Bilinsky P. Household Food Insecurity Access Scale (HFIAS) for measurement of food access: indicator guide. Version 3. Washington, DC: Academy for Educational Development;2006

Food Security Score

(Beegle 2017)

Scores HHs in terms of 4 levels of HH food insecurity, based on the frequency and severity of food insecurity experienced by HHs.

Ranges from –1 to –4; higher value indicates greater food security

World Food Programme

Resilience index

(Beegle 2017)

Based on the World Food Program Coping Strategy Index. Weighted sum of the number of days in the past 7 days that HHs had to reduce the quantity and quality food consumed.

Higher values indicate food security

Maxwell D, Caldwell R. The Coping Strategies Index: Field methods Manual. Cooperative for Assistance and Relief Everywhere, Inc. (CARE), January 2008.

Food Security Index (FSI)

(Pellerano 2014)

Study authors adapted the food security component of the Bristol Child Deprivation Index. It is a simple mean of 3 questions related to child food security.

Severe food deprivation: FSI > 2.

Gordon D, Nandy S, Pantazis C, Pemberton S, Townsend P. (2003), the Distribution of Child Poverty in the Developing World, Policy Press, Centre for International Poverty Research, University of Bristol, July 2003.

Food Security Index

(Haushofer 2013)

Weighted mean of 17 outcome measures of food security and hunger.

The higher the index, the greater the food security

No reference cited

HHdietary diversity indices

HDDS

(Alaofe 2019; Breisinger 2018; Brunie 2014; Daidone 2014; Hidrobo 2014; Jodlowski 2016a; Kurdi 2019; Merttens 2013; Olney 2016b)

Sum of the number of food groups consumed by a HH during the past day or week, or longer (e.g. 2 or 4 weeks). Food groups included cereals, roots and tubers, vegetables (included vitamin A‐rich vegetables and tubers, dark leafy vegetables and other), fruits (included vitamin A fruits and other), meat (includes organ meat and flesh meat), eggs, fish, pulses and legumes, fats and oil, sugar and sweets, milk and other milk product, and spices and beverages.

Score ranges from 0 to 12; higher score reflected higher level of dietary diversity.

Kennedy G, Ballard T, Dop M, 2011. Guidelines for Measuring Household and Individual Dietary Diversity. Food and Agriculture Organization, Rome.

Swindale A, Bilinsky P. Household dietary diversity score (HDDS) for measurement of household food access: indicator guide (v.2). Washington (DC): FHI 360/FANTA; 2006.

Dietary Diversity Index (DDI)

(Hoddinott 2013; Pellerano 2014)

or

Dietary Diversity Score (DDS)

(Asfaw 2014)

or

Food diversity composite score (Miller 2011)

Sum of the number of food groups consumed by a HH during the past week. Food groups included main staples, pulses, vegetables, fruit, meat (or fish or egg); dairy products, sugar and oil.

Score ranges from 0 to 8; higher score reflects higher level of dietary diversity.

Ruel M. 2003. Operationalizing dietary diversity: a review of measurement issues and research priorities. Journal of Nutrition 133, 3911S–3926S.

Dietary Diversity Index (DDI)

(Hoddinott 2013);

or

Dietary Diversity Score (DDS)

(Hidrobo 2014; Schwab 2013)

Sum of the number of distinct food items consumed by a HH during the previous week. Depended on the number of food items included in the dietary questionnaire.

Score ranges from 0 to 25 (Hoddinott 2013); 0 to 40 (Hidrobo 2014); 0 to 39 (Schwab 2013); higher score reflects higher level of dietary diversity.

Ruel M. 2003. Operationalizing dietary diversity: a review of measurement issues and research priorities. Journal of Nutrition 133, 3911S–3926S.

Food Consumption Score (FCS)

(Ahmed 2019a; Ahmed 2019b; Beegle 2017; Hidrobo 2014; Hoddinott 2013; Pellerano 2014; Ponce 2017)

Weighted sum of the consumption frequency of the 8 food groups consumed by a HH during the past week. Food groups include main staples, pulses, vegetables, fruit, meat (or fish or egg), dairy products, sugar and oil.

Maximum score is 112 or 126.

Acceptable food consumption: FCS ≥ 35;

Borderline food consumption:

FCS between 21 and 35;

Poor food consumption: FCS < 35

WFP, 2008. Food consumption analysis: Calculation and use of the food consumption score in food security analysis. World Food Programme, Rome

Individual dietary diversity indices

Individual Child Dietary Diversity score (IDDS)

(Darrouzet Nardi 2016; Hoddinott 2013; Marquis 2018; Pellerano 2014; Skoufias 2013; Tonguet Papucci 2015)

Sum of number of food groups consumed by a child aged 6–23 months or a child aged < 5 years during the past 24 hours calculated from 17 foods, aggregated into 7 food groups: starchy staples (grains and white potatoes); vitamin A‐rich fruits and vegetables; other fruits and vegetables; offal, meat, and fish; eggs; legumes, nuts, and seeds; milk and dairy products

Score ranges from 0 to 7; higher score reflects higher level of dietary diversity.

Minimum dietary diversity: Dietary Diversity Score ≥ 4

World Health Organization, 2010. Indicators for Assessing Infant and Young Child Feeding Practices. World Health Organization, Geneva.

Individual Child Dietary Diversity Score (IDDS)

(Brunie 2014)

Sum of the number of different food groups consumed during the past day by a child aged < 5 years (12 food groups).

Score ranges from 0 to 12; higher score reflects higher level of dietary diversity

Guidelines for measuring household and individual dietary diversity.

FAO Nutrition – 2007 – FAO, Rome (Italy)

Women's Dietary Diversity Score (WDDS‐10)

(Alaofe 2019)

Sum of the number of food groups consumed during the past 24 hours calculated from the following food groups: starchy staples; beans and peas; nuts and seeds; dairy; flesh foods; eggs; vitamin A‐rich dark green leafy vegetables; other vitamin A‐rich vegetables and fruits; other fruits and other vegetables.

Score ranges from 0 to 10; higher score reflects higher level of dietary diversity

Kennedy G, Ballard T, Dop M, 2011. Guidelines for Measuring Household and Individual Dietary Diversity. Food and Agriculture Organization, Rome.

aJodlowski 2016: modified HDDS to a total score out of 13.
bOlney 2016: the egg food group was not included because of an oversight during survey design.

HH: household.

Six studies reported adequacy of dietary intake (Ahmed 2019a; Ahmed 2019b; Andaleeb 2016; Brugh 2018; Jensen 2011; Kennedy 1989). Measures reported included the proportion of calorie‐deficient households and of preschool children meeting caloric requirements; mineral and vitamin sufficiency indices; calorie‐deficient households; and ratio of caloric, protein and fat intake to the dietary recommendations. Many studies reported intake in terms of calories or nutrients consumed without relating it to a measure of adequacy; these measures were not reported in this review.

A variety of anthropometric measures were reported in included studies. Twenty‐seven studies reported on measures of stunting in children (i.e. chronic undernutrition), such as the proportion stunted (HAZ < –2 standard deviations (SD)), severely stunted (HAZ < –3SD) or mean HAZ (Aguero 2006; Ahmed 2019a; Ahmed 2019b; Andersen 2015; Asfaw 2014; Daidone 2014; Darrouzet Nardi 2016; Doocy 2017; Evans 2014; Fenn 2015; Fernald 2011; Ferre 2014; Kandpal 2016; Kennedy 1989; Kurdi 2019; Kusuma 2017b; Kusuma 2017a; Leroy 2008 (PROGRESA); Lopez Arana 2016; Macours 2012; Maluccio 2005; Marquis 2018; Merttens 2013; Osei 2017; Renzaho 2017; Tonguet Papucci 2015; Verbowski 2018). Twenty studies reported on measures of wasting in children (i.e. acute undernutrition), such the proportion wasted (WHZ < –2SD), severely wasted (WHZ < –3SD) or mean WHZ (Ahmed 2019a; Ahmed 2019b; Asfaw 2014; Daidone 2014; Evans 2014; Fenn 2015; Ferre 2014; Kennedy 1989; Kurdi 2019; Kusuma 2017b; Kusuma 2017a; Leroy 2008 (PROGRESA); Lopez Arana 2016; Maluccio 2005; Marquis 2018; Merttens 2013; Osei 2017; Renzaho 2017; Tonguet Papucci 2015; Verbowski 2018). Twenty‐seven studies reported on measures of underweight in women and children, including WAZ or the proportion of underweight based on these (i.e. WAZ < –2SD), BMI for age or mean BMI, or mid‐upper arm circumference (MUAC) (Alaofe 2019; Andersen 2015; Asfaw 2014; Brunie 2014; Chen 2019; Daidone 2014; Darrouzet Nardi 2016; Doocy 2017; Evans 2014; Fenn 2015; Ferre 2014; Kandpal 2016;Katz 2001Kennedy 1989; Kusuma 2017b; Kusuma 2017a; Lopez Arana 2016; Macours 2012; Maluccio 2005; Marquis 2018; Merttens 2013; Olney 2016; Osei 2017; Pellerano 2014; Renzaho 2017; Verbowski 2018; Weinhardt 2017).

Six studies reported biochemical outcomes, including haemoglobin in five studies (Chen 2019; Fenn 2015; Fernald 2011; Osei 2017; Verbowski 2018) and vitamin A and iron deficiency in one study (Alaofe 2019).

Five studies reported cognitive function and development outcomes using a variety of measures including Ravens Colored Matrixes and other cognitive tests, Early Childhood Development Index, individual cognitive function measures such as language and memory, and grade attainment (Andersen 2015; Baird 2013; Daidone 2014; Fernald 2011; Macours 2012). Definitions for cognitive function and development measures reported in included studies are described in Table 7.

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Table 7. Summary of cognitive function indices reported by included studies

Index or scale

Definition/ measurement

Interpretation

Reference

Early Childhood Development Index (ECD)

(Daidone 2014)

Measures 4 developmental domains of children aged 3–7 years: physical (both gross and fine motor), language and cognition, socioemotional and approaches to learning.

Maximum score of 10; the higher the score the better functioning

Raven's Colored Progressive Matrices test score

(Baird 2013)

Non‐verbal test that measures abstract reasoning of children aged ≥ 5 years.

Maximum test score 60; the higher the score the better the abstract reasoning.

IDHC‐B test score

MacArthur‐Bates Communicative Development Inventory (adapted Spanish version)

(Fernald 2011)

Measures early language skills of children aged 12–35 months using parental report.

Scores range from 0 to 100 with 0 indicating that a child had not said any word on the checklist and 100 indicating that a child had said every word on the list.

Jackson‐Maldonado D, Thal D, Marchman V, Newton T, Fenson L, Conboy B. (2003). MacArthur Inventarios del Desarrollo de Habilidades Comunicativas. User's Guide and Technical Manual. Baltimore: Brookes Publishing.

TVIP test score

Peabody Picture Vocabulary Test (PPVT) (adapted Spanish version).

(Fernald 2011)

Measures receptive language/vocabulary of children aged ≥ 36 months.

Age‐adjusted norms: mean score of 100 and standard deviation of 15 at every age.

Woodcock‐Johnson‐Munoz battery test scores

(Fernald 2011)

WJ1 test measures long‐term memory in early childhood

Age‐adjusted percentile score

Woodcock, Richard, and Ana Munoz‐Sandoval. 1996. BaterıaWoodcock‐Munoz Pruebas de Aprovechamiento‐Revisada. Chicago: Riverside.

WJ2 test measures short‐term memory or immediate recall in early childhood

Age‐adjusted percentile score

WJ5 test measures visual integration, or visual‐spatial processing in early childhood

Age‐adjusted percentile score

Four studies reported mental health outcomes including measures such as depression score, stress, psychological distress and psychological well‐being (Baird 2013; Fernald 2011; Haushofer 2013; Hjelm 2017).

Seventeen studies reported morbidity outcomes (Ahmed 2019a; Ahmed 2019b; Alaofe 2019; Asadullah 2015; Chen 2019; Daidone 2014; Evans 2014; Fenn 2015; Gertler 2000 (PROGRESA); Kandpal 2016; Kennedy 1989; Macours 2012; Merttens 2013; Osei 2017; Pellerano 2014; Tonguet Papucci 2015; Verbowski 2018). Various measures of morbidity were reported including incidence of respiratory infections, diarrhoea and anaemia; the proportion of participants who were ill in a specified reference period or the number of days or percent of time ill.

No studies reported specific adverse events. We had specified that overweight and obesity would be considered adverse events in this review, and three studies reported this outcome in young and older children (Andersen 2015; Lopez Arana 2016; Pellerano 2014); however, not as adverse events per se.

Funding and conflicts of Interest

Most included studies were funded either by non‐profit organisations (including research institutes, world bank, non‐government organisations, etc) or governmental/intergovernmental agencies (or both) except for one study that was funded by a for‐profit organisation (Elanco Animal Health; Jodlowski 2016). One study did not disclose their funding (Gangopadhyay 2015).

Of 59 included studies, 39 did not report on potential conflicts of interest (COI) and 27 did. Of those that reported their COI, all declared that none of the authors had any potential COI.

Excluded studies

We excluded 297 studies. Of the excluded studies: 152 had an ineligible study design, seven did not have an eligible population or setting, 89 did not address an eligible intervention, 23 did not report on relevant outcomes and 26 were duplicates. A selection of 85 key excluded studies is reported in the Characteristics of excluded studies table.

Studies awaiting classification

We placed 39 studies awaiting classification because we could not assess their eligibility properly without access to the full text, or they were conference abstracts with insufficient data to include them in the review (see Characteristics of studies awaiting classification table).

Ongoing studies

We identified two studies that could potentially be included in the review once completed. Eleven studies were identified as ongoing. More details on these studies are available in the Characteristics of ongoing studies table.

Risk of bias in included studies

See the Characteristics of included studies table for more details for each domain of bias assessed for each study. Figure 3 presents a summary of the judgements per risk of bias items and Figure 4 presents the summary of the risk of bias judgments for each included study.


Risk of bias graph: review authors' judgements about each risk of bias item presented as percentages across all included studies.

Risk of bias graph: review authors' judgements about each risk of bias item presented as percentages across all included studies.


Risk of bias summary: review authors' judgements about each risk of bias item for each included study.

Risk of bias summary: review authors' judgements about each risk of bias item for each included study.

Allocation

Risk of selection bias is determined by whether participants were randomly allocated to the intervention groups (random sequence generation) and whether there was no possibility of altering the sequence while allocating participants to the intervention groups (allocation concealment).

Of the 36 included RCTs, 14 studies described adequate methods of random sequence generation and were at low risk of selection bias. Five studies described doing this using computer‐generated random numbers (Baird 2013; Beegle 2017; Fenn 2015; Marquis 2018; Verbowski 2018), one each used STATA software (Gertler 2000 (PROGRESA)) and a randomisation algorithm (Skoufias 2013), while the remaining seven studies randomised communities through public lottery events (Brugh 2018; Daidone 2014; Macours 2012; Maluccio 2005; Merttens 2013; Pellerano 2014; Tonguet Papucci 2015). Twenty‐two studies reported randomising individuals or clusters to intervention groups, but did not report how the random sequence had been generated and thus were at unclear risk of selection bias (Ahmed 2019a; Ahmed 2019b; Asfaw 2014; Chen 2019; Darrouzet Nardi 2016; Evans 2014; Fernald 2011; Gangopadhyay 2015; Haushofer 2013; Hidrobo 2014; Hjelm 2017; Hoddinott 2013; Jensen 2011; Kandpal 2016; Kurdi 2019; Kusuma 2017b; Kusuma 2017a; Miller 2011; Olney 2016; Osei 2017; Ponce 2017; Schwab 2013).

Of the 36 included RCTs, 20 reported that allocation was at the cluster level (communities, parishes, electoral divisions, etc.) and carried out at the beginning of the study, and these were classified at low risk of selection bias (Ahmed 2019a; Ahmed 2019b; Baird 2013; Beegle 2017; Brugh 2018; Chen 2019; Daidone 2014; Fenn 2015; Fernald 2011; Gertler 2000 (PROGRESA); Macours 2012; Maluccio 2005; Marquis 2018; Merttens 2013; Olney 2016; Osei 2017; Pellerano 2014; Schwab 2013; Skoufias 2013; Tonguet Papucci 2015). Three studies did not conceal allocation or report this, but household selection was done after villages had been allocated to each intervention group, and knowledge of allocation could have influenced the household selection process (Evans 2014; Hidrobo 2014; Kurdi 2019). In Gangopadhyay 2015, participants self‐selected into the intervention. These four studies were at high risk of selection bias. The remaining 12 studies did not report details on allocation concealment and were at unclear risk of selection bias (Asfaw 2014; Darrouzet Nardi 2016; Haushofer 2013; Hjelm 2017; Hoddinott 2013; Jensen 2011; Kandpal 2016; Kusuma 2017b; Kusuma 2017a; Miller 2011; Ponce 2017; Verbowski 2018).

All 23 PCS were at high risk of selection bias (both for random sequence and allocation concealment), according to EPOC's risk of bias tool guidance (EPOC 2017).

Baseline similarity in participants characteristics and outcome measures (selection bias)

Baseline imbalances in participant characteristics or outcome measures may occur in non‐randomised studies as well as in randomised studies in which the allocation procedure was not performed adequately.

Participants characteristics

In 46 included studies, there were no baseline imbalances reported for participant characteristics or, if there were, these were adjusted for in the analyses, and thus they were at low risk of selection bias (Figure 4). Four studies had significant differences at baseline that were not adjusted for in the analyses; thus they were at high risk of selection bias (Asadullah 2015; Beegle 2017; Kurdi 2019; Merttens 2013). Ten studies were at unclear risk of selection bias: six did not report any or some baseline characteristics (Aguero 2006; Breisinger 2018; Murshed E Jahan 2011; Ponce 2017; Porter 2016; Sturm 2013), one reported baseline characteristics but not whether these were balanced (Gangopadhyay 2015), one reported that characteristics were balanced at household level but not at province level (Jensen 2011), one reported discrepancies and it was unclear whether these were adjusted for in the analysis (Schwab 2013), and one only had baseline data for the group analysed, not for the entire sample (Chen 2019).

Outcome measures

Thirty‐one studies either were balanced at baseline with regards to outcome measures, or adjusted for any imbalance in the analyses, and were at low risk of selection bias (Figure 4). In seven studies there were significant baseline imbalances in relevant outcomes which were not controlled for in the analyses, and these were at high risk of selection bias (Andersen 2015; Asadullah 2015; Beegle 2017; Breisinger 2018; Merttens 2013; Renzaho 2017; Sturm 2013). The remaining 21 studies were classified at unclear risk of selection bias: 15 did not report any or relevant outcomes at baseline (Aguero 2006; Baird 2013; Chen 2019; Darrouzet Nardi 2016; Evans 2014; Gertler 2000 (PROGRESA); Hjelm 2017; Huerta 2006 (PROGRESA); Kandpal 2016; Kurdi 2019; Kusuma 2017b; Kusuma 2017a; Murshed E Jahan 2011; Porter 2016; Verbowski 2018); in three, the baseline data collection occurred after the intervention started, so true baseline data were not available (Hoddinott 2013; Lopez Arana 2016; Schwab 2013), in two studies it was unclear if reported imbalances were adjusted for (Doocy 2017; Osei 2017), and in one, although the outcomes were balanced at household level, there were imbalances at province level (Jensen 2011).

Blinding

Blinding participants and personnel to intervention allocation during the study helps prevent systematic differences in how participants are treated or behave during the trial due to knowledge of treatment allocation (performance bias). In the types of studies included in this review, blinding of participants and personnel was often not feasible; however, it is also unlikely that it would have influenced the behaviour of participants or personnel beyond that expected as part of the intervention, and thus less likely to be susceptible to performance bias. Thus, in all but one study included in this review, the risk of performance bias was low. One study was at high risk of performance bias as blinding was not possible and the delivery of the intervention, a nutrition subsidy to schools, was dependent on the school principal (Chen 2019).

Blinding of outcome assessors helps prevent systematic differences in how outcomes are assessed in either intervention groups due to knowledge of treatment allocation (detection bias). Fifteen studies were at low risk of detection bias; in 14 of these studies, blinding was not done; however, the outcomes measured and reported were objective and thus unlikely to have been influenced by knowledge of treatment allocation (Aguero 2006; Ahmed 2019a; Ahmed 2019b; Andersen 2015; Fernald 2011; Kusuma 2017b; Kusuma 2017a; Leroy 2008 (PROGRESA); Lopez Arana 2016; Marquis 2018; Osei 2017; Porter 2016; Renzaho 2017; Skoufias 2013). The other study was based on scanner sales data from supermarkets, which is not susceptible to detection bias due to lack of blinding (Sturm 2013). The remaining 44 studies were at high risk of detection bias either because there was no blinding or they included self‐reported or subjective outcomes that were susceptible to be influenced by knowledge of treatment allocation.

Protection against contamination (performance bias)

If the control group is exposed to the intervention intended for the intervention group, contamination occurs, introducing performance bias.

Thirty‐seven studies were at low risk of bias in this domain either because they reported evidence of no contamination, or because the intervention and control groups were allocated at the community/village/district level (i.e. in distinct geographical areas), which precludes contamination. Eight studies were at high risk of bias as they reported evidence of control group exposure to the intervention (Asfaw 2014; Doocy 2017; Ferre 2014; Huerta 2006 (PROGRESA); Jodlowski 2016; Kandpal 2016; Katz 2001; Kurdi 2019). The remaining 14 studies were at unclear risk (Aguero 2006; Andersen 2015; Asadullah 2015; Brunie 2014; Daidone 2014; Gertler 2000 (PROGRESA); Kangmennaang 2017; Lopez Arana 2016; Maluccio 2005; Murshed E Jahan 2011;Osei 2017; Ponce 2017; Porter 2016; Skoufias 2013). In these studies, the location or the distance between intervention and control communities was unclear, or the control and intervention households were in same community and there was potential for control households to have benefited from the intervention through interaction with intervention households (e.g. sharing), or communities were geographically near/adjacent.

Incomplete outcome data

Twenty‐five studies were at low risk of bias because they had low attrition (i.e. 10% or less) or because attrition between the groups was non‐differential or unrelated to the outcome. Seventeen studies were at high risk of attrition bias, because of high levels of attrition or they reported differential attrition between intervention groups or characteristics of those lost to follow‐up were different from those remaining in the study, or a combination of these (Andaleeb 2016; Asadullah 2015; Asfaw 2014; Chen 2019; Doocy 2017; Huerta 2006 (PROGRESA); Kandpal 2016; Kangmennaang 2017; Katz 2001; Kurdi Leroy 2008 (PROGRESA); Lopez Arana 2016; Merttens 2013; Ponce 2017; Schwab 2013; Skoufias 2013; Weinhardt 2017). Skoufias 2013 reported only a 5% difference in attrition between groups but lost one entire cluster and participants were excluded from the analysis were different than those included in the analysis, thus was classified at high risk. Seventeen studies were classified at unclear risk of attrition bias, as they either did not report attrition at all or did not report enough information to make this judgement (Aguero 2006; Alaofe 2016; Alaofe 2019; Beegle 2017; Brunie 2014; Chakrabarti 2018; Darrouzet Nardi 2016; Fernald 2011; Ferre 2014; Gertler 2000 (PROGRESA); Hjelm 2017; Kennedy 1989; Olney 2016; Osei 2017; Porter 2016; Sturm 2013; Verbowski 2018). Sturm 2013 analysed supermarket sales scanner data and did not report if any of these data were excluded or missing.

Selective reporting

Selective outcome reporting occurs when authors do not report on all outcomes prespecified and assessed in the study. Six studies were at low risk of bias, as they reported the same outcomes that were prespecified in the trial registry (Baird 2013; Fenn 2015; Haushofer 2013; Olney 2016; Verbowski 2018; Weinhardt 2017).

One study was at high risk of bias because some of the morbidity outcomes (oedema and measles) reported in protocol were not reported in the published paper (Tonguet Papucci 2015).

The remaining 52 studies were at unclear risk of bias as there were no protocols available.

Other potential sources of bias

Under other potential sources of bias we considered whether the study could have been influenced by 1. misclassification bias of the exposure (i.e. when exposure to the intervention was self‐reported); 2. measurement bias (i.e. whether outcomes were measured appropriately; 3. incorrect analysis, in the case of cRCTs (i.e. whether study data were adjusted for clustering. Such analyses do not lead to biased estimates of effect but in the meta‐analysis such studies receive undue weight leading to overprecision of the effect estimate); and 4. recruitment bias, in the case of cRCTs (i.e. whether recruitment of participants was done before allocation of clusters to intervention groups).

Twenty‐two studies were at low risk as no other potential sources of bias were identified. Six studies were at high risk of bias as at least one other potential source of bias was identified (Alaofe 2019; Asfaw 2014; Gertler 2000 (PROGRESA); Huerta 2006 (PROGRESA); Osei 2017; Schwab 2013). Asfaw 2014 was at high risk for misclassification bias as receipt of the intervention was based on self‐report. Three were at high risk of recruitment bias as clusters were assigned before households were recruited (Asfaw 2014; Gertler 2000 (PROGRESA); Osei 2017). Two studies were at high risk of measurement bias: in Huerta 2006 (PROGRESA) preliminary analyses showed evidence of reporting error regarding health outcomes, and in Alaofe 2019 dietary data were collected with only one 24‐hour recall. Schwab 2013 was at high risk of other bias due to the different timing of implementation of interventions in each group. The remaining 31 studies were at unclear risk of other bias; in these studies there was at least one of the other potential sources of bias for which there was insufficient information to make a judgement.

Effects of interventions

See: Summary of findings 1 Unconditional cash transfers compared to no intervention for food security; Summary of findings 2 Conditional cash transfers compared to no intervention for food security; Summary of findings 3 Income‐generation interventions compared to no intervention for food security; Summary of findings 4 Food vouchers compared to no intervention for food security; Summary of findings 5 Food and nutrition subsidies compared to no intervention for food security; Summary of findings 6 Social support compared to no intervention for food security

We present the effects of interventions on primary and secondary outcomes separately for each category of intervention as outlined below (see Table 2 for definitions of intervention categories and types).

  • Interventions that improved buying power:

    • Unconditional cash transfers

    • Conditional cash transfers

    • Income generation interventions

  • Interventions that addressed food prices:

    • Food prices – food vouchers

    • Food prices – food and nutrition subsidies

  • Interventions that addressed the social environment

    • Social support interventions (community grants/savings schemes)

We found no studies addressing the intervention category of infrastructure changes, which we had intended to include in the review.

The 'Summary of findings' tables provide an overview of effects on all primary outcomes and key secondary outcomes, for each comparison.

Comparison 1: unconditional cash transfers

Twenty‐one included studies assessed UCTs, where a specific amount of money was transferred to poor families monthly or once every two months, with no conditions regarding behaviours expected from the families. Fourteen cRCTs (Ahmed 2019a; Ahmed 2019b; Asfaw 2014; Baird 2013; Brugh 2018; Daidone 2014; Fenn 2015; Fernald 2011; Hjelm 2017; Merttens 2013; Miller 2011; Pellerano 2014; Skoufias 2013; Tonguet Papucci 2015), two RCTs (Gangopadhyay 2015; Haushofer 2013), and three PCS (Aguero 2006; Breisinger 2018; Renzaho 2017) assessed the effects of UCTs versus no intervention. Two cRCTs assessed UCTs versus food transfers (Hoddinott 2013; Schwab 2013).

Five cRCTs reported on the proportion of household expenditure on food (Asfaw 2014; Brugh 2018; Hjelm 2017; Merttens 2013; Miller 2011). Five cRCTs (Brugh 2018; Daidone 2014; Hjelm 2017; Miller 2011; Pellerano 2014) and one RCT (Haushofer 2013) reported on various food security measures, and 10 cRCTs reported on various dietary diversity measures (Ahmed 2019a; Ahmed 2019b; Asfaw 2014; Brugh 2018; Daidone 2014; Merttens 2013; Miller 2011; Pellerano 2014; Skoufias 2013; Tonguet Papucci 2015). Eight cRCTs (Asfaw 2014; Daidone 2014; Fernald 2011; Merttens 2013; Pellerano 2014; Tonguet Papucci 2015; Fenn 2015; Ahmed 2019a; Ahmed 2019b), and one PCS (Aguero 2006) reported various anthropometric measures. Two clusters RCT reported and biochemical indicators (Fernald 2011; Fenn 2015), and three cRCTs reported on cognitive function and development outcomes (Baird 2013; Daidone 2014; Fernald 2011). Three cRCTs (Baird 2013; Fernald 2011; Hjelm 2017) and one RCT (Haushofer 2013) reported on measures of mental well‐being. Seven cRCTs reported measures of morbidity (Ahmed 2019a; Ahmed 2019b; Daidone 2014; Fenn 2015; Merttens 2013; Pellerano 2014; Tonguet Papucci 2015), and one cRCT reported adverse effects (Pellerano 2014). Hoddinott 2013 and Schwab 2013, the cRCTs where the comparison group was food transfers, reported on measures of dietary diversity, and Schwab 2013 also reported measures of food security.

Further details about the studies in this comparison are presented in Table 8. Table 9 presents results of individual trials included and Table 10 presents results of individual PCS included, on all reported outcomes. The summary of findings Table 1 and the harvest plot in Figure 5 summarise the effects of UCTs on key outcomes.

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Table 8. Unconditional cash transfers – overview of included studies

Study ID (country)

Study design

Overall risk of biasa

Other key details of intervention

Population (sample size at baseline: intervention/control)

Outcome domains and measures with available data

Timepoint of measurement

UCTs vs no intervention

Baird 2013

(Malawi)

cRCT

Low

Programme name: Schooling, Income, and Health Risks study (SIHR). Includes unconditional and conditional groups.

Amount and frequency of payments: payments split between guardian and girl in each HH.

HH amount varied randomly (USD 4, USD 6, USD 8, USD 10 per month). Amount paid to girl beneficiaries varied randomly (USD 1, USD 2, USD 3, USD 4, USD 5 per month).

Provider: NGOs

Delivery: payments to girl beneficiaries at local distribution points

Co‐interventions: none reported

Adolescent girls who were never married, aged 13–22 years, in urban and rural HHs (526/1495)

Cognitive function and development:

  • Raven's Coloured Progressive Matrices

Anxiety and depression:

  • Psychological distress score (GHQ‐12)

1 and 2 years

Brugh 2018

(Malawi)

cRCT

Low

Programme name: Malawi Social Cash Transfer Scheme (SCTS)

Amount and frequency of payments: about USD 40 (depending on HH size and number of school‐aged children); monthly transfers. Top‐up payments made for children at primary and secondary school. At follow‐up, intervention HHs had received 5 or 6 bi‐monthly cash transfer payments, due to an administrative delay.

Provider: Government

Delivery: NR

Co‐interventions: None reported

Ultra‐poor and labour constrained HHs (1561/1729 HHs; Mangochi and Salima districts

HH expenditure on food:

  • Proportion of total HH expenditure per year

Food security:

  • Worried not enough food

  • Consume > 1 meal per day

Dietary diversity:

  • Household Dietary Diversity Score (HDDS)

Adequacy of dietary intake:

  • Food energy deficiency

  • Depth of hunger

1 year

Daidone 2014

(Zambia)

cRCT

Low

Programme name: Child Grant Programme (CGP)

Amount and frequency of payments: about USD 12 per month, regardless of HH size; payments made every other month

Provider: government

Delivery: payments through local pay point manager

Co‐interventions: none reported

1260 HHs (7254 individuals)/1259 HHs (7091 individuals)

Food security:

  • Consuming > 1 meal/day

  • HFIAS

Dietary diversity:

  • HDDS

Anthropometry:

  • WAZ

  • HAZ

  • WHZ

Cognitive function and development:

  • ECD index

Morbidity: children aged 0–60 months

  • ARI

  • Diarrhoea

2 years

Fenn 2015

(Pakistan)

cRCT

Low

Programme name: REFANI Pakistan standard cash transfer

Amount and frequency of payments: PKR 1500 (about USD 14) disbursed monthly for 6 consecutive months.

Provider: EU; DG ECHO; Action Against Hunger field staff.

Delivery: mobile banks in a central location or central banks serving a number of villages. Verbal messaging from Action Against Hunger field staff at distribution that children should benefit from the transfers.

Co‐interventions: WINS programme in all villages – provided outpatient treatment for children aged 6 (SD 59) months with SAM, micronutrient supplementation (children, pregnant and lactating women), and behaviour change communication.

Poor and very poor agrarian HHs (standard cash group: 31 villages/632 HHs; Double cash group: 24 villages/600 HHs; fresh food voucher group: 31 villages/632 HHs; control group: 28 villages/632 HHs

Anthropometric indicators:

  • BMI (mothers)

  • HAZ

  • Stunting (HAZ < –2SD) and severe stunting (HAZ < –3SD)

  • WHZ

  • Wasting (WHZ < –2SD) and severe wasting (WHZ < –3SD)

  • MUAC

Biochemical indicators:

  • Hb (children)

  • Hb (mothers)

  • anaemia (children)

  • anaemia (mothers)

Morbidity: child:

  • ARIs

  • Diarrhoea

6 and 12 months

Pellerano 2014

(Lesotho)

cRCT

Low

Programme name: Lesotho Child Grants Programme (CGP)

Amount and frequency of payments: about USD 12 per month every 3 months. From 2013 (after 2 years) transfer indexed to number of children in the HH. Payments not made as predicted; smaller number of payments made involving larger amounts.

Provider: government; UNICEF‐Lesotho

Delivery: cash‐in‐transit firm provided payments at community pay points.

Co‐interventions: all CGP HHs received bi‐monthly top‐up for a specific period for a Food Emergency Grant.

Ultra‐poor rural HHs with children 0–17 years (706/647 HHs)

Food security:

  • Severe food deprivation (FSI > 2)

Dietary diversity:

  • FCS

  • Acceptable food consumption (FCS > 35)

Anthropometry:

  • Underweight (WAZ < third percentile)

Morbidity: children aged 0–5 years:

  • Any illness in previous month

Adverse events:

  • Overweight (children)

2 years

Tonguet Papucci 2015

(Burkina Faso)

cRCT

Low

Programme name: Moderate Acute Malnutrition Out (MAM'Out) project.

Amount and frequency of payments: seasonal payments – about USD 17 from July to November.

Provider: European Commission Humanitarian Aid (ECHO) trained project staff

Delivery: mothers received card linked to electronic account and mobile phone. Payments provided via phones and cash withdrawal points.

Co‐interventions: ongoing national social protection policy that promoted social transfer mechanisms to the poorest and most vulnerable.

Poor rural HHs with ≥ 1 child aged < 1 year (644/634 children; 602/583 HHs)

Dietary diversity:

  • MDD

  • Minimum acceptable diet

Anthropometric indicators:

  • WHZ

  • Stunting (HAZ < –2SD)

  • MUAC

Morbidity: child:

  • Diarrhoea

  • ARIs

2.4 years

Ahmed 2019a; Ahmed 2019b

(Bangladesh)

cRCT

Unclear

Programme name: Transfer Modality Research Initiative (TMRI) (2 trials implemented in the North and South of Bangladesh reported in the same paper).

Amount and frequency of payments: Monthly payment of BDT 1500 (about USD 19) per HH.

Provider: United Nations' World Food Program (WFP); NGO (Eco‐Social Development Organization or ESDO)

Delivery: a mobile phone was provided to the mother who collected payments from distribution sites using mobile verification of identity.

Co‐interventions: none reported

Rural HHs in the northwest and southern regions (North: 458/450; South: 454/464 HHs)

Dietary diversity:

  • FCS

  • Poor food consumption (FCS < 35)

Adequacy of dietary intake:

  • Food poverty (daily caloric intake < 2122 kcal)

Anthropometric indicators:

  • WHZ

  • WAZ

Morbidity: children:

  • Diarrhoea in the previous 2 weeks

2 years

Fernald 2011

(Ecuador)

cRCT

Unclear

Programme name: Bono de Desarrollo Humano (BDH) programme

Amount and frequency of payments: USD 15 per month; could accumulate payments for up to 4 months.

Provider: government

Delivery: payments to mothers via the banking system.

Co‐interventions: none reported

Rural and urban parishes; poor families who had children aged 0–6 years at baseline (1388/681 children)

Anthropometry:

  • HAZ

Biochemical:

  • Hb

Cognitive function and development:

  • Language (TVIP score)

  • Language (IDHC‐B score)

Anxiety and Depression:

  • Mother's depression score (CES‐D)

  • Mother's Perceived Stress Scale

17 months

Haushofer 2013

(Kenya)

RCT

Unclear

Programme name: N/A

Amount and frequency of payments: total amount of KES 25,200 (USD 404). Either monthly (for 9 months) or a lump‐sum payment. A subgroup of intervention HHs received an additional KES 10,000 per month for 7 months (total KES 95,200 (USD 1525).

Provider: NGO (GiveDirectly)

Delivery: payments via mobile money service to recipients (women or men).

Co‐interventions: none reported

Poor villages and HHs (503/505 HHs)

Food security:

  • FSI

Anthropometry:

  • MUAC

Anxiety and depression:

  • Psychological well‐being index

2 and 3 years

Hjelm 2017

(Zambia)

cRCT

Unclear

Programme name: Zambia Multiple Category Cash Transfer Program (MCP)

Amount and frequency of payments: transfers made every second month. Monthly amount of transfer of ZMW 55,000 (USD 11), irrespective of HH size.

Provider: government

Delivery: payments made through a local paypoint manager.

Co‐interventions: none reported

Socially vulnerable HHs in 2 rural districts with extreme poverty (1571/1515 HHs)

HH expenditure on food:

  • Proportion of total per capita HH expenditure

Food security:

  • HFIAS

Anxiety/depression:

  • Cohen's Perceived Stress scale;

  • CES‐D

2 and 3 years

Miller 2011

(Malawi)

cRCT

Unclear

Programme name: Malawi Social Cash Transfer Scheme (SCTS)

Amount and frequency of payments: about USD 40 (depending on HH size and number of school aged children); monthly transfers. Top‐up payments made for children at primary and secondary school.

Provider: government

Delivery: NR

Co‐interventions: none reported

Ultra‐poor and labour constrained HHs (366/386 HHs), Mchinji district

HH expenditure on food:

  • Proportion of total HH expenditure per week

Food security:

  • Consuming > 1 meal/day

Dietary diversity:

  • Food diversity composite score

6 months, 1 year

Asfaw 2014

(Kenya)

cRCT

High

Programme name: Kenya Cash Transfer Programme for Orphans and Vulnerable Children (CT‐OVC)

Amount and frequency of payments: every 2 months (about USD 21) irrespective of HH size.

Conditionalities: although the programme was unconditional, some districts imposed conditions (e.g. school attendance) and penalties

Provider: Kenya government

Delivery: payments made through local post offices.

Co‐interventions: none reported.

Ultra‐poor HHs with orphans and vulnerable children (CT‐OVC) (1542 HHs/755 HHs)

HH expenditure on food:

  • Proportion of total HH expenditure per month

Dietary diversity:

  • DDS

Anthropometric indicators:

  • HAZ

  • WAZ

  • WHZ

  • Stunting (HAZ < –2SD)

  • Underweight (WAZ < –2SD)

  • Wasting (WHZ < –2SD)

2 and 4 years

Gangopadhyay 2015

(India)

RCT

High

Programme name: N/A

Amount and frequency of payments: monthly cash transfer of INR 1000 (about USD 18).

Provider: researchers

Delivery: transfers were made through bank accounts opened for women beneficiaries

Co‐interventions: none reported

Note: comparison included control group with no bank account and not receiving transfer

100 HHs/100 HHs

NR

Merttens 2013 (Kenya)

cRCT

High

Programme name: Hunger Safety Net Programme (HSNP) pilot programme

Amount and frequency of payments: transfer every 2 months of KES 2150 (at commencement) which increased to KES 3500 by the end of the intervention period. Some HHs had multiple nominated beneficiaries; the effective value of the transfer per HH member was smaller for larger HHs

Provider: Ministry of State for the Development of Northern Kenya and Other Arid Lands

Delivery: cash was loaded onto a biometric smartcard which could be used to collect the cash transfer from a range of paypoints (usually small shops). Several services providers contracted.

Co‐interventions: none reported

Impoverished rural HHs (1224/1212 HHs)

HH expenditure on food:

  • Proportion of total HH expenditure

Dietary diversity:

  • DDS

Anthropometric indicators:

  • Moderate (WHZ < –2SD) and severe wasting (WHZ < –3SD);

  • Moderate (HAZ < –2SD) and severe stunting (HAZ < –3SD);

  • Moderate (WAZ < –2SD) and severe underweight (WAZ < –3SD)

Morbidity: HHs

  • Illness/injury in previous 3 months

2 years

Skoufias 2013

(Mexico)

Other papers:

Ramirez‐Luzuriaga 2016

Leroy 2010

cRCT

High

Programme name: food support programme (PAL, Programa de Apoyo Alimentario). Included in‐kind and cash transfer groups. Health and nutrition education session offered but not compulsory. This review included cash + education group vs control group only.

Amount and frequency of payments: about USD 14/month; disbursed every 2 months. Same amount for all HHs.

Provider: Mexican Government's agency

Delivery: distribution through stored of the government's agency DICONSA.

Co‐interventions: none reported

Poor rural HHs (1687/1663 HHs; 279/289 children)

Dietary diversity:

  • MDD

Anthropometric indicators:

  • BMI

1 and 2 years

Aguero 2006

(South Africa)

Prospective cohort study

High

Programme name: Child Support Grant (CSG)

Amount and frequency of payments: monthly payments made to the primary carer of the child, with no recording of what the carer used the money for. The initial monthly benefit was SAR 100 in 1998 and during the time of the 2004 survey it was SAR 170 (about USD 25).

Provider: government

Delivery: NR

Co‐interventions: none reported

30% of poorest children. subsample of African and Indian HHs with ≥ 1 child.

245/154 children

Anthropometric indicators:

  • HAZ

6 years

Breisinger 2018

(Egypt)

Prospective controlled study

High

Programme name: Takaful cash transfer programme

Amount and frequency of payments: Payments changed from quarterly to monthly, originally starting from a basic amount of EGP 325 per HH, which increased depending on the number of children in the HHs and their educational level.

Conditionalities: programme had been designed to be conditional but not enforced yet at the time of the evaluation

Provider: government; World Bank

Delivery: some beneficiaries had to travel to collect the money

Co‐interventions: none reported

Poor HHs in districts where poverty rate was ≥ 50% (2190 beneficiaries/3813 non‐beneficiaries)

Diet diversity:

  • HDDS

  • Mother's DDS

  • Child's DDS

Anthropometric indicators:

  • LAZ or HAZ

  • Wasting (WHZ < –2SD)

  • Overweight (children)

Morbidity in children aged 0–5 years

  • Diarrhoea

  • Fever

11 months

Renzaho 2017

(Nepal)

Prospective controlled study

High

Programme name: Child Cash Grant (CCG)

Amount and frequency of payments: NPR 200 per month for up to 2 children for poor families with children aged < 5 years, as a complement to other government grants.

Provider: government; Asia Development Bank, UNICEF‐Nepal

Delivery: embedded within existing universal social transfer programmes

Co‐interventions: both intervention and control groups received targeted resources transfers from the government for senior citizens, single women, endangered communities and people with disabilities.

Poor communities and HHs with ≥ 1 child aged < 60 months (1500 HHs/1500 HHs)

Anthropometric indicators:

  • WAZ

  • Underweight (WAZ < –2SD)

  • WHZ

  • Wasting (WHZ < –2SD)

  • HAZ

  • Stunting (HAZ < –2SD)

5 years

UCTs vs food transfers

Hoddinott 2013

(Niger)

cRCT

Unclear

Programme name: N/A

Amount and frequency of payments: cash received for time worked for 3 months, followed by another 3 months where cash was received unconditionally. USD 2/day worked to maximum of USD 50/month. Transfers made twice monthly.

Provider: Nigerian NGOs contracted out to handle food transport, storage, distribution and cash payments

Delivery: public works committee set up in each village to liaise with NGOs. NGOs charged a fixed percentage of total cash amount distributed.

Co‐interventions: none reported but all receiving cash for work in previous 3 months

Poor rural HHs (total 2187)

Dietary diversity:

  • HDDS

  • FCS

  • DDI

  • CDS

3 months

Schwab 2013

(Yemen)

cRCT

High

Programme name: N/A

Amount and frequency of payments: HHs in cash group received 3 cash transfers of an amount equivalent to the local value of the food basket (about USD 50).

Provider: transfers distributed in co‐ordination with local partners: the Yemen Post and Postal Savings Corporation (PPSC) in the case of cash transfers and Ministry of Education in the case of food transfers.

Delivery: collection of cash at any time up to 25 days after disbursement. Initial meetings with beneficiaries to sensitise beneficiaries to the programme objectives and logistics. For cash transfer group, a second resensitisation campaign held after funds were transferred to reinforce messages. Transfers given out at district branches of the PPSC.

Co‐interventions: none reported

Poor HHs in rural communities (982/1001 HHs).

Food security:

  • Number of days with HH reduced meal frequency (last week)

  • Number of days adults ate less food (last week)

  • Number days children ate less food (last week)

  • Number of months had difficulty meeting food needs

Dietary diversity:

  • HDDS

  • DDI

  • FCS

  • Probability of a low FCS score

7 months

aOverall Risk of Bias based on risk of selection and attrition bias.

ARI: acute respiratory infection; BDT: Bangladeshi taka; BMI: body mass index; CDS: Child Diet Score; CES‐D: Center for Epidemiologic Studies Depression Scale; cRCT: cluster randomised controlled trial; DDI: Dietary Diversity Index; DDS: Dietary Diversity Score; ECD: Early Childhood Development; EGP: Egyptian pound; FCS: Food Consumption Score; FSI: Food Security Index; GHQ‐12: 12‐item General Health Questionnaire; HAZ: height‐for‐age z‐score; Hb: haemoglobin; HDDS: Household Dietary Diversity Score; HFIAS: Household Food Insecurity Access Scale; HH: household; IDHC‐B: Inventario do Desenvolvemento de Habilidades Comunicativas – B; KES: Kenyan shilling; LAZ: length‐for‐age z‐score; MDD: minimum dietary diversity; MUAC: mid‐upper arm circumference; N/A: not applicable/available; NGO: non‐governmental organisation; NPR: Nepalese rupee; PKR: Pakistani rupee; SAM: severe acute malnutrition; SAR: South African rand; SD: standard deviation; UCT: unconditional cash transfer; WAZ: weight‐for‐age z‐score; WHZ; weight‐for‐height z‐score; ZMW: Zambian kwacha.

Open in table viewer
Table 9. Unconditional cash transfers – results of included trials

Study ID

(risk of bias)

Study design (n)

Unconditional cash transfers

No intervention

Effect measure (time point)

Effect directiona

Meta‐analysis (yes/no)

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Outcome 1.2: proportion of HH expenditure on food

1.2.1 Outcome measure: proportion of total HH expenditure on food (weekly/monthly)

Brugh 2018 (+)

cRCT (3290 HHs)

0.77 (0.11)

0.70 (0.11)

1561

0.77 (0.11)

0.72 (0.11)

1729

pp –2 (SE 1) 95% CI –3.96 to –0.4; P < 0.1 (1 year)

Yes (excluding Merttens, Asfaw which are missing variance estimate)

Miller 2011 (?)

cRCT (HHs)

56%

68%

366 HHs

52%

48%

386 HHs

pp 12, P < 0.0001 (1 year), 95% CI 5.924 to 18.076, SE 3.1

Hjelm 2017 (?)

cRCT (3010 HHs)

74 (16)

77 (15)

pp 3.2, robust t‐statistic 1.815, 95% CI –0.328 to 6.728, SE 1.8 (2 years)

cRCT (2969 HHs)

74 (16)

74.5

1490 HHs

77 (15)

72.7

1479 HHs

pp 4.2 robust, SE 1.8, 95% CI 0.672 to 7.728, P < 0.05 (3 years)

Merttens 2013 (‐)

cRCT (2436 HHs)

76.5%

77.3%

1224 HHs

79.8%

81%

1212 HHs

pp –0.4, P > 0.1 (1 year)

Asfaw 2014 (‐)

cRCT (1824 HHs)

63%

69.6%

1286 HHs

61%

68.6%

538 HHs

pp –0.95, P > 0.1 (2 years)

Outcome 1.3: proportion of HHs who were food secure

1.3.1 Food security

1.3.1.1 Outcome measure: proportion consuming > 1 meal/day

Brugh 2018 (+)

cRCT (3290 HHs)

0.79 (0.40)

0.94 (0.24)

1561

0.82 (0.39)

0.88 (0.34)

1729

DD 0.11, SE 0.03, pp 11, 95% CI 0.0512 to 0.1688, P < 0.001 (1 year)

Yes

Miller 2011b (?)

cRCT (752 HHs)

About 45%

About 85%

366 HHs

About 45%

About 45%

386 HHs

pp 42, P < 0.0001 (1 year), SE 10.7

95% CI 21.028 to 62.972

1.3.1.2 Outcome measure: mean food security scores (HFIASc/FSId) (mean, SD)

Daidone 2014 (+)

HFIAS/Food Security Scale

cRCT (2299 HHs)

9.63

1158 HHs

12.36

1141 HHs

MD 2.498, SE 0.59, 95% CI 1.3416 to 3.6544, P < 0.05, SE 1.3 (2 years)

Yes

Haushofer 2013 (?)

(FSI)

RCT (940 HHs)

471 HHs

Mean –0.00 (SE 1.00)

469 HHs

MD 0.25, 95% CI 0.13 to 0.37, P < 0.01 (2 years), SE 0.1

Hjelm 2017 (?)

(HFIAS/food security scale)

cRCT (3010 HHs)

14.78 (5.49)

14.68 (5.71)

MD 1.78, robust t‐statistic 3.76, 95% CI 0.8 to 2.76 P < 0.05 (2 years), SE 0.5

cRCT (2970 HHs)

14.78 (5.49)

9.83

1490 HHs

14.68 (5.71)

12.47

1480 HHs

MD 2.69, robust t‐statistic 4.94, 95% CI 1.71 to 3.67, P < 0.05 (3 years), SE 0.5

1.3.1.3 Outcome measure: severe food deprivation (FSI > 2)

Pellerano 2014 (+)

cRCT (2220 children aged 0–5 years)

67.1%

53.4%

747 HHs

69.3%

72.2%

739 HHs

pp –16.63, P < 0.05 (2 years), SE 8.5

N/A. Outcomes from same study.

Pellerano 2014 (+)

cRCT (5384 children aged 6–17 years)

67.8%

58.6%

747 HHs

73.9%

70.7%

739 HHs

pp –6.103, P < 0.1 (2 years), SE 3.7

1.3.2 Dietary diversity

1.3.2.1 Outcome measure: dietary diversity scores, including composite FCS (weighted) (mean, SD) (scores refer to number food groups consumed; reference periods and scales vary)

Daidone 2014 (+)

HDDS 0–12

cRCT (2298 HHs)

6.73

1158

5.30

1141

MD 1.43 (2 years)

Yes (except for Daidone, Merttens, pellerano – missing variance estimate)

Pellerano 2014 (+)

FCS 0–112

cRCT (1486 HHs)

28.7

31.2

747 HHs

28.9

30.4

739 HHs

MD 0.946, P > 0.1 (2 years)

Brugh 2018 (+)

HDDS 0–12

cRCT (3290 HHs)

5.63 (1.78)

5.85 (1.54)

1561

5.64 (1.87)

5.34 (1.44)

1729

MD 0.23 (SE 0.32), 95% CI –0.3972 to 0.8572, P > 0.05 (1 year)

Miller 2011 (?)

FDCS 1–8

cRCT (752 HHs)

5

7

366 HHs

5

4

386 HHs

MD 2.4, P < 0.0001 (1 year), SE 0.6. 95% CI 1.224 to 3.576

Ahmed 2019a (?)

FCS 0–112

cRCT (HHs NR)

MD 6.84 points, SE 1.12, P < 0.01, 95% CI 4.6448 to 9.0352 (2 years)

Ahmed 2019b (?)

FCS 0–112

cRCT (HHs NR)

MD 2.62 points, SE 1.04, P < 0.05, 95% CI 0.5816 to 4.6584 (2 years)

Merttens 2013 (‐)

DDS 0–12

cRCT (2436 HHs)

6.7

7.2

1224 HHs

6.1

6.2

1212 HHs

MD 0.3, P > 0.1 (1 year)

Asfaw 2014 (‐)

DDS (0–8)

cRCT (2369 HHs)

5.225

6.177

1289 HHs

5.697

5.843

540 HHs

MD 0.821, SE 0.3, P < 0.01 (2 years)

1.3.2.2. Outcome measure: proportion with MDD (3–4 food groups)/acceptable food consumption (FCS > 35)

Tonguet Papucci 2015 (+)

cRCT (322 children aged 14–27 months)

65.6%

160

39.5%

162

OR 2.95, 95% CI 1.86 to 4.68, P < 0.001 (2 years)

SMD 0.6, SE 0.1

Yes

Skoufias 2013 (‐)

cRCT (568 children)

69.6%

66. 7%

279

72.7%

59.9%

289

pp 10.6, 95% CI –6.65 to 27.85, P > 0.05 (2 years), SE 8.8

SMD 0.1, SE 0.1

1.4 Change in adequacy of dietary intake

1.4.1 Food poverty (per capita daily caloric intake < 2122 calories; proportion)

Ahmed 2019a (?)

cRCT (n NR)

MD –0.05, SE 0.03, 95% CI –0.1088 to 0.0088, P > 0.05 (2 years)

Yes

Ahmed 2019b (?)

cRCT (n NR)

MD –0.04, SE 0.04, P > 0.05, 95% CI –0.1184 to 0.0384 (2 years)

1.4.2 Proportion food energy deficient (total HH caloric availability < total HH caloric requirements)

Brugh 2018 (+)

cRCT (3290 HHs)

1561

1729

DD –0.1, SE 0.04, 95% CI –0.1784 to –0.0216; P < 0.05 (1 year)

1.5Change in anthropometric indicators

1.5.1Stunting (chronic undernutrition)

1.5.1.1 Outcome measure: proportion stunted (HAZ < –2SD)

Tonguet Papucci 2015 (+)

cRCT

27.7%

630 children aged 0–15 months

27.2%

620 children aged 0–15 months

OR 0.73, 95% CI 0.47 to 1.14, P 0.17 (2 years)

Yes (except Asfaw, Merttens – no measure of variance)

Fenn 2015 (+)

cRCT (1683 children)

n (%): 457 (50.9)

NR

874 children

n (%): 437 (51.7)

NR

809 children

OR 0.36, 95% CI 0.22 to 0.59, P < 0.001 (6 months)

cRCT (1664 children)

n (%): 457 (50.9)

NR

849 children

n (%): 437 (51.7)

NR

815 children

OR 0.54, 95% CI 0.36 to 0.81, P = 0.003 (12 months)

Merttens 2013 (‐)

cRCT (1062 HHs)

26.7%

29.6%

35.6%

31.5%

pp 7.0, P > 0.1 (2 years)

Asfaw 2014 (‐)

cRCT

41.5%

35.7%

442 children aged 0–59 months

44%

37%

295 children aged 0–59 months

pp –4.63, P > 0.1 (2 years)

1.5.1.2 Outcome measure: proportion with severe stunting (HAZ < –3SD)

Fenn 2015 (+)

cRCT (1683 children)

NR

NR

874 children

NR

NR

809 children

OR 0.47, 95% CI 0.28 to 0.77, P = 0.003 (6 months)

No. SE not available for all studies.

cRCT (1664 children)

NR

NR

849 children

NR

NR

815 children

OR 0.59, 95% CI 0.38 to 0.92, P = 0.02 (12 months)

Merttens 2013 (‐)

cRCT (n = 1062)

11.6%

13.4%

15.2%

15.1%

pp 1.9, P > 0.1 (2 years)

1.5.1.3 Outcome measure: HAZ (mean, SD)

Daidone 2014 (+)

cRCT (2299 children aged 0–60 months)

–1.445

1158

–1.491

1141

MD 0.066, 95% CI –0.116 to 0.248, P > 0.05 (2 years)

Yes

Tonguet Papucci 2015 (+)

cRCT (1250 children aged 0–15 months

–1.18 (1.44)

–1.96 (1.03)

630

–1.33 (1.24)

–1.99,

SD 1.04)

620

MD –0.0005, 95% CI –0.004 to 0.003 z‐score/month, P = 0.78

Fenn 2015 (+)

cRCT (1683 children)

–1.98 (1.65)

NR

874 children

–1.97 (1.75)

NR

809 children

MD 0.24, 95% CI 0.17 to 0.32, P < 0.001 (6 months)

cRCT (1664 children)

–1.98 (1.65)

NR

849 children

–1.97 (1.75)

NR

815 children

MD 0.21, 95% CI 0.10 to 0.31, P < 0.001 (12 months)

Fernald 2011 (?)

cRCT (1196 children)

–0.5 (2.1)

–1.7 (1.2)

797

–0.7 (2.0)

–1.7 (1.2)

399

MD 0.01, 95% CI –0.18 to 0.19 (2 years)

Ahmed 2019a (?)

cRCT (n NR)

MD 0.132, SE 0.08, 95% CI –0.0248 to 0.2888, P > 0.05 (2 years)

Ahmed 2019b (?)

cRCT (n NR)

MD –0.097, SE 0.08, 95% CI –0.0598 to 0.2538, P > 0.05 (2 years)

Asfaw 2014 (‐)

cRCT (737 children aged 0–59 months)

–1.466

–1.279

442

–1.462

–1.248

295

MD –0.0272, 95% CI –0.503 to 0.449, P > 0.1 (2 years)

1.5.2Wasting (acute undernutrition)

1.5.2.1 Outcome measure: proportion wasted (WHZ < –2SD) (proportion)

Tonguet Papucci 2015 (+)

cRCT (1250 children aged 0–15 months

26%

630

192%

620

IRR 0.92, 95% CI 0,64 to 1.32; P = 0.66 (2 years)

No. SE not available for all studies and different effect size for 1 study.

Fenn 2015 (+)

cRCT (1683 children)

n (%): 196 (22.0)

NR

874 children

n (%): 184 (21.9)

NR

874 children

OR 1.09, 95% CI 0.64 to 1.87, P = 0.75 (6 months)

cRCT (1664 children)

n (%): 196 (22.0)

NR

849 children

n (%): 184 (21.9)

NR

849 children

OR 1.10, 95% CI 0.71 to 1.71, P = 0.66 (12 months)

Merttens 2013 (‐)

cRCT (1062 children)

25.3%

23.1%

24.2%

17.3%

pp 4.7, P > 0.1

Asfaw 2014 (‐)

cRCT (737 children aged 0–59 months)

6%

9%

648

9.4%

6.9%

341

pp 5.95, P > 0.1 (2 years)

1.5.2.2 Outcome measure: severe wasting (WHZ < –3SD) (proportion)

Fenn 2015 (+)

cRCT (1683 children)

69 (7.7)

874 children

62 (7.4)

874 children

OR 0.98, 95% CI 0.38 to 2.54, P = 0.97 (6 months)

No. Variance only available for 1 of the 2 studies.

Merttens 2013 (‐)

cRCT (1062 children)

6.8

6.2

8.0

3.5

pp 3.9, P > 0.1

1.5.2.3 Outcome measure: WHZ (mean, SD)

Daidone 2014 (+)

cRCT (2299 children aged 0–69 months)

–0.0961

1158

–0.154

1141

MD 0.118, 95% CI –0.015 to 0.251 (2 years)

Yes

Tonguet Papucci 2015 (+)

cRCT (1250 children aged 0–15 months)

–1.24

(1.23)

–0.56 (0.95)

630

–1.07 (1.12)

–0.61 (0.93)

620

MD –0.003 z‐score/month, 95% CI –0.008 to 0.0003, P = 0.07 (2 years)

Fenn 2015 (+)

cRCT (1683 children)

–1.11 (1.34)

NR

874 children

–1.15 (1.30)

NR

874 children

MD 0.04, 95% CI –0.07 to 0.14, P = 0.5 (6 months)

cRCT (1664 children)

–1.11 (1.34)

NR

849 children

–1.15 (1.30)

NR

849 children

MD –0.08, 95% CI –0.19 to 0.04, P = 0.21 (12 months)

Ahmed 2019a (?)

cRCT (n NR)

Coefficient –0.013, SE 0.07, 95% CI –0.1502 to 0.1242, P > 0.05 (2 years)

Ahmed 2019b (?)

cRCT (n NR)

Coefficient –0.088, SE 0.08, P > 0.05, 95% CI –0.2448 to 0.0688 (2 years)

Asfaw 2014 (‐)

cRCT (737 children aged 0–59 months)

–0.017

–0.332

442

0.065

–0.166

295

MD –0.0838, 95% CI –0.339 to 0.171, P > 0.1 (2 years)

1.5.3 Underweight

1.5.3.1 Weight for age z‐score

1.5.3.1.1 Outcome measure: proportion underweight (WAZ < –2SD)

Pellerano 2014 (+)

cRCT (total n: 6 month old 474; 12 month old 293)

6 month old: 29.2; 12 month old: 36.6

6 month old: 10.6;

12 month old: 16.4

6 month old: 11.0; 12 month old: 39.7

6 month old: 8.4

12 month old: 23.3

6 month old: pp –15.60, P < 0.05

12 month old: pp –3.637, P > 0.05 (2 years)

6 month old: ▲

12 month old: △

No. Variance not available for all studies.

Merttens 2013 (‐)

cRCT (1062)

30.7

24.9

33.7

24

pp 3.9, P > 0.1

Asfaw 2014 (‐)

cRCT (1435)

20.6

21

19.6

19.1

pp –0.62, P = 0.901 (2 years)

1.5.3.1.2 Outcome measure: proportion severely underweight (WAZ < –3SD)

Merttens 2013 (‐)

cRCT (1062)

9.8

8.9

10.9

6.9

pp 3.2, P > 0.1

N/A

1.5.3.1.3 Outcome measure: mean WAZ

Daidone 2014 (+)

cRCT (6825 children)

–0.900

–0.963

MD 0.128, 95% CI –0.05 to 0.261, P > 0.05 (2 years)

Yes

Asfaw 2014 (‐)

cRCT 752 children aged 0–59 months)

–0.879

–1.034

456

–0.923

–0.804

296

MD –0.274, 95% CI –0.633 to 0.085, P > 0.1 (2 years)

1.5.3.2 BMI (mean, SD)

Fenn 2015 (+)

cRCT 1208 HHs/mothers (flow diagram)

Median (IQR) 20.4 (18.3 to 23.5)

NR

607

median (IQR) 20.0 (18.1 to 22.7)

NR

601

Beta‐coefficient –0.10, 95% CI –0.36 to 0.16, P = 0.45 (6 months)

1.5.5 Mid‐upper arm circumference (MUAC) (mean, SD)

Fenn 2015 (+)

cRCT (1208 HHs/mothers)

24.4 (3.4)

NR

607

24.3 (3.2)

NR

601

Beta‐coefficient 0.09, 95% CI –0.13 to 0.30, P = 0.41 (6 months)

cRCT (1683 children)

13.5 (1.3)

NR

874

13.5 (1.2)

NR

809

beta‐coefficient 0.06, 95% CI –0.02 to 0.15, P = 0.15 (6 months)

1.6Change in biochemical indicators

1.6.1 Outcome measure: haemoglobin concentration (g/dL) (mean, SD)

Fenn 2015 (+)

cRCT (1208 HHs/mothers)

mean 103 (SD 18)

NR

607 mothers

mean 100 (SD 19)

NR

601 mothers

MD –0.42, 95% CI –0.63 to –0.20, P < 0.001 (6 months)

cRCT (1683 children)

mean 89 (17)

NR

874 children

mean 88 (16)

NR

809 children

MD –0.12, 95% CI –0.31 to 0.08, P = 0.24 (6 months)

Yes

Fernald 2011 (?)

cRCT (922 children)

9.7 (1.3)

10.4 (1.5)

9.5 (1.3)

10.3 (1.3)

MD 0.04, 95% CI –0.21 to 0.29, P > 0.1

1.7 Cognitive function and development

1.7.1 Outcome measure: cognitive and development scales/indices (mean, SD)

Baird 2013 (+)

cRCT (RCPM; 2057 adolescents)

MD 0.136, SE 0.119, 95% CI –0.097 to 0.369, P > 0.1 (2 years)

No (no n to calculate SMD)

Daidone 2014 (+)

cRCT (ECD Index; 5670 children)

5.174

4.926

MD 0.311, 95% CI –0.065 to 0.687, P > 0.1 (2 years)

1.7.2 Outcome measure: Individual cognitive function measures scores (mean, SD)

Fernald 2011 (?)

cRCT (Language: TVIP; 1894 children 36 months and older)

MD 0.013, 95% CI –0.076 to 0.102, P > 0.1 (2 years)

N/A

Language: IDHC‐B 1192 children aged 12–35 months)

45.0 (35.1)

42.3 (32.2)

MD 2.43, 95% CI –1.01 to 5.86, P > 0.1 (2 years)

1.8 Change in proportion of anxiety and depression

1.8.1 Outcome measure: depression score (CES‐D scale) (mean change in score, SD)

Fernald 2011 (?)

cRCT (1430 mothers)

19.6 (11.1)

18.9 (10.6)

MD 0.71, 95% CI –0.84 to 2.25, P > 0.1 (2 years)

Yes

Haushofer 2013 (?)

RCT (2140 adults)

471 HHs

26.48 (9.31)

469 HHs

MD –0.99, 95% CI –1.54 to –0.44, P < 0.1 (3 years)

Hjelm 2017 (?)

cRCT (1765 HHs with adolescents)

Effect estimate 0.00, robust t‐statistic 0.00, P not significant (2 years)

cRCT (2217 HHs with adolescents)

19.24

Effect estimate –0.54, 95% CI –1.80028 to 0.72028 (3 years)

1.8.2 Outcome measure: Perceived Stress Scale (mean, SD)

Fernald 2011 (?)

cRCT (n = 1430)

Top 3 income quartiles: MD 0.045, 95% CI –0.112 to 0.202, P > 0.1.

Bottom income quartile: MD 0.177, 95% CI –0.017 to 0.371, P < 0.1

(2 years)

Yes

Haushofer 2013 (?)

RCT (2140 adults)

0.00 (1.00)

MD –0.14, 95% CI –0.258 to –0.022, P < 0.05 (3 years)

Hjelm 2017 (?)

cRCT (2490 HHs)

9.58 (4.64)

9.92 (4.73)

Effect estimate –0.42, 95% CI –1.12364 to 0.28364 (3 years)

1.8.3 Outcome measure: proportion with psychological distress (psychological distress, anxiety and depression, social dysfunction, loss of confidence)

Baird 2013 (+)

cRCT (2089 adults)

0.374

pp –14.3, 95% CI –21.0 to –7.6, P < 0.001 (1 year)

N/A

0.308

pp –3.8, 95% CI –13.14 to 5.8 P > 0.1 (2 years)

1.8.4 Outcome measure: Psychological Well‐being Score (mean, SD)

Haushofer 2013 (?)

RCT (2140 adults)

–0.00 (1.00)

Coefficient 0.20 SD, 95% CI 0.082 to 0.318, P < 0.1 (2 years)

N/A

1.9 Morbidity

1.9.1 Outcome measure: incidence of respiratory infections (reference period: 1 and 2 weeks)

Daidone 2014 (+)

cRCT

Proportion children aged 0–60 months with ARI in previous 2 weeks (n = 7232)

0.0511

0.0832

pp –3.6, 95% CI –8.6 to 14.0, P > 0.05 (2 years)

No. 2 different measures of effect that could not be compared (IRR vs OR/pp).

Fenn 2015 (+)

cRCT (1683 children)

n (%): 310 (34.3)

NR

874 children

n (%): 273 (32.2)

NR

809 children

OR, 0.73, 95% CI 0.51 to 1.03, P = 0.07 (6 months)

Tonguet Papucci 2015 (+)

cRCT

Episodes/child‐month (1250 children aged 0–15 months)

N 0.87, 95% CI 0.84 to 0.89

N 0.95, 95% CI 0.92 to 0.97

IRR 0.79, 95% CI 0.78 to 0.81, P < 0.001 (2 years)

Asfaw 2014 (‐)

cRCT 957 children aged 0–7 years)

613 children

344 children

IRR 0.556, t‐statistics –2.40, P < 0.05 (2 years)

1.9.2 Outcome measure: incidence diarrhoeal disease

Fenn 2015 (+)

cRCT (1683 children)

n (%): 228 (25.2)

NR

874 children

n (%): 298 (35.0)

NR

809 children

OR 1.05, 95% CI 0.67 to 1.63, P = 0.84 (6 months)

No. Different measure of effect for one study (IRR vs OR/pp)

Daidone 2014 (+)

cRCT

Proportion children aged 0–60 months with diarrhoea in previous 2 weeks (n = 7232)

0.0684

0.0925

pp –4.9, 95% CI –8.9 to –0.9, P < 0.05 (2 years)

Tonguet Papucci 2015 (+)

cRCT

Episodes/child/month (1250 children aged 0–15 months)

n 0.85, 95% CI 0.82 to 0.88

n 0.83, 95% CI 0.80 to 0.85

IRR 1.00, 95% CI 0.97 to 1.03, P = 0.89 (2 years)

Ahmed 2019a (?)

cRCT (n NR)

Coefficient –0.003, pp –0.3, SE 0.02, 95% CI –0.0422 to 0.0362, P > 0.05 (2 years)

Ahmed 2019b (?)

cRCT (n NR)

Coefficient –0.009, pp –0.9, SE 0.02, 95% CI –0.0482 to 0.0302, P > 0.05

1.9.3 Outcome measure: proportion with any illness in previous reference period (1 month/3 months)

Pellerano 2014 (+)

cRCT (1996 children aged 0–5 years)

38.9

31.4

36.7

45.3

pp –15.38, P < 0.1 (2 years)

No. Variance estimates not available for all studies.

Merttens 2013 (‐)

cRCT (n = 14,342) (includes injury)

22.5

12.1

23.1

11.7

pp 1.0, P > 0.05 (2 years)

1.9.4 Proportion with anaemia (any)

Fenn 2015 (+)

cRCT (1683 children)

874 children

809 children

OR 1.13, 95% CI 0.68 to 1.86, P = 0.64 (6 months)

N/A

cRCT (1208 mothers)

607 mothers

601 mothers

OR 1.34, 95% CI 0.82 to 2.18, P = 0.24 (6 months)

1.10 Adverse events: proportion who were overweight (according to International standards and Bukana Health Card)

Pellerano 2014 (+)

cRCT (total n: 6 months old: 474; 12 months old: 293)

6 months old: 4.5; 12 months old: 6.0

6 months old: 2.2; 12 months old: 0.0

6 months old: 0.8; 12 months old: 0.0

6 months old: 2.0; 12 months old: 0.0

6 months old: pp –5.082, P > 0.05; 12 months old: pp –6.461, P > 0.05 (2 years)

N/A

aEach triangle represents one study; bValues are derived from graphs

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0; □: Effect measure is the null; (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias. FDCS: Food diversity consumption score; FCS: Food consumption score.

ARI: acute respiratory infection; CES‐D: Center for Epidemiologic Studies Depression Scale; CI: confidence interval; cRCT: cluster randomised controlled trial; DD: Diet diversity; DDS: Dietary Diversity Score; ECD: Early Childhood Development; FCS: Food Consumption Score; FDCS: Food Diversity Composite Score; FSI: Food Security Index; HAZ: height‐for‐age z‐score; HDDS: Household Dietary Diversity Score; HFIAS: Household Food Insecurity Access Scale; HH: household; IDHC‐B: Inventario do Desenvolvemento de Habilidades Comunicativas‐B; IQR: interquartile range; IRR: incidence rate ratio; MD: mean difference; MDD: minimum dietary diversity; n: number; NR: not reported; OR: odds ratio; pp: percentage point; RCPM: Ravens Coloured Progressive Matrices ; RCT: randomised controlled trial; SD: standard deviation; SE: standard error; SMD: standardised mean difference; TVIP: Test de Vocabulario en Imagenes Peabody; WHZ: weight‐for‐height z‐score.

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Table 10. Unconditional cash transfers – results of included prospective controlled studies

Study ID

(risk of bias)

Study design (n)

Unconditional cash transfers

No intervention

Effect measure (time point)

Effect directiona

Meta‐analysis (yes/no)

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

1.3.2 Dietary diversity

1.3.2.1 Outcome measure: Dietary diversity scores, including composite Food Consumption Score (FCS) (weighted) (mean, SD) (scores refer to number food groups consumed; reference periods and scales vary)

Breisinger 2018 (‐)

PCS (6003 HHs) – HDDS

NR

9.58 (1.38)

2190?

NR

9.48 (1.55)

3813?

MD (SE) 0.16 (0.117), 95% CI –0.06932 to 0.38932, P > 0.1 (1 year??)

N/A

Breisinger 2018 (‐)

PCS (5799 HHs) – mother DDS)

NR

4.21 (1.28)

2190?

NR

4.04 (1.26)

3813?

MD 0.011 (SE 0.100), 95% CI –0.185 to 0.207, P > 0.1 (1 year?),

Breisinger 2018 (‐)

PCS (1684 HHs) DDS children aged 6–23 months

NR

3.35 (1.73)

2190?

NR

3.39 (1.61)

3813?

MD –0.342 (SE 0.209) 95% CI –0.752 to 0.068, P > 0.1 (1 year)

Breisinger 2018 (‐)

PCS (3202 HHs) DDS children aged 24–59 months

NR

5.09 (1.37)

2190?

NR

4.89 (1.40)

3813?

MD –0.057 (SE 0.144) 95% CI –0.33924 to 0.22524, P > 0.1 (1 year)

1.5Change in anthropometric indicators

1.5.1Height‐for‐age z‐scores; chronic undernutrition)

1.5.1.1 Outcome measure: proportion stunted (HAZ < –2SD)

Renzaho 2017 (‐)

Prospective controlled study (n = 1491)

66.7

59.8

748

63

52.9

743

Adjusted DID (pp): –5.16, 95% CI –9.55 to –0.77 (5 years), SE 2.2

1.5.1.3 Outcome measure: HAZ (mean, SD)

Aguero 2006 (‐)

PCS

–0.84

–1.08

NR (MD 0.15 at 45%, and 0.25 at 80% of nutritional window; data derived from graph (6 years))

No. SE not available for all studies.

Renzaho 2017 (‐)

PCS (1491 children)

–2.6 (1.4)

–2.2 (1.4)

748

–2.3 (1.3)

–2.1 (1.3)

743

Adjusted DID: 0.18, 95% CI 0.09 to 0.27 (5 years)

1.5.2WHZ; acute undernutrition/wasting

1.5.2.1 Outcome measure: proportion wasted (WHZ < –2SD) (proportion)

Renzaho 2017 (‐)

PCS (1491 children)

12.7

5.7

748

5.8

6.4

743

Adjusted DID: pp –2.84, 95% CI –5.58 to –0.1 (5 years)

1.5.2.3 Outcome measure: WHZ (mean, SD)

Renzaho 2017 (‐)

PCS (1491 children)

–0.8 (1.1)

–0.4 (1.0)

748

–0.5 (0.9)

–0.4 (1.1)

743

Adjusted DID: MD 0.19, 95% CI 0.09 to 0.3 (5 years)

1.5.3 Weight‐for‐age z‐score (WAZ; underweight)

1.5.3.1 Outcome measure: proportion underweight (WAZ <2SD)

Renzaho 2017 (‐)

PCS (1491 children)

50.7

34.8

748

37.3

28.9

743

Adjusted DID: pp –7.35, 95% CI –11.62 to –3.08 (5 years)

N/A

1.5.3.3 Outcome measure: mean WAZ

Renzaho 2017 (‐)

PCS (1491 children)

–2.1 (1.1)

–1.6 (1.1)

748

–1.7 (1.0)

–1.4 (1.1)

743

Adjusted DID:

0.22, 95% CI 0.15 to 0.29 (5 years)

N/A

aEach triangle represents one study.

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

DDS: Dietary Diversity Score; DID: difference in differences; HAZ: height‐for‐age z‐score; HH: household; MD: mean difference; n: number; N/A: not applicable/available; NR: not reported; PCS: prospective controlled study; SD: standard deviation; SE: standard error; WAZ: weight‐for‐age z‐score; WHZ: weight‐for‐height z‐score.


Harvest plot: unconditional cash transfers.

Harvest plot: unconditional cash transfers.

Primary outcomes
1.1 Change in prevalence of undernourishment

None of the included studies measured prevalence of undernourishment.

1.2 Proportion of household expenditure on food

As household income increases, the share of household expenditure on food should decrease relative to other household expenditure (INDDEX Project 2018). Five cRCTs reported this outcome (Asfaw 2014; Brugh 2018; Hjelm 2017; Merttens 2013; Miller 2011), with evidence being very uncertain about the effects of UCTs on the proportion of household expenditure on food (5 trials, 11,271 households; very low certainty evidence; summary of findings Table 1). Effects varied across the five studies, with one study showing a clear effect favouring UCTs, two studies showing an unclear effect potentially favouring UCTs, and two studies show a clear effect favouring the control (P = 0.003; Figure 5). Three of these studies could be included in a forest plot but data could not be pooled due to high heterogeneity (I2 = 92%; Analysis 1.1). Miller 2011 and Brugh 2018 were two different studies assessing the Malawi cash transfer scheme and thus are the same in terms of the characteristics of participants and interventions although Miller 2011 was a pilot study. Hjelm 2017 assessed the Zambia cash transfer programme. These programmes differed in the amount provided by the cash transfer (USD 11 every second month which was fixed versus USD 40 monthly which varied depending on household size and the number of school‐aged children). As expected, the effect in Miller was worse than in the other studies.

Brugh 2018 reported a clear effect favouring UCTs, with a decrease in the proportion of household expenditure on food by 2 percentage points at 1 year (pp –2, 95% CI –3.96 to –0.4; 3290 households). This study was at low overall risk of bias.

Asfaw 2014 and Merttens 2013 reported an unclear effect potentially favouring UCTs, showing a decrease in the proportion of monthly amount spent on food: Merttens 2013 by –0.4 pp at one year (2435 participants; P > 0.1) and Asfaw 2014 by –0.95 pp at two years (1824 participants; P > 0.1). These studies were both at high overall risk of bias.

Miller 2011 and Hjelm 2017 reported a clear effect favouring the control. Miller 2011 reported an increase in the proportion of total weekly expenditures on food by 12 pp in the intervention group compared to the control at one year (P < 0.0001, 752 participants); Hjelm 2017 reported an increase in per capita share of food expenditure of 4.2 pp (95% CI 0.67 to 7.72). Miller 2011 is at low overall risk of bias and Hjelm 2017 at unclear overall risk of bias.

1.3 Proportion of households who were food secure

Twelve trials reported on different food security and dietary diversity measures. Food security measures reflect the frequency and severity of food insecurity experienced by households within a specific reference period (e.g. past month), with a higher number usually indicating more food insecurity. Dietary diversity refers to the number of food groups or food items consumed, by households or individuals within a specific reference period (e.g. past 24 hours), with a higher number indicating better dietary diversity. In our analysis, we included composite measures for food security and dietary diversity (e.g. reported scores or indices), in preference to single outcome measures; details of their definitions and interpretations are presented in Table 6.

1.3.1 Food security

Five cRCTs and one RCT reported this outcome, with evidence showing that UCTs improve food security (6 trials, 10,251 households and 7604 children; high‐certainty evidence; summary of findings Table 1). All six studies showed a clear effect favouring UCTs (P < 0.001; Figure 5). Miller 2011 and Brugh 2018 reported an increase in the proportion of people eating more than one meal per day with the UCTs: Miller 2011 reported an increase of 42 pp at one year follow‐up (P < 0.0001; 752 participants); Brugh 2018 reported an increase of 11 pp at one year (95% CI 5.12 to 16.9; 3290 households). Data from these studies could not be pooled due to high heterogeneity (I2 = 87%; Analysis 1.2). These two studies assessed cash transfers in Malawi and the characteristics of participants and interventions were the same, but Miller 2011 was a smaller pilot study and was at unclear overall risk of bias whereas Brugh 2018 was at low overall risk of bias. Daidone 2014, Haushofer 2013, and Hjelm 2017 reported food security scores; Hjelm 2017 using the FSI and Daidone 2014 and Haushofer 2013 using a food security scale based on the HFIAS, and for both of these the higher the value the more beneficial. A meta‐analysis of these three studies showed a slight improvement in scores (SMD 0.18, 95% CI 0.13 to 0.23; 6209 households; I2 = 0%; Analysis 1.3). Pellerano 2014 reported a decrease in severe food deprivation (FSI > 2) at two years in children aged from birth to five years (–16.63 pp; P < 0.05; 2220 children) and in children aged six to 17 years (82116.10 pp; P > 0.1; 5384 children) receiving the UCT compared to children in the control group. Brugh 2018, Daidone 2014, and Pellerano 2014 were at low overall risk of bias, whereas Haushofer 2013, Hjelm 2017, and Miller 2011 were at unclear overall risk of bias.

One cRCT at higher overall risk of bias compared food transfers with UCTs (Schwab 2013). They reported on the number of months of the previous six months that households had difficulty satisfying their food needs, with an unclear effect potentially favouring UCTs (MD –1.06; P > 0.05; 1983 households).

1.3.2 Dietary diversity

Ten cRCTs and one PCS reported dietary diversity measures.

Randomised controlled trials

Evidence showed that UCTs may increase dietary diversity (10 RCTs; 11,145 households and 3578 children; low‐certainty evidence; summary of findings Table 1). Five cRCTs reported a clear effect favouring UCTs and five cRCTs reported an unclear effect potentially favouring UCTs (P < 0.001; Figure 5).

Ahmed 2019a, Ahmed 2019b, Asfaw 2014, Miller 2011, and Tonguet Papucci 2015 reported clear effects favouring UCTs. Miller reported an increase in the food diversity composite score (scale: 1 to 8) of 2.4 points more with UCTs compared to the control group (95% CI 1.22 to 3.58; 752 households). Ahmed 2019a reported an increase in FCS (scale: 0 to 112) with UCTs at two years of 6.84 points (95% CI 4.64 to 9.03; n NR) and Ahmed 2019b of 2.62 points (95% CI 0.58 to 4.66; n NR). Asfaw 2014 reported a mean dietary diversity score (scale 0 to 8) higher by 0.82 at two years (1824 households; P < 0.01). Tonguet Papucci 2015 reported the odds of achieving MDD, which were approximately three‐fold higher in the children from the UCT group (OR 2.95, 95% CI 1.86 to 4.68; n = 322; P < 0.001). Of these studies, Ahmed 2019a, Ahmed 2019b, and Miller 2011 were at unclear overall risk of bias; Asfaw 2014 was at high overall risk of bias and Tonguet Papucci 2015 was at low overall risk of bias.

Brugh 2018, Daidone 2014, Merttens 2013, Pellerano 2014, and Skoufias 2013 reported unclear effects potentially favouring UCTs. Four of these cRCTs reported an increase in the mean dietary diversity score at household level (scale: 0 to 12) (Brugh 2018: 0.23 points, 95% CI –0.39 to 0.86; 3290 households; Daidone 2014: 1.43 points; 2298 households; P = NR; Merttens 2013: 0.3 points; 2436 households; P = NR; Pellerano 2014: 0.16; 1486 households; P > 0.1). Skoufias 2013 reported an increase of 10.6 percentage points in the proportion of children in the intervention group achieving MDD, which referred to consuming at least three to six food groups, compared to the control group at two years (pp 10.6, 95% CI –6.65 to 27.85; 568 children; P > 0.05).

Three studies reporting diet diversity scores had sufficient data for a meta‐analysis, but results were not pooled due to high heterogeneity (I2 = 83%; Analysis 1.4) (Asfaw 2014; Brugh 2018; Miller 2011). These studies reported slightly different measures of dietary diversity but, in all cases, a higher value indicated higher dietary diversity. Two studies assessed a cash transfer programme in Malawi and Asfaw 2014 assessed the Kenya cash transfer programme. Both studies in Malawi included ultra‐poor households, the one in Kenya included households with orphans and vulnerable children, which may be more vulnerable. Asfaw 2014 was also at high overall risk of bias whereas the others were at unclear and low overall risk of bias. Two studies reporting the proportion of children with MDD could not be pooled due to high heterogeneity (I2 = 94%, Analysis 1.5) (Skoufias 2013; Tonguet Papucci 2015). The interventions in these two studies differed somewhat; Skoufias 2013, which was at high overall risk of bias, made a payment of approximately USD 14 every two months and accompanied by health education sessions that were not compulsory. Tonguet Papucci 2015 assessed seasonal cash payments of USD 17 (from July to November only), with no educational sessions, and payments were made to mothers. The effect was larger for Tonguet Papucci 2015, which was at low overall risk of bias.

Two cRCTs compared UCTs with food transfers (Hoddinott 2013; Schwab 2013). Both reported a clear effect on the FCS favouring UCTs (Hoddinott 2013: MD 4.65, 95% CI 2.41 to 6.87, n = NR; Schwab 2013: MD 4.52, 95% CI 6.85 to 2.19; 1581 households). Hoddinott 2013 was at unclear overall risk of bias and Schwab 2013 was at high overall risk of bias.

Prospective controlled studies

One PCS reported an unclear effect on the HDDS potentially favouring UCTs (0.16, 95% CI –0.07 to 0.39; 6003 households) (Table 9) (Breisinger 2018). This study was at high overall risk of bias.

Secondary outcomes
1.4 Change in adequacy of dietary intake

Three cRCTs reported change in adequacy of dietary intake (Ahmed 2019a; Ahmed 2019b; Brugh 2018). One study reported a clear effect favouring UCTs and two studies reported an unclear effect potentially favouring UCTs (P = 0.047) (Table 9).

Brugh 2018 reported a clear effect favouring the intervention on the proportion of households who were food energy deficient (i.e. where the total household caloric availability was lower than the total household caloric requirement) (pp –10, 95% CI –17.8 to –2.16; 3290 households). This study was at low overall risk of bias.

Ahmed 2019a and Ahmed 2019b reported an unclear effect potentially favouring UCTs. A meta‐analysis of these two studies showed that UCTs may make no difference to the proportion of households with food poverty, defined as per capita daily caloric intake below 2122 calories (MD –4.64, 95% CI –9.34 to 0.06, n = NR). These studies were at low overall risk of bias.

1.5 Change in anthropometric indicators

Ten cRCTs (Ahmed 2019a; Ahmed 2019b; Asfaw 2014; Daidone 2014; Fenn 2015; Fernald 2011; Merttens 2013; Pellerano 2014; Skoufias 2013; Tonguet Papucci 2015) and two PCS (Aguero 2006; Renzaho 2017) reported various anthropometric measures.

1.5.1 Stunting: height‐for‐age z‐scores < –2SD; chronic undernutrition)

Randomised controlled trials

Four cRCTs reported on the proportion of children who were stunted (Asfaw 2014; Fenn 2015; Merttens 2013; Tonguet Papucci 2015), with evidence showing that UCTs may reduce stunting (4 trials, 4713 children; low‐certainty evidence; summary of findings Table 1). One study showed a clear effect favouring UCTs, two studies showed an unclear effect favouring UCTs, and one study showed an unclear effect favouring control (P = 0.047; Figure 5). A meta‐analysis of two of these studies showed a reduction in stunting with UCTs (OR 0.62, 95% CI 0.46 to 0.84; 2914 children; I2 = 0%; Analysis 1.7) (Fenn 2015; Tonguet Papucci 2015).

Fenn 2015 reported a clear effect favouring UCTs, with a reduction in the odds of stunting of 46% at 12 months (OR 0.54, 95% CI 0.36 to 0.81; 1664 children). There was a similar effect for the proportion of children who were severely stunted (HAZ < –3SD) (Table 9). This study was at low overall risk of bias.

Tonguet Papucci 2015 and Asfaw 2014 reported unclear effects potentially favouring UCTs. Tonguet Papucci 2015 reported a reduced likelihood of stunting in the intervention group compared to the control group at 24 months by 27% (OR 0.73, 95% CI 0.47 to 1.14; n = 1250; P = 0.17), but the effect ranged from a beneficial effect on the outcome to worsening the outcome. In Asfaw 2014, the intervention reduced the proportion of stunted children by 4.63 pp (P > 0.1). Tonguet Papucci 2015 was at low overall risk of bias whereas Asfaw 2014 was at high overall risk of bias.

Merttens 2013 reported an increase in the proportion of stunted children (pp 7.0; 1062 children; P > 0.1) and severely stunted children (pp 1.9; 1062 children; P > 0.1) among those in the UCT group compared to those in the control group. This study was at high overall risk of bias.

In addition to the proportion of stunting, six trials reported the effects on mean HAZ (Ahmed 2019a; Ahmed 2019b; Asfaw 2014; Daidone 2014; Fenn 2015; Fernald 2011); and one on mean z‐score per month (Tonguet Papucci 2015). A meta‐analysis showed an unclear effect of UCTs on HAZ (MD 0.07, 95% CI –0.04 to 0.18; I2 = 56%; Analysis 1.8). A sensitivity analysis performed using only studies at overall low risk of bias showed a clear effect favouring UCTs (MD 0.16, 95% CI 0.02 to 0.29; Appendix 4) (Daidone 2014; Fenn 2015).

Prospective controlled studies

One PCS reported a clear effect favouring UCTs on stunting at five years (pp –5.16, 95% CI –9.55 to –0.77; 1491 children; Table 9; Figure 5) (Renzaho 2017).

Renzaho 2017 and Aguero 2006 also reported on the effect of UCTs on mean HAZ (data not pooled as SE was not available for either study). Renzaho 2017 reported a clear effect favouring UCTs at five years (pp 18, 95% CI 9 to 27; 1491 children). Aguero 2006 reported that after 72 months, the mean z‐score was better in children receiving the intervention (–0.84) than in those who only received the intervention after they were aged three years (–0.91) (566 children) or those who were rejected or were not yet receiving the intervention (–1.08) (399 children). Both studies are at high overall risk of bias.

Aguero 2006 also reported the effects of the intervention on HAZ for children receiving the child care grant for different periods of the critical nutritional window aged between 0 and 36 months. They found that for children receiving the intervention for less than 20% of this period there was no effect on HAZ. Compared to receiving a 'small dose', they found a significant impact on HAZ for children receiving the intervention during 45% to 80% of the nutrition window (mean change in HAZ 0.15 at 45%, and 0.25 at 80% of nutritional window; data derived from graphs).

1.5.2 Wasting: weight‐for‐height z‐score < –2SD (acute undernutrition)

Randomised controlled trials

Four cRCTs reported effects of UCTs on wasting (Asfaw 2014; Fenn 2015; Merttens 2013; Tonguet Papucci 2015). Evidence showed that there was uncertainty about whether UCTs reduce wasting (4 trials, 6396 children; very low‐certainty evidence; summary of findings Table 1). One study showed an unclear effect potentially favouring UCTs and three studies showed an unclear effect potentially favouring the control (P = 0.016; Figure 5).

Tonguet Papucci 2015 reported an unclear effect potentially favouring the intervention, observing a 2% decrease in the risk of wasting at two years (incidence rate ratio (IRR) 0.92, 95% CI 0.64 to 1.32; 1250 children; P = 0.66). However, this effect ranged from a 36% reduction to a 32% increase in risk.

Asfaw 2014, Merttens 2013, and Fenn 2015 reported an unclear effect potentially favouring the control as they observed an increase in the proportion of wasting among children receiving UCTs, but reported a CI that crossed the null. In Fenn 2015, the odds of wasting was 10% higher among children receiving the intervention at one year (OR 1.10, 95% CI 0.71 to 1.71; 1664 children). At two years, the proportion of wasted children was 5.95 pp higher among those in the UCT group in Asfaw 2014 (989 children; P > 0.1) and 4.7 pp in Merttens 2013 (1062 children; P > 0.1). In Merttens 2013, the proportion of children younger than five years who were severely wasted also increased by 3.9 pp (P > 0.1). Both studies are at high overall risk of bias.

In addition to the effects on stunting, five cRCTs reported on the effect of UCTs on mean WHZ (Ahmed 2019a; Ahmed 2019b; Asfaw 2014; Daidone 2014; Fenn 2015) and one on mean WHZ/month (Tonguet Papucci 2015). A meta‐analysis of data from these studies showed that UCTs made no difference on WHZ (MD –0.02, 95% CI –0.10 to 0.06; I2 = 36%; Analysis 1.9). Two of these studies were at unclear overall risk of bias and one at high overall risk of bias. A sensitivity analysis of studies at low overall risk of bias studies changed the direction of effect to unclearly favour UCTs (MD 0.02, 95% CI –0.18 to 0.21; Appendix 4) (Daidone 2014; Fenn 2015).

Prospective controlled studies

One PCS reported on stunting, with effects clearly favouring UCTs (pp –2.84, 95% CI –5.58 to –0.1; 1491 children) (Figure 5) (Renzaho 2017). This study was at high overall risk of bias.

This study also reported on effect of UCTs on mean WHZ, also reporting a clear effect favouring UCTs (MD 0.19, 95% CI 0.09 to 0.03; 1491 children) (Table 10).

1.5.3 Underweight

1.5.3.1 Weight‐for‐age z‐scores < –2SD

Three cRCTs reported unclear effects of UCTs on the proportion of underweight children (Asfaw 2014; Merttens 2013; Pellerano 2014). Two trials reported unclear effects potentially favouring UCTs and one trial reported unclear effects potentially favouring the control (P = 0.047) (Table 9; Figure 5).

The two studies favouring the intervention reported an unclear effect potentially favouring UCTs on underweight (Asfaw 2014; Pellerano 2014). Pellerano 2014 reported a reduction in proportion of children aged from birth to 36 months who were underweight at two years by 3.64 pp when they were aged 12 months (P > 0.05), Asfaw 2014 reported a reduction of 0.62 pp in the proportion of children aged under five years who were underweight (pp –0.62; 1491 children; P > 0.1).

Merttens 2013 reported an unclear effect favouring the control, as they reported an increase in the proportion of children who were underweight or severely underweight in the UCT group (3.9 pp with UCT versus 3.2 pp with control; 1062 children; P > 0.1).

In addition to the effects of UCTs on underweight, two trials also reported on the effects of UCTs on mean WAZ (Asfaw 2014; Daidone 2014). A meta‐analysis of these two studies showed that UCTs may have no effect on underweight (MD –0.04, 95% CI –0.43, 0.35; 7577 children: I2 = 74%; Analysis 1.10). Daidone 2014 was at low overall risk of bias, whereas Asfaw 2014 was at high overall risk of bias. Daidone 2014 assessed a child grant programme and Asfaw 2014 assessed a cash transfer programme for households with orphans and vulnerable children, where in some districts some conditions, such as school attendance, and penalties were imposed even though the programme was unconditional.

1.5.3.2 Body mass index

One cRCT reported unclear effects potentially favouring the control on BMI of mothers at six months (MD –0.1, 95% CI –0.36 to 0.16; 1208 mothers; Table 9) (Fenn 2015).

1.5.3.3 Mid‐upper arm circumference

One cRCT reported unclear effects potentially favouring UCTs on MUAC measures for mothers and children (MD 0.09, 95% CI –0.13 to 0.3; 1208 mothers; MD 0.06, 95% CI –0.02 to 0.15; 1683 children; Table 9) (Fenn 2015).

1.6 Change in biochemical indicators
1.6.1 Haemoglobin concentration

Two cRCTs reported on the effects of UCTs on haemoglobin concentration (Fenn 2015; Fernald 2011). A meta‐analysis of these two studies showed an unclear effect of UCTs on haemoglobin in children (MD –0.06, 95% CI –0.21 to 0.09; 2605 children; Analysis 1.11). Fenn 2015 also reported on the effects in mothers, finding a clear effect favouring the control on haemoglobin in mothers receiving UCTs (MD –0.42, 95% CI –0.63 to –0.20; 1208 mothers). Both studies were at low overall risk of bias.

1.7 Cognitive function and development

Three cRCTs reported different measures of cognitive function and development (Baird 2013; Daidone 2014; Fernald 2011). Evidence showed that UCTs make little or no difference on cognitive function and development (3 cRCTs; 10,813 children; high‐certainty evidence; summary of findings Table 1). All three trials reported an unclear effect favouring the intervention (P = 0.016; Figure 5).

Baird 2013 reported an increase in the cognitive test score based on a version of Raven's Colored Progressive matrices at two years (MD 0.14 SDs, 95% CI 0.02 to 0.26; 2057 children) and Daidone 2014 reported an increase in the Early Childhood Development score among children receiving UCTs at two years (MD 0.31; 5670 children; P > 0.1) (Table 9). Fernald 2011 reported on scores after two years for language development in young children receiving UCTs, using two different measures: scores for early language skills of children aged 12 to 35 months using the Inventario do Desenvolvemento de Habilidades Comunicativas (IDHC)‐B tool (MD 2.43, 95% CI –1.01 to 5.86; 1192 children; P > 0.1), and scores for the receptive vocabulary test (Test de Vocabulario en Imagenes Peabody (TVIP)) in children older than 36 months (MD 0.01, 95% CI –0.08 to 0.10; 1894 children; P > 0.1). However, for both of these, the effect ranged from a decrease to an increase in scores. All cognitive measures reported in the included studies are summarised in Table 7.

1.8 Change in proportion of anxiety or depression (mental health indicators)

Three cRCTs (Baird 2013; Fernald 2011; Hjelm 2017) and one RCT (Haushofer 2013) reported different measures of mental health.

1.8.1 Depression

Fernald 2011, Haushofer 2013, and Hjelm 2017 reported effects of UCTs on depressive symptoms scores using the Center for Epidemiologic Studies Depression Scale (CES‐D) (higher scores indicate worse symptoms). The meta‐analysis indicated that UCTs do not make a difference in depression scores at two years (MD –0.41, 95% CI –1.31 to 0.49; 5787 participants; I2 = 36%; Analysis 1.12). Fernald 2011 assessed the effect of the intervention on men and women, Haushofer 2013 on mothers, and Hjelm 2017 on adolescents. Fernald 2011 and Haushofer 2013 were at low overall risk of bias and Hjelm 2017 was at unclear overall risk of bias.

1.8.2 Perceived stress

Two cRCTs (Fernald 2011; Hjelm 2017) and one RCT (Haushofer 2013) reported on the effects of UCTs on perceived stress using Cohen's Perceived Stress Scale (PSS) (lower values correspond to less stress). A meta‐analysis of Haushofer 2013 and Hjelm 2017 indicated that UCTs may reduce perceived stress (MD –0.15, 95% CI –0.26 to –0.03; n = 3570; I2 = 0%; Analysis 1.13). Fernald 2011 reported an increase in the perceived stress z‐score of mothers in the intervention group at two years, both for those in the bottom quartile of baseline expenditure (MD 0.18, 95% CI –0.02 to 0.37; n = 1430; P < 0.1) and for those in the top three quartiles of baseline expenditure at two years (MD 0.05, 95% CI –0.11 to 0.20; P > 0.1).

1.8.3 Psychological distress

One cRCT reported on psychological distress, a binary measure of psychological distress, anxiety and depression; social dysfunction; and loss of confidence based on the 12‐item General Health Questionnaire (GHQ‐12) (Baird 2013). Among girls who were attending school and exposed to a UCT compared to girls in the control group, the proportion of psychological distress was smaller by 14.3 pp at one year (95% CI –21.0 to –7.6), and by 3.8 pp at two years (95% CI –13.14 to 5.8; n = 2089; P > 0.1), but the effect at two years was imprecise.

1.9 Morbidity

Seven cRCTs reported on different morbidity measures (Ahmed 2019a; Ahmed 2019b; Daidone 2014; Fenn 2015; Merttens 2013; Pellerano 2014; Tonguet Papucci 2015).

1.9.1 Respiratory infections

Four cRCTs reported on the effects of UCTs on respiratory infections (Asfaw 2014; Daidone 2014; Fenn 2015; Tonguet Papucci 2015). Two trials reported a clear effect favouring the intervention and two trials reported an unclear effect potentially favouring the intervention (P = 0.023; Table 9). Data could not be pooled.

Asfaw 2014 and Tonguet Papucci 2015 reported clear effects favouring UCTs. Tonguet Papucci 2015 reported a 21% reduced incidence of acute respiratory tract infection episodes among children aged from birth to 15 months in the previous seven days, as refereed by mothers (IRR 0.79, 95% CI 0.78 to 0.81; 1250 children; P < 0.001). Asfaw 2014 reported a reduced risk of respiratory infections of 44% among children aged from birth to seven years (957 children; P < 0.05). Asfaw 2014 was at high overall risk of bias whereas Tonguet Papucci 2015 was at low overall risk of bias.

Daidone 2014 and Fenn 2015 reported unclear effects potentially favouring UCTs. Daidone 2014 reported that the proportion of children aged from birth to 60 months with acute respiratory tract infection in a two‐week reference period was lower by 3.6 pp in the intervention group compared to the control group (effect estimate –0.036, 95% CI –0.061 to –0.011; P > 0.05). In Fenn 2015, the odds of respiratory infections was 27% lower among children in the UCT group (OR 0.73, 95% CI 0.51 to 1.03; 1683 children). Both studies were at low overall risk of bias.

1.9.2 Diarrhoeal disease

Five cRCTs reported on the effects of UCTs on diarrhoeal disease (Ahmed 2019a; Ahmed 2019b; Daidone 2014; Fenn 2015; Tonguet Papucci 2015). One study reported a clear effect favouring UCTs, two studies reported an unclear effect potentially favouring UCTs, one study reported an unclear effect potentially favouring the control, and one study reported no effect (P = 0.047) (Table 9).

Daidone 2014 reported clear effects favouring UCTs. Among children aged from birth to 60 months in the UCT group, the proportion with diarrhoea in the previous two weeks reduced by 4.9 pp at one year (pp –4.9, 95% CI –8.9 to –0.9; 7232 children; P < 0.05).

Ahmed 2019a and Ahmed 2019b reported unclear effects potentially favouring UCTs. In Ahmed 2019a, there was a reduction in the proportion of children with diarrhoea of 0.3 pp (95% CI –0.04 to 0.04, n = NR), and in Ahmed 2019b of 0.9 pp (95% CI –0.05 to 0.03, n = NR). All studies were at low overall risk of bias.

Tonguet Papucci 2015 reported no difference between the groups in the incidence of diarrhoeal episodes in the previous seven days, as reported by the mother, after one year of the intervention (IRR 1.00, 95% CI 0.97 to 1.03; 1250 children; P = 0.89). This study was at low overall risk of bias.

1.9.3 Any illness

Two cRCTs reported on the effects of UCTs on the proportion of children or people who were ill (Merttens 2013; Pellerano 2014), one reporting a clear effect favouring UCTs and the other unclear effects favouring the control (P = 0.125) (Table 9).

Pellerano 2014 reported a reduction in the proportion of children who were ill in the previous month in the intervention compared to the control group by 15 pp at one year (from 39% to 31%) (pp –15,38; 1996 children; P < 0.1).

In Merttens 2013, there was no difference between the intervention and control groups in the proportion of people who reported being ill or injured in the previous three months, after one year of the intervention (pp 1.0; 14,342 participants; P > 0.1). In this study, these proportions reduced significantly in both intervention and control groups, and injuries were also included as an 'illness'. Pellerano 2014 was at low overall risk of bias whereas Merttens 2013 was at high overall risk of bias due to high risk of selection and attrition bias.

1.10 Adverse outcomes (proportion of participants overweight or obese)

Pellerano 2014, a cRCT, reported unclear effects potentially favouring UCTs on the proportion of infants who were overweight when they were aged six and 12 months, at two years of the intervention (6 months old: pp –5.08; 474 children; P > 0.05; 12 months old: pp –6.46; 293 children; P > 0.05) (Table 9).

Comparison 2: conditional cash transfers

Fourteen included studies assessed CCTs, where a specified amount of money was transferred to poor families regularly as long as they meet specific conditions. Nine were cRCTs (Baird 2013; Evans 2014; Gertler 2000 (PROGRESA); Hidrobo 2014; Kandpal 2016; Kurdi 2019; Kusuma 2017a; Macours 2012; Maluccio 2005) and five were PCS (Andersen 2015; Ferre 2014; Huerta 2006 (PROGRESA); Leroy 2008 (PROGRESA); Lopez Arana 2016). All studies compared CCTs with no intervention. However, requirements and other intervention components differed across studies. Requirements (or conditions) included regular check‐ups for children, school enrolment and regular attendance, vaccination, micronutrient supplementation for children or for pregnant women, and attending nutrition education sessions.

Two cRCTs (Macours 2012; Maluccio 2005) and one PCS (Ferre 2014) reported the proportion of household expenditure on food. Two cRCTs (Hidrobo 2014; Kurdi 2019) and one PCS (Ferre 2014) reported on dietary diversity measures. Seven cRCTs (Evans 2014; Gertler 2000 (PROGRESA); Kandpal 2016; Kusuma 2017a; Kurdi 2019; Macours 2012; Maluccio 2005; ) and four PCS (Andersen 2015Leroy 2008 (PROGRESA); Lopez Arana 2016; Ferre 2014) reported on various anthropometric measures. Two cRCTs (Baird 2013; Macours 2012) and one PCS (Andersen 2015) reported on cognitive function and development measures. One cRCT (Baird 2013) reported on psychological distress. Four cRCTs reported measures of morbidity (Evans 2014; Gertler 2000 (PROGRESA); Kandpal 2016; Macours 2012). Two PCS reported adverse measures of overweight (Andersen 2015; Lopez Arana 2016).

Further details about the studies in this comparison are presented in Table 11. Table 12 presents the results of cRCTs and Table 13 of PCS. summary of findings Table 2 and harvest plot in Figure 6 summarise the results of CCTs on key outcomes.

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Table 11. Conditional cash transfers – overview of included studies

Study name (year) country of conduct

Study design

Overall risk of biasa

Other key detail of intervention

Population (sample size at baseline: intervention/ control)

Outcome domains and measures with available data

Timepoint of measurement

Baird 2013

(Malawi)

cRCT

Low

Programme name: Schooling, Income, and Health Risks study (SIHR). Includes unconditional and conditional groups.

Type, amount and frequency of payments: payments were split between guardian and girl in each HH. HH amount varied randomly from USD 4, USD 6, USD 8, to USD 10 per month. Amount paid to girl beneficiaries varied randomly from USD 1, USD 2, USD 3, USD 4, to USD 5 per month.

Conditionalities: school attendance for 80% of the days during the previous month.

Provider: 2 NGOs

Delivery: payments to girl beneficiaries at local distribution points

Co‐interventions: NR

Adolescent girls who were never married from urban and rural HHs (1211/1495 girls)

Cognitive function and development:

  • Cognitive test score (Raven's Coloured matrices and other)

Anxiety/depression:

  • Psychological distress test score (GHQ‐12)

1 and 2 years

Macours 2012

(Nicaragua)

cRCT

Low

Programme name: Atención a Crisis

Amount and frequency of payments: Standard payment of USD 145 per HH every 2 months. 3 intervention groups:

1. Standard transfer + education: additional USD 145 per HH and USD 25 per child for HHs with children aged 7–15 years; 2. Standard transfer + scholarship for vocational training; and 3. Standard transfer + lump sum to start non‐agricultural activity.

Conditionalities: 1. Regular health check‐ups for children aged 0–5 years, school enrolment; 2. regular attendance, however not monitored in practice; and 3. developing a business plan.

Provider: government

Delivery: payments to child's primary carer.

Co‐interventions: NR

Poor rural HHs with 2377 children aged < 6 years (3002/1019 HHs)

HH expenditure on food:

  • Percentage of total expenditure

Anthropometric indicators:

  • WAZ

  • HAZ

Anxiety/depression:

  • Depression score (CES‐D)

Cognitive function and development:

  • Language test score (TVIP score)

Morbidity – child

  • Number of days ill in bed in the past month

9 months

(12 months for CES‐D)

Maluccio 2005

(Nicaragua)

cRCT

Low

Programme name: Red de Protección Social

Amount and frequency of payments: amount NR; payments every 2 months.

Conditionalities: school attendance; preventive health care visits for children for growth and development monitoring, vaccination, and provision of antiparasites, vitamins, and iron supplements.

Provider: government. Preventive health services provided by private healthcare providers.

Delivery: NR

Co‐interventions: NR

Poor, rural HHs (1396 HHs)

HH expenditure on food:

  • Percentage of total expenditure

Anthropometric indicators:

  • HAZ

  • WAZ

  • WHZ

1 and 2 years

Kusuma 2017a

(Indonesia)

cRCT

Unclear

Programme name: Program Keluarga Harapan (PKH)

Amount and frequency of payments: USD 60–220 per HH per year, depending on the number and age of children in the HH.

Conditionalities: health: pre‐ and postnatal visits, iron supplementation and assisted deliveries for pregnant women, growth monitoring, immunisation and vitamin A supplementation of children aged < 5 years. Education: primary and junior secondary school enrolment and attendance rates of 85%.

Provider: government

Delivery: payment to mothers through local post offices

Co‐interventions: NR

Very poor urban HHs with children aged 24–36 months (1395 HHs)

Anthropometry:

  • Underweight (WAZ < –2SD)

  • Severe underweight (WAZ < –3SD)

  • Wasting (WHZ < –2SD)

  • Severe wasting (WHZ < –3SD)

  • Stunting (HAZ < –2SD)

  • Severe stunting (HAZ < –3SD)

2 years

Gertler 2000 (PROGRESA)

(Mexico)

cRCT

Unclear

Programme name: Oportunidades (previously known as PROGRESA)

Type, amount and frequency of payments: scholarships of up to MXN 490 (January–June 98) and MXN 625 per HH (July–December 1999), every 2 months; payments for school supplies; and monthly payments for food.

Conditionalities: health: attendance of preventive health services by every family member; growth monitoring and immunisation of children aged 0–5 years; nutrition supplements (for lactating women, children aged 6–23 months or low‐weight children), antenatal care for pregnant women. Education: school enrolment and school attendance > 85%.

Provider: government

Delivery: lump sum payment to mothers once completed forms were submitted by HHs to verify school attendance.

Co‐interventions: NR

Poor rural HHs

(506 villages; 320/186)

Anthropometric indicators:

  • HAZ

  • Stunting (HAZ < –2SD)

  • BMIZ

Biochemical indicators:

  • Anaemia

Cognitive function and development:

  • Cognitive test scores (verbal, cognitive, behavioural)

Morbidity – Child

  • Illness during past 4 weeks

8, 12, 15, 20 months, 10 years

Evans 2014

(Tanzania)

cRCT

High

Programme name: N/A

Amount and frequency of payments: USD 12–36, depending on the number of people in the HH, every 2 months.

Conditionalities: education: primary school enrolment and attendance for children aged 7–15 years; health: health facility visits for growth monitoring 6 times a year for children aged 0–5 years; vaccination and growth monitoring for children 0–2 years; yearly visit to health facility for elderly people (aged ≥ 60 years).

Provider: Tanzania Social Action Fund (TASAF), World bank

Delivery: payments disbursed by TASAF to bank accounts managed by local government authorities. Funds disbursed directly to community‐managed accounts who made payments to mothers.

Co‐interventions: transfers from government/TASAF or from NGOs/religious organisation

Poor HHs with vulnerable children or elderly people, or both

(80 villages; 40/40)

Anthropometric indicators: NR

30 and 42 month

Hidrobo 2014

(Colombia)

cRCT

High

Programme name: N/A

Amount and frequency of payments: USD 40 per month per HH.

Conditionalities: attendance of monthly nutrition sensitisation training sessions by HH members.

Provider: World Food Programme (NPO)

Delivery: money transferred on to pre‐programmed debit cards.

Co‐interventions: NR

Poor urban HHs (2357 HHs)

HH expenditure on food:

  • Proportion of total expenditure per month

Dietary diversity:

  • DDI

  • HDDS

  • FCS

7 months

Kandpal 2016

(Philippines)

cRCT

High

Programme name: Pantawid Pamilyang Pilipino Programme

Type, amount and frequency of payments: health grant of PHP 500 (USD 11) per HH per month; education grant of PHP 300 (USD 6.50) per child per month for ≤ 10 months/year, and for ≤ 3 children in the HH. Payments every 2 months.

Conditionalities: health: clinic visits for immunisation and vaccination, growth monitoring, and management of childhood disease in children aged < 5 years; antenatal care for pregnant women, starting from the first trimester; school‐aged children (6–14 years) to receive deworming tablets 2 times/year; and HHs with children 0–14 years, the HH grantee (mother) or spouse (or both) had to attend family development sessions monthly. Education: enrolment of children aged 6–14 years in primary or secondary school and 85% school attendance every month.

Provider: government

Delivery: NR

Co‐interventions: NR

Poor HHs with children aged 0–14 years or pregnant women (714/ 704 HHs)

Anthropometric indicators:

  • WAZ

  • Underweight (WAZ < –2SD)

  • Severely underweight (WAZ < –3SD)

  • HAZ

  • Stunted (HAZ < –2SD)

  • Severely stunted (HAZ < –3SD)

Morbidity – child:

  • Seeking treatment for child for fever, cough or diarrhoeal disease in past 2 weeks

36 months

Kurdi 2019

(Yemen)

cRCT

High

Programme name: Cash for Nutrition programme

Amount and frequency of payments: payments every 3 months (YER 30,000 per month for 9 months in 2015; YER 10,000 (USD 30) per month for 12 months in 2016/2017) to mothers of children aged 2 years of age and pregnant women.

Conditionalities: attending monthly nutrition‐focused trainings, complying with child monitoring and treatment of malnutrition. Attendance tracked but conditionality not strictly enforced.

Provider: government, Yemen Emergency Crisis Response Project (funded by the World Bank)

Delivery: nutrition sessions delivered by trained local women. Details of cash transfer not reported.

Co‐interventions: unspecified other food distribution programmes.

Women from poor and vulnerable (1001/999 women)

Diet diversity:

  • HDDS

Anthropometric indicators:

  • HAZ

  • WHZ

2.5 years

Andersen 2015

(Peru)

Prospective controlled study

High

Programme name: Juntos

Amount and frequency of payments: PEN 100 (30 US dollars) each month regardless of HH composition.

Conditionalities: regular health visits for children aged < 5 years, or pregnant and lactating women. Children aged 6–14 years with primary school attendance ≥ 85%.

Provider: Peruvian government

Delivery: NR

Co‐interventions: NR

Poor HHs with children aged 6–18 months (374/586 children)

Anthropometric indicators:

  • HAZ

  • Stunting (HAZ < –2SD)

  • BMIZ

Cognitive function and development:

  • Language (TVIP) score

  • Grade attainment

Adverse effects:

  • Overweight (BMIZ > 2SD)

< 2 years and ≥ 2 years

Ferre 2014

(Bangladesh)

Prospective controlled study

High

Programme name: Shombhob project

Amount and frequency of payments: BDT 400 per months for HHs with children 0–36 months and BDT 400 per month for HHs with primary school children (6–15 years).

Conditionalities: Health: Attending growth monitoring of children aged 0 – 36 months, and nutrition session for mother/carer. Education: school attendance of at least 80% every month.

Provider: Government

Delivery: Cash cards provided to beneficiary mothers. Electronic transfer to their accounts with the Bangladesh Post Office (BPO). Withdrawal from mobile machines on a designated day during each payment cycle in each village, or from Upazila BPO branch office at any time point.

Rural HHs (700/1587)

HH expenditure on food:

  • Proportion of total expenditure

Dietary diversity:

  • MDD

Anthropometric indicators:

  • Stunting (HAZ < –2SD)

  • Wasting (WHZ < –2SD)

  • Underweight (WAZ < –2SD)

13 months

Huerta 2006 (PROGRESA) (Mexico)

Prospective controlled study

High

Programme name: Oportunidades (previously known as PROGRESA)

Type, amount and frequency of payments: SeeGertler 2000 (PROGRESA)

Conditionalities: seeGertler 2000 (PROGRESA)

Provider: Mexican government

Delivery: seeGertler 2000 (PROGRESA)

Co‐interventions: NR

Poor rural HHs with ≥ 1 child aged < 5 years (205/142 communities)

Anthropometric indicators:

  • LAZ or HAZ

  • WAZ

  • WLZ or WHZ

Biochemical indicators:

  • Anaemia

  • Hb

Morbidity – child:

  • Respiratory infection during the past 2 weeks

  • Diarrhoeal disease during the past 2 weeks

14 and 26 months

Leroy 2008 (PROGRESA)

(Mexico)

Prospective controlled study

High

Programme name: Oportunidades (previously known as PROGRESA)

Type, amount and frequency of payments: USD 32.5–41.3 per month (see Gertler 2000 (PROGRESA))

Conditionalities: seeGertler 2000 (PROGRESA)

Provider: government of Mexico

Delivery: see Gertler 2000 (PROGRESA)

Co‐interventions: NR

Poor and vulnerable urban HHs

(733 children aged 0–24 months)

Anthropometric indicators:

  • HAZ

  • WHZ

2 years

Lopez Arana 2016

(Colombia

Prospective controlled study

High

Programme name: Familias en Acción

Type, amount and frequency of payments:

COP 40,000 for children aged < 7 years; COP 14,000 per primary school and COP 28,000 per secondary school child. Periodic payments.

Conditionalities: children aged < 7 years to attend vaccination programmes and growth and development check‐ups regularly; children aged 7–17 years to attend ≥ 80% of school lessons.

Provider: government, World Bank and Inter‐American Development Bank

Delivery: transfer of cash to mothers into the HH bank account.

Co‐interventions: some children participated in a childcare supplementary nutrition and psychosocial stimulation programme (Hogares Comunitarios programme).

Poor HHs with children aged 0–17 years (9293/4424)

Anthropometric indicators:

  • HAZ

  • Stunting (HAZ < –2SD)

  • BMIZ

  • Thinness (BMIZ < –2SD)

Adverse events:

  • Overweight (BMIZ > 1)

  • Obesity (BMIZ > 2)

About 4 years

aOverall Risk of Bias based on risk of selection and attrition bias.

BMIZ: body mass index‐for‐age z‐score; CES‐D: Center for Epidemiologic Studies Depression Scale; COP: Colombian peso; cRCT: cluster randomised controlled trial; DDI: Dietary Diversity Index; FCS: Food Consumption Score; GHQ‐12: 12‐item General Health Questionnaire; HAZ: height‐for‐age z‐score; Hb: haemoglobin; HDDS: Household Dietary Diversity Score; HH: household; LAZ: length‐for‐age z‐score; MXN: Mexican peso; N/A: not applicable/available; non‐governmental organisation; NPO: non‐profit organisation; NR: not reported; PEN: Yemeni rial; PHP: Philippine peso; TVIP: Test de Vocabulario en Imagenes Peabody; WAZ: weight‐for‐age z‐score; WHZ: weight‐for‐height z‐score; WLZ: weight‐for‐length z‐score; YER: Yemeni rial.

Open in table viewer
Table 12. Conditional cash transfers – results of included trials

Study ID (risk of bias)

Study design (n)

Conditional cash transfers

No intervention

Effect measure (time point)

Effect directiona

Meta‐analysis (yes/no)

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

2.2: Proportion of HH expenditure on food

2.2.1 Outcome measure: proportion of HH expenditure on food (weekly/monthly)

Maluccio 2005 (+)

cRCT (1490 HHs)

69.8

70

766

70.2

66.5

724

pp 3.9, SE 1.7, 95% CI 0.568 to 7.232, P < 0.01 (1 year)

N/A

cRCT (1434 HHs)

722

712

pp 4.1, SE 1.3, 95% CI 1.552 to 6.648, P < 0.01 (2 years)

2.2.2 Outcome measure: proportion of food in total expenditures (SDs)

Macours 2012 (+)

cRCT (3326 HHs)

70%

70.7%

Effect 0.005, SD, SE 0.009, 95% CI –0.013 to 0.023, P > 0.1 (9 months)

2.3: Proportion of HHs who were food secure

2.3.2 Dietary diversity

2.3.2.2 Outcome measure: HDDS (012) (mean)

Hidrobo 2014 (‐)

cRCT (2087 HHs)

9.23

9.11

MD 0.46, SE 0.11, 95% CI 0.244 to 0.676, P < 0.01 (7 months)

Yes

Kurdi 2019 (‐)

cRCT (1850 HHs)

935 HHs

915 HHs

MD 0.374, SE 0.262, 95% CI –0.13952 to 0.88752 (2.5 years)

Secondary outcomes

2.5Change in anthropometric indicators

2.5.1 Stunting (chronic undernutrition)

2.5.1.1 Outcome measure: proportion stunted (HAZ < –2SD)

Maluccio 2005 (+)

cRCT (722 children aged < 5 years)

41.9

37.1

40.9

41.5

pp –5.3, 95% CI –11.376 to 0.776, P < 0.1 (2 years)

Yes; this subset.

This was entered as MD: difference in percentage stunted

Gertler 2000 (PROGRESA) (?)

cRCT (n at follow‐up 1062)

0.396

0.410

OR 0.914, P = 0.495 (20 months)

Kusuma 2017a (?)

cRCT (1394 children aged 24–36 months)

mean 0.55

DID 0.035, SE 0.046, 95% CI –0.05516 to 0.12516

pp 3.5, 95% CI –5.5 to 12.5, P > 0.05 (2 years)

Yes; this subset.

This was entered as MD: difference in percentage stunted

Kandpal 2016 (‐)

cRCT (351 children aged < 36 months)

49.701

pp –3.768, 95% CI –13.830 to 6.294, P > 0.1 (36 months)

2.5.1.2 Outcome measure: proportion with severe stunting (HAZ < –3SD)

Kusuma 2017a (?)

cRCT (1394 children aged 24–36 months)

mean 0.29

DID 0.047, SE 0.053, 95% CI –0.05688 to 0.15088.

pp 4.7, 95% CI –5.7 to 15.1, P > 0.05 (2 years)

Yes

Kandpal 2016 (‐)

cRCT (351 children aged < 36 months)

–10.189

23.952

pp –10.189, 95% CI –18.769 to –1.607 (3 years)

2.5.1.3 Outcome measure: HAZ (mean, SD)

Maluccio 2005 (+)

cRCT (1036 children aged < 5 years)

–1.79 (1.14)

–1.65 (1.15)

479

–1.76 (1.15)

–1.80 (1.18)

557

MD 0.17, 95% CI 0.0132 to 0.327, P < 0.05 (2 years)

Yes

Macours 2012 (+)

cRCT (3082 children aged < 6 years)

–1.27b

–1.08b

MD 0.072, 95% CI 0.005 to 0.139, P < 0.05 (9 months)

Evans 2014 (‐)

cRCT (102 children aged 0–4 years)

MD 0.86, 95% CI –2.358 to 3.718, P > 0.1 (1.5 years)

Kandpal 2016 (‐)

cRCT (351 children)

0.284

–1.903

MD 0.284, 95% CI –0.034 to 0.600, P < 0.1 (3 years)

Kurdi 2019 (‐)

cRCT (1048 children)

MD 0.109, SE 0.146, 95% CI –0.18 to 0.395 (2.5 years)

2.5.2Wasting (acute undernutrition)

2.5.2.1 Outcome measure: proportion wasted (WHZ < –2SD)

Maluccio 2005 (+)

cRCT (722 children aged < 5 years)

1.0%

0.4%

479

0.3

0.2

557

pp –0.4, SE 0.5, 95% CI –1.38 to 0.58, P > 0.1 (2 years)

Yes

Kusuma 2017a (?)

cRCT (1394 children aged 24–36 months)

Mean 0.19

DID –0.063, SE 0.032, 95% CI –0.12572 to –0.00028, P < 0.05

2.5.2.2 Outcome measure: proportion severely wasted (WHZ < –3SD

Kusuma 2017a (?)

cRCT (1394 children aged 24–36 months)

Mean 0.09

Beta –0.037, SE 0.022, 95% CI –0.08012 to 0.00612, P < 0.1

2.5.2.3 Outcome measure: WHZ (mean, SD)

Evans 2014 (‐)

cRCT (63 children aged 0–4 years)

MD –0.03, SE 0.45, 95% CI –0.9120 to 0.852, P > 0.1 (1.5 years)

Yes

Kurdi 2019 (‐)

cRCT (1048 children)

MD 0.190, SE 0.148, 95% CI –0.10008 to 0.48008 (2.5 years)

2.5.3 Underweight

2.5.3.1 Outcome measure: proportion underweight (WAZ < –2SD)

Maluccio 2005 (+)

cRCT (722 children aged < 5 years)

15.3

10.4

14.7

15.8

pp –6, SE 2.6, P < 0.05 (2 years)

Yes

Kusuma 2017a (?)

cRCT (1394 children aged 24–36 months)

Mean 0.38

DID –0.040, SE 0.036, 95% CI –0.11056 to 0.03056, P > 0.05

Kandpal 2016 (‐)

cRCT (390 children aged < 36 months)

28.72

pp –2.57, 95% CI –11.980 to 6.839 (3 years)

2.5.3.2 Outcome measure: proportion severely underweight (WAZ < –3SD)

Kusuma 2017a (?)

cRCT (1394 children aged 24–36 months)

Mean 0.10

DID –0.025, SE 0.024, 95% CI –0.07204 to 0.02204

Kandpal 2016 (‐)

cRCT (390 children aged < 36 months)

8.51

pp 1.075, 95% CI –4.72 to 6.87, P > 0.1 (3 years)

Yes

2.5.3.3 Outcome measure: weight‐for‐age z‐score (WAZ) (mean standard deviation)

Macours 2012 (+)

cRCT (3082 children aged < 6 years)

–1.06

–0.88

MD 0.036, SE 0.037, 95% CI –0.037 to 0.109, P > 0.1 (9 months)

Yes

Evans 2014 (‐)

cRCT (76 children 0–4 years)

MD –0.29, SE 1.25, 95% CI –2.74 to 2.16, P > 0.1 (1.5 years)

Kandpal 2016 (‐)

cRCT (390 children < 36 months)

0.14

MD 0.140, 95% CI –0.161 to 0.438, P > 0.1 (3 years)

2.5.3.4 Outcome measure: BMI‐for‐age z‐score

Evans 2014 (‐)

cRCT (64 children aged 0–4 years)

MD –1.55, 95% CI –4.43 to 1.33, P > 0.1 (1.5 years)

2.7 Cognitive function and development

2.7.1 Outcome measure: cognitive test scores/cognitive and socioemotional outcomes (mean, SD)

Macours 2012 (+)

cRCT (3326 children)

MD 0.1211, SE 0.028, 95% CI 0.066 to 0.176 P < 0.01 (9 months)

Yes

Baird 2013 (+)

cRCT (2057 schoolgirls)

MD 0.174, 95% CI 0.0799 to 0.268, SE 0.048, P < 0.01 (2 years)

2.8 Change in proportion of anxiety and depression

2.8.1 Outcome measure: proportion with psychological distress

Baird 2013 (+)

cRCT (2089 schoolgirls)

Mean 0.374, SE 0.02, P < 0.01

pp –0.063, SE 0.03, P < 0.05 (1 year)

N/A

Mean 0.308, SE 0.017, P < 0.01

pp –0.039, SE 0.047, P > 0.1 (2 years)

2.9 Morbidity

2.9.1 Outcome measure: proportion reporting being ill in past 4 weeks/parents seeking care for illness in past 2 weeks

Gertler 2000 (PROGRESA) (?)

cRCT (7703 children aged 0–35 months)

OR 0.777, P = 0.000 (20 months)

Yes. Gertler subgroup 3–5 years selected as converting OR to SMD not possible due to missing group sizes.

cRCT (19,939 children aged 3–5 years at baseline)

0.280

0.097

0.263

0.127

Estimate –0.021, 95% CI –0.045 to 0.003 (20 months)

Evans 2014 (‐)

cRCT (18,192 participants)

Estimate –0.04, 95% CI –0.099 to 0.019, P > 0.1 (32 months)

Kandpal 2016 (‐)

cRCT (456 children aged 6–36 months)

229

41.85

227

pp 9.830, 95% CI 0.179 to 19.481, P < 0.05 (36 months)

2.9.2 Outcome measure: number of days ill in bed (SD)

Macours 2012 (+)

cRCT (3326 children)

0.669

MD –0.357 SD, SE 0.133, 95% CI –0.6178 to –0.096, P < 0.01 (9 months)

2.9.3 Outcome measure: proportion with anaemia

Gertler 2000 (PROGRESA) (?)

cRCT (2010 children)

0.410

0.483

OR 0.745, P = 0.012 (20 months)

aEach triangle represents one study.
bValues derived from graphs

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

CI: confidence interval; cRCT: cluster randomised controlled trial; DID: difference in differences; HAZ: height‐for‐age z‐score; HDDS: Household Dietary Diversity Score; HH: household; MD: mean difference; n: number; N/A: not applicable/available; OR: odds ratio; pp: percentage point; SD: standard deviation; SE: standard error; SMD: standardised mean difference; WAZ: weight‐for‐age z‐score; WHZ: weight‐for‐height z‐score.

Open in table viewer
Table 13. Conditional cash transfers – results of included prospective controlled studies

Study ID (risk of bias)

Study design (n)

Conditional cash transfers

No intervention

Effect measure (time point)

Effect directiona

Meta‐analysis (yes/no)

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

2.2 Proportion of HH expenditure on food

2.2.1 Outcome measure: proportion of HH expenditure on food (weekly/monthly)

Ferre 2014 (‐)

PCS (n NR)

3168/5548 = 0.57

3153/5780 = 0.55

Proportion after study period is 337.0/378.8 = 0.89 (not impact) (13 months)

N/A

2.3: Proportion of HHs who were food secure

2.3.2 Dietary diversity

2.3.2.1 Proportion with MDD

Ferre 2014 (‐)

Prospective controlled study (n = 1318 children)

12.1

12.5

MD 0.031, SE 0.05, 95% CI –0.067 to 0.129 (13 months)

Secondary outcomes

2.5Change in anthropometric indicators

2.5.1 Stunting (chronic undernutrition)

2.5.1.1 Outcome measure: proportion stunted (HAZ < –2SD)

Ferre 2014 (‐)

Prospective controlled study (1580 children)

47.2

43.3

MD 0.034, SE 0.05, 95% CI –0.064 to 0.132 (13 months)

Yes. Subset. (except Lopez‐Arana as OR could not be converted to SMD due to missing group sizes)

Andersen 2015 (‐)

Prospective controlled study (n = 188 children)

91 (48.4%)

72 (38.3%)

80 (42.6%)

76 (40.4%)

Treatment effect: –7.98, 95% CI –22.3 to 6.34, P = 0.27 (< 2 years)

Prospective controlled study (n = 169 children)

101 (59.8%)

67 (39.6%)

84 (49.7%)

81 (47.9%)

Treatment effect –18.3, 95% CI –38.3 to 1.59, P = 0.07 (≥ 2 years)

Lopez Arana 2016 (‐)

Prospective controlled study (2874 children)

391 (30.3%)

442 (27.9%)

OR 0.92, 95% CI 0.82 to 1.05, P > 0.05 (4 years)

2.5.1.2 Outcome measure: height‐for‐age z‐score (HAZ) (mean, SD)

Leroy 2008 (PROGRESA) (‐)

Prospective controlled study (432 children)

–1.29 (1.36)

–1.4 (1.16)

MD 0.1, 95% CI –0.086 to 0.306, P = 0.13 (2 years)

Yes

Andersen 2015 (‐)

Prospective controlled study (n = 188 children)

–1.97 (1.1)

–1.76 (0.864)

–1.80 (1.02)

–1.71 (0.757)

MD 0.12, 95% CI –0.10 to 0.33, P = 0.28 (< 2 years)

Prospective controlled study (n = 169 children)

–2.11 (1.24)

–1.85 (0.829)

–2.08 (1.12)

–1.95 (0.813)

MD 0.14, 95% CI –0.20 to 0.49, P = 0.41 (≥ 2 years)

Lopez Arana 2016 (‐)

Prospective controlled study (2874 children)

–1.47 (1.21)

–1.42 (1.13)

MD 0.00, 95% CI –0.10 to 0.11, P > 0.05 (4 years)

2.5.2:Wasting (acute undernutrition)

2.5.2.1 Outcome measure: proportion wasted (WHZ < –2SD)

Ferre 2014 (‐)

Prospective controlled study (2244 children)

27.8

22.9

MD/DID –0.036, SE 0.04, 95% CI –0.1144 to 0.0424 (ages 22–46 months when enrolled)

MD –0.125, SE 0.07, 95% CI –0.2622 to 0.0122 (aged 10–22 months when enrolled) pp –12.5

(13 months)

No. Lopez‐Arana/Ferre 2014 could not be converted to SMD due to missing group sizes.

Lopez Arana 2016 (‐)

Prospective controlled study (2874 children)

25 (1.9%)

14 (0.9%)

OR 0.25, 95% CI 0.09 to 0.74, P < 0.05 (4 years)

2.5.2.2 Outcome measure: WHZ (mean, SD)

Leroy 2008 (PROGRESA) (‐)

Prospective controlled study (432 children)

0.30 (1.07)

0.33 (1.00)

MD 0.085, 95% CI –0.113 to 0.283, P = 0.2 (2 years)

2.5.3 Underweight

2.5.3.1 Outcome measure: proportion underweight (WAZ <2SD)

Ferre 2014 (‐)

Prospective controlled study (1638 children)

47.1

42.9

MD/DID 0.046, SE 0.05, 95% CI –0.052 to 0.144

pp 4.6 (13 months)

N/A

2.5.3.2 Outcome measure: BMIZ (mean, SD)

Andersen 2015 (‐)

Prospective controlled study (n = 188 children)

0.527 (1.15)

0.145 (0.833)

0.790 (0.986)

0.436 (0.739)

MD –0.028, 95% CI –0.31 to 0.25, P = 0.84 (< 2 years)

Yes

Prospective controlled study (n = 169 children)

0.613 (1.23)

0.248 (0.788)

0.622 (1.3)

0.622 (0.773)

MD –0.36, 95% CI –0.79 to 0.06, P = 0.09 (≥ 2 years)

Lopez Arana 2016 (‐)

Prospective controlled study (2874 children)

MD 0.14, 95% CI 0.00 to 0.27, P < 0.05 (4 years)

2.7 Cognitive function and development

2.7.1 Outcome measure: language score (TVIP) (mean, SD)

Andersen 2015 (‐)

Prospective controlled study (n = 243 children)

–0.538 (0.782)

–0.718 (0.959)

–0.531 (0.761)

–0.552 (1.03)

Coefficient –0.15, 95% CI –0.37 to 0.066, P = 0.17 (≥ 2 years)

N/A

2.10: Adverse outcomes: overweight/obesity

2.10.1 Outcome measure: overweight (BMI z‐score >2SD)

Andersen 2015 (‐)

Prospective controlled study (n = 188 children)

n = 65, 34.6%

n = 24, 12.8%

n = 81, 43.1%

n = 34, 18.1%

pp 3.19, 95% CI –9.93 to 16.3, P = 0.63 (< 2 years)

Yes

Prospective controlled study (n = 169 children)

n = 65, 37.9%

n = 28, 16.6%

n = 64, 37.9%

n = 42, 24.9%

pp –8.89, 95% CI –24.7 to 7.0, P = 0.27 (≥ 2 years); log OR –0.2784

Lopez Arana 2016 (‐)

Prospective controlled study (2874 children)

OR 1.30, 95% CI 0.83 to 2.03, P > 0.05 (4 years)

2.10.2 Obesity

Lopez Arana 2016 (‐)

Prospective controlled study (2874 children)

41 (3.2%)

37 (2.3%)

OR 0.56, 95% CI 0.20 to 1.53, P > 0.05

aEach triangle represents one study.

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

BMIZ: body mass index‐for‐age z‐score; CI: confidence interval; DID: difference in differences; HAZ: height‐for‐age z‐score; HH: household; MD: mean difference; n: number; N/A: not applicable/available; NR: not reported; OR: odds ratio; PCS: prospective controlled study; SD: standard deviation; SE: standard error; TVIP: Test de Vocabulario en Imagenes Peabody; WAZ: weight‐for‐age z‐score; WHZ: weight‐for‐height z‐score.


Harvest plot: conditional cash transfers.

Harvest plot: conditional cash transfers.

Primary outcomes
2.1 Change in the prevalence of undernourishment

None of the included studies measured prevalence of undernourishment.

2.2 Proportion of household expenditure on food

Two cRCTs (Macours 2012; Maluccio 2005) and one PCS (Ferre 2014) reported proportion of household expenditure on food.

Randomised controlled trials

Evidence from two cRCTs indicated that CCTs result in little to no difference in the proportion of household expenditure on food (4760 households, 2 RCTs; high‐certainty evidence; summary of findings Table 2). One study reported a clear effect favouring the control and one study reported an unclear effect potentially favouring the control (P = 0.125) (Figure 6). Both studies were at low overall risk of bias.

Maluccio 2005 reported clear effects favouring the control, with an increase in the proportion of household expenditure of 3.9 pp at one year (1490 households; P < 0.01) and of 4.1 pp at two years (1434 households; P < 0.01) (Table 12).

Macours 2012 reported an unclear effect potentially favouring the control because among those in the intervention group the proportion of household expenditure increased very slightly by 0.01 SDs (95% CI –0.01 to 0.02; 3326 households).

Prospective controlled studies

Ferre 2014 reported on this outcome but did not report an effect estimate (Table 13).

2.3 Proportion of households who were food secure
2.3.1 Food security

None of the studies in this comparison reported food security.

2.3.2 Dietary diversity

Randomised controlled trials

Evidence from two cRCTs indicated that CCTs probably slightly increase dietary diversity (3937 households; 2 cRCTs; moderate‐certainty evidence; summary of findings Table 2; Figure 6) (Hidrobo 2014; Kurdi 2019). A meta‐analysis of these two studies, which reported FCS (scale: 0 to 112, higher score indicating better dietary diversity) showed a clear effect favouring CCTs (MD 0.45, 95% CI 0.25 to 0.65; 3937 households; I2 = 0%). Both trials were at high overall risk of bias.

Prospective controlled trials

Ferre 2014 reported an unclear effect potentially favouring CCT on the proportion of children with MDD (i.e. proportion of children aged six months and above fed from at least four food groups) (Figure 6). The proportion of children with MDD increased by 3.1 pp with CCTs compared to the control group at 13 months (MD 0.03, 95% CI –0.07 to 0.13, n = 1318) (Table 13).

Secondary outcomes
2.4 Change in adequacy of dietary intake

No included study reported the adequacy of dietary intake. Some studies reported caloric availability and intake; we have not reported these data as they do not relate to measures of adequacy.

2.5 Change in anthropometric indicators

Seven cRCTs (Evans 2014; Gertler 2000 (PROGRESA); Kandpal 2016; Kusuma 2017a; Kurdi 2019; Macours 2012; Maluccio 2005) and four PCS (Andersen 2015; Ferre 2014; Leroy 2008 (PROGRESA); Lopez Arana 2016) reported on various anthropometric measures.

2.5.1 Stunting: height‐for‐age z‐scores < –2SD (chronic undernutrition)

Cluster randomised controlled trials

Evidence from four cRCTs (Gertler 2000 (PROGRESA); Kandpal 2016; Kusuma 2017a; Maluccio 2005) showed that CCTs may make little or no difference to the proportion of stunted children (4 RCTs, 3529 children; low‐certainty evidence; summary of findings Table 2). Three studies showed an unclear effect favouring CCTs and one study showed an unclear effect potentially favouring the control (P = 0.016) (Figure 6).

Gertler 2000 (PROGRESA), Kandpal 2016, and Maluccio 2005 reported an unclear effect potentially favouring CCTs. The proportion of stunted children was reduced among those receiving CCTs in these three studies; however, all the 95% CI crossed the null effect (Table 12). In Maluccio 2005, the proportion reduced by 5.3 pp at two years (95% CI –11.38 to 0.78; 722 children aged under 5 years); in Gertler 2000 (PROGRESA), the odds of children being stunted was lower by 8.6% at 1.6 years (OR 0.91; 1062 children; P = 0.495); and in Kandpal 2016 the proportion of stunted children was lower by 3.77 pp at three years (95% CI –13.83 to 6.29; 351 children younger than 36 months; P > 0.1). Kandpal 2016 also reported that, in the CCT group, the proportion of children who were severely stunted was reduced by 10.19 pp compared to the control group at three years (95% CI –18.77 to –1.61; 351 children).

Kusuma 2017a reported an unclear effect potentially favouring the control. Among children aged 24 to 36 months in the CCT group, the proportion of stunting increased by 3.5 pp (95% CI –5.5 to 12.5; 1394 children) (Table 12). This study reported a similar effect on the proportion of children who were severely stunted (HAZ < –3SD).

A meta‐analysis of three of these studies showed an unclear effect favouring CCTs (MD –2.51, 95% CI –7.78 to 2.75; 2467 children; I2 = 22%; Analysis 2.2) (Kandpal 2016; Kusuma 2017a; Maluccio 2005). The two studies that reported on the effects of CCTs on severe stunting could not be pooled due to high heterogeneity (I2 = 78%; Analysis 2.3) (Kandpal 2016; Kusuma 2017a). The cash transfer programmes evaluated in these studies are similar in the cash transfer amount and programme conditions; however, Kusuma 2017a included children aged 24 to 36 months whereas Kandpal 2016 included children under 36 months. They also differed in their overall risk of bias, with Kandpal 2016 at high and Kusuma 2017a at unclear risk.

In addition to reporting the effects of CCTs on stunting, five cRCTs reported on mean HAZ (Evans 2014; Kandpal 2016; Kurdi 2019; Macours 2012; Maluccio 2005). A meta‐analysis of these studies indicated that CCTs improve mean HAZ (MD 0.09, 95% CI 0.04 to 0.15; 5619 children; I2 = 0%; Analysis 2.4). The follow‐up period ranged from nine months to three years. Three of the studies in this comparison were at high overall risk of bias (Evans 2014; Kandpal 2016; Kurdi 2019), and the others at low overall risk of bias. A sensitivity analysis of the studies at overall low risk of bias did not affect the results (Appendix 4) (Macours 2012; Maluccio 2005).

Prospective controlled studies

Three PCS reported on stunting (Lopez Arana 2016, Andersen 2015; Ferre 2014). Two reported unclear effects potentially favouring CCTs and one reported unclear effects potentially favouring the control (P = 0.047; Figure 6; Table 13). All studies were at high overall risk of bias.

Lopez Arana 2016 and Andersen 2015 reported unclear effects potentially favouring CCTs. Lopez Arana 2016 reported a reduction in stunting among 2874 children in the intervention group at four years but the CIs overlapped with the null effect (OR 0.92, 95% CI 0.82 to 1.05; P > 0.05). In Andersen 2015, there was a smaller proportion of stunted children in the intervention group, both among those receiving the intervention for less than two years (treatment effect: –7.98, 95% CI –22.3 to 6.34; 188 children; P = 0.27) as well as those receiving the intervention for longer than two years (treatment effect –18.3, 95% CI –38.3 to 1.59; 169 children; P = 0.07). Both of these studies were at high overall risk of bias.

Ferre 2014 reported unclear effects potentially favouring the control, with a higher proportion of stunted children in the CCT group by 3.4 pp at approximately one year (95% CI –6.4 to 13.2).

A meta‐analysis of two of these studies indicated an unclear effect potentially favouring CCTs (MD –5.63, 95% CI –26.59 to 15.34; 1749 children; I2 = 73%; Analysis 2.5) (Andersen 2015; Ferre 2014).

In addition to reporting the effects of CCTs on stunting, three PCS, all at high overall risk of bias, reported on mean HAZ (Andersen 2015; Leroy 2008 (PROGRESA); Lopez Arana 2016). The pooled analysis indicated that CCTs may or may not increase HAZ, as the effect could range from a small reduction to a significant increase in HAZ (MD 0.03, 95% CI –0.06 to 0.12; 3475 children, I2 = 0%; Analysis 2.6).

2.5.2. Wasting: weight‐for‐height z‐scores < –2SD (acute undernutrition)

Four cRCTs (Evans 2014; Kurdi 2019; Kusuma 2017a; Maluccio 2005) and three PCS (Ferre 2014; Leroy 2008 (PROGRESA); Lopez Arana 2016) reported measures related to wasting.

Cluster randomised controlled trials

Evidence from two cRCTs indicated that CCTs may make little or no difference to wasting (2 trials, 2116 children; low‐certainty evidence; summary of findings Table 2; Figure 6) (Maluccio 2005; Kusuma 2017a). A meta‐analysis showed an unclear effect favouring CCTs (MD –2.50, 95% CI –8.04 to 3.04; I2 = 70%; Analysis 2.7).

Two other trials reported on the effects of CCTs on mean WHZ (Evans 2014; Kurdi 2019). A meta‐analysis indicated an unclear effect potentially favouring CCTs (MD 0.17, 95% CI –0.11 to 0.44; 1111 children; I2 = 0%; Analysis 2.8).

Prospective controlled studies

Two PCS reported on the effects of CCTs on wasting (Lopez Arana 2016; Ferre 2014). One study reported clear effects favouring CCTs and one study unclear effects potentially favouring CCTs (P = 0.125) (Figure 6; Table 13). Data could not be pooled. Both studies were at high overall risk of bias.

Lopez Arana 2016 reported a clear effect favouring CCTs. In this study the odds of wasting were reduced by 75% among children in the CCT group at four years (OR 0.25, 95% CI 0.09 to 0.74; 2874 children).

Ferre 2014 reported an unclear effect potentially favouring CCTs. The proportion of wasted children was lower in the CCT group at 13 months, by 3.6 pp for those that were aged 22 to 46 months when enrolled (MD –0.04, 95% CI –0.11 to 0.04) and by 13 pp for those aged 10 to 22 months when enrolled (MD –0.13, 95% CI –0.26 to 0.01). However, effects ranged from potential benefit to potential harm.

In addition, one PCS, at high overall risk of bias, reported an unclear effect on WHZ potentially favouring CCTs (Leroy 2008 (PROGRESA)). The mean WHZ was higher by 0.085 SDs at two years (95% CI –0.11 to 0.28; 432 children; P = 0.2).

2.5.3 Underweight

2.5.3.1 Weight‐for‐age z‐scores < –2SD)

Three cRCTs reported on the effects of CCTs on the proportion of children who were underweight (Kandpal 2016; Kusuma 2017a; Maluccio 2005). One study reported a clear effect favouring CCTs, and two studies reported unclear effects potentially favouring CCTs (Table 12). A meta‐analysis of these studies indicated that CCTs can help reduce underweight (MD –4.87, 95% CI –8.65 to –1.09; 2506 children; I2 = 0%; Analysis 2.9). The study clearly favouring CCTs was at low overall risk of bias. Two of these studies also reported the effects on severe stunting, showing the CCTs may not make a difference to this outcome (MD –1.08, 95% CI –4.73 to 2.57; 1784 children; I2 = 0%; Analysis 2.10) (Kandpal 2016; Kusuma 2017a).

In addition, Evans 2014, Kandpal 2016, and Macours 2012 reported on the effects of CCTs on mean WAZ. The pooled analysis indicated that CCTs slightly increased WAZ by 0.04 SDs in a period ranging from nine months to three years after the intervention (MD 0.04, 95% CI –0.03 to 0.11; 3 trials, 3,548 children; I2 = 0%; Analysis 2.11). Two of these studies were at high overall risk of bias (Evans 2014; Kandpal 2016).

Prospective controlled studies

One PCS, at high overall risk of bias, reported an unclear effect favouring the control (Ferre 2014). The proportion of children who were underweight increased by 4.6 pp at 13 months among those in the CCT group compared to the control group, but the effects ranged from a decrease to an increase (MD 0.05, 95% CI –0.05 to 0.14; 1638 children) (Table 13).

2.5.3.2 Body mass index‐for‐age z‐score

Cluster randomised controlled trials

One cRCT at high overall risk of bias reported unclear effects on body mass index‐for‐age z‐score (BMIZ) potentially favouring the control at 1.5 years (MD –1.55, 95% CI –4.43 to 1.33; P > 0.1; 64 children aged 0 to 4 years; Table 12) (Evans 2014).

Prospective controlled studies

Two PCS, both at high overall risk of bias, reported on BMIZ, with effects ranging from unclear potentially favouring CCTs to unclear potentially favouring the control (P = 0.125) (Table 13) (Andersen 2015; Lopez Arana 2016). Studies could not be pooled due to high heterogeneity (I2 = 79%; Analysis 2.12). Both studies are at high overall risk of bias. In Lopez Arana 2016, some of the children also participated in a separate childcare supplementary nutrition programme, which could have influenced the effects of the cash transfer. This study included children aged from birth to 17 years, whereas Andersen 2015 included children aged six to 18 months only.

Lopez Arana 2016 reported unclear effects potentially favouring CCTs, with BMIZ increasing for children in the intervention group compared to those in the control group (MD 0.14, 95% CI 0.00 to 0.27; P < 0.05; Table 13).

Andersen 2015 reported unclear effects on BMIZ potentially favouring the control, both for those receiving the intervention for less than two years (MD –0.03, 95% CI –0.31 to 0.25, P = 0.84) and those receiving it for longer than two years (MD –0.36, 95% CI –0.79 to 0.06; P = 0.09) (Table 13).

2.6 Change in biochemical indicators

No included study addressing this comparison reported biochemical indicators.

2.7 Cognitive function and development

Two cRCTs (Baird 2013; Macours 2012) and one PCS (Andersen 2015) reported on cognitive function and development.

Cluster randomised controlled trials

Evidence from two cRCTs indicated that CCTs slightly improve cognitive function in children (2 RCTs, 5383 children; high‐certainty evidence; summary of findings Table 2; Figure 6) (Baird 2013; Macours 2012). Pooled effects indicated that CCTs slightly improve different measures of cognitive function compared to control (SMD 0.13, 95% CI 0.09 to 0.18; I2 = 0%; Analysis 2.13). The measure used in Macours 2012 was a combined measure averaging the effect across two language tests, short‐ and long‐term memory tests, and two behavioural tests. Baird 2013 used the Ravens Coloured Progressive Matrices test score, which is a measure of abstract reasons in children from the age of five years. For both of these, the higher the score, the more beneficial the effect.

Prospective controlled studies

Andersen 2015 reported small unclear effects potentially favouring the control on the TVIP score, which is the Spanish‐speaking version of the Peabody Picture Vocabulary Test (PPVT), a test of receptive vocabulary that can be applied to children 36 months and older. The TVIP score was reduced by 0.15 SDs in the CCT group compared to the control group (MD –0.15, 95% CI –0.37 to 0.07; 243 children; P = 0.17; Table 13).

2.8 Change in proportion of anxiety or depression

One cRCT, at low overall risk of bias, reported on psychological distress, with effects ranging from clear effects favouring CCTs at one year to unclear effects potentially favouring CCTs at two years (Table 12) (Baird 2013). Psychological distress was assessed with the General Health Questionnaire 12, a tool used widely in clinical settings, in which psychological distress is a binary measure of psychological distress, anxiety and depression; social dysfunction; and loss of confidence. The study authors reported that the proportion of school girls with psychological distress reduced in the intervention group both at one year (pp –0.06; 2089 girls; P < 0.05) and at two years (pp –0.04; 2089 girls; P > 0.1). However, the change was very small and unlikely to be meaningful. This study was at low overall risk of bias.

2.9 Morbidity

Four cRCTs report on various morbidity measures (Table 12) (Evans 2014; Gertler 2000 (PROGRESA); Kandpal 2016; Macours 2012).

2.9.1 Illness

Three cRCTs reported the effects of CCTs on illness. A meta‐analysis of three of these studies indicated that CCTs may not make a difference to the proportion of people reporting being ill or that reporting seeking care for illness in the past two to four weeks (MD –0.28, 95% CI –5.92 to 5.35; 38,587 participants; Analysis 2.14).

Macours 2012 reported a clear effect favouring CCTs on the number of days ill in bed, which was lower among children in the CCT group compared to the control by 0.357 SDs (MD –0.36, 95% CI –0.62 to –0.10; 3326 children; Table 12).

2.9.2 Anaemia

Gertler 2000 (PROGRESA) reported a clear effect favouring CCTs on anaemia. After 20 months of receiving the intervention, the odds of children being anaemic were 25.5% smaller in children receiving CCTs compared to those receiving no intervention (OR 0.75; 2010 children; P = 0.012). This study was at unclear overall risk of bias (Table 12).

2.10 Adverse outcomes (proportion of overweight/obesity)

A meta‐analysis of two PCS, both at high overall risk of bias, showed that CCTs make no difference to the proportion of overweight children aged under 18 years at two to four years of the intervention (OR 1.00, 95% CI 0.59 to 1.71; 3042 children; I2 = 60%; Analysis 2.15; Table 13) (Andersen 2015; Lopez Arana 2016).

Lopez Arana 2016 also reported on the effects of CCTs on obesity. This study reported an unclear effect favouring CCTs; the risk of obesity was reduced by 44% among children in the intervention group at four years (OR 0.56, 95% CI 0.20 to 1.53; 2874 children; P > 0.05). However, effects were uncertain as the CIs crossed the null effect.

Comparison 3: income‐generation interventions

Six cRCTs and 11 PCS assessed a variety of interventions aimed at generating income as a means to improve food security through increased economic access to food. Interventions included broad community development programmes that comprised training on livestock management, citizen empowerment, poverty alleviation delivered to women or women's self‐help groups (Darrouzet Nardi 2016; Doocy 2017; Osei 2017), training programmes to improve farming practices and sustainable agriculture (Doocy 2017; Kangmennaang 2017), and access to savings and investments and building capacity of local governance structures (Weinhardt 2017). Other interventions aimed to generate income through one‐off transfers of livestock with ongoing training (Asadullah 2015; Jodlowski 2016), and in some cases with additional intervention components such as health visits, access to community savings and technical advice (Asadullah 2015). Other studies focused on agriculture‐related interventions as a means to generate income: through an integrated agriculture and nutrition programme including training and input provision for crop farming or animal rearing (Marquis 2018; Olney 2016), development of a sustainable integrated agriculture‐aquaculture approaches (Murshed E Jahan 2011; Verbowski 2018), a sugarcane farmers scheme (Kennedy 1989), and implementation of a solar‐powered irrigation systems (Alaofe 2016; Alaofe 2019). Three PCS evaluated employment interventions, including public works programmes (Porter 2016; Beegle 2017), and part‐time employment for women (Katz 2001).

Four PCS assessed effects on food security outcomes (Asadullah 2015; Doocy 2017; Kangmennaang 2017; Weinhardt 2017). Three cRCTs (Beegle 2017; Darrouzet Nardi 2016; Olney 2016) and three PCS (Alaofe 2019; Doocy 2017; Jodlowski 2016) reported various measures of dietary diversity. Four cRCTs (Darrouzet Nardi 2016; Marquis 2018; Olney 2016; Verbowski 2018) and five PCS (Alaofe 2019; Doocy 2017; Katz 2001; Kennedy 1989; Weinhardt 2017) reported on various anthropometric measures. One cRCT (Verbowski 2018) and three PCS (Alaofe 2019; Asadullah 2015; Kennedy 1989) reported on morbidity outcomes.

Further details about the studies in this comparison are provided in Table 5. Results of individual trials included in this comparison are presented in Table 14 and PCS in Table 15. summary of findings Table 3 and the harvest plot in Figure 7 summarise the effects on key outcomes.

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Table 14. Income‐generation interventions – results of included trials

Study ID (risk of bias)

Study design (n)

Income‐generation interventions

No intervention

Effect measure (time point)

Effect direction

Meta‐analysis (yes/no)

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

3.3 Proportion of HHs who were food secure

3.3.1 Food security

3.3.1.1 Outcome measure: proportion experiencing food security (based on HFIAS)

Osei 2017 (?)

cRCT (2614 HHs)

79.7, 95% CI 77.2 to 82.0

53.6, 95% CI 51.0 to 56.1

87.4, 95% CI 85.3 to 89.3

78.3, 95% CI 76.0 to 80.4

— (2.5 years)

No n for individual groups to calculate MD.

3.3.1.2 Outcome measure: HH food security score (mean, SD)

Beegle 2017 (‐)

RCT (2193 HHs)

1083 HHs

–3.12 (1.29)

1110 HHs

MD –0.060, SE 0.080, 95% CI –0.2168 to 0.0968 (3/4 months)

3.3.1.3 Outcome measure: Resilience Index (mean, SD)

Beegle 2017 (‐)

RCT (2195 participants)

–9.32 (9.84)

MD –0.224, SE 0.630 (3/4 months)

3.3.1.4 Outcome measure: Principal Components Analysis index (mean, SD)

Beegle 2017 (‐)

RCT (2123 participants)

0.15 (2.08)

MD –0.029, SE 0.135 (3/4 months)

3.3.2 Dietary diversity

3.3.2.1 Outcome measure: odds of consuming an additional food group based on the DDS

Darrouzet Nardi 2016 (?)

(DDS 0–7)

cRCT (2584 children)

OR 1.524, 95% CI 1.45 to 4.38, P = 0.001 (2 years)

3.3.2.2 Outcome measure: HDDS (mean, SD)/Food Consumption Score

Olney 2016 (?)

(HDDS 0–11)

cRCT (1476 HHs)

5.6 (1.93)

5.6 (2.07)

880 HHs

5.8 (1.70)

5.2 (2.11)

596 HHs

MD 0.7, SE 0.44, 95% CI –0.1624 to 1.5624, P = 0.17 (2 years)

Yes. SMD.

Beegle 2017 (‐)

(FCS 0–126)

RCT (2201 HHs)

1191 HHs

38.82 (16.01)

1110 HHs

MD –0.708, SE 1.072, 95% CI –2.80912 to 1.39312 (3/4 months)

3.3.2.3 Outcome measure: MDD (n, %)

Marquis 2018 (+)

cRCT (428 children)

30.9

80.2

247

33.8

69.5

181

OR 1.65, SE 0.41, 95% CI 0.8464 to 2.4536, P < 0.05 (12 months)

Yes. Olney groups combined.

Darrouzet Nardi 2016 (?)

cRCT (2604 children)

OR 1.146, 95% CI 1.02 to 1.29, P = 0.021 (2 years)

Olney 2016 (?)

cRCT (758 children)

OWL: 7 (3.0)

HC: 4 (1.7)

OWL: 35 (15.0)

HC: 43 (18.2)

OWL: 220

HC: 231

8 (2.6)

20 (6.3)

307

OWL villages vs control: pp 8.3, P = 0.17

HC villages vs control: pp 12.6, P = 0.08

(2 years)

Combined effect: MD pp 10.08, 95% CI 1.02 to 19.14

Secondary outcomes

3.5Change in anthropometric indicators

3.5.1 Stunting

3.5.1.1 Outcome measure: Height‐for‐Age z‐score (HAZ) (mean, SD or SE)

Marquis 2018 (+)

cRCT (428 children)

–0.88 (1.27)

247

–0.78 (1.30)

181

MD 0.22, SE 0.06, P < 0.01, 95% CI 0.10 to 0.34 (12 months)

No. Effect sizes calculated for Darrouzet (2 years) and Osei from group estimates.

Darrouzet Nardi 2016 (?)

cRCT (303 children)

–1.47 (0.07)

–1.38 (0.06)

–1.48 (0.06)

–1.41 (0.06)

MD 0.109, 95% CI 0.000 to 0.218, P = 0.048 (12 months)

609 children

–1.47 (0.07)

–1.30 (0.06)

305

–1.48 (0.06)

–1.33 (0.06)

304

MD 0.03, SE 0.0049, 95% CI 0.020 to 0.040 (2 years)

Osei 2017 (?)

cRCT (2569 children)

–2.23 (0.03)

–2.1 (0.03)

1299

–2.4 (0.04)

–2.32 (0.03)

1297

MD 0.22, SE 0.0012, 95% CI 0.218 to 0.222 (2.5 years)

3.5.1.2 Outcome measure: proportion stunted (HAZ < –2SD) (CI)

Osei 2017 (?)

cRCT (2569 children)

57.7

55.1

1299

65.8

63.5

1297

OR 0.94, 95% CI 0.74 to 1.19 (2.5 years)

Yes. Verbowski groups combined

Verbowski 2018 and aquaculture (?)

cRCT (597 children)

27.9

29.9

299

29.3

32.0

298

MD pp –0.62, P = 0.927 (1.8 years)

MD pp 2.2, 95% CI –5.64 to 10.05

Verbowski 2018 (?)

cRCT (598 children)

22.7

28.9

300

29.3

32.0

298

MD pp 3.73, P = 0.453 (1.8 years)

3.5.2Wasting

3.5.2.1 Outcome measure: WHZ (mean, SD or SE)

Marquis 2018 (+)

cRCT (428 children)

–0.37 (1.08)

247

–0.31 (1.24)

181

MD 0.07, SE 0.08, 95% CI –0.087 to 0.227, P > 0.10 (12 months)

No. Effect for Osei calculated from group estimates.

Osei 2017 (?)

cRCT (2603 children)

–0.91 (0.03)

–0.85 (0.03)

1300

–0.93 (0.03)

–0.71 (0.03)

1303

MD –0.14, SE 0.0012, 95% CI –0.142 to –0.138 (2.5 years)

3.5.2.2 Outcome measure: proportion wasted (WHZ < –2SD)

Osei 2017 (?)

cRCT (2603 children)

10.6

10.5

1300

10.1

9.7

1303

OR 1.03, 95% CI 0.70 to 1.52 (2.5 years)

Yes. Verbowski groups combined.

Verbowski 2018 and aquaculture (?)

cRCT (597 children)

6.7

10.2

299

8.3

8.9

298

MD pp 2.75, P = 0.424 (22 months)

MD pp 3.19, 95% CI –1.95 to 8.33

Verbowski 2018 (?)

cRCT (598 children)

8.4

13.0

300

8.3

8.9

298

MD pp 3.80, P = 0.348 (22 months)

3.5.3 Underweight

3.5.3.1 Outcome measure: Weight‐for‐age z‐score (WAZ) (mean, SD or SE)

Marquis 2018 (+)

cRCT (428 children)

–0.78 (1.12)

247

–0.68 (1.27)

181

MD 0.15, SE 0.07, P < 0.05 (12 months)

Yes. Effect estimates calculated using group estimates.

Darrouzet Nardi 2016 (?)

cRCT (634 children)

–2.04 (0.07)

–1.97 (0.06)

301

–1.94 (0.06)

–1.89 (0.06)

333

NR (1 year)

–2.04 (0.07)

–1.97 (0.06)

–1.94 (0.06)

–2.07 (0.06)

MD 0.10, 95% CI 0.09 to 0.11 (2 years)

Osei 2017 (?)

cRCT (2613 children)

–1.87 (0.03)

–1.77 (0.03)

1306

–1.97 (0.03)

–1.77 (0.03)

1307

MD 0.00, 95% CI –0.00 to 0.00 (2.5 years)

3.5.3.2 Outcome measure: percentage underweight (WAZ < 80% standard/ < –2SD) (includes severe underweight)

Osei 2017 (?)

cRCT (2613 children)

43.4

41.0

1306

48.0

40.6

1307

OR 1.15, 95% CI 0.91 to 1.46 (2.5 years)

Yes. Verbowski groups combined.

Verbowski 2018 and aquaculture (?)

cRCT (597 children)

23.5

32.0

299

23.0

28.8

298

MD pp 2.75, P = 0.670 (22 months)

MD pp –1.16, 95% CI –9.02 to 6.70

Verbowski 2018 (?)

cRCT (598 children)

26.1

28.8

300

23.0

28.8

298

MD pp –3.63, P = 0.479 (22 months)

3.5.3.3 Outcome measure: BMI (kg/m2) (mean, SD or SE)

Olney 2016 (?)

cRCT (1297 women)

20.2 (2.22)

20.7 (2.34)

787

20.6 (2.27)

21.1 (2.70)

510

MD 0.2, 95% CI –0.192 to 0.592, SE 0.20, P = 0.26 (2 years)

Yes. Effect estimate for Osei calculated from group estimates.

Osei 2017 (?)

cRCT (2614 mothers)

19.6 (0.07)

19.8 (0.05)

1182

20.1 (0.06)

19.9 (0.05)

1303

MD –0.10, 95% CI –0.10 to –0.10 (2.5 years)

3.5.3.4 Proportion of women who were underweight (BMI < 18.5 kg/m2)

Olney 2016 (?)

cRCT (1297 women)

23

15

787

15

16

510

pp –8.7, P = 0.01 (2 years)

No. Verbowski groups combined.

Osei 2017 (?)

cRCT (2614 mothers)

28.2

28.6

1182

17.5

19.9 (0.05)

1303

OR 0.61, 95% CI 0.46 to 0.82 (2.5 years)

Verbowski 2018 and aquaculture (?)

cRCT (541 women)

14.2

9.0

270

16.6

9.4

271

MD pp 1.19, P = 0.920 (22 months)

MD 3.88, 95% CI –4.36 to 12.12

Verbowski 2018 (?)

cRCT (541 women)

13.4

13.5

270

16.6

9.4

271

MD pp 4.27, P = 0.347 (22 months)

3.6 Change in biochemical indicators

3.6.1 Mean haemoglobin concentration (children) (mean, SE)

Osei 2017 (?)

cRCT (2614 children)

115.3 (0.1)

114.3 (0.1)

1307

113.6 (0.1)

110.8 (0.1)

1307

MD 3.5, SE 0.0039, 95% CI 3.492 to 3.507 (2.5 years)

Yes. Verbowski groups combined.

Verbowski 2018 and aquaculture (?)

cRCT (597 children)

104.5 (13.7)

108.4 (13.1)

298

105.7 (13.6)

107.1 (12.9)

299

MD 2.54, SE 1.43, P = 0.076 (22 months)

MD 2.48, 95% CI 0.51 to 4.46

Verbowski 2018 (?)

cRCT (597 children)

104.1 (13.8)

108.0 (12.3)

298

105.7 (13.6)

107.1 (12.9)

299

MD 2.43, SE 1.42, P = 0.088 (22 months)

3.6.2 Mean haemoglobin concentration (women) (mean, SD or SE)

Osei 2017 (?)

cRCT (2614 mothers)

129.3 (0.1)

126.5 (0.1)

1307

129.6 (0.1)

121.9 (0.1)

1307

MD 4.6, SE 0.0039, 95% CI 4.592 to 4.608 (2.5 years)

Yes. Verbowski groups combined.

Verbowski 2018 and aquaculture (?)

cRCT (541 women)

122.4 (12.1)

122.9 (12.9)

270

121.5 (12.5)

121.1 (12.1)

271

MD 0.49, SE 1.33, P = 0.714 (22 months)

MD –0.07, 95% CI –1.92 to 1.78

Verbowski 2018 (?)

cRCT (541 women)

121.7 (13.7)

121.0 (11.9)

270

121.5 (12.5)

121.1 (12.1)

271

MD –0.63, SE 1.34, P = 0.637 (22 months)

3.9 Morbidity

3.9.1 Prevalence of anaemia (children)

Osei 2017 (?)

cRCT (2614 children)

28.2

30.8

1307

31.6

42.5

1307

OR 0.76, 95% CI 0.59 to 0.98 (2.5 years)

Yes. Verbowski groups combined.

Verbowski 2018 and aquaculture (?)

cRCT (597 children)

63.1

54.3

298

59.2

59.5

299

MD pp –9.74, P = 0.119 (22 months)

MD pp –11.90, 95% CI –20.47 to –3.33

Verbowski 2018 (?)

cRCT (597 children)

65.4

52.6

298

59.2

59.5

299

MD pp –14.0, P = 0.023 (22 months)

3.9.2 Prevalence of anaemia (women)

Osei 2017 (?)

cRCT (2614 mothers)

19.6

24.6

1307

21.1

35.8

1307

OR 0.62, 95% CI 0.48 to 0.82 (2.5 years)

Yes. Verbowski groups combined.

Verbowski 2018 and aquaculture (?)

cRCT (541 women)

38.9

35.8

270

40.4

38.7

271

MD pp –1.10, P = 0.865 (22 months)

MD pp 1.34, 95% CI –7.94 to 10.61

Verbowski 2018 (?)

cRCT (541 women)

41.9

43.5

270

40.4

38.7

271

MD pp 4.14, P = 0.551 (22 months)

aEach triangle represents one study.

(+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias. = Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0.

BMI: body mass index; cRCT: cluster randomised controlled trial; CI: confidence interval; DDS: Dietary Diversity Score; EHFP: enhanced homestead food production; FCS: Food Consumption Score; HAZ: height‐for‐age z‐score; HC: health committee; HDDS: Household Dietary Diversity Score; HFIAS: Household Food Insecurity Score; HH: household; MD: mean difference; MDD: Minimum Dietary Diversity; n: number; NR: not reported; OR: odds ratio; OWL: older women leaders; pp: percentage point; SD: standard deviation; SE: standard error; SMD: standardised mean difference; WAZ: weight‐for‐age z‐score; WHZ: weight‐for‐height z‐score.

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Table 15. Income‐generation interventions – results of included prospective controlled studies

Study ID (risk of bias)

Study design (n)

Income‐generation interventions

No intervention

Effect measure (time point)

Effect of combined groups/calculated effect

Effect directiona

Meta‐analysis (yes/no)

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

3.2: Proportion of HH expenditure on food

3.2.1 Outcome measure: proportion of HH expenditure on food

Kennedy 1989 (?)

Prospective controlled study (378 HHs)

(2 years)

N/A

Alaofe 2016 (?)

Prospective controlled study (56 HHs)

(1 year)

3.3: Proportion of HHs who were food secure

3.3.1 Food security

3.3.1.1 Outcome measure: proportion experiencing food security (0 months with insufficient food in past 12 months)/ Doocy: based on HFIAS

Weinhardt 2017 (?)

Prospective controlled study (827 participants)

165/564 (29.3%)

309/564 (54.8%)

564

71/262 (27.1%)

117/263 (44.5%)

263

OR 1.36, 95% CI 0.93 to 1.97, P = 0.108 (1.5 years)

N/A no effect measure for Doocy

165/564 (29.3%)

36 months: 308/531 (58.0%)

71/262 (27.1%)

129/245 (52.7%)

OR 1.12, 95% CI 0.75 to 1.67, P = 0.585 (3 years)

Doocy 2017 – FFS (‐)

Prospective controlled study (571 HHs)

1.90%

27.80%

317 HHs

0.40%

14.60%

254 HHs

— (3.5 years)

Doocy 2017– WEG (‐)

Prospective controlled study (548 HHs)

0,3%

29.9%

0.4%

14.6%

— (3.5 years)

3.3.1.2 Proportion experiencing food deficit always

Asadullah 2015 (‐)

Prospective controlled study (4038 HHs)

60.1

15.3

2098

41.91

28.87

1940

pp –28.85, P < 0.01 (3 years)

60.1

21.02

41.91

28.45

pp –17.15, P < 0.01 (6 years)

60.1

42.9

41.91

44.38

pp –13.91, P < 0.01 (9 years)

3.3.1.3 Outcome measure: HFIAS (mean, SD or SE)

Doocy 2017 – FFS (‐)

Prospective controlled study (571 HHs)

14.4 (4.6)

5.7 (5.1)

317

14.8 (5.3)

10.1 (6.1)

254

MD –4.6, 95% CI –5.0 to –4.2, P < 0.001 (3.5 years)

MD –4.23, 95% CI –4.96 to –3.49

No

Doocy 2017 – WEG (‐)

Prospective controlled study (548 HHs)

15.3 (5.3)

6.3 (5.5)

294

14.8 (5.3)

10.1 (6.1)

254

MD –3.85, 95% CI –4.26 to –3.43, P < 0.01 (3.5 years)

Kangmennaang 2017 (‐)

Prospective controlled study (1000 HHs)

1.255 (0.029)

1.173 (0.033)

571

1.136 (0.044)

1.359 (0.071)

429

MD –0.304, SE 0.095, P < 0.01 (about 2 years)

3.3.1.4 Outcome measure: proportion of HHs improving a HFIAS category (95% CI)

Doocy 2017 – FFS (‐)

Prospective controlled study (571 HHs)

55.3 (48.8 to 61.9)

317

32.4 (24.6 to 40.3)

254

MD 22.9, 95% CI 12.7 to 33.1, P < 0.001 (3.5 years)

MD pp 24.21, 95% CI 16.67 to 31.76

N/A

Doocy 2017 – WEG (‐)

Prospective controlled study (548 HHs)

59.5

294

31.5

254

MD 25.8, 95% CI 14.6 to 36.9, P < 0.001 (3.5 years)

3.3.2 Dietary diversity

3.3.2.1 Outcome measure: probability weighted DDS (mean, SD)

Jodlowski 2016 (+)

Prospective controlled study (283 HHs)

105 HHs

178 HHs

MD –0.123, 95% CI –0.43 to 0.18, P > 0.1 (18 months)

3.3.2.2 Outcome measure: HDDS (mean, SD)

Jodlowski 2016 (+)

Prospective controlled study (283 HHs)

5.86 (1.848)

105 HHs

5.747 (1.774)

178 HHs

MD 0.267, 95% CI –0.13 to 0.66, P > 0.1 (18 months)

Yes. (Doocy groups combined)

Alaofe 2019b (?)

Prospective controlled study (423 HHs)

6.07 (1.26)

6.50 (1.23)

282

6.05 (1.26)

6.24 (1.24)

214

MD 0.94, SE 0.24, 95% CI 0.4696 to 1.4104, P < 0.01 (1 year)

Doocy 2017 – FFS (‐)

Prospective controlled study (571 HHs)

3.4 (1.4)

3.4 (1.5)

317

3.4 (1.5)

4.8 (2.1)

254

MD 0.9, 95% CI 0.5 to 1.3, P < 0.001 (3.5 year)

MD 0.80, 95% CI 0.51 to 1.09

Doocy 2017 – WEG (‐)

Prospective controlled study (548 HHs)

3.4 (1.7)

5.5 (2.2)

294

3.4 (1.5)

4.8 (2.1)

254

MD 0.69, 95% CI 0.27 to 1.10, P = 0.001 (3.5 year)

3.3.2.3 Outcome measure: Women's Household Dietary Diversity Score (WDDS‐10) (mean, SD)

Alaofe 2019b (?)

Prospective controlled study (430 women)

4.58 (1.04)

4.91 (0.97)

286

4.83 (0.97)

4.01 (1.12)

220

MD 0.83, SE 0.19, P < 0.01, 95% CI 0.46 to 1.20 (1 year)

3.3.2.4 Outcome measure: proportion achieving target dietary diversity at endline according to HDDS

Doocy 2017 – FFS (‐)

Prospective controlled study (571 HHs)

21.3

69.7

317

18.1

67.6

254

MD 21.7, 95% CI 12.3 to 31.1, P < 0.001 (3.5 year)

MD 17.03, 95% CI 7.81 to 26.24

N/A

Doocy 2017 – WEG (‐)

Prospective controlled study (548 HHs)

18.7

62.2

294

18.1

67.6

254

MD 12.3, 95% CI 2.8 to 21.8, P = 0.011 (3.5 years)

3.4 Change in adequacy of dietary intake

3.4.1 Outcome measure: percentage of calorie‐deficient HHs (< 80% of caloric requirement/adult equivalent)

Kennedy 1989 (?)

Prospective controlled study (374 HHs)

30.7

28.1

30

28.7

(2 years)

3.4.2 Outcome measure: percentage of preschool‐aged children meeting caloric requirements

Kennedy 1989 (?)

Prospective controlled study (1297 children)

69

66

58

62

(2 years)

Secondary outcomes

3.5Change in anthropometric indicators

3.5.1 Stunting

3.5.1.1 Outcome measure: HAZ (mean, SD or SE)

Kennedy 1989 (?)

Prospective controlled study (746 children)

–1.34

–1.67

–1.50

–1.76

NR

(2 years)

3.5.1.2 Outcome measure: proportion stunted (HAZ <2SD) (CI)

Kennedy 1989 (?)

Prospective controlled study (222 children)

25.3

94

25.7

128

NR (2 years)

N/A

Doocy 2017 – FFS (‐)

Prospective controlled study (471 children)

60.2 (50.8 to 69.6)

265

58.8 (50.1 to 67.5)

206

(adjusted) MD 1.4, 95% CI –10.7 to 13.6, P = 0.81 (3.5 year)

3.5.2:Wasting

3.5.2.1 Outcome measure: WHZ (mean, SD or SE)

Kennedy 1989 (?)

Prospective controlled study (651 children)

–0.22

–0.15

–0.31

–0.04

NR (2 years)

3.5.2.2 Outcome measure: proportion wasted (WHZ < –2SD)

Kennedy 1989 (?)

Prospective controlled study (118 children)

13.0

48

14.1

70

NR (2 years)

3.5.3 Underweight

3.5.3.1 Outcome measure: WAZ (mean, SD or SE)

Kennedy 1989 (?)

Prospective controlled study (198 children)

–1.03

–1.14

–1.17

–1.10

NR (2 years)

3.5.3.2 Outcome measure: percentage underweight (WAZ < 80% standard/ < –2SD) (includes severe underweight)

Kennedy 1989 (?)

Prospective controlled study (198 children)

19.7

74

24.1

124

NR (2 years)

No. Subset. Except Kennedy – effect could not be calculated.

Weinhardt 2017 (?)

Prospective controlled study (509 children)

14.8%

16.8%

322

22.5%

19.8%

187

OR 1.52, 95% CI 0.80 to 2.90, P = 0.205 (1.5 years)

Prospective controlled study (538 children)

14.8%

18.6%

344

22.5%

24.2%

194

OR 1.27, 95% CI 0.54 to 3.01, P = 0.585 (3 years)

Doocy 2017 – FFS (‐)

Prospective controlled study (471 children)

22.3 (14.8 to 29.8)

265

29.8 (22.0 to 37.7)

206

(adjusted)

MD –7.6, CI –17.7 to 2.5, P = 0.13 (3.5 year)

3.5.3.3 Outcome measure: BMI (kg/m2) (mean, SD or SE)

Kennedy 1989 (?)

Prospective controlled study (753 women)

22.3

22.2

NR (2 years)

No. No effect estimate for Kennedy and variance estimate cannot be calculated for Asadullah (missing group sizes)

Alaofe 2019b (?)

Prospective controlled study (359 women)

23.01 (2.94)

22.95 (3.73)

256

22.03 (3.14)

21.69 (3.24)

167

MD 0.43, SE 0.24, 95% CI –0.0504 to 0.8904, P < 0.1 (1 year)

Asadullah 2015 (‐)

Prospective controlled study (3547 women)

19.0

18.95

19.17

18.98

MD 0.14, P = 0.29

3.5.3.4 Proportion of women who were underweight (BMI < 18.5 kg/m2)

Alaofe 2019b (?)

Prospective controlled study (359 women)

4.88

3.10

256

6.57

14.08

167

MD –0.22, SE 0.27, 95% CI –0.749 to 0.309, P > 0.1 (1 year)

3.5.3.5 Outcome measure: mid‐upper arm circumference (mean, SD)

Katz 2001 (‐)

Prospective controlled study (718 women)

22.8 (2.0)

335

23.0 (2.2)

383

MD in intervention group –0.20 cm

MD in control group –0.25 cm,

P = 0.67 (2 years)

3.6 Change in biochemical indicators

3.6.1 Proportion with iron deficiency

Alaofe 2019b (?)

Prospective controlled study (68 women)

15.3%

13.5%

17.9%

12.8%

DID –0.11, SE 0.83, 95% CI –0.94 to 0.72, P > 0.05 (1 year)

3.6.2 Proportion with vitamin A deficiency

Alaofe 2019b (?)

Prospective controlled study (60 women)

14.3%

5.8%

20.2%

10.8%

DID 0.54, SE 0.95, 95% CI –0.41 to 1.49, P > 0.05 (1 year)

3.9 Morbidity

3.9.1 Outcome measure: proportion seriously ill in past year

Asadullah 2015 (‐)

Prospective controlled study (4038 HHs)

23.38%

15.89

24.24

17.17

pp –1.72, P > 0.1 (3 years)

23.38

12.93

24.24

12.53

pp –0.78, P > 0.1 (6 years)

23.38

22.16

24.24

22.37

pp –0.70, P > 0.1 (9 years)

3.9.2 Outcome measure: % time ill

Kennedy 1989 (?)

Prospective controlled study (1055 children)

29.8

31.2

NR (2 years)

Prospective controlled study (420 women)

23.8

24.3

NR (2 years)

3.9.3 Outcome measure: % time ill with diarrhoea

Kennedy 1989 (?)

Prospective controlled study (1055 children)

4.6

4.0

NR (2 years)

3.9.4 Prevalence of anaemia (women)

Alaofe 2019b (?)

Prospective controlled study (126 women)

49.3%

36.9%

49%

53.2%

MD –1.25, SE 0.58, 95% CI –1.83 to –0.67, P < 0.05 (1 year)

3.9.5 Prevalence of iron‐deficiency anaemia (women)

Alaofe 2019b (?)

Prospective controlled study (564 women)

6.6%

4.2%

13.8%

8.4%

MD –0.99, SE 1.40, 95% CI –2.39 to 0.41, P > 0.05 (1 year)

aEach triangle represents one study.
bThis study also has a component comparing the intervention plus a working group versus a comparison group with a working group. Results are not presented here.

(+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias; = Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0.

CI: confidence interval; DDS: Dietary Diversity Score; DID: difference in differences; FFS: Farmer Field School; HAZ: height‐for‐age z‐score; HDDS: Household Dietary Diversity Score; HFIAS: Household Food Insecurity Scale; HH: household; MD: mean difference; N/A: not applicable/available; NR: not reported; OR: odds ratio; PCS: prospective controlled study; SD: standard deviation; SE: standard error; WAZ: weight‐for‐age z‐score; WEG: Women Empowerment Group; WHZ: weight‐for‐height z‐score.


Harvest plot: income‐generation interventions.

Harvest plot: income‐generation interventions.

Primary outcomes
3.1 Change in the prevalence of undernourishment

None of the included studies measured prevalence of undernourishment.

3.2 Proportion of household expenditure on food

We found no evidence about the effect of income‐generation interventions on the proportion of household expenditure on food. Although two PCS mentioned this outcome in their manuscript, they did not report relevant numerical data or indicate clearly the direction of the effect (Alaofe 2016; Kennedy 1989). Four other PCS reported total expenditure on food, not in relation to income or total expenditure and these results are thus not reported here (Asadullah 2015; Jodlowski 2016; Katz 2001; Murshed E Jahan 2011).

3.3 Proportion of households who were food secure

Two cRCTs reported the effects on food security of interventions where households received training for activities such as livestock management, aquaculture interventions and community development through women's self‐help groups (Darrouzet Nardi 2016; Osei 2017), whereas two cRCTs assessed the effects of an integrated agriculture and nutrition programme (Olney 2016; Marquis 2018). In one cRCT, households participated in a public works programme in Malawi (Beegle 2017). Six PCS assessing different interventions reported different food security or dietary diversity measures (Alaofe 2019; Asadullah 2015; Doocy 2017; Jodlowski 2016; Kangmennaang 2017; Weinhardt 2017).

3.3.1 Food security

Cluster randomised controlled trials

Evidence from one cRCT suggested that income‐generation interventions may result in little to no difference in food security of households who receive these interventions, compared to households who do not (MD –0.06, 95% CI –0.22 to 0.1; 2193 households; low‐certainty evidence; summary of findings Table 3). A cRCT from Malawi showed an unclear effect potentially favouring the control on household food security scores, after three to four months (Figure 7) (Beegle 2017). One RCT did not report an overall effect estimate for the proportion of households experiencing food security (based on HFIAS) after 2.5 years, following the implementation of an agricultural and nutrition training programme in women from rural villages In Nepal (Osei 2017).

Prospective controlled studies

Three PCS reported clear effects favouring the intervention on measures of food security and one PCS reported unclear effects favouring the intervention (P = 0.01) (Figure 7).

Asadullah 2015, Doocy 2017, and Kangmennaang 2017 reported a clear effect favouring the intervention. In Asadullah 2015, among participants receiving a multicomponent intervention including training and transfer of productive assets for an income‐generation enterprise, the proportion of households that reported always experiencing a food deficit decreased compared to the control group over a period of nine years (at 3 years: pp –28.85; P < 0.01; at 6 years: pp –17.15; P < 0.0; at 9 years: pp –13.91; P < 0.01; all 4038 households). In Doocy 2017, the combined effect of both groups of the interventions (where farmer field schools and women empowerment groups in farming villages) showed a clear effect favouring the intervention, with a decrease in mean HFIAS compared to control villages not receiving any intervention (MD –4.23, 95% CI –4.96 to –3.49; 1119 households). In Kangmennaang 2017, the implementation of a training and development programme for farmers resulted in a clear effect favouring the intervention, with a decrease in mean HFIAS in intervention households compared to control households (MD –0.30; 1000 households; P < 0.01) (Table 15). These studies could not be pooled due to high heterogeneity (I2 = 99%; Analysis 3.1). Both studies are at high overall risk of bias. The interventions differed. Doocy 2017 assessed a programme including women empowerment groups, where weekly meetings and training were provided regularly as well as start‐up materials, and farmer field schools, where farmers received semi‐monthly training in farming practices as well as business and administration. Kangmennaang 2017 assessed an intervention in which farmers experimented with agroecological innovations and which also included sharing knowledge and training on various aspects including leadership.

Weinhardt 2017 reported an unclear effect potentially favouring the intervention. This study assessed a multilevel health and development intervention including training on farming practices, and access to VSLs groups. The odds of being food secure (i.e. household that had zero months where there was insufficient food to meet their needs in the previous 12 months) increased by 36% at 1.5 years (OR 1.36, 95% CI 0.93 to 1.97; 827 participants; P = 0.108) and 12% at three years (OR 1.12, 95% CI 0.75 to 1.67; 827 participants; P = 0.585).

3.3.2 Dietary diversity

Four cRCTs (Beegle 2017; Darrouzet Nardi 2016; Marquis 2018; Olney 2016) and three PCS (Alaofe 2019; Doocy 2017; Jodlowski 2016) reported on seven different measures of dietary diversity. Definitions and explanations of dietary diversity measures reported here are provided in Table 6.

Cluster randomised controlled trials

Evidence from four RCTs suggests that income‐generation interventions may improve dietary diversity in children and may result in little or no difference to household dietary diversity (4 cRCTs, 3677 households and 3790 children; low‐certainty evidence; summary of findings Table 3). Two cRCTs reported a clear effect favouring income‐generation interventions (Darrouzet Nardi 2016; Marquis 2018), one reported an unclear effect favouring the intervention (Olney 2016), and one reported an unclear effect favouring the control group (Beegle 2017) (P = 0.047; Figure 7).

A meta‐analysis of two of these trials, assessing a public works programme in Malawi and an integrated agriculture and nutrition programme including provision of inputs and training, showed that income‐generation interventions make no difference to the HDDSs at three months to two years (SMD 0.02, 95% CI –0.09 to 0.13; 3677 households; I2 = 63%; Analysis 3.2) (Beegle 2017; Olney 2016). Beegle 2017 was at high and Olney 2016 at unclear overall risk of bias. Beegle 2017 measured dietary diversity using the FCS (scale: 0 to 126) and Olney 2016 used the HDDS (scale: 0 to 11); for both, the higher the score, the higher the food diversity. Another meta‐analysis of three of these cRCTs, assessing the implementation of women's groups, agricultural and nutritional training, and community development, showed that children in intervention households were 1.28 times more likely to achieve MDD, compared to children from control households, one to two years after the implementation of the interventions (OR 1.28, 95% CI 1.11 to 1.47; 3790 children; Analysis 3.3) (Darrouzet Nardi 2016; Marquis 2018; Olney 2016). Marquis 2018 was at low overall risk of bias and Darrouzet Nardi 2016 and Olney 2016 were at unclear overall risk of bias.

Prospective controlled studies

Two PCS reported a clear effect favouring income‐generation interventions and one study reported an unclear effect potentially favouring income‐generation interventions on dietary diversity (Figure 7). A meta‐analysis of these studies indicated that income‐generation interventions increase the HDDS (MD 0.67, 95% CI 0.29 to 1.05; 1571 households; I2 = 67%; Analysis 3.4) (Alaofe 2019; Doocy 2017; Jodlowski 2016). Doocy 2017 assessed farmer field schools or women's empowerment groups at 3.5 years, Alaofe 2019 assessed the installation of a low‐pressure drip irrigation system, combined with a solar‐powered water pump in each intervention village, and Jodlowski 2016 assessed a livestock transfer with training support. All studies were at high overall risk of bias.

Secondary outcomes
3.4 Change in adequacy of dietary intake
Prospective controlled studies

Although one PCS reported two measures of dietary intake adequacy among participants of an intervention where smallholder sugarcane growers were enrolled in a scheme to provide sugarcane to a new factory, the study authors did not report any effect measures (Kennedy 1989).

3.5 Change in anthropometric indicators

Four cRCTs (Darrouzet Nardi 2016; Olney 2016; Osei 2017; Verbowski 2018) and six PCS (Alaofe 2019; Asadullah 2015; Doocy 2017; Katz 2001; Kennedy 1989; Weinhardt 2017) reported nine different anthropometric measures in children and women.

3.5.1 Stunting: height‐for‐age z‐scores < –2SD (chronic undernutrition)

Four cRCTs (Darrouzet Nardi 2016; Osei 2017; Marquis 2018; Verbowski 2018) and two PCS (Doocy 2017; Kennedy 1989) reported on stunting.

Cluster randomised controlled trials

Evidence from two trials indicated that income‐generation interventions probably make little or no difference to wasting (2 trials, 3500 children; moderate‐certainty evidence; summary of findings Table 3) (Osei 2017; Verbowski 2018). A meta‐analysis of these two studies showed no difference to stunting (OR 1.00, 95% CI 0.84 to 1.19; I2 = 0%; Analysis 3.5).

In addition to reporting the proportion of children who are stunted, Marquis 2018, Darrouzet Nardi 2016, and Osei 2017 reported on the effect of income‐generation interventions on mean HAZ. Data from these studies could not be pooled due to high heterogeneity (I2 = 100%; Analysis 3.6). Marquis 2018 was at low overall risk of bias whereas Darrouzet Nardi 2016 and Osei 2017 were at unclear risk. All three studies assessed some form of training on agricultural practices or livestock management but, in all but one (Darrouzet Nardi 2016), nutrition and health education sessions were also provided, with which the most beneficial effects were observed. If this study was removed from the meta‐analysis, heterogeneity reduced to 0%. All three studies reported a clear effect favouring income‐generation interventions at 1 to 2.5 years of follow‐up. In Marquis 2018, the mean HAZ increased by 0.22 SD with the intervention at 12 months (95% CI 0.10 to 0.34; 428 children); the study assessed an integrated package of agricultural inputs and training as well as education in nutrition, health care and child stimulation for participants. In Darrouzet Nardi 2016, it increased by 0.03 SD in the intervention group, which included training for poverty alleviation, citizen empowerment, community development and optimisation of livestock management as means to generate income (95% CI 0.02 to 0.04; 609 children). However, the effect was unclear potentially favouring the intervention at one year. In Osei 2017, which assessed an enhanced homestead food production (EHFP) programme encompassing training in improved gardening and poultry‐rearing practices, among others, it increased by 0.22 SD at 2.5 years (95% CI 0.22 to 0.22; 2569 children) (Table 14).

Prospective controlled studies

Two PCS reported on stunting (Doocy 2017; Kennedy 1989). One study, assessing an income‐generation intervention with women's groups and farmer field schools, reported an unclear effect potentially favouring the control, with an increase in the proportion of stunted children at 3.5 years (MD 1.4, 95% CI –10.7 to 13.6; P = 0.81, 471 children) (Table 15) (Doocy 2017). The other study did report any effect measures (Kennedy 1989).

3.5.2 Wasting: weight‐for‐height z‐scores < –2SD (acute undernutrition)

Three cRCTs (Marquis 2018; Osei 2017; Verbowski 2018) and one PCS (Kennedy 1989) reported on wasting.

Cluster randomised controlled trials

Evidence indicated that income‐generation interventions probably make little or no difference to wasting (2 cRCTs, 3500 children; moderate‐certainty evidence; summary of findings Table 3). A meta‐analysis of these two cRCTs showed an unclear effect potentially favouring the control, with an increased risk of wasting in children in the intervention group at two years (OR 1.13 95% CI 0.92 to 1.40; I2 = 0%; Analysis 3.7) (Osei 2017; Verbowski 2018).

In addition, Marquis 2018 and Osei 2017 reported on the effects of income‐generation interventions on the mean WHZ. Data could not be pooled due to high heterogeneity (I2 = 85%; Analysis 3.8). Marquis 2018 reported a clear effect favouring the control (MD 0.07, 95% CI –0.087 to 0.227; 429 children), and Osei 2017 reported an unclear effect favouring the income‐generation intervention (MD –0.14, 95% CI –0.142 to –0.138; 2603 children) (Table 14).

3.5.3 Underweight

3.5.3.1 Weight‐for‐age z‐scores < –2SD

Three cRCTs (Darrouzet Nardi 2016; Marquis 2018; Osei 2017) and three PCS (Doocy 2017; Kennedy 1989; Weinhardt 2017) reported on weight‐for‐age measures.

Cluster randomised controlled trials

A meta‐analysis of two cRCTs showed that income‐generation interventions make little or no difference to the percentage of children who are underweight in households that receive the intervention compared to households that did not, after two years follow‐up (MD 1.06, 95% CI 0.89 to 1.26; 3808 children; I2 = 4%; Analysis 3.9) (Osei 2017; Verbowski 2018).

In addition, three cRCTs reported on the effect of income‐generation interventions on WAZ (Darrouzet Nardi 2016; Marquis 2018; Osei 2017). Data could not be pooled due to high heterogeneity (I2 = 99%; Analysis 3.10). Two studies reported a clear effect favouring income‐generation interventions and Osei 2017 reported no effect at 2.5 years (MD 0.00, 95% CI –0.00 to 0.00; 2613 children).

Prospective controlled studies

Two studies reported on the effects of income‐generation interventions on the percentage of children who were underweight (Doocy 2017; Weinhardt 2017). A meta‐analysis of these studies showed that these interventions make no difference to the percentage of children who are underweight (OR 0.83, 95% CI 0.61 to 1.12; 909 children; I2 = 16%; Analysis 3.11). No effect measures could be calculated for Kennedy 1989.

3.5.3.2 Body mass index

Two cRCTs (Olney 2016; Osei 2017) and three PCS (Alaofe 2019; Asadullah 2015; Kennedy 1989) reported on BMI measures in women.

Cluster randomised controlled trials

Two cRCTs reported a clear effect favouring income‐generation interventions on the proportion of women who were underweight (BMI< 18.5 kg/m2) (Olney 2016; Osei 2017) and one study reported an unclear effect potentially favouring the control (Verbowski 2018) (P = 0.047). Data could not be pooled due to high heterogeneity (I2 = 80%; Analysis 3.12). Heterogeneity seemed to be driven by Verbowski 2018, which was the only study with an aquaculture component in the intervention. All three studies were a variation of the EHFP intervention, so similar in other characteristics.

Olney 2016 and Osei 2017 reported a clear effect favouring income‐generation interventions. Olney 2016 reported that the proportion of underweight women in the intervention group was 8.7 pp lower compared to the control group at two years (1297 women; P = 0.01). Osei 2017 reported reduced odds of underweight among women in the intervention group by 39% at two years (OR 0.61, 95% CI 0.46 to 0.82; 2614 mothers) (Table 14). Both studies were at unclear overall risk of bias.

Verbowski 2018 reported an unclear effect potentially favouring the control, with the proportion of underweight women being higher in the intervention group by 3.88 pp (95% CI –4.36 to 12.12; 911 women) (Table 14). This study was at unclear overall risk of bias.

In addition Olney 2016 and Osei 2017 reported on the mean BMI in women. A meta‐analysis of these two cRCTs reported little or no effect on the mean BMI of women from households who received income‐generation interventions, such as integrated agriculture and nutrition programmes or community development programmes, compared to women from households who did not, after two years of follow‐up (MD –0.02, 95% CI –0.28 to 0.25; 2 RCTs, 3911 women; Analysis 3.13).

Prospective controlled studies

One PCS reported an unclear effect on the proportion of underweight women in the villages with the intervention, compared to women from villages who did not, after one year of follow‐up (MD –0.22, 95% CI –0.75 to 0.31; 359 women) (Table 15) (Alaofe 2019).

Three studies reported on the effect on mean BMI in women. Effect measures for mean BMI could not be calculated for one PCS (Kennedy 1989). The other two studies reported an unclear effect potentially favouring the intervention (P = 0.063). Asadullah 2015 did not report the variance of effect for mean BMI of women (MD 0.14; P = 0.29). Alaofe 2019 reported an unclear effect on the mean BMI of women, favouring the intervention (the installation of solar‐powered irrigation systems), compared to the mean BMI of women from households in villages where the technology was not available, after one year of follow‐up (difference in differences (DID) 0.43, 95% CI –0.05 to 0.89; 359 women; P < 0.1).

3.5.4 Mid‐upper arm circumference

One PCS reported no difference in the mean MUAC of women who were part of an employment intervention compared to women in the control group, in Nepal at two years; the mean change in the intervention group was –0.20 cm and in the control group was –0.25 cm (718 women; P = 0.67) (Katz 2001).

3.6 Change in biochemical indicators

Two cRCTs reported biochemical indicators, such as haemoglobin levels in women and children (Osei 2017; Verbowski 2018), and one PCS reported iron and vitamin A deficiency in women (Alaofe 2019).

Cluster randomised controlled trials

A meta‐analysis of two cRCTs showed a clear effect on mean haemoglobin levels in children, favouring income‐generation interventions (MD 3.49, 95% CI 3.25 to 3.72; 2 RCTs, 3808 children; Analysis 3.14) (Osei 2017; Verbowski 2018). We could not pool the data for women due to high heterogeneity (I2 = 96%; Analysis 3.15). Osei 2017 reported a clear effect on haemoglobin levels favouring income‐generation interventions at 2.5 years (MD 4.6, 95% CI 4.59 to 4.61; 2614 mothers), whereas Verbowski 2018 reported an unclear effect potentially favouring the control (MD –0.07, 95% CI –1.92 to 1.78; 811 women) (Table 14). Both studies were at unclear overall risk of bias.

Prospective controlled studies

Alaofe 2019 reported unclear effects favouring the intervention (the installation of solar‐powered irrigation systems) for the proportion of women with iron deficiency (MD –0.11, 95% CI –0.94 to 0.72; 68 women; P > 0.05), and favouring the control for the proportion of women with vitamin A deficiency (MD 0.54, 95% CI –0.41 to 1.49; P > 0.05; 60 women) (Table 15).

3.7 Cognitive function and development

None of the studies included in this comparison reported cognitive function and development.

3.8 Change in proportion of anxiety or depression

None of the studies included in this comparison reported proportion of anxiety or depression.

3.9 Morbidity
Cluster randomised controlled trials

Two cRCTs report on morbidity measures, such as the prevalence of anaemia in women and children (Osei 2017; Verbowski 2018). A meta‐analysis of these showed a clear effect favouring income‐generation interventions on the proportion of children with anaemia, after two years of follow‐up (OR 0.73, 95% CI 0.61 to 0.88; 2 RCTs, 3808 children; Analysis 3.16). However, in women, these interventions resulted in an unclear effect favouring the control after follow‐up for two years (OR 1.06, 95% CI 0.82 to 1.38; 2 RCTs, 3696 women; Analysis 3.17).

Prospective controlled studies

Three PCS reported unclear effects different morbidity measures (Alaofe 2019; Asadullah 2015; Kennedy 1989).

Asadullah 2015 reported an unclear effect potentially favouring the intervention in the proportion of household members reporting serious illness in the previous year, over nine years of the intervention, with this proportion reducing both in the intervention and the control group (3 years: pp –1.72, P > 0.1; 6 years: pp –0.78, P > 0.1; 9 years: pp –0.70, P > 0.1; 4038 households). This study is at high overall risk of bias. Kennedy 1989 reported a higher percentage of time being ill among those in the intervention group (sugarcane growers scheme) at two years; both for children (29.8% with intervention versus 31.2% with control; 1055 children) and for women (23.8 with intervention versus 24.3% with control; 420 women). In terms of the percent of time children were ill with diarrhoea, this was higher in the intervention group at two years (4.6 with intervention versus 4.0% with control; 1055 children). However, the study reported no baseline values or effect measures, which was at unclear overall risk of bias. Alaofe 2019 reported a clear effect favouring income‐generation interventions on the prevalence of anaemia in women from intervention households, compared to control households (at 1 year: MD –1.25, 95% CI –1.83 to –0.67; 126 women; P = 0.05); the effect was unclear favouring the intervention for the prevalence of iron‐deficiency anaemia in women from intervention households compared to control households (at 1 year: MD –0.99, 95% CI –2.39 to 0.41; 546 women; P > 0.05).

3.10 Adverse outcomes (proportion of overweight/obesity)

None of the studies included in this comparison reported proportion of overweight/obesity.

Comparison 4: food prices – food vouchers

Three cRCTs (Fenn 2015; Hidrobo 2014; Ponce 2017) and one RCT (Jensen 2011) reported the effects of food vouchers compared to no intervention. Food vouchers are provided to households or individuals for the purchase of food, which could be specific to particular foods or for any foods, and usually can be redeemed at specific vendors. Hidrobo 2014 provided vouchers to households, to the value of USD 40 per month, that could be redeemed at specific supermarkets in urban areas in Ecuador for nutritionally approved foods within 30 days of receiving the vouchers. Participants in this study also had to attend monthly nutrition sensitisation sessions. Fenn 2015 provided monthly fresh food vouchers with a cash value of 1500 PKR (approximately USD 14), which could be exchanged for specified fresh foods (fruits, vegetables, milk and meat) in nominated shops in Pakistan. Vouchers were distributed at specific distribution points either by mobile banks that travelled to a central location serving some of the participating villages or through central banks that served a number of villages. In Ponce 2017, households in Ecuador received a food voucher of USD 40 monthly. In Jensen 2011, a month's supply of vouchers entitled participants to a price reduction of the local staple food to the value of 750 g per person per day of that staple food. More details on these studies are available in Table 16 and in the Characteristics of included studies table.

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Table 16. Food vouchers, subsidies, social support: overview of included studies

Study ID (country)

Study design

Overall risk of biasa

Other key details of intervention

Population (sample size at baseline: intervention/ control)

Outcome domains and measures with available data

Timepoint of measurement

Comparison 4: food vouchers

Fenn 2015

(Pakistan)

cRCT

Low

Programme name: REFANI Pakistan

Intervention description and frequency: 3 intervention groups all disbursed at the same time every month for 6 consecutive months:

  • Unconditional transfer (see OSIS Table comparison 1);

  • Unconditional transfer (see OSIS table comparison 2) and

  • Fresh food vouchers with a cash value of PKR 1500 (approximately USD 14), which could be exchanged for specified fresh foods (fruits, vegetables, milk and meat) in nominated shops.

Provider: Action Against Hunger field staff

Delivery: food vouchers disbursed monthly at distribution points. Verbal messaging from Action Against Hunger field staff at distribution that children should benefit from the transfers.

Co‐interventions: WINS programme in all villages provided outpatient treatment for children aged 6 (SD 59) months with SAM, micronutrient supplementation (children, pregnant and lactating women), and behaviour change communication.

Poor and very poor HHs in agrarian district

(food voucher intervention/control: 632/632 HHs)

Anthropometric indicators:

  • Wasting (WHZ < –2SD)

  • Severe wasting (WHZ < –3SD)

  • WHZ

  • Stunting (HAZ < –2SD)

  • Severe stunting (HAZ < –3SD)

  • HAZ

  • MUAC

  • BMI

Biochemical indicators:

  • Hb

Morbidity:

  • ARI

  • Diarrhoea

  • Anaemia

6 and 12 months

Jensen 2011

(China)

RCT

Unclear

Programme name: N/A

Intervention description and frequency: 1‐month supply of vouchers entitling HHs to a price reduction of CNY 0.10, CNY 0.20 or CNY 0.30 (Rmb; 1 Rmb = USD 0.13) off the price of 1 jin (1 jin = 500 g) of the local staple (rice or wheat flour) to the value of 750 g per person per day.

Provider: employees of the provincial‐level agencies of the Chinese National Bureau of Statistics.

Delivery: printed vouchers redeemed by HHs at local grain shops. Shop owners reimbursed for the cost of the vouchers and given a fixed payment for complying with implementation guidelines. Re‐sale of vouchers or goods purchased with vouchers not permitted.

Co‐interventions: NR

Poor urban HHs (969/324)

Adequacy of dietary intake

  • Mineral Sufficiency index

  • Vitamin Sufficiency index

6–7 months

Hidrobo 2014

(Ecuador)

cRCT

High

Programme name: N/A

Intervention description and frequency: included a CCT group (see OSIS table comparison 2) and a food voucher group. Value of USD 40 per month per HH, given in denominations of USD 20. Participants were required to attend monthly nutrition sensitisation training sessions by HH members.

Provider: World Food Programme (NPO)

Delivery: printed serialised vouchers redeemed at central supermarkets in urban centres for a list of nutritionally approved foods, within 30 days of receipt.

Co‐interventions: NR

Poor urban HHs (2087 HHs)

Dietary diversity:

  • DDI;

  • HDDS;

  • FCS

7 months

Ponce 2017

(Ecuador)

cRCT

High

Programme name: N/A

Intervention description and frequency: 2 intervention groups:

  • HHs received a food voucher of USD 40 monthly;

  • HHs received a food voucher of USD 40 monthly + monthly training sessions on topics that included malnutrition, food preparation, children's health, mother's health, women's rights and women's empowerment.

Provider: NR

Delivery: NR

Co‐interventions: NR

HHs based in 3 provinces in Ecuador (food voucher only group/food voucher + training on health and nutrition/control: 171/401/201 HHs)

Dietary diversity:

  • FCS

12 months

Comparison 5: food and nutrition subsidies

Chen 2019

(China)

cRCT

High

Programme name: N/A

Intervention description and frequency: Schools in 2 intervention groups received a one‐off nutrition subsidy with a monetary equivalent of CNY 225 (USD 33) per enrolled student. Schools could use these for nutrition‐related expenses, e.g. buying food. Schoolmasters received information about the proportion of enrolled students who were anaemic, elective methods for reducing iron‐deficient anaemia, and details about anaemia's relation with school attendance, educational performance, and cognitive development. Schoolmasters in treatment group 1 were given a general policy target of 'malnutrition reduction' and in treatment group 2 a specific policy target of 'anaemia reduction', with a potential monetary bonus tied to a reduction in anaemia prevalence (CNY 150/USD 22 per student whose anaemia status changed).

Provider: project team and local government

Delivery: CNY 225 (equivalent to USD 33) per student was transferred into the school's bank account. Incentive payment for treatment group 2 was only calculated and transferred after the intervention period.

Co‐interventions: NR

Primary schools in rural areas (nutritional subsidy only/nutritional subsidy + monetary incentive/control: 15/15/29 schools)

Dietary diversity:

  • Dietary Diversity Score

Anthropometric indicators:

  • BMIZ

  • Underweight

Biochemical indicators:

  • Hb

Morbidity:

  • Anaemia

6 months

Andaleeb 2016

(India)

Prospective controlled study

High

Programme name: PDS

Intervention description and frequency: universal access to the PDS. All HHs that possess a ration card were eligible for 25 kg of subsidised rice, whether they are the poorest of the poor, below the poverty line or above the poverty line.

Provider: state government

Delivery: a ration card was a document issued by the government which entitled an individual/family to purchase from the PDS. Ration cards classified HHs based upon their poverty status and were also used as an identity card to avail many of the other government schemes.

Co‐interventions: other government schemes (not specified)

Rural HHs (3819 HHs)

Adequacy of dietary intake

  • Ratio of nutrient intake to RDA

7 years

Chakrabarti 2018

(India)

Prospective controlled study

High

Programme name: PDS

Intervention description and frequency: subsidising a variety of pulses in different districts as part of the PDS, in addition to the usual subsidising of rice, wheat, sugar and kerosene oil.

Provider: state governments (subsiding of pulses) and central Indian government (subsiding of rice, wheat, sugar and kerosene).

Delivery: government‐issued ration cards are given to poor HHs enabling them to purchase from the PDS.

Co‐interventions: NR

Rural and urban HHs in selected states (23,558/101,086 HHs)

No relevant outcome measures reported

5 years

Sturm 2013

(South Africa)

Prospective controlled study

High

Programme name: HealthyFood Program

Intervention description and frequency: provided a rebate of up to 25% on healthy food purchases in > 400 designated supermarkets across South Africa, for members of the private Discovery Health Insurance and their Vitality programme.

Provider: Discovery Health Insurance company in collaboration with Pick n Pay (brand) supermarkets.

Delivery: members had specific Discovery credit cards that they use for shopping. Scanner data from pay points available every time the card was swiped when purchasing certain healthy food items at Pick n Pay supermarket. These data were collated monthly.

Co‐interventions: NR

169,485 Discovery Vitality members who shopped at Pick n Pay supermarkets with linkable purchasing data (100,344 activated participants and 69,141 non‐participants, i.e. who were not actively using their benefits.)

Proportion of HH expenditure on food

  • Ratio of healthy to total food expenditure: for 10%/25% rebate group compared to control

Maximum 28 months (period November 2009 to March 2012)

Comparison 6: Social support interventions

Kusuma 2017b

(Indonesia)

cRCT

Unclear

Programme name: Generasi

Intervention description and frequency: block payments to villages of USD 8500 (2007) and USD 18200 (2009) per village.

Provider: government

Delivery: trained facilitators advised village management team on allocation of funds (41% villages implemented financial incentives for health worker outreach, 79% villages implemented SFP, and 96% villages implemented financial assistance for mothers)

Co‐interventions: NR

Rural HHs 1481 children aged 24–36 months

Anthropometric indicators:

  • Stunting (HAZ < –2SD)

  • Severe stunting (HAZ < –3SD)

  • Wasting (WHZ < –2SD)

  • Severe wasting (WHZ < –3SD)

  • Underweight (WAZ < –2SD)

  • Severe underweight (WAZ < –3SD)

1 year

Brunie 2014

(Mozambique)

Prospective controlled study

High

Programme name: VSL or a combination of VSL and Ajuda Mútua.

Intervention description and frequency: VSLs are self‐managed and capitalised microfinance programmes where members pool savings and can borrow from the pool and repay with interest. Programmes work in cycles which terminate in paying out the accumulated savings and interest to members proportional to their initial deposit. The Ajuda Mútua rotating labour scheme operates with groups of HHs working together on each family's land or enterprise on a rotational basis.

Provider: Save the Children (NGO)

Delivery: NR

Co‐interventions: SANA (Segurança Alimentar de Nutrição e Agricultura) – food security through nutrition and agriculture multiyear assistance programme targeting aspects of food utilisation. Communities are mobilised to adopt good nutrition practices, and pregnant women and carers are taught to prevent malnutrition in young children.

Interested HHs in randomised district (VSL: 395; VSL+Ajuda Mútua: 401; control: 480)

Food security:

  • Self‐reported months of food sufficiency in previous year

Dietary diversity:

  • HDDS

  • IDDS

Anthropometric indicators:

  • WAZ

3 years

aOverall risk of bias based on key domains: selection and attrition bias. If any of these were high, overall risk of bias was considered high.

ARI: acute respiratory infection; BMI: body mass index; BMIZ: body mass index‐for‐age z‐score; CCT: conditional cash transfer; CNY: Chinese yuan; cRCT: cluster randomised controlled trial; DDI: Dietary Diversity Index; FCS: Food Consumption Score; HAZ: height‐for‐age z‐score; Hb: haemoglobin; HDDS: Household Dietary Diversity Score; HH: household; IDDS: Individual Dietary Diversity Score; MUAC: mid‐upper arm circumference; N/A: not applicable/available; NPO: non‐profit organisation; NR: not reported; PDS: Public Distribution System; PKR: Pakistani rupee; RCT: randomised controlled trial; RDA: recommended daily allowance; SAM: severe acute malnutrition; SD: standard deviation; SFP: Supplementary Feeding Programme; VSL: village savings and loan; WAZ: weight‐for‐age z‐score; WINS: Women and Children/Infants Improved Nutrition in Sindh; WHZ: weight‐for‐height z‐score.

Hidrobo 2014 and Ponce 2017 reported dietary diversity measures. Fenn 2015 reported anthropometric measures. Jensen 2011 reported adequacy of dietary intake measures. Results from individual studies are reported in Table 17 and the harvest plot is presented in Figure 8.

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Table 17. Food vouchers – results of included trials

Study ID (risk of bias)

Study design (n)

Food vouchers

No intervention

Effect measure (time point)

Effect direction a

Meta‐analysis

Notes

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

4.3 Proportion of HHs who were food secure

4.3.1 Dietary diversity

4.3.1.1 Outcome measure: Food Consumption Score (mean): different scales (out of 112 and 8)

Hidrobo 2014 (‐)

cRCT (2087 HHs)

59.75

59.05

Coefficient 9.40, 95% CI 6.6 to 12.2, P < 0.01 (7 months)

No. SMD needed as scales are different. SMD could not be calculated due to missing group sizes for Hidrobo – MV to email authors.

SE calculated from CI

Ponce 2017 food voucher alone (‐)

cRCT (372 HHs)

5.96

NR

171 HHs

5.89

NR

201 HHs

Coefficient 0.394, SE 0.05, 95% CI 0.296 to 0.492, P < 0.01 (1 year)

SE available

Ponce 2017 food voucher + education (‐)

cRCT (602 HHs)

5.83

NR

401 HHs

5.89

NR

201 HHs

Coefficient 0.291, SE 0.081, P < 0.01 (1 year)

SE available

Secondary outcomes

4.4 Change in adequacy of dietary intake

4.4.1 Outcome measure: Mineral Sufficiency Index (mean, SD)

Jensen 2011 (?)

RCT (1265 HHs)

1.02 (0.36)

969

1.00 (0.34)

% change –0.061, 95% CI –0.219 to 0.098 (5 months)

4.4.2 Outcome measure: Vitamin Sufficiency Index (mean, SD)

Jensen 2011 (?)

RCT (1265 HHs)

1.2 (0.44)

1.17 (0.38)

% change –0.051, 95% CI –0.218 to 0.116 (5 months)

4.5 Change in anthropometric indicators

4.5.1 Stunting

4.5.1.1 Outcome measure: % stunted (HAZ < –2SD), n (%)

Fenn 2015 (+)

cRCT (1643 children)

473 (54.9)

NR

834 children

437 (51.7)

NR

809 children

OR 0.41, 95% CI 0.25 to 0.67, P < 0.001 (6 months)

Fenn 2015 (+)

cRCT (1633 children)

473 (54.9)

NR

818 children

437 (51.7)

NR

815 children

OR 0.48, 95% CI 0.31 to 0.73, P = 0.001 (12 months)

4.5.1.2 Outcome measure: % severely stunted (HAZ < –3SD)

Fenn 2015 (+)

cRCT (1643 children)

NR

NR

834 children

NR

NR

809 children

OR 0.38, 95% CI 0.23 to 0.63, P < 0.001 (6 months)

Fenn 2015 (+)

cRCT (1633 children)

NR

NR

818 children

NR

NR

815 children

OR 0.51, 95% CI 0.33 to 0.79, P = 0.003 (12 months)

4.5.1.3 Outcome measure: HAZ, mean (SD)

Fenn 2015 (+)

cRCT (1643 children)

–2.12 (1.69)

NR

834 children

–1.97 (1.75)

NR

809 children

Beta‐coefficient 0.27, 95% CI 0.19 to 0.34, P < 0.001 (6 months)

Fenn 2015 (+)

cRCT (1633 children)

–2.12 (1.69)

NR

818 children

–1.97 (1.75)

NR

815 children

Beta‐coefficient 0.29, 95% CI 0.19 to 0.40, P < 0.001 (12 months)

4.5.2 Wasting

4.5.2.1 Outcome measure: % wasted (WHZ <2SD), n (%)

Fenn 2015 (+)

cRCT (1643 children)

165 (19.3)

NR

834 children

184 (21.9)

NR

809 children

OR 1.16, 95% CI 0.67 to 2.01, P = 0.6 (6 months)

Fenn 2015 (+)

cRCT (1633 children)

165 (19.3)

NR

818 children

184 (21.9)

NR

815 children

OR 1.17, 95% CI 0.75 to 1.82, P = 0.5 (12 months)

4.5.2.2 Outcome measure: % severely wasted (WHZ) < –3SD

Fenn 2015 (+)

cRCT (1643 children)

46 (5.4)

NR

834 children

62 (7.4)

NR

809 children

OR 1.27, 95% CI 0.45 to 3.55, P = 0.66 (6 months)

4.5.2.3 Outcome measure: WHZ, mean (SD)

Fenn 2015 (+)

cRCT (1643 children)

–1.08 (1.14)

NR

834 children

–1.15 (1.30)

NR

809 children

Beta‐coefficient 0.16, 95% CI 0.05 to 0.26, P = 0.004 (6 months)

Fenn 2015 (+)

cRCT (1633 children)

–1.08 (1.14)

NR

818 children

–1.15 (1.30)

NR

815 children

Beta‐coefficient 0.02, 95% CI –0.10 to 0.14, P = 0.79 (12 months)

4.5.3 Underweight

4.5.3.1 Outcome measure: MUAC, mean (SD)

Fenn 2015 (+)

cRCT (1643 children)

13.8 (1.2)

NR

834 children

13.5 (1.2)

NR

809 children

Beta‐coefficient –0.05, 95% CI –0.14 to 0.04, P = 0.27 (6 months)

Fenn 2015 (+)

cRCT (1204 women)

25.2 (3.2)

NR

603 mothers

24.3 (3.2)

NR

601 mothers

Beta‐coefficient –0.16, 95% CI –0.38 to 0.05, P = 0.14 (6 months)

4.5.3.2 Outcome measure: BMI, mean (SD)

Fenn 2015 (+)

cRCT (1204 women)

20.8 (18.5 ± 24.0)

NR

603 mothers

20.0 (18.1 ± 22.7)

NR

601 mothers

Beta‐coefficient 0.29, 95% CI 0.03 to 0.54, P = 0.03 (6 months)

aEach triangle represents one study.

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

CI: confidence interval; cRCT: cluster randomised controlled trial; FCS: Food Consumption Score; HAZ: height‐for‐age z‐score; HH: household; MUAC: mid‐upper arm circumference; n: number; NR: not reported; OR: odds ratio; RCT: randomised controlled trial; SD: standard deviation; SE: standard error; SMD: standardised mean difference.


Harvest plot: food vouchers.

Harvest plot: food vouchers.

Primary outcomes
4.1 Change in the prevalence of undernourishment

None of the included trials reported prevalence of undernourishment.

4.2 Proportion of household expenditure on food

None of the included trials reported household expenditure on food.

4.3 Proportion of households who were food secure
4.3.1 Dietary diversity

Evidence from two trials reported that food vouchers may improve dietary diversity slightly (2 RCT, 2459 households; low‐certainty evidence; summary of findings Table 4) (Hidrobo 2014; Ponce 2017). Both studies reported clear effects favouring the intervention (P = 0.063; Figure 8).

In Hidrobo 2014, at seven months, among households in the food voucher group the FCS was higher by 9.4 points (out of maximum score of 112) (95% CI 6.6 to 12.2; 2087 households; P < 0.01). Other measures of dietary diversity reported in Hidrobo 2014 also indicate clear effects favouring food vouchers (Table 17). In Ponce 2017, the FCS increased by 0.39 points in the intervention group (95% CI 0.30 to 0.49). Pooled analysis was not possible because numbers per group were not reported for Hidrobo 2014 and thus SMD could not be calculated (Analysis 4.1). Both studies were at high overall risk of bias.

Secondary outcomes
4.4 Change in adequacy of dietary intake

Jensen 2011 reported an unclear effect potentially favouring the control on the mineral and vitamin sufficiency indices (Table 17). These indices reflect the mean intake per person relative to the Dietary Reference Intake (DRI). Among households in the intervention group, there was a reduction in the mineral sufficiency index compared to the control group (percentage change –0.06, 95% CI –0.22 to 0.10), and a reduction in the vitamin sufficiency index (percentage change –0.05, 95% CI –0.22 to 0.12) at five months. However, the CIs crossed the null. This study was at low overall risk of bias.

4.5 Change in anthropometric indicators

One cRCT reported on stunting, wasting and underweight (Fenn 2015). This study was at low overall risk of bias.

4.5.1 Stunting: height‐for‐age z‐scores < –2SD

Fenn 2015 reported that food vouchers probably reduce stunting (1 trial; moderate‐certainty evidence; summary of findings Table 4). At 12 months' follow‐up, the odds of stunting was 52% less in the food voucher group compared to control. This study reported a similar effect on other measures of stunting; it reported a reduction in the proportion of children who are severely stunted (OR 0.51, 95% CI 0.33 to 0.79; 1633 children), and an increase in the mean HAZ at 12 months (MD 0.29, 95% CI 0.19 to 0.40; 1633 children; Table 17).

4.5.2 Wasting: weight‐for‐height z‐scores < –2SD

Fenn 2015 reported that food vouchers may result in little to no difference in wasting (1 trial, 1633 children; low‐certainty evidence; summary of findings Table 4). At 12 months of follow‐up, it reported an unclear effect potentially favouring the control (Figure 8); the odds of stunting were 17% higher in the food voucher group compared to the control; however, this effect ranged from 25% reduction to an 82% increased odds of stunting. Similar effects are reported for severe wasting at six months (OR 1.27, 95% CI 0.45 to 3.55; 1643 children); however, for mean WHZ they reported unclear effect favouring the food vouchers at 12 months (coefficient 0.02, 95% CI –0.1 to 0.14; 1633 children; Table 17).

4.5.3 Underweight

Fenn 2015 reported two different measures of underweight. In children, it reported an unclear effect on MUAC at six months (MD –0.05, 95% CI –0.14 to 0.04; 1643 children). In mothers, they reported a clear effect on BMI favouring food vouchers (MD 0.29 kg/m2, 95% CI 0.03 to 0.54; 1204 mothers; Table 17).

4.6 Change in biochemical indicators

None of the included trials reported biochemical indicators.

4.7 Cognitive function and development

None of the included trials reported cognitive function and development.

4.8 Change in proportion of anxiety or depression

None of the included trials reported proportion of anxiety or depression.

4.9 Morbidity

None of the included trials reported morbidity.

4.10 Adverse outcomes (proportion of overweight/obesity)

None of the included trials reported proportion of overweight/obesity.

Comparison 5: food prices – food and nutrition subsidies

One cRCT (Chen 2019) and three PCS (Andaleeb 2016; Chakrabarti 2018; Sturm 2013) assessed the effects of food and nutrition subsidies. These interventions aim to address rising food prices by reducing the price of the foods for the consumer, and are usually provided by the government. Chen 2019 assessed the provision of a one‐off nutrition subsidy with a monetary equivalent of CYN 225 (USD 33) per enrolled student to schools in China, which they could use for nutrition‐related expenses (e.g. buying food). Schoolmasters received information about the proportion of enrolled students who were anaemic; elective methods for reducing iron‐deficient anaemia; and details about anaemia's relation with school attendance, educational performance, and cognitive development. Andaleeb 2016 assessed the public distribution system (PDS) in India, in which households with a ration card were eligible for 25 kg of subsidised rice. A ration card was a document issued by the government which entitled an individual/family to purchase from the PDS, and which was also used as an identity card for other government schemes. Chakrabarti 2018 also assessed the PDS in India, but this study subsidised a variety of pulses in different districts as part of the PDS, in addition to the usual subsidising of rice, wheat, sugar and kerosene oil. Sturm 2013 assessed cash rebates on food purchases. It reports on the HealthyFood programme, which provides a rebate between 10% and 25% on healthy food purchases in designated supermarkets in South Africa for members of the Vitality programme of Discovery Health Insurance scheme. More details about this study are available in Table 11 and in the Characteristics of included studies table.

Chen 2019 reported on dietary diversity, anthropometric, biochemical and morbidity measures. Sturm 2013 reported on the proportion of household expenditure on food. Andaleeb 2016 reported on adequacy of dietary intake. Although Chakrabarti 2018 reported food security, dietary diversity and the proportion of household expenditure on food, it did not report any relevant measure under any of these outcome domains.

Further details of these studies are presented in Table 16. Results of included trials are presented in Table 18 and PCS in Table 19. The harvest plot is presented in Figure 9.

Open in table viewer
Table 18. Food and nutrition subsidies – results of included trials

Study ID (risk of bias)

Study design (n)

Food rebate/subsidy

No intervention

Effect measure (timepoint)

Effect direction

Meta‐analysis

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

5.3 Proportion of HHs who were food secure

5.3.1 Dietary diversity

5.3.1.1 Outcome measure: DDS for nutrition subsidy only (general target: malnutrition reduction) group vs control (mean, SD)

Chen 2019 – nutrition subsidy (‐)

DDS 0–10

cRCT (656 students)

4.75 (2.17)

5.21 (2.18)

219 students

5.33 (2.32)

4.82 (2.36)

437 students

MD 0.956, robust SE 0.255, 95% CI 0.4562 to 1.4558, P < 0.01 (6 months)

N/A

Chen 2019 – nutrition subsidy+monetary incentive (‐)

cRCT

4.65 (2.20)

5.32 (2.09)

210 students

5.33 (2.32)

4.82 (2.36)

437 students

Mean score 1.263, robust SE 0.224, P < 0.01 (6 months)

Secondary outcomes

5.5 Change in anthropometric indicators

5.5.1 Outcome measure: BMI‐for‐age z‐score (mean, SD)

Chen 2019 – nutrition subsidy (‐)

cRCT

–0.70 (0.91)

–0.71 (0.95)

219 students

–0.68 (0.94)

–0.76 (0.97)

437 students

Mean score 0.080, robust SE 0.058

No significant difference from control (6 months)

N/A

Chen 2019 – nutrition subsidy+monetary incentive (‐)

cRCT

–0.63 (0.91)

–0.60 (0.89)

210 students

–0.68 (0.94)

–0.76 (0.97)

437 students

Mean score 0.123, robust SE 0.047, P < 0.01 (6 months)

5.5.2 Outcome measure: proportion underweight (mean, SD)

Chen 2019 – nutrition subsidy (‐)

cRCT

0.07 (0.25)

0.07 (0.26)

219 students

0.08 (0.26)

0.11 (0.32)

437 students

Mean proportion –0.032, robust SE 0.024, 95% CI –0.079 to 0.015 (6 months)

N/A

Chen 2019 – nutrition subsidy+monetary incentive (‐)

cRCT

0.06 (0.24)

0.06 (0.23)

210 students

0.08 (0.26)

0.11 (0.32)

437 students

Mean proportion –0.041, robust SE 0.022, 95% CI –0.084 to 0.002 (6 months)

5.6 Change in biochemical indicators

5.6.1 Outcome measure: haemoglobin concentration in children in nutrition subsidy only (general target: malnutrition reduction) group vs control (mean, SD)

Chen 2019 – nutrition subsidy (‐)

cRCT

128.51 (12.63)

128.11 (15.86)

219 students

128.03 (12.95)

127.93 (14.86)

437 students

Mean concentration 0.512, robust SE 1.348, 95% CI –2.130 to 3.154 (6 months)

N/A

Chen 2019 – nutrition subsidy+monetary incentive (‐)

cRCT

127.84 (12.80)

130.95 (15.66)

210 students

128.03 (12.95)

127.93 (14.86)

437 students

Mean concentration 4.490, robust SE 1.241, 95% CI 2.058 to 6.922, P < 0.01 (6 months)

5.9 Morbidity

5.9.1 Outcome measure: proportion of anaemic children in nutrition subsidy only (general target: malnutrition reduction) group vs control (mean, SD)

Chen 2019 – nutrition subsidy (‐)

cRCT

0.18 (0.38)

0.22 (0.42)

219 students

22 (0.42)

0.23 (0.42)

437 students

Mean proportion –0.005, robust SE 0.048, 95% CI –0.099 to 0.089, P > 0.01 (6 months)

N/A

Chen 2019 – nutrition subsidy+monetary incentive (‐)

cRCT

0.23 (0.42)

0.16 (0.36)

210 students

0.22 (0.42)

0.23 (0.42)

437 students

Mean proportion –0.120, robust SE 0.046, 95% CI –0.210 to –0.029, P < 0.01 (6 months)

cRCT: cluster randomised controlled trial; DDS: Dietary Diversity Score; MD: mean difference; n: number; N/A: not applicable/available; SD: standard deviation; SE: standard error.

Open in table viewer
Table 19. Food and nutrition subsidies – results of included prospective controlled studies

Study ID (risk of bias)

Study design (n)

Food rebate

No intervention

Effect measure (time point)

Effect directiona

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

5.2 Proportion of HH expenditure on food

5.2.1 Outcome measure: ratio of healthy to total food expenditure (mean, SD)

Sturm 2013 – 10% rebate (‐)

Prospective controlled study (169,485 HHs)

0.21 (0.11)

67,343 HHs

0.17 (0.13)

69,141 HHs

Increase by 6.0%, 95% CI 5.3% to 6.8% (3 years)

Sturm 2013 – 25% rebate (‐)

Prospective controlled study (136,484 HHs)

0.21 (0.12)

0.17 (0.13)

Increase by 9.3%, 95% CI 8.5% to 10.0% (2 years and 4 months)

5.4 Change in adequacy of dietary intake

5.4.1 Outcome measure: ratio of current caloric intake to the RDA (multiplied by 100)

Andaleeb 2016 (‐)

Controlled before‐after study

NR

NR

1134 HHs

NR

NR

NR

DID estimate 2.55, SE 1.31, 95% CI –0.018 to 5.118, P < 0.1 (7 years)

5.4.2 Outcome measure: ratio of current protein intake to the RDA (multiplied by 100)

Andaleeb 2016 (‐)

Controlled before‐after study

NR

NR

1134 HHs

NR

NR

NR

DID estimate 3.75, SE 1.65, 95% CI 0.516 to 6.984, P < 0.05 (7 years)

5.4.3 Outcome measure: ratio of current fat intake to the RDA (multiplied by 100)

Andaleeb 2016 (‐)

Controlled before‐after study

NR

NR

1134 HHs

NR

NR

NR

DID estimate –0.1, SE 0.00, P > 0.1 (7 years)

aEach triangle represents one study.

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

CI: confidence interval; DID: difference in differences; HH: household; n: number; NR: not reported; RDA: recommended daily allowance; SD: standard deviation; SE: standard error.


Harvest plot: food and nutrition subsidies.

Harvest plot: food and nutrition subsidies.

Primary outcomes
5.1 Change in the prevalence of undernourishment

None of the included studies measured prevalence of undernourishment.

5.2 Proportion of household expenditure on food

Evidence from one study was very uncertain about the effects of food rebates on household expenditure on healthy foods (1 study, 169,485 households; very low‐certainty evidence; summary of findings Table 5) (Sturm 2013). This study reported clear effects favouring cash rebates on proportion of healthy to total household food expenditure (Figure 9). The study authors reported that food rebates increased the ratio of healthy to total food expenditure: the 10% rebated increased it by 6% at three years (95% CI 5.3 to 6.8; 169,485 households), and the 25% rebate increased it by 9.3% at two years and four months (95% CI 8.5 to 10.0; 136,484 households) (Table 19). This study was at high overall risk of bias due to high risk of selection bias.

5.3 Proportion of households who were food secure

One cRCT reported the effects of nutrition subsidies on dietary diversity (Chen 2019).

5.3.1 Dietary diversity

Evidence from one trial indicated that nutrition subsidies may improve dietary diversity among school children (1 RCT, 656 children; low‐certainty evidence; summary of findings Table 5) (Chen 2019). This study reported a clear effect favouring nutrition subsidies (Figure 9); at six months, the dietary diversity score of school children in the subsidy group increased 0.956 points more (almost one more food group) more than in the control group (MD 0.96, 95% CI 0.46 to 1.45). This study was at high overall risk of bias.

Secondary outcomes
5.4 Change in adequacy of dietary intake

Andaleeb 2016 reported on the adequacy of dietary intake for energy, protein and fat (Table 19). At seven years, it reported a clear effect favouring food subsidies for the ratio of protein intake to the recommended daily allowance (RDA) (DID 3.75, 95% CI 0.52 to 6.98; n = NR), an unclear effect potentially favouring food subsidies for the ratio of current caloric intake to the RDA (DID 2.55, 95% CI –0.02 to 5.12; n = NR), and unclear effects potentially favouring the control on the ratio of fat intake to the RDA (DID –0.1, SE 0.00; P > 0.1).

5.5 Change in anthropometric indicators

Chen 2019 reported on anthropometric indicators.

5.5.1 Underweight (body mass index z‐score < –2SD)

Chen 2019 reported an unclear effect potentially favouring nutrition subsidies (Table 18). The proportion of underweight children reduced in the schools receiving the nutrition subsidy by 3.2 pp compared to the control schools; however, the CIs crossed the null (MD –0.03, 95% CI –0.08 to 0.02, 656 children). There was a similar effect in mean BMIZ (Table 18). In the group where a monetary incentive was provided if the school achieved the desired targets, the effect was similar for the proportion of children who were underweight, but it clearly favoured nutrition subsidies for BMIZ. This study was at high overall risk of bias.

5.6 Change in biochemical indicators
5.6.1 Haemoglobin

Chen 2019 reported an unclear effect potentially favouring the intervention at six months on the mean concentration of haemoglobin in school children (MD 0.51, 95% CI –2.13 to 3.15; n = 656; Table 18). Adding a monetary incentive resulted in a clear effect favouring nutrition subsidies (MD 4.49, 95% CI 2.06 to 6.92).

5.7 Cognitive function and development

None of the included studies measured cognitive function and development.

5.8 Change in proportion of anxiety or depression

None of the included studies measured anxiety or depression.

5.9 Morbidity
5.9.1 Anaemia

Chen 2019 reported an unclear effect potentially favouring nutrition subsidies on the proportion of school children with anaemia at six months (MD –0.005, 95% CI –0.1 to 0.09). Adding a monetary incentive resulted in a clear effect favouring nutrition subsidies (Table 18). This study was at high overall risk of bias.

5.10 Adverse outcomes (proportion of overweight/obesity)

None of the included studies measured overweight/obesity.

Comparison 6: social support

Two included studies assessed social support interventions. One cRCT in Indonesia randomised subdistricts to receive a community cash grant or to a control group, which were linked to health and education conditionalities (Kusuma 2017b). Two intervention groups were implemented, one with and one without a performance incentive, but the effect of both is reported together. One PCS randomised households either to a VSL group or to a VSL and Ajuda Mutua (AM) group, or to a control group (Brunie 2014). VSLs are self‐managed and capitalised microfinance programmes where members pool savings and can borrow from the pool and repay with interest. AM is a rotating labour scheme, where groups of households work together on each family's land or enterprise on a rotational basis. A combined effect of both groups is reported.

Kusuma 2017b reported effects on anthropometric indicators, and Brunie 2014 reported effects on measures of food security, dietary diversity and anthropometry.

Further details about these studies are presented in Table 16 and in the Characteristics of included studies table. Results from these studies are presented in Table 20 and Table 21, and in the harvest plot in Figure 10.

Open in table viewer
Table 20. Social support interventions – results of included trials

Study ID (risk of bias)

Study design (n)

Village savings/grants

No intervention

Effect measure (time point)a

Effect direction

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Secondary outcomes

6.5 Change in anthropometric indicators

6.5.1 Stunting

6.5.1.1 Outcome measure: proportion stunted (HAZ < –2SD)

Kusuma 2017b (?)

cRCT (1481 children aged 24–36 months)

Mean 0.48

DID 0.034, SE 0.055, 95% CI –0.074 to 0.142, P > 0.05 (2 years)

1.1.2 Outcome measure: proportion severely stunted (HAZ < –3SD)

Kusuma 2017b (?)

cRCT (1481 children aged 24–36 months)

Mean 0.29

DID –0.06, SE 0.053, 95% CI –0.164 to 0.044, P > 0.05 (2 years)

6.5.2 Wasting

6.5.2.1 Outcome measure: proportion wasted (WHZ < –2SD)

Kusuma 2017b (?)

cRCT (1481 children aged 24–36 months)

Mean 0.19

DID –0.010, SE 0.035, 95% CI –0.079 to 0.059

pp –1.0, 95% CI –7.86 to 5.86, P > 0.05 (2 years)

6.5.2.2 Outcome measure: proportion severely wasted (WHZ < –3SD)

Kusuma 2017b (?)

cRCT (1481 children aged 24–36 months)

Mean 0.10

DID –0.021, SE 0.025, 95% CI –0.07 to 0.028, P > 0.05 (2 years)

6.5.3 Underweight

6.5.3.1 Outcome measure: proportion underweight (WAZ < –2SD)

Kusuma 2017b (?)

cRCT (1481 children aged 24–36 months)

Mean 0.34

Beta –0.020, SE 0.051, 95% CI –0.120 to 0.080, P > 0.05 (2 years)

6.5.3.2 Outcome measure: proportion severely underweight (WAZ < –3SD)

Kusuma 2017b (?)

cRCT (1481 children aged 24–36 months)

Mean 0.12

Beta –0.056, SE 0.034, 95% CI –0.123 to 0.011, P < 0.1 (2 years)

aEach triangle represents one study.

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

DID: difference in differences; HAZ: height‐for‐age z‐score; n: number; pp: percentage point; SD: standard deviation; SE: standard error; WAZ: weight‐for‐age z‐score; WHZ: weight‐for‐height z‐score.

Open in table viewer
Table 21. Social support interventions – results of included prospective controlled studies

Study ID (risk of bias)

Study design (n)

Village savings/grants

No intervention

Effect measure (time point)a

Combined group effect

Effect direction

Meta‐analysis

Results

at baseline

Results

at follow‐up

n

Results

at baseline

Results

at follow‐up

n

Primary outcomes

6.3 Proportion of HH who were food secure

6.3.1 Food security

6.3.1.1 Outcome measure: self‐reported months of food sufficiency in previous year (mean, SD)

Brunie 2014 – VSL (‐)

Prospective controlled study (851 HHs)

10.41

10.52

10.58

10.21

DID estimate 0.47, 95% CI –0.04 to 0.98, P < 0.1 (3 years)

MD 1.25, 95% CI –0.28 to 2.79

N/A

Brunie 2014 – VSL+AM (‐)

836 HHs

9.27

11.18

10.47

10.35

DID estimate 2.04, 95% CI 1.53 to 2.55, P < 0.1 (3 years)

6.3.2 Dietary diversity

6.3.2.1 Outcome measure: HDDS (mean, SD)

Brunie 2014 – VSL (‐)

Prospective controlled study (802 HHs)

4.06

5.44

3.73

4.84

DID estimate 0.27, 95% CI –0.16 to 0.70, P > 0.1 (3 years)

MD –0.30, 95% CI –1.46 to 0.87

N/A

Brunie 2014 – VSL+AM (‐)

813 HHs

4.2

4.56

3.82

5.11

DID estimate −0.92, 95% CI –1.567 to –0.273, P < 0.001

6.3.2.2 Outcome measure: IDDS (mean, SD)

Brunie 2014 – VSL (‐)

Prospective controlled study (542 children)

2.51

3.43

2.87

2.97

DID estimate 0.81, 95% CI 0.36 to 1.26, P < 0.01 (3 years)

MD 0.52, 95% CI –0.18 to 1.23

N/A

Brunie 2014 – VSL+AM (‐)

(579 children)

2.99

3.46

2.82

3.22

DID estimate 0.07, 95% CI –0.7532 to 0.8932, P > 0.01 (3 years)

Secondary outcomes

6.5 Change in anthropometric indicators

6.5.1 Outcome measure: weight‐for‐age z‐scores (WAZ)

Brunie 2014 – VSL (‐)

Prospective controlled study (503 children)

–1.21

–0.91

–1.25

–0.83

DID estimate –0.11, 95% CI –0.561 to 0.341, P > 0.1 (3 years)

MD 0.05, 95% CI –0.37 to 0.48

N/A

Brunie 2014 – VSL+AM (‐)

(550 children)

–0.96

–0.93

–1.15

–0.78

DID estimate 0.34, 95% CI –0.31 to 0.99, P > 0.01

aEach triangle represents one study.

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

AM: Ajuda Mútua; CI: confidence interval; DID: difference in differences; HDDS: Household Dietary Diversity Score; HH: household; IDDS: Individual Dietary Diversity Score; MD: mean difference; n: number; N/A: not applicable/available; SD: standard deviation; VSL: village savings and loan; WAZ: weight‐for‐age z‐score.


Harvest plot: social support interventions.

Harvest plot: social support interventions.

Primary outcomes
6.1 Change in the prevalence of undernourishment

None of the included studies measured prevalence of undernourishment.

6.2 Proportion of household expenditure on food

None of the included studies measured household expenditure on food.

6.3 Proportion of households who were food secure

One PCS reported on food security and dietary diversity measures (Brunie 2014).

6.3.1 Food security

The evidence was very uncertain about the effects of VSLs on food security (MD 1.25, 95% CI –0.28 to 2.79; 851 households; very low‐certainty evidence; summary of findings Table 6; Figure 10). Brunie 2014 reported an unclear effect potentially favouring the VSL intervention on the number of self‐reported months of food sufficiency in the previous year, with an increase by 1.25 months in the intervention group at three years (Table 21).

6.3.2 Dietary diversity

The evidence was very uncertain about the effects of VSLs on dietary diversity (1 study, 802 households; very low‐certainty evidence; summary of findings Table 6; Figure 10). Brunie 2014 reported an unclear effect on household dietary diversity favouring the control (MD –0.30, 95% CI –1.46 to 0.87; 1615 households) (Table 21).

Brunie 2014 also reported on Individual Dietary Diversity Scores (IDDS) among children: IDDS was slightly higher by 0.81 points (out of 12) in the VSL group (MD 0.52, 95% CI –0.18 to 1.23; 1121 children) (Table 21).

Secondary outcomes
6.4 Change in adequacy of dietary intake

None of the included studies measured adequacy of dietary intake.

6.5 Change in anthropometric indicators

One trial reported on stunting, wasting and underweight measures (Kusuma 2017b). One PCS reported on underweight measures (Brunie 2014).

6.5.1 Stunting (height‐for‐age z‐scores < –2SD)

Community grants may make little or no difference to stunting (1 trial, 1481 children; low‐certainty evidence; summary of findings Table 6; Figure 10). Kusuma 2017b reported an unclear effect on stunting favouring the control (3.4 pp, 95% CI –7.4 to 14.2; 1481 children aged 24 to 36 months). The effect on severe stunting (HAZ < –3SD) was different: they reported an unclear effect favouring the community cash grants (–6 pp, 95% CI –16.4 to 4.4; 1481 children aged 24 to 36 months; Table 20).

6.5.2 Wasting (weight‐for‐height z‐scores < –2SD)

Community grants probably make little or no difference to wasting (1 RCT, 1481 children; moderate‐certainty evidence; summary of findings Table 6; Figure 10). Kusuma 2017b reported an unclear effect favouring the intervention at two years (–1.0 pp, 95% CI –7.9 to 5.9). The effect on severe wasting (WHZ < –3SD) was similar (–2.1 pp, 95% CI –7 to 2.8; Table 20).

6.5.3 Underweight: weight‐for‐age z‐scores < –2SD

Randomised controlled trials

Kusuma 2017b reported an unclear effect on stunting favouring community cash grants (–2 pp, 95% CI –11.9 to 7.9; 1481 children). The effect on severe underweight (WAZ < –3SD) was similar (Table 20). This study was at unclear overall risk of bias.

Prospective controlled studies

Brunie 2014 reported an unclear effect on WAZ potentially favouring the VSL intervention (with or without a rotating labour scheme). Among children in the intervention group, the mean WAZ increased slightly by 0.05 SDs compared to the control group at three years (MD 0.05, 95% CI –0.37 to 0.48; 1053 children; Table 21).

6.6 Change in biochemical indicators

None of the included studies measured biochemical indicators.

6.7 Cognitive function and development

None of the included studies measured cognitive function and development.

6.8 Change in proportion of anxiety or depression

None of the included studies measured anxiety or depression.

6.9 Morbidity

None of the included studies measured morbidity.

6.10 Adverse outcomes (proportion of overweight/obesity)

None of the included studies measured proportion of overweight/obesity.

Discussion

Summary of main results

Fifty‐nine studies, addressing six intervention types, met the criteria for inclusion in this review. Some studies evaluated the same programme. None of the studies included assessed the primary outcome of prevalence of undernourishment.

Sixteen cRCTs, two parallel‐group RCTs and three PCS assessed UCTs. Available evidence indicates that UCTs improve food security (six RCTs) and make little or no difference to cognitive function and development (three RCTs) (high‐certainty evidence); UCTs may increase dietary diversity (10 RCTs) and may reduce stunting (four RCTs) (low‐certainty evidence); and that the evidence regarding the effects of UCTs on the proportion of household expenditure on food (five RCTs) and wasting (four RCTs) is very uncertain (very low‐certainty evidence). Regarding adverse outcomes, evidence from one trial indicates that UCTs reduce the proportion of infants who are overweight.

Nine cRCTs and five PCS assessed CCTs. None of these studies reported on food security measures. Available evidence indicates that CCTs result in little to no difference in the proportion of household expenditure on food (two RCTs) and that they slightly improve cognitive function in children (two RCTs) (high‐certainty evidence); that CCTs probably slightly improve dietary diversity (two RCTs) (moderate‐certainty evidence); and that they may make little to no difference to stunting (four RCTs) or wasting (two RCTs) (low‐certainty evidence). Evidence on adverse outcomes (two PCS) shows that CCTs make no different to the proportion of overweight children.

Six cRCTs and 11 PCS assessed income‐generation interventions. None of these studies reported on cognitive function and development, or the proportion of household expenditure on food. Available evidence indicates that income‐generation interventions make little or no difference to stunting (two RCTs) or wasting (two RCTs) (moderate‐certainty evidence); and that they may results in little to no difference to food security (two RCTs) and may improve dietary diversity in children but not for households (four RCTs) (low‐certainty evidence).

Four trials reported on food vouchers. None reported on the proportion of households expenditure on food, food security, or cognitive function and development. Available evidence indicates that food vouchers probably reduce stunting (one RCT) (moderate‐certainty evidence), and that they may improve dietary diversity slightly (two RCTs) and may result in little to no difference in wasting (one RCT) (low‐certainty evidence).

One RCT and three PCS reported the effects offood and nutrition subsidies. None of these studies reported on food security, stunting, wasting, or cognitive function and development. Available evidence indicates that food and nutrition subsidies may improve dietary diversity among school children (one RCT) (low‐certainty evidence), and the evidence is very uncertain about the effects on household expenditure on healthy foods as a proportion of total expenditure on food (very low‐certainty evidence).

One RCT and one PCS reported on the effects of social environment interventions. None of the studies reported on the proportion of household expenditure on food, or on cognitive function and development. Available evidence indicates that community grants probably make little to no difference to wasting (one RCT) (moderate‐certainty evidence) and that they make little or no difference to stunting (one RCT) (low‐certainty evidence); and the evidence is very uncertain about the effects of VSLs on food security (one PCS) and dietary diversity (one PCS) (very low‐certainty evidence).

Overall completeness and applicability of evidence

We considered the differences between the evidence identified and our prespecified eligibility criteria, including relevant gaps identified with the harvest plots regarding outcomes with no data, when assessing the completeness and applicability of the evidence.

Participants in included studies ranged from households to individuals, including adults and children. A few studies specifically targeted women as recipients of the intervention. Most studies targeted poor households, and mostly in rural areas, based on specific criteria to identify poor and vulnerable households. Although we had planned to assess effects within specific disadvantaged subgroups, this was not possible because of unclear reporting about these types of characteristics in the included studies.

Our logic model showed that there is a wide range of interventions that could address access to food. Across all the possible interventions, we included mostly studies aiming to increase buying power, including UCTs, CCTs, and income‐generation interventions. Fewer studies addressed food prices and only two assessed social environment interventions, namely a community cash grant programme, and a VSLs programme. We found no studies assessing infrastructure interventions that aimed to improve physical access to food. Some of the studies we excluded from this review assessed some of these relevant interventions, but they were conducted in high‐income countries. For example, one before‐after study that assessed the implementation of a fruit and vegetable market in low‐income neighbourhoods (Gorham 2015), which addressed lack of infrastructure; a randomised trial that assessed financial incentives to increase fruit and vegetable intake among participants in the USA Food Stamp programme (Olsho 2016), which addresses high food prices. This illustrates, perhaps, a lack of such interventions being implemented in LMIC settings. Regarding the variation in type and intensity of interventions, particularly of CCTs and how the conditions and the enforcement of these components vary across interventions, we were unable to distinguish which specific conditionalities were linked to the outcome based on available data. In these interventions, conditionalities ranged from attending clinic visits, educational sessions and school attendance. For other interventions such as income‐generation interventions, intervention components also varied significantly, and it is difficult to specify which intervention components are associated with the observed outcome.

In majority of studies, the intervention was not compared with another intervention. For some larger studies of government programmes, such as cash transfers, it was common for a delayed control to be used as a comparator. In this case, the control group also received the intervention, but at a later stage, as it would be unethical to randomise communities to no intervention in these types of programmes. One issue with this was that many long‐term outcomes were not eligible for reporting in this review as, by that time, both the intervention and control groups were receiving the intervention.

In terms of outcomes, no included study reported on the primary outcome of prevalence of undernourishment. Prevalence of Undernourishment (PoU) is a national‐level model‐based indicator used to understand access to food in terms of dietary energy inadequacy and can be measured at national or household level (INDDEX Project 2018). It measures the percentage of the population whose dietary energy intake is below the MDER. In line with this, the adequacy of dietary intake at the individual level was also not reported in most studies, and only six studies reported this outcome. Most studies reported on actual energy intake (i.e. calories) or intake of specific nutrients (i.e. grams), without assessing this intake against some measure of adequacy, such as the DRIs. Besides these outcome categories, fewer studies reported on child cognitive function and development compared to other outcomes. We had intended to assess not only if adequacy of dietary intake improved, but also if diet quality increased. Included studies did not report on dietary quality (i.e. whether they were refined and high in saturated fat or healthier foods such as legumes, fruit and vegetables). However, many studies reported on dietary diversity, which is an approximate measure of diet quality. Greater dietary diversity should indicate better overall dietary quality, as it means that foods from more food groups are being consumed. For most outcomes, data were available at two and three years, so there is not much evidence on longer‐term impact of included interventions. Only three studies reported on the adverse outcome of overweight and obesity.

Due to the lack of information in many included studies, we were unable to address our first secondary objective – to identify features of interventions that enable or impair the effective implementation. We believe this warrants a separate study assessing each intervention more in‐depth and using different study designs, such as qualitative studies.

Quality of the evidence

We assessed the certainty of the evidence using the GRADE approach and presented our findings in a 'Summary of findings' table for each comparison. For all comparisons except comparison 6, the 'Summary of findings' tables included only data from RCTs, as there were data from at least one RCT for the key outcomes. The 'Summary of findings' tables for comparisons 5 and 6 include data from RCTs and PCS, as for specific outcomes there were no data from RCTs.

For UCTs, the certainty of the evidence ranged from very low to high across outcomes. Reasons for downgrading included inconsistency due to wide variance of point estimates, imprecision due to wide CIs and due to high overall risk of bias.

For CCTs, the certainty of the evidence ranged from low to high across outcomes. Reasons for downgrading included inconsistency due to wide variation in point estimates, high overall risk of bias and imprecision due to wide CIs.

For income‐generation interventions, the certainty of the evidence ranged from low to moderate across outcomes. Reasons for downgrading included imprecision due to wide CIs, indirectness because the evidence was from a single study, high overall risk of bias and inconsistency due to wide variation in point estimates.

The certainty of the evidence on food voucher interventions ranged from low to moderate across outcomes. Reasons for downgrading included high overall risk of bias, inconsistency as CIs had minimal overlap, indirectness as findings were from one single study and imprecision due to findings ranging from an important harm to important benefit.

The certainty of the evidence on food and nutrition subsidies ranged from very low to low across outcomes. Reasons for downgrading included high overall risk bias and indirectness as the results were from a single study.

For VSLs, the certainty of the evidence ranged from very low to moderate. Reasons for downgrading included indirectness, as the results were from a single study, and imprecision, due to wide CIs.

All but one included study was funded by a for‐profit organisation and 66.1% did not report on potential COI.

Potential biases in the review process

We followed Cochrane Review methodology to prevent potential biases from being introduced into the review process. Nevertheless, potential biases could have been introduced due to the nature of subjective decisions that had to be made while conducting the review and because the protocol was outdated and new methods had emerged since its publication (Durao 2015).

Multiple outcome measures concerning the same outcome category were reported across included studies. Since there is no guidance in the literature on what measures are considered as 'gold standard' for measuring food and nutrition security, we selected the most comprehensive or largest scale measure reported for the same outcome domain in the same study. For example, we reported changes in z‐scores for height or weight but we did not report actual height (in centimetre) or weight (kilogram) measures. We judged changes in z‐scores to be more useful and easier to interpret as they are assessing standardised height and weight attainment adjusted for age and sex. Still, multiplicity of outcome measures made it difficult to include all studies reporting the same outcome domain in meta‐analyses.

We also could not include all studies in meta‐analyses due to incomplete reporting of the required data (e.g. variance measures). Therefore, we made post‐hoc decisions about synthesising the evidence using vote counting based on effect direction, using harvest plots to visually illustrate the results. We used the point estimates and the 95% CI to decide how to categorise the effects of the studies. However, data for these were sometimes not available or could not be calculated, and thus we had to base decisions regarding whether the effect was clear or unclear based on the P value.

As we had not prespecified the outcomes to be assessed using GRADE in the protocol (Durao 2015), we had to make this decision post‐hoc. The author team discussed and agreed through consensus which outcomes we considered best for informing decision‐making. However, it may be that another group could have made different decisions regarding which outcomes to highlight in the 'Summary of findings' tables. All outcomes are, however, reported in the review text and in the tables of individual results. We also had to prioritise outcome measures to report in the review as often there were multiple outcome measures reported in the same study for the same outcome domain. We attempted to prevent introduction of biases by selecting an approach that was independent of the effect measure reported (i.e. prioritising the most comprehensive outcome measure).

Our interpretation of the primary outcome 'proportion of household expenditure on food' was based on Engel's Law "…according to which the household decreases its budget share of food as its income increases". This is the interpretation used in one of the included studies (Brugh 2018). Not all included studies interpreted this outcome in the same way, with some interpreting an increase in this proportion as 'good' while others interpreting a decrease as 'good'. Furthermore, not all studies clearly interpreted their findings. This variability led us to interpret it based on Engel's law. While food expenditure is expected to increase with higher income, the proportion of expenditure on food in relation to other expenditure should decrease. The use of this approach may explain some of the diversity in effect measures observed for this outcome.

Due to our high search yield we were unable to complement the electronic database search with screening reference lists of included studies, reference lists of identified relevant systematic reviews or websites of specific organisations, as planned. Thus, we may have missed some relevant studies. However, since our search was very comprehensive, we believe that the chance of this was small.

One limitation of our review was that the synthesis was unable to draw conclusions about the mean effect size for many outcomes, due to limited availability of suitable data. However, we have tried to calculate all necessary information for meta‐analysis where this was possible.

Agreements and disagreements with other studies or reviews

Other recent reviews on food access tend to focus on individual interventions rather than on a comprehensive review of the evidence base of interventions addressing access to food like ours.

We identified six published reviews among the results of the updated search for this review which addressed access to food or included similar interventions to those included in this review. Three reviews addressed cash transfers (Baird 2014; Hunter 2017; Melo 2016); two reviews evaluated interventions related to the income‐generation category (Bird 2019; Pullar 2018); two reviews focused on interventions addressing food prices (Mizdrak 2015; Alagiyawanna 2015); and one review addressed interventions addressing infrastructure, for which we did not find any studies to include in our review (Hsiao 2019).

Of the reviews addressing cash transfers, two had a different focus to our review: one on the effect of cash transfers on educational outcomes (Baird 2014), and one on use and quality of maternity care services (Hunter 2017). The third review included 10 studies of cash transfer interventions in Latin America and reported a positive association of cash transfers with children's anthropometric status (Melo 2016). However, they also reported that improvements may differ by age and that the included studies, which varied in design, were of questionable methodological quality.

Other systematic reviews on cash transfers had a different focus than that of our review; they assessed the effects of such interventions on improving infant vaccination (Munk 2019), women economic empowerment (Leite 2019), clinical outcomes for pulmonary tuberculosis (Richterman 2018), or social determinants of health (Owusu‐Addo 2018). Of those that had a similar focus, some reported positive effects or no effects. One review of cash transfer programmes including different types of literature assessed the evidence of the impact of cash transfers on a range of individual‐ or household‐level outcomes in 201 included studies, of which 89 reported on health and nutrition outcomes, including the use of health services, dietary diversity and anthropometry (Bastagli 2016). They reported a greater proportion of significant results for dietary diversity than for anthropometric measures, but positive impacts in relation to the cash transfer interventions overall. Another review assessed universal and targeted UCTs and targeted CCTs and reported mostly positive effects of these interventions on birth weight, infant mortality, among other outcomes (Siddiqi 2018). Another review assessed the effects of CCTs on child health in LMICs (Owuso‐Addo 2014), and included 16 studies predominantly from Latin America. The review authors reported that programmes improved nutritional status of children in intervention compared to control groups in terms of, for example, growth in height and weight, decreased chance of being underweight, and improved dietary intake of protein and vegetables. However, some of the programmes did not always find an effect on anthropometric outcomes such as childhood wasting or stunting. Of note, these two reviews included the same studies as this Cochrane Review. Therefore, it is perhaps unsurprising that their findings were similar to ours. In one Cochrane Review of UCTs for reducing poverty and vulnerabilities and its effects on health services use and health outcomes in LMICs, the authors also reported that UCTs had beneficial effects on food security and dietary diversity, with evidence certainty ranging from low to moderate, and uncertain effects on stunting due to very low‐certainty evidence (Pega 2017).

Of the two reviews addressing income‐generation interventions, results were similar, with some potential positive effect on diet intake and diversity. Bird 2019 assessed agriculture interventions on nutrition outcomes in specific countries in South Asia (India, Bangladesh, Nepal, Pakistan and Afghanistan) where agriculture activities are major sources for the livelihoods of large sections of the population. They included six studies assessing interventions of provision of seeds, plants and training, or livestock and training, or both, and reported a positive impact on intermediate outcomes, such as diet quality and diversity, but mixed results regarding impact on nutritional outcomes, such as anthropometry and anaemia. No meta‐analyses were carried out due to high heterogeneity. Pullar 2018 assessed the effects of poverty reduction and development interventions on non‐communicable disease (NCD) prevalence and risk. They included 29 studies, mostly of agricultural interventions, and reported limited methodological quality in included studies and high heterogeneity of outcome measures, similarly to our review. Included studies failed to measure and report on NCD prevalence and risk, but they reported that intensive agricultural interventions were associated with improved calorie, vitamin, fruit and vegetable intake, with the effects being dependent on other factors such as land ownership and infection status. However, the findings had poor generalisability because of small sample sizes and use of convenience samples of population with the highest need.

Both reviews of interventions addressing food prices focused on obesity reduction and not on undernutrition. Alagiyawanna 2015 assessed the effects of fiscal interventions implemented at national or local levels to improve diets and reduce obesity, assessing effects on consumption and health outcomes in adults and children. They included 18 studies, mostly from high‐income countries. Nine of these studies assessed the impact of taxes and these were all were from high‐income countries. They reported that the effects of taxation of soft drinks and its consumption was mixed, as was the effect on BMI among children and adolescents. Existing taxation studies tend to be from high‐income countries, which is likely the reason why we did not find any taxation‐related studies to include in our review, which only focused on LMICs. Regarding subsidies, in high‐income countries, Alagiyawanna 2015 reported positive associations with fruit and vegetable intake, maternal weight gain, increase in mean haemoglobin levels, consumption of healthy foods and height‐for‐age, but no association with BMI, low birthweight or fetal survival. One study in a low‐ to middle‐income country reported a negative association of subsidies with increased obesity. The Mizdrak 2015 review assessed fiscal interventions, but its scope was to specifically assess which personal characteristics influence differential impact of fiscal interventions, as this is considered a barrier to implementation. They included eight studies from high‐income countries, reporting high heterogeneity between studies and population groups concerning the effects of fiscal measures on healthy diets. Although they reported that the evidence pointed towards a differential impact depending on personal characteristics, the data were limited and underpowered to detect effects according to personal characteristics.

Hsiao 2019 assessed the barriers and facilitators of mobile produce markets in the US. This is a category of interventions for which we did not find studies to include in our review (i.e. infrastructure interventions), likely because these types of studies currently tend to be from high‐income countries. They reported a positive association of mobile produce markets and fruit and vegetable intake, but noted that the quality of the evidence was problematic as the studies were found not to be rigorous in their design and had high potential for selection and other types of bias.

An important aspect about this review is that it included interventions addressing upstream factors affecting access to food, that do not only rely on individual agency. This has been globally recognised as the best approach to address over‐ and undernutrition, and cash transfers specifically are high on the agenda of many countries as they address the social determinants of health (Hawkes 2020; Owusu‐Addo 2018). These types of interventions fall under the category of double duty actions that address both under‐ and overnutrition at the same time, especially if complemented by education and behaviour change communication and regular check‐ups as part of the intervention (Hawkes 2020).

Food security logic model: how interventions influence food and nutritional security.
Figures and Tables -
Figure 1

Food security logic model: how interventions influence food and nutritional security.

Study flow diagram.

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Figure 2

Study flow diagram.

Risk of bias graph: review authors' judgements about each risk of bias item presented as percentages across all included studies.

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Figure 3

Risk of bias graph: review authors' judgements about each risk of bias item presented as percentages across all included studies.

Risk of bias summary: review authors' judgements about each risk of bias item for each included study.

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Figure 4

Risk of bias summary: review authors' judgements about each risk of bias item for each included study.

Harvest plot: unconditional cash transfers.

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Figure 5

Harvest plot: unconditional cash transfers.

Harvest plot: conditional cash transfers.

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Figure 6

Harvest plot: conditional cash transfers.

Harvest plot: income‐generation interventions.

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Figure 7

Harvest plot: income‐generation interventions.

Harvest plot: food vouchers.

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Figure 8

Harvest plot: food vouchers.

Harvest plot: food and nutrition subsidies.

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Figure 9

Harvest plot: food and nutrition subsidies.

Harvest plot: social support interventions.

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Figure 10

Harvest plot: social support interventions.

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 1: Proportion of household expenditure on food

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Analysis 1.1

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 1: Proportion of household expenditure on food

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 2: Proportion consuming > 1 meal/day

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Analysis 1.2

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 2: Proportion consuming > 1 meal/day

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 3: Food security scores

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Analysis 1.3

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 3: Food security scores

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 4: Dietary Diversity Score including composite food consumption score (FCS) (weighted)

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Analysis 1.4

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 4: Dietary Diversity Score including composite food consumption score (FCS) (weighted)

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 5: Proportion with minimum dietary diversity

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Analysis 1.5

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 5: Proportion with minimum dietary diversity

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 6: Proportion of food poverty (per capita daily caloric intake < 2122 calories)

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Analysis 1.6

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 6: Proportion of food poverty (per capita daily caloric intake < 2122 calories)

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 7: Proportion stunted (height‐for‐age z‐score (HAZ) < ‐2SD)

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Analysis 1.7

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 7: Proportion stunted (height‐for‐age z‐score (HAZ) < ‐2SD)

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 8: HAZ

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Analysis 1.8

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 8: HAZ

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 9: Weight‐for‐height z‐score (WHZ)

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Analysis 1.9

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 9: Weight‐for‐height z‐score (WHZ)

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 10: Weight‐for‐age z‐score (WAZ)

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Analysis 1.10

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 10: Weight‐for‐age z‐score (WAZ)

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 11: Haemoglobin concentration (g/dL)

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Analysis 1.11

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 11: Haemoglobin concentration (g/dL)

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 12: Depression score (CES‐D scale)

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Analysis 1.12

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 12: Depression score (CES‐D scale)

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 13: Perceived Stress Scale (PSS)

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Analysis 1.13

Comparison 1: Unconditional cash transfers (UCT) versus no intervention, Outcome 13: Perceived Stress Scale (PSS)

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 1: Household Dietary Diversity Score (HDDS)

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Analysis 2.1

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 1: Household Dietary Diversity Score (HDDS)

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 2: Proportion stunted (height‐for‐age z‐score (HAZ) < ‐2SD) – RCTs

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Analysis 2.2

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 2: Proportion stunted (height‐for‐age z‐score (HAZ) < ‐2SD) – RCTs

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 3: Proportion with severe stunting (HAZ < ‐3 SD) – RCTs

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Analysis 2.3

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 3: Proportion with severe stunting (HAZ < ‐3 SD) – RCTs

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 4: HAZ – RCTs

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Analysis 2.4

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 4: HAZ – RCTs

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 5: Proportion stunted (HAZ < ‐2 SD) – PCS

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Analysis 2.5

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 5: Proportion stunted (HAZ < ‐2 SD) – PCS

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 6: HAZ – PCS

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Analysis 2.6

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 6: HAZ – PCS

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 7: Proportion wasted (weight‐for‐height z‐score (WHZ) < ‐2 SD) – RCTs

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Analysis 2.7

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 7: Proportion wasted (weight‐for‐height z‐score (WHZ) < ‐2 SD) – RCTs

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 8: WHZ – RCTs

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Analysis 2.8

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 8: WHZ – RCTs

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 9: Proportion underweight (weight‐for‐age z‐score (WAZ) < ‐2SD) – RCTs

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Analysis 2.9

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 9: Proportion underweight (weight‐for‐age z‐score (WAZ) < ‐2SD) – RCTs

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 10: Proportion severely underweight (WAZ < ‐3 SD) – RCTs

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Analysis 2.10

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 10: Proportion severely underweight (WAZ < ‐3 SD) – RCTs

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 11: WAZ – RCTs

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Analysis 2.11

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 11: WAZ – RCTs

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 12: BMI‐for‐age z‐score – PCS

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Analysis 2.12

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 12: BMI‐for‐age z‐score – PCS

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 13: Cognitive test scores – RCTs

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Analysis 2.13

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 13: Cognitive test scores – RCTs

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 14: Proportion reporting being ill in past 4 weeks/parents seeking care for illness past 2 weeks – RCTs

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Analysis 2.14

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 14: Proportion reporting being ill in past 4 weeks/parents seeking care for illness past 2 weeks – RCTs

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 15: Overweight (BMI z‐score > 2 SD)_PCS

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Analysis 2.15

Comparison 2: Conditional cash transfers (CCT) versus no intervention, Outcome 15: Overweight (BMI z‐score > 2 SD)_PCS

Comparison 3: Income generation (IG) versus no intervention, Outcome 1: HFIAS – PCS

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Analysis 3.1

Comparison 3: Income generation (IG) versus no intervention, Outcome 1: HFIAS – PCS

Comparison 3: Income generation (IG) versus no intervention, Outcome 2: HDDS – RCTs

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Analysis 3.2

Comparison 3: Income generation (IG) versus no intervention, Outcome 2: HDDS – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 3: Minimum dietary diversity (MDD) – RCTs

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Analysis 3.3

Comparison 3: Income generation (IG) versus no intervention, Outcome 3: Minimum dietary diversity (MDD) – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 4: HDDS – PCS

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Analysis 3.4

Comparison 3: Income generation (IG) versus no intervention, Outcome 4: HDDS – PCS

Comparison 3: Income generation (IG) versus no intervention, Outcome 5: Proportion stunted (HAZ < ‐2 SD) – RCTs

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Analysis 3.5

Comparison 3: Income generation (IG) versus no intervention, Outcome 5: Proportion stunted (HAZ < ‐2 SD) – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 6: HAZ – RCTs

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Analysis 3.6

Comparison 3: Income generation (IG) versus no intervention, Outcome 6: HAZ – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 7: Proportion wasted (WHZ < ‐2 SD) – RCTs

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Analysis 3.7

Comparison 3: Income generation (IG) versus no intervention, Outcome 7: Proportion wasted (WHZ < ‐2 SD) – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 8: WHZ – RCTs

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Analysis 3.8

Comparison 3: Income generation (IG) versus no intervention, Outcome 8: WHZ – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 9: Percentage underweight – RCTs

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Analysis 3.9

Comparison 3: Income generation (IG) versus no intervention, Outcome 9: Percentage underweight – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 10: WAZ – RCTs

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Analysis 3.10

Comparison 3: Income generation (IG) versus no intervention, Outcome 10: WAZ – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 11: Percentage underweight – PCS

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Analysis 3.11

Comparison 3: Income generation (IG) versus no intervention, Outcome 11: Percentage underweight – PCS

Comparison 3: Income generation (IG) versus no intervention, Outcome 12: Proportion of women underweight – RCTs

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Analysis 3.12

Comparison 3: Income generation (IG) versus no intervention, Outcome 12: Proportion of women underweight – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 13: BMI – RCTs

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Analysis 3.13

Comparison 3: Income generation (IG) versus no intervention, Outcome 13: BMI – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 14: Haemoglobin concentration (children) – RCTs

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Analysis 3.14

Comparison 3: Income generation (IG) versus no intervention, Outcome 14: Haemoglobin concentration (children) – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 15: Haemoglobin concentration (women) – RCTs

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Analysis 3.15

Comparison 3: Income generation (IG) versus no intervention, Outcome 15: Haemoglobin concentration (women) – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 16: Prevalence of anaemia (children) – RCTs

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Analysis 3.16

Comparison 3: Income generation (IG) versus no intervention, Outcome 16: Prevalence of anaemia (children) – RCTs

Comparison 3: Income generation (IG) versus no intervention, Outcome 17: Prevalence of anaemia (women) – RCTs

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Analysis 3.17

Comparison 3: Income generation (IG) versus no intervention, Outcome 17: Prevalence of anaemia (women) – RCTs

Comparison 4: Food vouchers vs no intervention, Outcome 1: Food consumption score

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Analysis 4.1

Comparison 4: Food vouchers vs no intervention, Outcome 1: Food consumption score

Summary of findings 1. Unconditional cash transfers compared to no intervention for food security

Unconditional cash transfers compared to no intervention for food security

Patient or population: children, adults, households
Setting: poor rural and urban households in LMICs
Intervention: UCTs
Comparison: no intervention

Outcomes

Impact

№ of participants
(studies)

Certainty of the evidence
(GRADE)

Comments

Prevalence of undernourishment

0 included studies measured this outcome.

Proportion of household expenditure on food
follow‐up: range 1–2 years

1 study showed a clear effect favouring UCTs, 2 studies showed unclear effect potentially favouring UCTs and 2 studies showed clear effect favouring the control. Data not pooled.

11271 households
(5 RCTs)

⊕⊝⊝⊝
Very lowa,b,c

Evidence is very uncertain about the effects of UCTs on the proportion of household expenditure on food.

Food security
assessed with: proportion of households consuming > 1 meal per day; modified HFIAS; FSI
follow‐up: range 1–2 years

6 studies showed a clear effect favouring UCTs.

A meta‐analysis of 3 of these studies showed a small improvement in food security scores (SMD 0.18, 95% CI 0.13 to 0.23; 6209 households)

10,251 households, 7604 children (6 RCTs)

⊕⊕⊕⊕
High

UCTs improve food security.

Dietary diversity
assessed with: dietary diversity scores (i.e. number of food groups consumed); proportion with minimum dietary diversity
follow‐up: range 1–2 years

5 studies showed a clear effect favouring UCTs and 5 studies show an unclear effect potentially favouring UCTs.

Data not pooled.

12,631 households, 890 children (10 RCTs)

⊕⊕⊝⊝
Lowa,b

UCTs may increase dietary diversity.

Stunting
assessed with: HAZ < –2SD
follow‐up: 2 years

1 study showed a clear effect favouring UCTs, 2 studies showed an unclear effect favouring UCTs and 1 study showed an unclear effect favouring the control.

A meta‐analysis of 2 of these studies showed a reduction in stunting with UCTs (OR 0.62, 95% CI 0.46 to 0.84; 2914 children)

4713 children
(4 RCTs)

⊕⊕⊝⊝
Lowa,b

UCTs may reduce stunting.

Wasting
assessed with: WHZ < –2SD
follow‐up: range 2 years

1 study showed an unclear effect potentially favouring UCTs and 3 studies showed an unclear effect potentially favouring the control. Data not pooled.

6396 children
(4 RCTs)

⊕⊝⊝⊝
Very lowa,b,c

We are uncertain whether UCTs reduce wasting.

Cognitive function and development
assessed with: cognitive test scores, language scores
follow‐up: 2 years

3 studies reported unclear effect potentially favouring intervention.

10,813 children

(3 RCTs)

⊕⊕⊕⊕
High

UCTs make little or no difference on cognitive function and development.

*No meta‐analyses carried out.

CI: confidence interval; FSI: Food Security Index; HAZ: height‐for‐age z‐score; HFIAS: Household Food Insecurity Access Scale; LMIC: low‐ and middle‐income country; OR: odds ratio; RCT: randomised controlled trial; SD: standard deviation; SMD: standardised mean difference; UCT: unconditional cash transfer; WHZ: weight‐for‐height z‐score.

GRADE Working Group grades of evidence
High certainty: we are very confident that the true effect lies close to that of the estimate of the effect.
Moderate certainty: we are moderately confident in the effect estimate: the true effect is likely to be close to the estimate of the effect, but there is a possibility that it is substantially different.
Low certainty: our confidence in the effect estimate is limited: the true effect may be substantially different from the estimate of the effect.
Very low certainty: we have very little confidence in the effect estimate: the true effect is likely to be substantially different from the estimate of effect.

aDowngraded one level for risk of bias: at least one study was at high overall risk of bias due to selection or attrition bias, or both.
bDowngraded one level for inconsistency: there was wide variance of point estimates.
cDowngraded one level for imprecision: wide confidence intervals.

Figures and Tables -
Summary of findings 1. Unconditional cash transfers compared to no intervention for food security
Summary of findings 2. Conditional cash transfers compared to no intervention for food security

Conditional cash transfers compared to no intervention for food security

Patient or population: children, adults, households
Setting: poor urban and rural communities in LMICs
Intervention: CCTs
Comparison: no intervention

Outcomes

Impact

№ of participants
(studies)

Certainty of the evidence
(GRADE)

Comments

Prevalence of undernourishment

0 included studies measured this outcome.

Proportion of household expenditure spent on food
follow‐up: 9 months to 2 years

1 study showed a clear effect potentially favouring the control and 1 study showed an unclear effect favouring the control. Data not pooled.

4760 households
(2 RCTs)

⊕⊕⊕⊕
High

CCTs result in little to no difference in the proportion of household expenditure on food.

Food security

0 included studies measured this outcome.

Dietary diversity
assessed with: Food Consumption Score
follow‐up: 7 months to 2.5 years

Meta‐analysis of 2 studies showed a clear effect favouring CCTs (MD 0.45, 95% CI 0.25 to 0.65)

3937 households
(2 RCTs)

⊕⊕⊕⊝
Moderatea

CCTs probably slightly improve dietary diversity

Stunting
assessed with: HAZ < –2SD
follow‐up: range 20 months to 3 years

3 studies showed an unclear effect potentially favouring CCTs and 1 study showed an unclear effect potentially favouring the control.

A meta‐analysis of 3 of these studies showed an unclear effect favouring CCTs (MD –2.51, 95% CI –7.78, 2.75)

3529 children
(4 RCTs)

⊕⊕⊝⊝
Lowa,b

CCTs may make little or no difference to the proportion of stunted children.

Wasting
assessed with: WHZ < –2SD
follow‐up: 2 years

A meta‐analysis of 2 studies showed an unclear effect favouring CCTs (MD –2.50 95% CI –8.04 to 3.04)

2116 children
(2 RCTs)

⊕⊕⊝⊝
Lowb,c

CCTs may make little or no difference in wasting.

Cognitive function and development
assessed with: cognitive test scores; cognitive and socioemotional outcomes scores
follow‐up: range 9 months to 2 years

A meta‐analysis of 2 studies showed a slight improvement with CCTs (SMD 0.13, 95% CI 0.09 to 0.18)

5383 children
(2 RCTs)

⊕⊕⊕⊕
High

CCTs slightly improve cognitive function in children.

*No meta‐analyses carried out.

CCT: conditional cash transfer; CI: confidence interval; HAZ: height‐for‐age z‐score; MD: mean difference; RCT: randomised controlled trial; SD: standard deviation; SMD: standardised mean difference; WHZ: weight‐for‐height z‐score.

GRADE Working Group grades of evidence
High certainty: we are very confident that the true effect lies close to that of the estimate of the effect.
Moderate certainty: we are moderately confident in the effect estimate: the true effect is likely to be close to the estimate of the effect, but there is a possibility that it is substantially different.
Low certainty: our confidence in the effect estimate is limited: the true effect may be substantially different from the estimate of the effect.
Very low certainty: we have very little confidence in the effect estimate: the true effect is likely to be substantially different from the estimate of effect.

aDowngraded one level for risk of bias: at least one study was at high overall risk of bias due to selection or attrition bias, or both.
bDowngraded one level imprecision: wide confidence intervals.
cDowngraded one level for inconsistency: wide variation in point estimates.

Figures and Tables -
Summary of findings 2. Conditional cash transfers compared to no intervention for food security
Summary of findings 3. Income‐generation interventions compared to no intervention for food security

Income‐generation interventions compared to no intervention for food security

Patient or population: children, adults, households
Setting: poor rural communities in LMICs
Intervention: income‐generation interventions (e.g. livestock transfers, community development programmes)
Comparison: no intervention

Outcomes

Impact

№ of participants
(studies)

Certainty of the evidence
(GRADE)

Comments

Prevalence of undernourishment

0 included studies reported this outcome.

Proportion of household expenditure on food
follow‐up: range 1–2 years

2 studies reported this outcome but did not provide relevant numerical data or indicated clearly the direction of effect.

434 households (2 prospective controlled studies)

Food security
assessed with: proportion experiencing food security; Household food security score
follow‐up: 3–4 months

1 trial reported no effect measure and 1 trial showed an unclear effect potentially favouring the control.

2193 households (1 trial)

⊕⊕⊝⊝
Lowa,b

Income‐generation interventions may result in little to no difference in food security.

Dietary diversity
assessed with: DDS, HDDS, MDD
follow‐up: 2 years

2 trials showed a clear effect favouring income‐generation interventions, 1 trial showed an unclear effect favouring the intervention and 1 trial showed an unclear effect favouring control.

A meta‐analysis of 3 of these studies showed that the intervention improved the proportion of children achieving MDD (OR 1.28, 95% CI 1.11 to 1.47)

3677 households and 3790 children (4 RCTs)

⊕⊕⊝⊝
Lowa,c

Income‐generation interventions may improve dietary diversity in children and may result in little or no difference to household dietary diversity.

Stunting
assessed with: HAZ
follow‐up: 12 months

Meta‐analysis of 2 studies showed no difference to stunting (OR 1.00, 95% CI 0.84 to 1.19)

3466 children (2 RCTs)

⊕⊕⊕⊝
Moderated

Income‐generation interventions probably make little or no difference to stunting.

Wasting
assessed with: WHZ
follow‐up: 2 years

Meta‐analysis of 2 studies showed unclear effect favouring the intervention (OR 1.13, 95% CI 0.92 to 1.40)

3500 children (2 trials)

⊕⊕⊕⊝
Moderated

Income‐generation interventions probably make little or no difference to wasting.

Cognitive function and development

0 included studies reported this outcome.

CI: confidence interval; DDS: Dietary Diversity Score; HAZ: height‐for‐age z‐score; HDDS: Household Dietary Diversity Score; MDD: minimum dietary diversity; OR: odds ratio; RCT: randomised controlled trial; WHZ: weight‐for‐height z‐score.

GRADE Working Group grades of evidence
High certainty: we are very confident that the true effect lies close to that of the estimate of the effect.
Moderate certainty: we are moderately confident in the effect estimate: the true effect is likely to be close to the estimate of the effect, but there is a possibility that it is substantially different.
Low certainty: our confidence in the effect estimate is limited: the true effect may be substantially different from the estimate of the effect.
Very low certainty: we have very little confidence in the effect estimate: the true effect is likely to be substantially different from the estimate of effect.

aDowngraded one level for risk of bias: at least one study was at high overall risk of bias due to selection or attrition bias, or both.
bDowngraded one level for indirectness: results are from a single study which assessed a public works programme and the effects may be different from other types of income generation interventions. Additionally public works programmes are often implemented in different ways in different settings.
cDowngraded one level for inconsistency: wide variation in point estimates.
dDowngraded one level for imprecision: wide confidence intervals.

Figures and Tables -
Summary of findings 3. Income‐generation interventions compared to no intervention for food security
Summary of findings 4. Food vouchers compared to no intervention for food security

Food vouchers compared to no intervention for food security

Patient or population: poor households
Setting: urban and agrarian communities in LMICs
Intervention: food vouchers
Comparison: no intervention

Outcomes

Impact

№ of participants
(studies)

Certainty of the evidence
(GRADE)

Comments

Prevalence of undernourishment

0 included studies reported this outcome.

Proportion of household expenditure on food

0 included studies reported this outcome.

Food security

0 included studies reported this outcome.

Dietary diversity
assessed with: FCS
follow‐up: 7 months to 1 year

2 studies reported improved dietary diversity (not pooled).

2459 households (2 RCT)

⊕⊕⊝⊝
Lowa,b

Food vouchers may improved dietary diversity slightly.

Stunting (HAZ < –2SD)

follow‐up: 12 months

1 study reported reduced stunting (OR 0.48, 95% CI 0.31 to 0.73)

1633 children (1 RCT)

⊕⊕⊕⊝

Moderatec

Food vouchers probably reduce stunting.

Wasting (WHZ < –2SD)

follow‐up: 12 months

1 study reports an unclear effect potentially favouring the control (OR 1.17, 95% CI 0.75, 1.82)

1633 children (1 RCT)

⊕⊕⊝⊝

Lowc,d

Food vouchers may result in little to no difference in wasting

Cognitive function and development

0 included studies reported this outcome.

CI: confidence interval; FCS: Food Consumption Score; HAZ: height‐for‐age z‐score; OR: odds ratio; RCT: randomised controlled trial; SD: standard deviation; WHZ: weight‐for‐height z‐score.

GRADE Working Group grades of evidence
High certainty: we are very confident that the true effect lies close to that of the estimate of the effect.
Moderate certainty: we are moderately confident in the effect estimate: the true effect is likely to be close to the estimate of the effect, but there is a possibility that it is substantially different.
Low certainty: our confidence in the effect estimate is limited: the true effect may be substantially different from the estimate of the effect.
Very low certainty: we have very little confidence in the effect estimate: the true effect is likely to be substantially different from the estimate of effect.

aDowngraded one level for overall risk of bias: two studies at high risk of selection and attrition bias.
bDowngraded one level for inconsistency: confidence intervals had minimal overlap.
cDowngraded one level for indirectness: findings are from one single study that assessed a programme of fresh food vouchers redeemed at designated vendors. Food vouchers may be implemented in different ways across different settings, e.g. for staple foods alone, or with, no vendor‐ restrictions.
dDowngraded one level for imprecision: findings ranged from an important harm to important benefit.

Figures and Tables -
Summary of findings 4. Food vouchers compared to no intervention for food security
Summary of findings 5. Food and nutrition subsidies compared to no intervention for food security

Food and nutrition subsidies compared to no intervention for food security

Patient or population: primary schools and households and members of healthcare plan
Setting: urban and rural settings in LMICs
Intervention: food and nutrition subsidies
Comparison: no intervention

Outcomes

Impact

№ of participants
(studies)

Certainty of the evidence
(GRADE)

Comments

Prevalence of undernourishment

0 included studies reported this outcome.

Proportion of household expenditure on food
assessed with: ratio of healthy to total food expenditure
follow‐up: 28 months

1 study reported that food rebates of 10% improved the ratio of healthy, to total food expenditure

169,485 households (1 prospective controlled study)

⊕⊝⊝⊝
Very lowa,b

The evidence is very uncertain about the effects of food rebates on household expenditure on healthy foods.

Food security

0 included studies reported this outcome.

Dietary diversity

1 study reported a clear effect favouring nutrition subsidies.

656 children (1 RCT)

⊕⊕⊝⊝

Lowc ,d

Nutrition subsidies may improve dietary diversity among school children

Stunting

0 included studies reported this outcome.

Wasting

0 included studies reported this outcome.

Cognitive function and development

0 included studies reported this outcome.

LMIC: low‐ and middle‐income country; RCT: randomised controlled trial.

GRADE Working Group grades of evidence
High certainty: we are very confident that the true effect lies close to that of the estimate of the effect.
Moderate certainty: we are moderately confident in the effect estimate: the true effect is likely to be close to the estimate of the effect, but there is a possibility that it is substantially different.
Low certainty: our confidence in the effect estimate is limited: the true effect may be substantially different from the estimate of the effect.
Very low certainty: we have very little confidence in the effect estimate: the true effect is likely to be substantially different from the estimate of effect.

aDowngraded one level for risk of bias: high risk of selection bias due to disparate baseline expenditure on healthy food as a ratio of total expenditure between households in the intervention and control group.
bDowngraded one level for indirectness: results are from a single study that assessed food rebates at a supermarket in South Africa. The population was restricted to members of the health insurance company's program, who are usually healthier and wealthier in general. Effects in other populations may differ.

cDowngraded one level for indirectness: results are from a single study that assessed the effects of providing nutrition subsidies to schools. Subsidies to individuals or households may have different effects.
dDowngraded one level for risk of bias: study was at high overall risk of bias due to attrition bias.

Figures and Tables -
Summary of findings 5. Food and nutrition subsidies compared to no intervention for food security
Summary of findings 6. Social support compared to no intervention for food security

Social support compared to no intervention for food security

Patient or population: households at risk of food insecurity
Setting: poor communities in LMICs
Intervention: village savings and loans groups and community cash transfers
Comparison: no intervention

Outcomes

Impact

№ of participants
(studies)

Certainty of the evidence
(GRADE)

Comments

Prevalence of undernourishment

0 included studies reported this outcome.

Proportion of household expenditure on food

0 included studies reported this outcome.

Food security
assessed with: self‐reported months of food sufficiency
follow‐up: 3 years

1 study reported an unclear effect favouring village savings and loans

1687 households (1 prospective controlled study)

⊕⊝⊝⊝
Very lowa

The evidence is very uncertain about the effects of village savings and loan on food security.

Dietary diversity
assessed with: HDDS
follow‐up: 3 years

1 study showed an unclear effect favouring the control.

1615 households (1 prospective controlled study)

⊕⊝⊝⊝
Very lowa

The evidence is very uncertain about the effects of village savings and loan on dietary diversity.

Stunting

assessed with: HAZ < –2SD

follow‐up: 2 years

1 study showed an unclear effect favouring the control.

1481 children (1 RCT)

⊕⊕⊝⊝

Lowb ,c

Community grants may make little or no difference to stunting.

Wasting

assessed with: WHZ < –2SD

follow‐up: 2 years

1 study showed an unclear effect favouring a community grant programme.

1481 children (1 RCT)

⊕⊕⊕⊝

Moderateb

Community grants probably make little or no difference to wasting.

Cognitive function and development

0 included studies reported this outcome.

*No meta‐analyses carried out.
CI: confidence interval; HAZ: height‐for‐age z‐score; HDDS: Household Dietary Diversity Score; LMIC: low‐ and middle‐income country; RCT: randomised controlled trial; SD: standard deviation; WHZ: weight‐for‐height z‐score.

GRADE Working Group grades of evidence
High certainty: we are very confident that the true effect lies close to that of the estimate of the effect.
Moderate certainty: we are moderately confident in the effect estimate: the true effect is likely to be close to the estimate of the effect, but there is a possibility that it is substantially different.
Low certainty: our confidence in the effect estimate is limited: the true effect may be substantially different from the estimate of the effect.
Very low certainty: we have very little confidence in the effect estimate: the true effect is likely to be substantially different from the estimate of effect.

aDowngraded one level for indirectness: results from a single study which assessed the effects of microfinance program to villages in Mozambique. Effects of other types of social support interventions may be different. As this was a prospective controlled study the certainty of evidence started at low.
bDowngraded one level for indirectness: results are from a single study which assessed the effects of a community cash transfer programme implemented in rural villages in Indonesia. Village management teams allocated funds to different types of social support interventions, Effects in urban populations and with different intervention implementation may differ.

cDowngraded one level for imprecision: wide confidence interval.

Figures and Tables -
Summary of findings 6. Social support compared to no intervention for food security
Table 1. Summary of PICOS and of AMSTAR scores of included systematic reviews, and how existing reviews informed the PICOS of a new Cochrane Review

Domain

Finding

How it informed our review question or methods

Setting

  • 12 reviews did not specify the setting

  • 11 reviews stated the community as the setting

  • 3 reviews stated the setting was LMICs

  • 3 reviews specified a school as the setting

We chose the community as the setting, defined as a group of people with diverse characteristics who were linked by social ties, share common perspectives and engage in joint action in geographical locations or settings (MacQueen 2001).

Participants

  • 5 reviews did not specify the types of participants for inclusion

  • 11 reviews included infants and children (up to school‐aged children)

  • 1 review included adults and adolescents

  • 6 reviews included pregnant women or mothers in the immediate postpartum period. 1 of these also targeted other adults who could be linked to women who may have breastfed. Many of these were assessing interventions on breastfeeding or complementary feeding.

  • 1 review included only parents of children aged 2–5 years, as it assessed influence of parenting practices on children's dietary habits

  • 2 reviews included all people living in a community

  • 3 reviews included only poor people who were recipients of some service, e.g. recipients of a government conditional cash‐transfer programme

As existing reviews specifically addressed specific high‐risk groups, we did not focus on these. Instead, we included all individuals across all ages that belonged to the community where relevant interventions had been implemented.

Intervention (including its duration)

  • 14 reviews addressed interventions related to the availability of food, 5 of which also assessed interventions influencing utilisation of food, such as nutrition education

  • 13 reviews assessed interventions addressing food utilisation

  • 7 reviews assessed interventions addressing access to food (2 of which had a low AMSTAR score of 4)

  • 28 reviews did not specify the duration of the intervention, and only 1 included interventions with a minimum duration of 3 months. As a result, the duration and the follow‐up times of the interventions varied considerably within and across reviews

Of the 14 reviews that addressed food availability, 5 also assessed food utilisation (e.g. combination of community gardens and nutrition education). As fewer reviews addressed food access, we included interventions that had addressed this dimension of food security.

We included interventions with any duration but extracted outcomes that were measured ≥ 3 months after implementation.

Control

  • 18 reviews did not specify a control group

  • 6 reviews compared the intervention with either no intervention, an alternative intervention or placebo

  • 3 reviews did not have any control group

  • 2 reviews stated that included studies needed to have a control group, but did not specify further

We included studies in which these interventions, individually or in combination, were compared to no intervention or to other eligible intervention.

Outcomes assessed

The specific outcomes assessed across the included reviews varied considerably and often they were not clearly specified at the outset.

The most common and important outcomes reported in these reviews were related to dietary intake, anthropometric measurements, and biochemical and clinical indicators, to describe the impact of the intervention on nutritional status. Other outcomes measured included food purchase or expenditure, food production, morbidity and mortality, and breastfeeding initiation rates or duration.

Often, reviews measured the same outcome in different ways. For example, anthropometric indicators assessed differed, as did their classifications, across the included reviews. This makes it difficult to compare results across reviews and to reach a conclusion about the effectiveness of a specific intervention.

The most commonly specified outcomes measured food and nutrition security, and nutritional status. We also focused on these outcomes. Examples included: diet diversity scores and hunger measures; and anthropometric, biochemical and dietary intake indicators. We clearly defined, a priori, the specific outcome measures and metrics that we included in our review.

Study designs

  • 11 reviews did not specify which study designs they would include

  • 3 reviews included only RCTs

  • 1 review included only CCTs

  • 1 review included only impact evaluations

  • 13 reviews included a variety of study designs, which included ≥ 2 of: RCTs, BAS, quasi‐RCTs, analytical cohort studies, ITS, CCTs, randomised field trials and CSS

However, the definitions of the study design labels used were not always clear and varied across the included reviews.

The study design labels used varied across included reviews and were not always clearly defined.

We included both randomised and non‐randomised studies, as we expect that existing RCTs in the area of food security would be scarce. We wanted to include the best available evidence for our review question. We clearly defined the type of study designs included in our review.

Search strategies

Most reviews ran comprehensive searches. They used a comprehensive set of keywords and searched a variety of relevant databases. Only 5 reviews did not indicate search terms either in the text or in an appendix.

  • 2 reviews conducted searches until 2012

  • 11 reviews searched until 2010–2011

  • 9 reviews searched before 2010

  • 7 reviews did not specify the date of the last search

Our review included updated searches across a variety of relevant databases and websites. We drew on common keywords used across these included reviews.

Reporting

The methods sections of most reviews were often not reported clearly. The reporting of results in these reviews, in terms of characteristics of included studies, was also poor.

Poor reporting of the characteristics of included studies makes it difficult to assess the context in which these results were obtained. Thus, it is difficult to generalise the results.

We clearly reported on the characteristics of included studies, so that the context in which the interventions were implemented was clearly understood.

AMSTAR scores

  • 9 reviews were of low quality (AMSTAR score: 0–4)

  • 11 reviews were of moderate quality (AMSTAR score: 5–8)

  • 8 reviews were of high quality (AMSTAR score: 9–11)

  • 1 review did not have a score as it did not include any studies

Of the 8 high‐quality reviews, 5 assessed interventions that aimed to improve food availability or utilisation (or both), and 3 assessed interventions addressing food access. The other 2 included reviews that addressed food access were of low quality (AMSTAR = 4).

We contributed to the evidence base on interventions addressing food access by producing a high‐quality systematic review that assessed the effectiveness of the interventions on relevant outcomes, such as nutritional status.

BAS: before‐and‐after study; CCT: controlled clinical trial; CSS: cross‐sectional study; ITS: interrupted time series; LMIC: low‐ and middle‐income country; RCT: randomised controlled trial.

Figures and Tables -
Table 1. Summary of PICOS and of AMSTAR scores of included systematic reviews, and how existing reviews informed the PICOS of a new Cochrane Review
Table 2. Definition of interventions included in the review

Category of intervention

Definition

Types of interventions

Improve buying power

Interventions that generate/increase/maintain income to ensure economic access to food and other basic needs.

  • Cash transfers (conditional or unconditional)

  • Other income generation interventions, e.g.

    • Cash‐for‐work programmes

    • Microcredit/microenterprise development – facilitation of small business development through credit‐provision and training in specific business skills

    • Employment generating activities, that will generate/increase income

    • Agriculture‐related interventions – training /cash cropping/livestock ownership/other. These interventions are only included if they aim to increase income of households. Agricultural interventions only aiming to increase/ensure enough food for consumption are excluded.

Food prices

Interventions that reduce price of food and thus increase economic access to food.

  • Food stamps or vouchers (distribution of coupons or stamps that can be used to purchase foods in local markets, etc.)

  • Food subsidies/discounts

  • Policies/regulations that reduce/regulate food prices

Infrastructure/transport

Interventions that ensure people/communities have physical access to food/food outlets.

  • Rural infrastructure development; e.g. roads that enable access to shops/ markets

  • Interventions that ensure affordable transportation to markets/food outlets

  • Adequate food storage facilities

Social environment/support

Interventions that ensure people have social support/support network they can resort to for money/food in times of need, or access to adequate storage facilities (e.g. shared fridge) or services (e.g. transport/childcare) – leading to increased economic or physical access to food

Social support can be instrumental, emotional, informational, or companionship. We were interested in instrumental social support, i.e. practical help that can be accessed in times of need.

  • Childcare so parents can go to work

  • Borrowing money/food from neighbours/relatives

  • Community fund/village savings loans

  • Shared fridge/storage facilities

  • Shared transport

Figures and Tables -
Table 2. Definition of interventions included in the review
Table 3. Summary of included studies

Intervention category

Intervention type

Studies and study designs

Improve buying power

Unconditional cash transfers

18 RCTs: Ahmed 2019a; Ahmed 2019b; Asfaw 2014; Baird 2013a; Brugh 2018; Daidone 2014; Fenn 2015; Fernald 2011; Gangopadhyay 2015; Haushofer 2013; Hjelm 2017; Hoddinott 2013; Merttens 2013; Miller 2011; Pellerano 2014; Schwab 2013; Skoufias 2013; Tonguet Papucci 2015

3 prospective controlled studies: Aguero 2006; Breisinger 2018; Renzaho 2017

Conditional cash transfers

9 RCTs: Baird 2013a; Evans 2014; Gertler 2000 (PROGRESA); Hidrobo 2014c; Kandpal 2016; Kurdi 2019; Kusuma 2017a; Macours 2012; Maluccio 2005

5 prospective controlled studies: Andersen 2015; Ferre 2014; Huerta 2006 (PROGRESA); Leroy 2008 (PROGRESA); Lopez Arana 2016

Income generationd

6 RCTs: Beegle 2017; Darrouzet Nardi 2016; Marquis 2018; Olney 2016; Osei 2017; Verbowski 2018

11 prospective controlled studies: Alaofe 2016; Alaofe 2019; Asadullah 2015; Doocy 2017; Jodlowski 2016; Kangmennaang 2017; Katz 2001; Kennedy 1989; Murshed E Jahan 2011; Porter 2016e; Weinhardt 2017

Food prices

Food vouchers

4 RCTs: Fenn 2015b; Hidrobo 2014c; Jensen 2011; Ponce 2017

0 prospective controlled studies

Food rebates/subsidies

1 RCT: Chen 2019

3 prospective controlled study: Andaleeb 2016; Chakrabarti 2018; Sturm 2013

Infrastructure changes

0 identified

Social environment

Village savings and loans

1 RCT: Kusuma 2017b

1 prospective controlled study: Brunie 2014

aBaird 2013 assesses both conditional and unconditional cash transfers.
bFenn 2015 assesses both unconditional cash transfers and food vouchers.
cHidrobo 2014 assesses both conditional cash transfers and food vouchers.
dThis includes different interventions that aimed to generate income of participants (e.g. integrated agricultural programmes, community development programmes).
ePorter 2016 assessed a public works (80%) (cash/food‐for‐work) or unconditional cash transfer government programme (20%). Results were reported for the entire population, not disaggregated according to intervention received.

RCT: randomised controlled trial.

Figures and Tables -
Table 3. Summary of included studies
Table 4. Description of included studies assessing the effects of Mexico's PROGRESA/Oportunidades conditional cash transfer programme

Study ID

Linked references

Study design and duration

Description of intervention

Sampling

Outcomes reported

Gertler 2000 (PROGRESA)

Gertler 2004; Hoddinott 2000; Hoddinott 2003a; Hoddinott 2004(?); Skoufias 2001; Skoufias 2007; Fernald 2008; Fernald 2009

Cluster‐RCT conducted between 1998 and 2000, where communities were randomly allocated to either receive the intervention immediately (intervention group) or to receive the intervention 2 years later (control group). In reality, control communities started receiving the intervention in late 1999, about 1.5 years after the intervention communities.

Timepoints of data collection (through household surveys – ENCEL):

  • March 1998 (pre‐intervention)

  • October/November 1998

  • May/June 1999

  • October/November 1999

  • September/December 2003 (follow‐up)

  • September/December 2007 (follow‐up)

'Oportunidades' (previously called Progresa) is a conditional cash transfer programme implemented by the Mexican government since April 1998.

Women in eligible households receive cash transfers every 2 months (a food and an education transfer) if they adhered to specific conditionalities: all family members attend preventive health services regularly; children aged 0–5 years and lactating mothers attended nutrition monitoring clinics for growth monitoring, immunisation, to obtain nutrition supplements, and for nutrition and hygiene education; pregnant women attend antenatal care, receive nutritional supplements and health education.

The education transfers included scholarships for school attendance and school supplies, and was dependent on children's school attendance.

The value of the transfers was about 20–30% to the household consumption expenditure preintervention.

506/50,000 eligible rural villages were randomly selected based on the index level of community poverty. Of these, 320 communities were allocated to the intervention group and 186 to the control group. Within each community, households were selected by proxy means testing and selection validated in a community assembly.

Some studies assessed outcomes in a subsample of the study population.

Fernald 2008 followed up on a sample of children in 2003: children aged 24–72 months in the 'Early intervention' group (from 144 communities), and children aged 2–5 years in the 'Late intervention' group (from 108 communities).

Fernald 2009, followed up a sample of children in 2007: 1093 children aged 8–10 years in the 'Early intervention' group, and 700 children aged 9–10 years in the 'Late intervention' group.

  • Household food consumption (Hoddinott 2000)

  • Dietary diversity (Hoddinott 2000)

  • Total caloric availability (Hoddinott 2000; 2003a)

  • Morbidity (children aged 0–5 years) (Gertler 2004)

  • Fernald 2008 and Fernald 2009 only assessed data that included the period when both the control and intervention groups were receiving the intervention (i.e. early vs late intervention). These data were not extracted for the review but were mentioned in the Discussion.

Huerta 2006 (PROGRESA)

Rivera 2004; Gertler 2004; Behrman 2001

Nested cohort study conducted on a subset of the larger cRCT sample (described above), including a random selection of 205 of original intervention communities and 142 of original 186 control communities. Additional household surveys conducted on health and nutrition indicators.

Time points of data collection:

  • August/September 1998 (i.e. no true baseline data available as by this time all intervention households were already receiving transfers);

  • September/December 1999;

  • November/December 2000 (both groups exposed to the programme for approximately 1 year)

As above

Subsample of children selected.

Behrman 2005 (?)

Rivera 2004: children aged < 12 months (461 children from 175 communities in the intervention and 334 children from 107 communities in the control).

Gertler 2004 and Huerta 2006: sample sizes not reported

  • Height (Behrman 2005; Gertler 2004)

  • Stunting (Gertler 2004)

  • Anaemia (Gertler 2004)

No outcome data reported for exposed vs non‐exposed groups after 1 year of follow‐up (Rivera 2004; Huerta 2006) (?).

Leroy 2008 (PROGRESA)

N/A

CBA: urban communities randomly selected for expansion of Oportunidades into 149 urban areas. The control group comprised eligible households that did not enrol in the programme.

Time points of data collection through household surveys:

  • September/December 2002 (preintervention)

  • July/November 2004

As above

Children aged < 24 months in 2002: 574 in intervention and 159 in control

  • Height

  • HAZ

  • Weight

  • WHZ

CBA: controlled before‐after study; cRCT: cluster randomised controlled trial; HAZ: height‐for‐age z‐score; N/A: not applicable/available; RCT: randomised controlled trial; WHZ: weight‐for‐height z‐score.

Figures and Tables -
Table 4. Description of included studies assessing the effects of Mexico's PROGRESA/Oportunidades conditional cash transfer programme
Table 5. Income‐generation interventions – overview of included studies

Study (country of conduct)

Study design

Overall risk of biasa

Other key details of intervention

Population (sample size at baseline: Intervention/ Control)

Outcome domains and measures with available data

Time point of measurement

Darrouzet Nardi 2016 (Nepal)

cRCT

Unclear

Programme name: Heifer training curriculum

Programme description and frequency: participation in programme that focused on training regarding poverty alleviation, citizen empowerment, community development and optimisation of livestock management as means to generate income.

Provider: NGO (Heifer International)

Delivery: women's self‐help groups which met with a trained facilitator, supplemented by specific interactive instruction, workshops, guidance, and training. Biweekly meetings

Co‐interventions: none reported

Rural farming communities; HHs: 201/214; children (aged 6–60 months): 283/324

Dietary diversity:

  • Household dietary diversity index

  • Child minimum dietary diversity

Anthropometry

  • HAZ;

  • WAZ

1 and 2 years

Doocy 2017 (Democratic Republic of the Congo)

Prospective controlled study

High

Programme name: Intervention implemented as part of the Jenga Jamaa II project

Programme description and frequency: WEGs met weekly and meetings served as a delivery mechanism for a variety of interventions including literacy and numeracy, business and marketing training, and income‐generation activities. Savings and credit groups were started in each WEG. Beneficiaries were provided with a starter kit of basic materials for their income‐generation activity. Many WEG participants also received goats and energy‐efficient stoves. The FFS intervention provided farmers with experience‐based education on farming practices and postharvest handling as well as business and natural resource management skills. Each FFS group received semi‐monthly training sessions for 2 years. Each FFS group had a community demonstration plot, and group members also received starter packages of seeds and tools for use on individual farms. The FFS programmes focused on a variety of common crops in the region. The first year of training focused on knowledge of production systems and technologies; adoption of techniques and technologies and behaviour change were the focus in the second year

Provider: ADRA

Delivery: FFS – training sessions on agriculture techniques and other content by ADRA field agents.

Co‐interventions: after they finished the FFS intervention (2 years) some transitioned to farmer business associations, which were intended to improve access to credit and marketing opportunities.

Farming villages; HHs (WEG: 390/324; FFS: 338/324)

Food security:

  • HFIAS

  • Proportion of HHs improving a HFIAS category

Dietary diversity:

  • HDDS

  • Achieving target dietary diversity (based on HDDS)

3.5 years

Weinhardt 2017 (Malawi)

Prospective controlled study (non‐equivalent control group)

Unclear

Programme name: support to able‐bodied vulnerable groups to achieve food security (SAFE) programme

Programme description and frequency: programme comprised 4 components

  • Improving farming practices and sustainable agriculture through Farmer Field Schools

  • Increasing access to savings and investment through Village Savings and Loans Groups

  • Building capacity of local governance structures

  • Integrating HIV education and gender empowerment into programmes through training and education

Provider: NGO (CARE Malawi)

Delivery: community‐based programme

Co‐interventions: agricultural education programme for a few intervention and control participants

Rural HHs (598/301)

Food security:

  • Mean number of months with less food than necessary to meet needs

Anthropometry:

  • WAZ

  • HAZ

  • Moderate and severe underweight (< –2SD WAZ)

  • Child BMI

18 and 36 months

Jodlowski 2016 (Zambia)

Prospective controlled study

Low

Programme name: Copperbelt Rural Livelihoods Enhancement Support Project (CRLESP)

Programme description and frequency: ongoing training and one‐off transfer of livestock contingent on training participation. 1 female livestock offspring per transferred female had to be donated to a Pass‐on‐the‐Gift HH.

Provider: NGO (Heifer International)

Delivery: NR

Co‐interventions: none reported

Rural households (105/178)

Dietary diversity:

  • Household Dietary Diversity Index

  • Probability weighted dietary diversity score

6, 12 and 18 months

Asadullah 2015 (Bangladesh)

Prospective controlled study

High

Programme name: challenging the frontiers of poverty reduction – targeting the ultra‐poor (CFPR‐TUP)

Programme description and frequency: multicomponent intervention including orientation training, selection of income‐generation microenterprise by female participants with one‐off transfer of productive assets worth BDT 10,000 to support it (90% of households chose livestock combination), community savings, monthly health worker visits, weekly follow‐up for technical advice, building social capital (village support networks and sponsorship of community leaders), and weekly stipends (BDT 70).

Provider: NGO (Bangladesh Rural Advancement Committee (BRAC))

Delivery: NGO staff deliver training and assets

Co‐interventions: none reported

Ultra‐poor households (2633/2993)

Food security

  • Proportion experiencing food deficit always

Morbidity:

  • Perceived health status

  • Perceived health improvement

3, 6 and 9 years

Marquis 2018 (Ghana)

cRCT

Low

Programme name: Nutrition Links (NL)

Programme description and frequency: 12‐month intervention was an integrated package of agricultural inputs and training as well as education in nutrition, health care and child stimulation for participants. The intervention had 4 main components

  • Poultry for egg production

  • Home gardens

  • Weekly group education sessions throughout the year

  • Community‐wide education

Provider: "Heifer's Passing on the Gift (POG) community development programme, project staff, district agricultural extension officers, district government staff, University of Ghana's Nutrition Research and Training Centre

Delivery:

  • 4‐day training received chickens and initial feed for 1 month and vaccinations, and weekly technical assistance by the project staff

  • Training, received planting materials, and weekly technical assistance

  • Weekly group education sessions

  • Training that was accessible to all residents

Co‐interventions: none reported

Mother–infant pairs in rural communities (287/213).

Dietary diversity

  • Minimal diet diversity

Anthropometry:

  • WAZ;

  • LAZ/HAZ;

  • WLZ/WHZ

1 year

Olney 2016 (Burkina Faso)

cRCT

Unclear

Programme name: enhanced‐homestead food production (EHFP)

Programme description and frequency: integrated agriculture and nutrition programme. Agriculture interventions included provision of land with inputs (crops, animals and implements) and training. Nutrition intervention included behaviour change communication strategy for health and nutrition behaviours, delivered through visits by community volunteers twice per month.

Provider: NGO (Helen Keller International – HKI)

Delivery: agriculture interventions rolled out first to female village farm leaders, who then trained other mothers. Nutrition education carried out by older women leaders or health committee members.

Co‐interventions: none reported

Villages with agricultural homesteads (30/25). HHs: 514 (health committee); 512 (older women leaders); 741 (control)

Dietary diversity:

  • Household Dietary Diversity Index

  • Proportion of mothers consuming individual food groups in past 7 days

Anthropometry:

  • BMI (adult)

  • Underweight (adults) (BMI < 18.5 kg/m2)

2 years

Osei 2017 (Nepal)

cRCT

Unclear

Programme name: Enhanced Homestead Food Production (EHFP) programme

Programme description and frequency: training in improved gardening and poultry‐rearing practices; hosting of a village model farm, which served as a site for purchasing inputs and ongoing training for all the beneficiary women. For every season (rainy and winter) of the first year, each woman was given a one‐off free supply of seeds, saplings and locally bred chicks to establish their home gardens and poultry production. Throughout the period of the intervention, the women met monthly at the farm to refresh lessons on agriculture techniques and nutrition through social and behaviour change communications. During monthly home visits, the project staff and the female community health volunteers also reinforced the educational messages on breastfeeding and complementary feeding to all mothers.

Provider: NGO (Helen Keller International – HKI)

Delivery: 1 woman per group of intervention villages (5 or 6) was selected and trained by HKI and this woman then trained 20 other beneficiary women; meetings at farm; home visits by trained project staff, female community health volunteers and agriculture extension officers.

Co‐interventions: none reported.

Homesteads: mothers (1055/1051), children (1055/1051)

Food security

  • Prevalence of HH food insecurity

Anthropometry:

  • HAZ

  • Stunting (HAZ < –2SD)WAZ

  • Underweight (child) (WAZ < –2SD) and mother (BMI < 18.5 kg/m2)

  • WHZ

  • Wasting (WHZ < –2SD)

  • BMI (mother)

Biochemical indicators:

  • Mean haemoglobin concentration (child and mother)

Morbidity:

  • Prevalence of anaemia (child and mother)

2.5 years

Verbowski 2018 (Cambodia)

cRCT

Unclear

Programme name: Fish on Farms (FoF) project using the Enhanced Homestead Food Production (EHFP) programme

Programme description and frequency: basic agricultural inputs and training, and nutrition and hygiene education. The education focused on optimal nutrition for women and infants and young child practices, and the use of nutrient‐dense produce grown by farmers were demonstrated. The purpose of EHFP was to increase production and intakes of various types of vegetables, herbs and tree fruit. The aquaculture intervention was designed to increase the production of 3 types of small fish, which typically were consumed whole, as well as 3 types of large fish (typically sold for income or fillets consumed).

Provider: NGO (Helen Keller International – HKI, local)

Delivery: trained village health volunteers provided education sessions, through small group and 1‐to‐1 counselling. Cooking demonstrations were also conducted. Support was provided through village model farms (1 in each village).

Co‐interventions: none reported.

Rural HHs: EHFP + aquaculture (100), EHFP (100) and control (100)

Anthropometry:

  • Underweight (women) (BMI <18.5 kg/m2) and children (WAZ < –2SD);

  • Stunting (HAZ < –2SD);

  • Wasting (WHZ < –2SD)

Biochemical indicators:

  • Haemoglobin (non‐pregnant women)

  • Haemoglobin (children)

Morbidity:

  • Anaemia (non‐pregnant women)

  • Anaemia (children)

22 months

Murshed E Jahan 2011 (Bangladesh)

Prospective controlled study

Unclear

Programme name: Development of Sustainable Aquaculture Project (DSAP)

Programme description and frequency: farmers received support to efficiently implement integrated aquaculture‐agriculture (IAA) approaches under 2 models – 1 with a one‐off provision of a small grant for purchasing inputs (value not reported) and 1 without, with training provided (3 sessions in the first year, 2 in the second year and 1 in the third year).

Provider: NGO; WorldFish Center

Delivery: farmers trained in recording required information which was collected bi‐monthly by research assistants.

Co‐interventions: none reported

Small‐scale farmers (260/126).

Within intervention farmers: 127 grant farmers, 133 non‐grant farmers

Proportion of HH expenditure on food

3 years

Kennedy 1989 (Kenya)

Prospective controlled study

Unclear

Programme name: South Nyanza Sugar Factory (Sony) smallholder sugarcane outgrowers' scheme

Programme description and frequency: farmers were enrolled into the scheme to provide sugarcane to a new factory, with payments to farmers after every harvest (24 months after planting)

Provider: Kenyan government

Delivery: contract agreement between farmers and factory.

Co‐interventions: none reported

Smallholder farm HHs (181/231).

Within intervention: 139 sugar farmers and 42 new entrant

  • Proportion of HH expenditure on food

Adequacy of dietary intake

  • Percentage of HHs with caloric deficiency

  • Caloric adequacy of preschool children

Anthropometry

  • WAZ

  • Underweight (< 80% of standard for WAZ)

  • HAZ

  • Stunted (< 90% of standard for HAZ)

  • WHZ

  • Wasting (< 90% of standard for WHZ)

  • BMI (adult)

Morbidity:

  • Illness of women and children (all‐cause and diarrhoea)

2 years

Alaofe 2016 (Benin)

Prospective controlled study

Unclear

Programme name: Solar Market Gardens (SMG)

Programme description and frequency: drip irrigation powered by solar water pump, using a perennial stream or borehole, with continued maintenance and training to farmers provided.

Provider: NGO (Solar Electric Light Fund – SELF)

Delivery: installation of system and training of local technicians carried.

Co‐interventions: women's agriculture group activities.

Rural HHs (116/98)

In both intervention and control groups, HHs included women who participated in women's agriculture groups (59/38) or not (60/60)

Proportion of HH expenditure on food

1 year

Alaofe 2019 (Benin)

Prospective controlled study

Unclear

Programme name: Solar Market Garden (SMG)

Programme description and frequency: Installation of a low‐pressure drip irrigation system, combined with a solar‐powered water pump in each intervention village. Each SMG was used jointly by 30–35 women belonging to the local women's agriculture group (each woman farmed her own land of 120 m2).

Provider: NGO (Solar Electric Light Fund – SELF)

Delivery: expanded installation of SMG systems (from programme reported in Alaofe 2016).

Co‐interventions: women's agriculture group activities.

Women in rural HHs (415/359).

In both intervention and control groups, HHs included women who participated in women's agriculture groups (184/126) or not (228/233)

Dietary diversity:

  • HDDS

  • Women's Dietary Diversity Score

Anthropometry

  • BMI (adult);

  • Underweight (adult) (BMI <18.5 kg/m2)

Biochemical indictors:

  • Iron deficiency

  • Vitamin A deficiency

Morbidity:

  • Anaemia

  • Iron‐deficiency anaemia

1 year

Kangmennaang 2017 (Malawi)

Prospective controlled study

High

Programme name: the Malawi Farmer to Farmer Agroecology project (MAFFA).

Programme description and frequency: farmers do their own experimentation with agroecological methods. Farmers are also encouraged to share knowledge gained with other farmers. MAFFA encourages farmers to adopt a suit of innovations rather than just a single innovation and to encourage farmer‐led learning. In addition to crop diversification, many farmers increased or began to apply compost and manure to their rain‐fed fields. Some farmers also experimented with botanical pesticides. Also, MAFFA goes beyond agroecological training to focus on knowledge sharing, leadership support, nutrition and attention to social inequalities.

Provider: Soils, Food and Healthy Communities organisation of Ekwendeni Hospital, Chancellor College, University of Malawi as well as Malawian and Canadian scientists.

Delivery: training, educational activities, campaigns, provision of seeds. Farmers shared knowledge with other farmers.

Co‐interventions: none reported.

Smallholder farm HHs (793/408)

Food security:

  • HFIAS score

About 2 years

Beegle 2017 (Malawi)

cRCT

High

Programme name: Malawi Social Action Fund's Public Works Programme (MASAF PWP).

Programme description and frequency: the MASAF PWP aims to provide short‐term labour‐intensive activities. The programme was designed to be interlinked with Malawi's large‐scale fertiliser input subsidy programme through the implementation of the PWP in the planting months of the main agricultural season when the fertiliser distribution also occurs. Projects were mostly road rehabilitation or construction, with some afforestation and irrigation projects. The wage rate was USD 0.92/day for a total payment of USD 11.01 for a 12‐day wave, total of 4 waves.

Provider: Malawi government

Delivery: payments in the study districts were facilitated by the research team for the purposes of the evaluation, with physical delivery of the cash in conjunction with the district officials.

Co‐interventions: the national fertiliser subsidy programme provided fertiliser coupons that allow two bags of fertiliser to be purchased for MK 500 each. These coupons are more likely to be available to treated HHs.

10 poor and able‐bodied HHs per community were offered the programme; communities (144/38)

Food security:

  • Food Security Score

Dietary diversity:

  • Food Consumption Score

  • Number of food groups consumed

  • Food Security Score

3/4 months

Porter 2016 (Ethiopia)

Prospective controlled study

High

Programme name: Productive Safety Net Program (PSNP)

Programme description and frequency: 80% public works programme (food/cash‐for‐work; USD 0.56/day in 2008) and 20% unconditional transfers to those unable to work (value NR). Programme operated seasonally but predictably, i.e. not emergency.

Provider: Ethiopian government, with donor funding

Delivery: centrally co‐ordinated by Government

Co‐interventions: none reported

Poor and food insecure rural HHs (682/924)

Anthropometry (results presented for all programme participants; not disaggregated according to type of intervention received)

  • HAZ

  • WAZ

5 and 7 years

Katz 2001 (Nepal)

Prospective controlled study

High

Programme name: N/A

Programme description and frequency: part‐time (5 hours/week) employment for women; distributing weekly supplements to and recording data on married women of child‐bearing age in own or neighbouring communities. Monthly income valued at USD 15

Provider: Joint undertaking by USAID, academic institutions (Johns Hopkins University), NGOs (National Society for the Prevention of Blindness, Kedia Seva Mandir) and the Nepalese government

Delivery: NR

Co‐interventions: approximately 31% of women employed by the project reported having additional cash employment, but amounts are unknown

Women living in rural areas (350/520)

Anthropometry:

  • MUAC

2 years

aOverall risk of bias based on risk for selection and attrition bias

ADRA: Adventist Development and Relief Agency; BDT: Bangladeshi taka; BMI: body mass index; FFS: Farmer Field School; HAZ: height‐for‐age z‐score; HDDS: Household Dietary Diversity Score; HFIAS: Household Food Insecurity Access Scale; HH: household; LAZ: length‐for‐age z‐score; MUAC: mid‐upper arm circumference; NGO: non‐governmental organisation; NR: not reported; RCT: randomised controlled trial; SD: standard deviation; WLZ: weight‐for‐length z‐score; WAZ: weight‐for‐age z‐score; WEG: Women Empowerment Group.

Figures and Tables -
Table 5. Income‐generation interventions – overview of included studies
Table 6. Food security and dietary diversity indices reported by included studies

Index/scale (study ID of studies reporting this measure)

Definition

Interpretation

Reference cited

Household food security indices

Household Food Insecurity Access Scale (HFIAS)

(Daidone 2014; Hjelm 2017; Kangmennaang 2017)

or

Household Food Insecurity Access Prevalence (HFIAP)

(Doocy 2017; Osei 2017; Weinhardt 2017)

HFIAS: sum of responses to 9 questions related to 4 domains of food security of a HH during the past 4 weeks.

HFIAP: categorises HHs into 4 levels of HH food insecurity, based on the frequency and severity of food insecurity experienced by HHs.

HFIAS: score ranges from 0 to 27. The higher the score the more food insecure the HH.

HFIAP: categorised as: food secure, and mild, moderately and severely food insecure.

Coates J, Swindale A, Bilinsky P. Household Food Insecurity Access Scale (HFIAS) for measurement of food access: indicator guide. Version 3. Washington, DC: Academy for Educational Development;2006

Food Security Score

(Beegle 2017)

Scores HHs in terms of 4 levels of HH food insecurity, based on the frequency and severity of food insecurity experienced by HHs.

Ranges from –1 to –4; higher value indicates greater food security

World Food Programme

Resilience index

(Beegle 2017)

Based on the World Food Program Coping Strategy Index. Weighted sum of the number of days in the past 7 days that HHs had to reduce the quantity and quality food consumed.

Higher values indicate food security

Maxwell D, Caldwell R. The Coping Strategies Index: Field methods Manual. Cooperative for Assistance and Relief Everywhere, Inc. (CARE), January 2008.

Food Security Index (FSI)

(Pellerano 2014)

Study authors adapted the food security component of the Bristol Child Deprivation Index. It is a simple mean of 3 questions related to child food security.

Severe food deprivation: FSI > 2.

Gordon D, Nandy S, Pantazis C, Pemberton S, Townsend P. (2003), the Distribution of Child Poverty in the Developing World, Policy Press, Centre for International Poverty Research, University of Bristol, July 2003.

Food Security Index

(Haushofer 2013)

Weighted mean of 17 outcome measures of food security and hunger.

The higher the index, the greater the food security

No reference cited

HHdietary diversity indices

HDDS

(Alaofe 2019; Breisinger 2018; Brunie 2014; Daidone 2014; Hidrobo 2014; Jodlowski 2016a; Kurdi 2019; Merttens 2013; Olney 2016b)

Sum of the number of food groups consumed by a HH during the past day or week, or longer (e.g. 2 or 4 weeks). Food groups included cereals, roots and tubers, vegetables (included vitamin A‐rich vegetables and tubers, dark leafy vegetables and other), fruits (included vitamin A fruits and other), meat (includes organ meat and flesh meat), eggs, fish, pulses and legumes, fats and oil, sugar and sweets, milk and other milk product, and spices and beverages.

Score ranges from 0 to 12; higher score reflected higher level of dietary diversity.

Kennedy G, Ballard T, Dop M, 2011. Guidelines for Measuring Household and Individual Dietary Diversity. Food and Agriculture Organization, Rome.

Swindale A, Bilinsky P. Household dietary diversity score (HDDS) for measurement of household food access: indicator guide (v.2). Washington (DC): FHI 360/FANTA; 2006.

Dietary Diversity Index (DDI)

(Hoddinott 2013; Pellerano 2014)

or

Dietary Diversity Score (DDS)

(Asfaw 2014)

or

Food diversity composite score (Miller 2011)

Sum of the number of food groups consumed by a HH during the past week. Food groups included main staples, pulses, vegetables, fruit, meat (or fish or egg); dairy products, sugar and oil.

Score ranges from 0 to 8; higher score reflects higher level of dietary diversity.

Ruel M. 2003. Operationalizing dietary diversity: a review of measurement issues and research priorities. Journal of Nutrition 133, 3911S–3926S.

Dietary Diversity Index (DDI)

(Hoddinott 2013);

or

Dietary Diversity Score (DDS)

(Hidrobo 2014; Schwab 2013)

Sum of the number of distinct food items consumed by a HH during the previous week. Depended on the number of food items included in the dietary questionnaire.

Score ranges from 0 to 25 (Hoddinott 2013); 0 to 40 (Hidrobo 2014); 0 to 39 (Schwab 2013); higher score reflects higher level of dietary diversity.

Ruel M. 2003. Operationalizing dietary diversity: a review of measurement issues and research priorities. Journal of Nutrition 133, 3911S–3926S.

Food Consumption Score (FCS)

(Ahmed 2019a; Ahmed 2019b; Beegle 2017; Hidrobo 2014; Hoddinott 2013; Pellerano 2014; Ponce 2017)

Weighted sum of the consumption frequency of the 8 food groups consumed by a HH during the past week. Food groups include main staples, pulses, vegetables, fruit, meat (or fish or egg), dairy products, sugar and oil.

Maximum score is 112 or 126.

Acceptable food consumption: FCS ≥ 35;

Borderline food consumption:

FCS between 21 and 35;

Poor food consumption: FCS < 35

WFP, 2008. Food consumption analysis: Calculation and use of the food consumption score in food security analysis. World Food Programme, Rome

Individual dietary diversity indices

Individual Child Dietary Diversity score (IDDS)

(Darrouzet Nardi 2016; Hoddinott 2013; Marquis 2018; Pellerano 2014; Skoufias 2013; Tonguet Papucci 2015)

Sum of number of food groups consumed by a child aged 6–23 months or a child aged < 5 years during the past 24 hours calculated from 17 foods, aggregated into 7 food groups: starchy staples (grains and white potatoes); vitamin A‐rich fruits and vegetables; other fruits and vegetables; offal, meat, and fish; eggs; legumes, nuts, and seeds; milk and dairy products

Score ranges from 0 to 7; higher score reflects higher level of dietary diversity.

Minimum dietary diversity: Dietary Diversity Score ≥ 4

World Health Organization, 2010. Indicators for Assessing Infant and Young Child Feeding Practices. World Health Organization, Geneva.

Individual Child Dietary Diversity Score (IDDS)

(Brunie 2014)

Sum of the number of different food groups consumed during the past day by a child aged < 5 years (12 food groups).

Score ranges from 0 to 12; higher score reflects higher level of dietary diversity

Guidelines for measuring household and individual dietary diversity.

FAO Nutrition – 2007 – FAO, Rome (Italy)

Women's Dietary Diversity Score (WDDS‐10)

(Alaofe 2019)

Sum of the number of food groups consumed during the past 24 hours calculated from the following food groups: starchy staples; beans and peas; nuts and seeds; dairy; flesh foods; eggs; vitamin A‐rich dark green leafy vegetables; other vitamin A‐rich vegetables and fruits; other fruits and other vegetables.

Score ranges from 0 to 10; higher score reflects higher level of dietary diversity

Kennedy G, Ballard T, Dop M, 2011. Guidelines for Measuring Household and Individual Dietary Diversity. Food and Agriculture Organization, Rome.

aJodlowski 2016: modified HDDS to a total score out of 13.
bOlney 2016: the egg food group was not included because of an oversight during survey design.

HH: household.

Figures and Tables -
Table 6. Food security and dietary diversity indices reported by included studies
Table 7. Summary of cognitive function indices reported by included studies

Index or scale

Definition/ measurement

Interpretation

Reference

Early Childhood Development Index (ECD)

(Daidone 2014)

Measures 4 developmental domains of children aged 3–7 years: physical (both gross and fine motor), language and cognition, socioemotional and approaches to learning.

Maximum score of 10; the higher the score the better functioning

Raven's Colored Progressive Matrices test score

(Baird 2013)

Non‐verbal test that measures abstract reasoning of children aged ≥ 5 years.

Maximum test score 60; the higher the score the better the abstract reasoning.

IDHC‐B test score

MacArthur‐Bates Communicative Development Inventory (adapted Spanish version)

(Fernald 2011)

Measures early language skills of children aged 12–35 months using parental report.

Scores range from 0 to 100 with 0 indicating that a child had not said any word on the checklist and 100 indicating that a child had said every word on the list.

Jackson‐Maldonado D, Thal D, Marchman V, Newton T, Fenson L, Conboy B. (2003). MacArthur Inventarios del Desarrollo de Habilidades Comunicativas. User's Guide and Technical Manual. Baltimore: Brookes Publishing.

TVIP test score

Peabody Picture Vocabulary Test (PPVT) (adapted Spanish version).

(Fernald 2011)

Measures receptive language/vocabulary of children aged ≥ 36 months.

Age‐adjusted norms: mean score of 100 and standard deviation of 15 at every age.

Woodcock‐Johnson‐Munoz battery test scores

(Fernald 2011)

WJ1 test measures long‐term memory in early childhood

Age‐adjusted percentile score

Woodcock, Richard, and Ana Munoz‐Sandoval. 1996. BaterıaWoodcock‐Munoz Pruebas de Aprovechamiento‐Revisada. Chicago: Riverside.

WJ2 test measures short‐term memory or immediate recall in early childhood

Age‐adjusted percentile score

WJ5 test measures visual integration, or visual‐spatial processing in early childhood

Age‐adjusted percentile score

Figures and Tables -
Table 7. Summary of cognitive function indices reported by included studies
Table 8. Unconditional cash transfers – overview of included studies

Study ID (country)

Study design

Overall risk of biasa

Other key details of intervention

Population (sample size at baseline: intervention/control)

Outcome domains and measures with available data

Timepoint of measurement

UCTs vs no intervention

Baird 2013

(Malawi)

cRCT

Low

Programme name: Schooling, Income, and Health Risks study (SIHR). Includes unconditional and conditional groups.

Amount and frequency of payments: payments split between guardian and girl in each HH.

HH amount varied randomly (USD 4, USD 6, USD 8, USD 10 per month). Amount paid to girl beneficiaries varied randomly (USD 1, USD 2, USD 3, USD 4, USD 5 per month).

Provider: NGOs

Delivery: payments to girl beneficiaries at local distribution points

Co‐interventions: none reported

Adolescent girls who were never married, aged 13–22 years, in urban and rural HHs (526/1495)

Cognitive function and development:

  • Raven's Coloured Progressive Matrices

Anxiety and depression:

  • Psychological distress score (GHQ‐12)

1 and 2 years

Brugh 2018

(Malawi)

cRCT

Low

Programme name: Malawi Social Cash Transfer Scheme (SCTS)

Amount and frequency of payments: about USD 40 (depending on HH size and number of school‐aged children); monthly transfers. Top‐up payments made for children at primary and secondary school. At follow‐up, intervention HHs had received 5 or 6 bi‐monthly cash transfer payments, due to an administrative delay.

Provider: Government

Delivery: NR

Co‐interventions: None reported

Ultra‐poor and labour constrained HHs (1561/1729 HHs; Mangochi and Salima districts

HH expenditure on food:

  • Proportion of total HH expenditure per year

Food security:

  • Worried not enough food

  • Consume > 1 meal per day

Dietary diversity:

  • Household Dietary Diversity Score (HDDS)

Adequacy of dietary intake:

  • Food energy deficiency

  • Depth of hunger

1 year

Daidone 2014

(Zambia)

cRCT

Low

Programme name: Child Grant Programme (CGP)

Amount and frequency of payments: about USD 12 per month, regardless of HH size; payments made every other month

Provider: government

Delivery: payments through local pay point manager

Co‐interventions: none reported

1260 HHs (7254 individuals)/1259 HHs (7091 individuals)

Food security:

  • Consuming > 1 meal/day

  • HFIAS

Dietary diversity:

  • HDDS

Anthropometry:

  • WAZ

  • HAZ

  • WHZ

Cognitive function and development:

  • ECD index

Morbidity: children aged 0–60 months

  • ARI

  • Diarrhoea

2 years

Fenn 2015

(Pakistan)

cRCT

Low

Programme name: REFANI Pakistan standard cash transfer

Amount and frequency of payments: PKR 1500 (about USD 14) disbursed monthly for 6 consecutive months.

Provider: EU; DG ECHO; Action Against Hunger field staff.

Delivery: mobile banks in a central location or central banks serving a number of villages. Verbal messaging from Action Against Hunger field staff at distribution that children should benefit from the transfers.

Co‐interventions: WINS programme in all villages – provided outpatient treatment for children aged 6 (SD 59) months with SAM, micronutrient supplementation (children, pregnant and lactating women), and behaviour change communication.

Poor and very poor agrarian HHs (standard cash group: 31 villages/632 HHs; Double cash group: 24 villages/600 HHs; fresh food voucher group: 31 villages/632 HHs; control group: 28 villages/632 HHs

Anthropometric indicators:

  • BMI (mothers)

  • HAZ

  • Stunting (HAZ < –2SD) and severe stunting (HAZ < –3SD)

  • WHZ

  • Wasting (WHZ < –2SD) and severe wasting (WHZ < –3SD)

  • MUAC

Biochemical indicators:

  • Hb (children)

  • Hb (mothers)

  • anaemia (children)

  • anaemia (mothers)

Morbidity: child:

  • ARIs

  • Diarrhoea

6 and 12 months

Pellerano 2014

(Lesotho)

cRCT

Low

Programme name: Lesotho Child Grants Programme (CGP)

Amount and frequency of payments: about USD 12 per month every 3 months. From 2013 (after 2 years) transfer indexed to number of children in the HH. Payments not made as predicted; smaller number of payments made involving larger amounts.

Provider: government; UNICEF‐Lesotho

Delivery: cash‐in‐transit firm provided payments at community pay points.

Co‐interventions: all CGP HHs received bi‐monthly top‐up for a specific period for a Food Emergency Grant.

Ultra‐poor rural HHs with children 0–17 years (706/647 HHs)

Food security:

  • Severe food deprivation (FSI > 2)

Dietary diversity:

  • FCS

  • Acceptable food consumption (FCS > 35)

Anthropometry:

  • Underweight (WAZ < third percentile)

Morbidity: children aged 0–5 years:

  • Any illness in previous month

Adverse events:

  • Overweight (children)

2 years

Tonguet Papucci 2015

(Burkina Faso)

cRCT

Low

Programme name: Moderate Acute Malnutrition Out (MAM'Out) project.

Amount and frequency of payments: seasonal payments – about USD 17 from July to November.

Provider: European Commission Humanitarian Aid (ECHO) trained project staff

Delivery: mothers received card linked to electronic account and mobile phone. Payments provided via phones and cash withdrawal points.

Co‐interventions: ongoing national social protection policy that promoted social transfer mechanisms to the poorest and most vulnerable.

Poor rural HHs with ≥ 1 child aged < 1 year (644/634 children; 602/583 HHs)

Dietary diversity:

  • MDD

  • Minimum acceptable diet

Anthropometric indicators:

  • WHZ

  • Stunting (HAZ < –2SD)

  • MUAC

Morbidity: child:

  • Diarrhoea

  • ARIs

2.4 years

Ahmed 2019a; Ahmed 2019b

(Bangladesh)

cRCT

Unclear

Programme name: Transfer Modality Research Initiative (TMRI) (2 trials implemented in the North and South of Bangladesh reported in the same paper).

Amount and frequency of payments: Monthly payment of BDT 1500 (about USD 19) per HH.

Provider: United Nations' World Food Program (WFP); NGO (Eco‐Social Development Organization or ESDO)

Delivery: a mobile phone was provided to the mother who collected payments from distribution sites using mobile verification of identity.

Co‐interventions: none reported

Rural HHs in the northwest and southern regions (North: 458/450; South: 454/464 HHs)

Dietary diversity:

  • FCS

  • Poor food consumption (FCS < 35)

Adequacy of dietary intake:

  • Food poverty (daily caloric intake < 2122 kcal)

Anthropometric indicators:

  • WHZ

  • WAZ

Morbidity: children:

  • Diarrhoea in the previous 2 weeks

2 years

Fernald 2011

(Ecuador)

cRCT

Unclear

Programme name: Bono de Desarrollo Humano (BDH) programme

Amount and frequency of payments: USD 15 per month; could accumulate payments for up to 4 months.

Provider: government

Delivery: payments to mothers via the banking system.

Co‐interventions: none reported

Rural and urban parishes; poor families who had children aged 0–6 years at baseline (1388/681 children)

Anthropometry:

  • HAZ

Biochemical:

  • Hb

Cognitive function and development:

  • Language (TVIP score)

  • Language (IDHC‐B score)

Anxiety and Depression:

  • Mother's depression score (CES‐D)

  • Mother's Perceived Stress Scale

17 months

Haushofer 2013

(Kenya)

RCT

Unclear

Programme name: N/A

Amount and frequency of payments: total amount of KES 25,200 (USD 404). Either monthly (for 9 months) or a lump‐sum payment. A subgroup of intervention HHs received an additional KES 10,000 per month for 7 months (total KES 95,200 (USD 1525).

Provider: NGO (GiveDirectly)

Delivery: payments via mobile money service to recipients (women or men).

Co‐interventions: none reported

Poor villages and HHs (503/505 HHs)

Food security:

  • FSI

Anthropometry:

  • MUAC

Anxiety and depression:

  • Psychological well‐being index

2 and 3 years

Hjelm 2017

(Zambia)

cRCT

Unclear

Programme name: Zambia Multiple Category Cash Transfer Program (MCP)

Amount and frequency of payments: transfers made every second month. Monthly amount of transfer of ZMW 55,000 (USD 11), irrespective of HH size.

Provider: government

Delivery: payments made through a local paypoint manager.

Co‐interventions: none reported

Socially vulnerable HHs in 2 rural districts with extreme poverty (1571/1515 HHs)

HH expenditure on food:

  • Proportion of total per capita HH expenditure

Food security:

  • HFIAS

Anxiety/depression:

  • Cohen's Perceived Stress scale;

  • CES‐D

2 and 3 years

Miller 2011

(Malawi)

cRCT

Unclear

Programme name: Malawi Social Cash Transfer Scheme (SCTS)

Amount and frequency of payments: about USD 40 (depending on HH size and number of school aged children); monthly transfers. Top‐up payments made for children at primary and secondary school.

Provider: government

Delivery: NR

Co‐interventions: none reported

Ultra‐poor and labour constrained HHs (366/386 HHs), Mchinji district

HH expenditure on food:

  • Proportion of total HH expenditure per week

Food security:

  • Consuming > 1 meal/day

Dietary diversity:

  • Food diversity composite score

6 months, 1 year

Asfaw 2014

(Kenya)

cRCT

High

Programme name: Kenya Cash Transfer Programme for Orphans and Vulnerable Children (CT‐OVC)

Amount and frequency of payments: every 2 months (about USD 21) irrespective of HH size.

Conditionalities: although the programme was unconditional, some districts imposed conditions (e.g. school attendance) and penalties

Provider: Kenya government

Delivery: payments made through local post offices.

Co‐interventions: none reported.

Ultra‐poor HHs with orphans and vulnerable children (CT‐OVC) (1542 HHs/755 HHs)

HH expenditure on food:

  • Proportion of total HH expenditure per month

Dietary diversity:

  • DDS

Anthropometric indicators:

  • HAZ

  • WAZ

  • WHZ

  • Stunting (HAZ < –2SD)

  • Underweight (WAZ < –2SD)

  • Wasting (WHZ < –2SD)

2 and 4 years

Gangopadhyay 2015

(India)

RCT

High

Programme name: N/A

Amount and frequency of payments: monthly cash transfer of INR 1000 (about USD 18).

Provider: researchers

Delivery: transfers were made through bank accounts opened for women beneficiaries

Co‐interventions: none reported

Note: comparison included control group with no bank account and not receiving transfer

100 HHs/100 HHs

NR

Merttens 2013 (Kenya)

cRCT

High

Programme name: Hunger Safety Net Programme (HSNP) pilot programme

Amount and frequency of payments: transfer every 2 months of KES 2150 (at commencement) which increased to KES 3500 by the end of the intervention period. Some HHs had multiple nominated beneficiaries; the effective value of the transfer per HH member was smaller for larger HHs

Provider: Ministry of State for the Development of Northern Kenya and Other Arid Lands

Delivery: cash was loaded onto a biometric smartcard which could be used to collect the cash transfer from a range of paypoints (usually small shops). Several services providers contracted.

Co‐interventions: none reported

Impoverished rural HHs (1224/1212 HHs)

HH expenditure on food:

  • Proportion of total HH expenditure

Dietary diversity:

  • DDS

Anthropometric indicators:

  • Moderate (WHZ < –2SD) and severe wasting (WHZ < –3SD);

  • Moderate (HAZ < –2SD) and severe stunting (HAZ < –3SD);

  • Moderate (WAZ < –2SD) and severe underweight (WAZ < –3SD)

Morbidity: HHs

  • Illness/injury in previous 3 months

2 years

Skoufias 2013

(Mexico)

Other papers:

Ramirez‐Luzuriaga 2016

Leroy 2010

cRCT

High

Programme name: food support programme (PAL, Programa de Apoyo Alimentario). Included in‐kind and cash transfer groups. Health and nutrition education session offered but not compulsory. This review included cash + education group vs control group only.

Amount and frequency of payments: about USD 14/month; disbursed every 2 months. Same amount for all HHs.

Provider: Mexican Government's agency

Delivery: distribution through stored of the government's agency DICONSA.

Co‐interventions: none reported

Poor rural HHs (1687/1663 HHs; 279/289 children)

Dietary diversity:

  • MDD

Anthropometric indicators:

  • BMI

1 and 2 years

Aguero 2006

(South Africa)

Prospective cohort study

High

Programme name: Child Support Grant (CSG)

Amount and frequency of payments: monthly payments made to the primary carer of the child, with no recording of what the carer used the money for. The initial monthly benefit was SAR 100 in 1998 and during the time of the 2004 survey it was SAR 170 (about USD 25).

Provider: government

Delivery: NR

Co‐interventions: none reported

30% of poorest children. subsample of African and Indian HHs with ≥ 1 child.

245/154 children

Anthropometric indicators:

  • HAZ

6 years

Breisinger 2018

(Egypt)

Prospective controlled study

High

Programme name: Takaful cash transfer programme

Amount and frequency of payments: Payments changed from quarterly to monthly, originally starting from a basic amount of EGP 325 per HH, which increased depending on the number of children in the HHs and their educational level.

Conditionalities: programme had been designed to be conditional but not enforced yet at the time of the evaluation

Provider: government; World Bank

Delivery: some beneficiaries had to travel to collect the money

Co‐interventions: none reported

Poor HHs in districts where poverty rate was ≥ 50% (2190 beneficiaries/3813 non‐beneficiaries)

Diet diversity:

  • HDDS

  • Mother's DDS

  • Child's DDS

Anthropometric indicators:

  • LAZ or HAZ

  • Wasting (WHZ < –2SD)

  • Overweight (children)

Morbidity in children aged 0–5 years

  • Diarrhoea

  • Fever

11 months

Renzaho 2017

(Nepal)

Prospective controlled study

High

Programme name: Child Cash Grant (CCG)

Amount and frequency of payments: NPR 200 per month for up to 2 children for poor families with children aged < 5 years, as a complement to other government grants.

Provider: government; Asia Development Bank, UNICEF‐Nepal

Delivery: embedded within existing universal social transfer programmes

Co‐interventions: both intervention and control groups received targeted resources transfers from the government for senior citizens, single women, endangered communities and people with disabilities.

Poor communities and HHs with ≥ 1 child aged < 60 months (1500 HHs/1500 HHs)

Anthropometric indicators:

  • WAZ

  • Underweight (WAZ < –2SD)

  • WHZ

  • Wasting (WHZ < –2SD)

  • HAZ

  • Stunting (HAZ < –2SD)

5 years

UCTs vs food transfers

Hoddinott 2013

(Niger)

cRCT

Unclear

Programme name: N/A

Amount and frequency of payments: cash received for time worked for 3 months, followed by another 3 months where cash was received unconditionally. USD 2/day worked to maximum of USD 50/month. Transfers made twice monthly.

Provider: Nigerian NGOs contracted out to handle food transport, storage, distribution and cash payments

Delivery: public works committee set up in each village to liaise with NGOs. NGOs charged a fixed percentage of total cash amount distributed.

Co‐interventions: none reported but all receiving cash for work in previous 3 months

Poor rural HHs (total 2187)

Dietary diversity:

  • HDDS

  • FCS

  • DDI

  • CDS

3 months

Schwab 2013

(Yemen)

cRCT

High

Programme name: N/A

Amount and frequency of payments: HHs in cash group received 3 cash transfers of an amount equivalent to the local value of the food basket (about USD 50).

Provider: transfers distributed in co‐ordination with local partners: the Yemen Post and Postal Savings Corporation (PPSC) in the case of cash transfers and Ministry of Education in the case of food transfers.

Delivery: collection of cash at any time up to 25 days after disbursement. Initial meetings with beneficiaries to sensitise beneficiaries to the programme objectives and logistics. For cash transfer group, a second resensitisation campaign held after funds were transferred to reinforce messages. Transfers given out at district branches of the PPSC.

Co‐interventions: none reported

Poor HHs in rural communities (982/1001 HHs).

Food security:

  • Number of days with HH reduced meal frequency (last week)

  • Number of days adults ate less food (last week)

  • Number days children ate less food (last week)

  • Number of months had difficulty meeting food needs

Dietary diversity:

  • HDDS

  • DDI

  • FCS

  • Probability of a low FCS score

7 months

aOverall Risk of Bias based on risk of selection and attrition bias.

ARI: acute respiratory infection; BDT: Bangladeshi taka; BMI: body mass index; CDS: Child Diet Score; CES‐D: Center for Epidemiologic Studies Depression Scale; cRCT: cluster randomised controlled trial; DDI: Dietary Diversity Index; DDS: Dietary Diversity Score; ECD: Early Childhood Development; EGP: Egyptian pound; FCS: Food Consumption Score; FSI: Food Security Index; GHQ‐12: 12‐item General Health Questionnaire; HAZ: height‐for‐age z‐score; Hb: haemoglobin; HDDS: Household Dietary Diversity Score; HFIAS: Household Food Insecurity Access Scale; HH: household; IDHC‐B: Inventario do Desenvolvemento de Habilidades Comunicativas – B; KES: Kenyan shilling; LAZ: length‐for‐age z‐score; MDD: minimum dietary diversity; MUAC: mid‐upper arm circumference; N/A: not applicable/available; NGO: non‐governmental organisation; NPR: Nepalese rupee; PKR: Pakistani rupee; SAM: severe acute malnutrition; SAR: South African rand; SD: standard deviation; UCT: unconditional cash transfer; WAZ: weight‐for‐age z‐score; WHZ; weight‐for‐height z‐score; ZMW: Zambian kwacha.

Figures and Tables -
Table 8. Unconditional cash transfers – overview of included studies
Table 9. Unconditional cash transfers – results of included trials

Study ID

(risk of bias)

Study design (n)

Unconditional cash transfers

No intervention

Effect measure (time point)

Effect directiona

Meta‐analysis (yes/no)

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Outcome 1.2: proportion of HH expenditure on food

1.2.1 Outcome measure: proportion of total HH expenditure on food (weekly/monthly)

Brugh 2018 (+)

cRCT (3290 HHs)

0.77 (0.11)

0.70 (0.11)

1561

0.77 (0.11)

0.72 (0.11)

1729

pp –2 (SE 1) 95% CI –3.96 to –0.4; P < 0.1 (1 year)

Yes (excluding Merttens, Asfaw which are missing variance estimate)

Miller 2011 (?)

cRCT (HHs)

56%

68%

366 HHs

52%

48%

386 HHs

pp 12, P < 0.0001 (1 year), 95% CI 5.924 to 18.076, SE 3.1

Hjelm 2017 (?)

cRCT (3010 HHs)

74 (16)

77 (15)

pp 3.2, robust t‐statistic 1.815, 95% CI –0.328 to 6.728, SE 1.8 (2 years)

cRCT (2969 HHs)

74 (16)

74.5

1490 HHs

77 (15)

72.7

1479 HHs

pp 4.2 robust, SE 1.8, 95% CI 0.672 to 7.728, P < 0.05 (3 years)

Merttens 2013 (‐)

cRCT (2436 HHs)

76.5%

77.3%

1224 HHs

79.8%

81%

1212 HHs

pp –0.4, P > 0.1 (1 year)

Asfaw 2014 (‐)

cRCT (1824 HHs)

63%

69.6%

1286 HHs

61%

68.6%

538 HHs

pp –0.95, P > 0.1 (2 years)

Outcome 1.3: proportion of HHs who were food secure

1.3.1 Food security

1.3.1.1 Outcome measure: proportion consuming > 1 meal/day

Brugh 2018 (+)

cRCT (3290 HHs)

0.79 (0.40)

0.94 (0.24)

1561

0.82 (0.39)

0.88 (0.34)

1729

DD 0.11, SE 0.03, pp 11, 95% CI 0.0512 to 0.1688, P < 0.001 (1 year)

Yes

Miller 2011b (?)

cRCT (752 HHs)

About 45%

About 85%

366 HHs

About 45%

About 45%

386 HHs

pp 42, P < 0.0001 (1 year), SE 10.7

95% CI 21.028 to 62.972

1.3.1.2 Outcome measure: mean food security scores (HFIASc/FSId) (mean, SD)

Daidone 2014 (+)

HFIAS/Food Security Scale

cRCT (2299 HHs)

9.63

1158 HHs

12.36

1141 HHs

MD 2.498, SE 0.59, 95% CI 1.3416 to 3.6544, P < 0.05, SE 1.3 (2 years)

Yes

Haushofer 2013 (?)

(FSI)

RCT (940 HHs)

471 HHs

Mean –0.00 (SE 1.00)

469 HHs

MD 0.25, 95% CI 0.13 to 0.37, P < 0.01 (2 years), SE 0.1

Hjelm 2017 (?)

(HFIAS/food security scale)

cRCT (3010 HHs)

14.78 (5.49)

14.68 (5.71)

MD 1.78, robust t‐statistic 3.76, 95% CI 0.8 to 2.76 P < 0.05 (2 years), SE 0.5

cRCT (2970 HHs)

14.78 (5.49)

9.83

1490 HHs

14.68 (5.71)

12.47

1480 HHs

MD 2.69, robust t‐statistic 4.94, 95% CI 1.71 to 3.67, P < 0.05 (3 years), SE 0.5

1.3.1.3 Outcome measure: severe food deprivation (FSI > 2)

Pellerano 2014 (+)

cRCT (2220 children aged 0–5 years)

67.1%

53.4%

747 HHs

69.3%

72.2%

739 HHs

pp –16.63, P < 0.05 (2 years), SE 8.5

N/A. Outcomes from same study.

Pellerano 2014 (+)

cRCT (5384 children aged 6–17 years)

67.8%

58.6%

747 HHs

73.9%

70.7%

739 HHs

pp –6.103, P < 0.1 (2 years), SE 3.7

1.3.2 Dietary diversity

1.3.2.1 Outcome measure: dietary diversity scores, including composite FCS (weighted) (mean, SD) (scores refer to number food groups consumed; reference periods and scales vary)

Daidone 2014 (+)

HDDS 0–12

cRCT (2298 HHs)

6.73

1158

5.30

1141

MD 1.43 (2 years)

Yes (except for Daidone, Merttens, pellerano – missing variance estimate)

Pellerano 2014 (+)

FCS 0–112

cRCT (1486 HHs)

28.7

31.2

747 HHs

28.9

30.4

739 HHs

MD 0.946, P > 0.1 (2 years)

Brugh 2018 (+)

HDDS 0–12

cRCT (3290 HHs)

5.63 (1.78)

5.85 (1.54)

1561

5.64 (1.87)

5.34 (1.44)

1729

MD 0.23 (SE 0.32), 95% CI –0.3972 to 0.8572, P > 0.05 (1 year)

Miller 2011 (?)

FDCS 1–8

cRCT (752 HHs)

5

7

366 HHs

5

4

386 HHs

MD 2.4, P < 0.0001 (1 year), SE 0.6. 95% CI 1.224 to 3.576

Ahmed 2019a (?)

FCS 0–112

cRCT (HHs NR)

MD 6.84 points, SE 1.12, P < 0.01, 95% CI 4.6448 to 9.0352 (2 years)

Ahmed 2019b (?)

FCS 0–112

cRCT (HHs NR)

MD 2.62 points, SE 1.04, P < 0.05, 95% CI 0.5816 to 4.6584 (2 years)

Merttens 2013 (‐)

DDS 0–12

cRCT (2436 HHs)

6.7

7.2

1224 HHs

6.1

6.2

1212 HHs

MD 0.3, P > 0.1 (1 year)

Asfaw 2014 (‐)

DDS (0–8)

cRCT (2369 HHs)

5.225

6.177

1289 HHs

5.697

5.843

540 HHs

MD 0.821, SE 0.3, P < 0.01 (2 years)

1.3.2.2. Outcome measure: proportion with MDD (3–4 food groups)/acceptable food consumption (FCS > 35)

Tonguet Papucci 2015 (+)

cRCT (322 children aged 14–27 months)

65.6%

160

39.5%

162

OR 2.95, 95% CI 1.86 to 4.68, P < 0.001 (2 years)

SMD 0.6, SE 0.1

Yes

Skoufias 2013 (‐)

cRCT (568 children)

69.6%

66. 7%

279

72.7%

59.9%

289

pp 10.6, 95% CI –6.65 to 27.85, P > 0.05 (2 years), SE 8.8

SMD 0.1, SE 0.1

1.4 Change in adequacy of dietary intake

1.4.1 Food poverty (per capita daily caloric intake < 2122 calories; proportion)

Ahmed 2019a (?)

cRCT (n NR)

MD –0.05, SE 0.03, 95% CI –0.1088 to 0.0088, P > 0.05 (2 years)

Yes

Ahmed 2019b (?)

cRCT (n NR)

MD –0.04, SE 0.04, P > 0.05, 95% CI –0.1184 to 0.0384 (2 years)

1.4.2 Proportion food energy deficient (total HH caloric availability < total HH caloric requirements)

Brugh 2018 (+)

cRCT (3290 HHs)

1561

1729

DD –0.1, SE 0.04, 95% CI –0.1784 to –0.0216; P < 0.05 (1 year)

1.5Change in anthropometric indicators

1.5.1Stunting (chronic undernutrition)

1.5.1.1 Outcome measure: proportion stunted (HAZ < –2SD)

Tonguet Papucci 2015 (+)

cRCT

27.7%

630 children aged 0–15 months

27.2%

620 children aged 0–15 months

OR 0.73, 95% CI 0.47 to 1.14, P 0.17 (2 years)

Yes (except Asfaw, Merttens – no measure of variance)

Fenn 2015 (+)

cRCT (1683 children)

n (%): 457 (50.9)

NR

874 children

n (%): 437 (51.7)

NR

809 children

OR 0.36, 95% CI 0.22 to 0.59, P < 0.001 (6 months)

cRCT (1664 children)

n (%): 457 (50.9)

NR

849 children

n (%): 437 (51.7)

NR

815 children

OR 0.54, 95% CI 0.36 to 0.81, P = 0.003 (12 months)

Merttens 2013 (‐)

cRCT (1062 HHs)

26.7%

29.6%

35.6%

31.5%

pp 7.0, P > 0.1 (2 years)

Asfaw 2014 (‐)

cRCT

41.5%

35.7%

442 children aged 0–59 months

44%

37%

295 children aged 0–59 months

pp –4.63, P > 0.1 (2 years)

1.5.1.2 Outcome measure: proportion with severe stunting (HAZ < –3SD)

Fenn 2015 (+)

cRCT (1683 children)

NR

NR

874 children

NR

NR

809 children

OR 0.47, 95% CI 0.28 to 0.77, P = 0.003 (6 months)

No. SE not available for all studies.

cRCT (1664 children)

NR

NR

849 children

NR

NR

815 children

OR 0.59, 95% CI 0.38 to 0.92, P = 0.02 (12 months)

Merttens 2013 (‐)

cRCT (n = 1062)

11.6%

13.4%

15.2%

15.1%

pp 1.9, P > 0.1 (2 years)

1.5.1.3 Outcome measure: HAZ (mean, SD)

Daidone 2014 (+)

cRCT (2299 children aged 0–60 months)

–1.445

1158

–1.491

1141

MD 0.066, 95% CI –0.116 to 0.248, P > 0.05 (2 years)

Yes

Tonguet Papucci 2015 (+)

cRCT (1250 children aged 0–15 months

–1.18 (1.44)

–1.96 (1.03)

630

–1.33 (1.24)

–1.99,

SD 1.04)

620

MD –0.0005, 95% CI –0.004 to 0.003 z‐score/month, P = 0.78

Fenn 2015 (+)

cRCT (1683 children)

–1.98 (1.65)

NR

874 children

–1.97 (1.75)

NR

809 children

MD 0.24, 95% CI 0.17 to 0.32, P < 0.001 (6 months)

cRCT (1664 children)

–1.98 (1.65)

NR

849 children

–1.97 (1.75)

NR

815 children

MD 0.21, 95% CI 0.10 to 0.31, P < 0.001 (12 months)

Fernald 2011 (?)

cRCT (1196 children)

–0.5 (2.1)

–1.7 (1.2)

797

–0.7 (2.0)

–1.7 (1.2)

399

MD 0.01, 95% CI –0.18 to 0.19 (2 years)

Ahmed 2019a (?)

cRCT (n NR)

MD 0.132, SE 0.08, 95% CI –0.0248 to 0.2888, P > 0.05 (2 years)

Ahmed 2019b (?)

cRCT (n NR)

MD –0.097, SE 0.08, 95% CI –0.0598 to 0.2538, P > 0.05 (2 years)

Asfaw 2014 (‐)

cRCT (737 children aged 0–59 months)

–1.466

–1.279

442

–1.462

–1.248

295

MD –0.0272, 95% CI –0.503 to 0.449, P > 0.1 (2 years)

1.5.2Wasting (acute undernutrition)

1.5.2.1 Outcome measure: proportion wasted (WHZ < –2SD) (proportion)

Tonguet Papucci 2015 (+)

cRCT (1250 children aged 0–15 months

26%

630

192%

620

IRR 0.92, 95% CI 0,64 to 1.32; P = 0.66 (2 years)

No. SE not available for all studies and different effect size for 1 study.

Fenn 2015 (+)

cRCT (1683 children)

n (%): 196 (22.0)

NR

874 children

n (%): 184 (21.9)

NR

874 children

OR 1.09, 95% CI 0.64 to 1.87, P = 0.75 (6 months)

cRCT (1664 children)

n (%): 196 (22.0)

NR

849 children

n (%): 184 (21.9)

NR

849 children

OR 1.10, 95% CI 0.71 to 1.71, P = 0.66 (12 months)

Merttens 2013 (‐)

cRCT (1062 children)

25.3%

23.1%

24.2%

17.3%

pp 4.7, P > 0.1

Asfaw 2014 (‐)

cRCT (737 children aged 0–59 months)

6%

9%

648

9.4%

6.9%

341

pp 5.95, P > 0.1 (2 years)

1.5.2.2 Outcome measure: severe wasting (WHZ < –3SD) (proportion)

Fenn 2015 (+)

cRCT (1683 children)

69 (7.7)

874 children

62 (7.4)

874 children

OR 0.98, 95% CI 0.38 to 2.54, P = 0.97 (6 months)

No. Variance only available for 1 of the 2 studies.

Merttens 2013 (‐)

cRCT (1062 children)

6.8

6.2

8.0

3.5

pp 3.9, P > 0.1

1.5.2.3 Outcome measure: WHZ (mean, SD)

Daidone 2014 (+)

cRCT (2299 children aged 0–69 months)

–0.0961

1158

–0.154

1141

MD 0.118, 95% CI –0.015 to 0.251 (2 years)

Yes

Tonguet Papucci 2015 (+)

cRCT (1250 children aged 0–15 months)

–1.24

(1.23)

–0.56 (0.95)

630

–1.07 (1.12)

–0.61 (0.93)

620

MD –0.003 z‐score/month, 95% CI –0.008 to 0.0003, P = 0.07 (2 years)

Fenn 2015 (+)

cRCT (1683 children)

–1.11 (1.34)

NR

874 children

–1.15 (1.30)

NR

874 children

MD 0.04, 95% CI –0.07 to 0.14, P = 0.5 (6 months)

cRCT (1664 children)

–1.11 (1.34)

NR

849 children

–1.15 (1.30)

NR

849 children

MD –0.08, 95% CI –0.19 to 0.04, P = 0.21 (12 months)

Ahmed 2019a (?)

cRCT (n NR)

Coefficient –0.013, SE 0.07, 95% CI –0.1502 to 0.1242, P > 0.05 (2 years)

Ahmed 2019b (?)

cRCT (n NR)

Coefficient –0.088, SE 0.08, P > 0.05, 95% CI –0.2448 to 0.0688 (2 years)

Asfaw 2014 (‐)

cRCT (737 children aged 0–59 months)

–0.017

–0.332

442

0.065

–0.166

295

MD –0.0838, 95% CI –0.339 to 0.171, P > 0.1 (2 years)

1.5.3 Underweight

1.5.3.1 Weight for age z‐score

1.5.3.1.1 Outcome measure: proportion underweight (WAZ < –2SD)

Pellerano 2014 (+)

cRCT (total n: 6 month old 474; 12 month old 293)

6 month old: 29.2; 12 month old: 36.6

6 month old: 10.6;

12 month old: 16.4

6 month old: 11.0; 12 month old: 39.7

6 month old: 8.4

12 month old: 23.3

6 month old: pp –15.60, P < 0.05

12 month old: pp –3.637, P > 0.05 (2 years)

6 month old: ▲

12 month old: △

No. Variance not available for all studies.

Merttens 2013 (‐)

cRCT (1062)

30.7

24.9

33.7

24

pp 3.9, P > 0.1

Asfaw 2014 (‐)

cRCT (1435)

20.6

21

19.6

19.1

pp –0.62, P = 0.901 (2 years)

1.5.3.1.2 Outcome measure: proportion severely underweight (WAZ < –3SD)

Merttens 2013 (‐)

cRCT (1062)

9.8

8.9

10.9

6.9

pp 3.2, P > 0.1

N/A

1.5.3.1.3 Outcome measure: mean WAZ

Daidone 2014 (+)

cRCT (6825 children)

–0.900

–0.963

MD 0.128, 95% CI –0.05 to 0.261, P > 0.05 (2 years)

Yes

Asfaw 2014 (‐)

cRCT 752 children aged 0–59 months)

–0.879

–1.034

456

–0.923

–0.804

296

MD –0.274, 95% CI –0.633 to 0.085, P > 0.1 (2 years)

1.5.3.2 BMI (mean, SD)

Fenn 2015 (+)

cRCT 1208 HHs/mothers (flow diagram)

Median (IQR) 20.4 (18.3 to 23.5)

NR

607

median (IQR) 20.0 (18.1 to 22.7)

NR

601

Beta‐coefficient –0.10, 95% CI –0.36 to 0.16, P = 0.45 (6 months)

1.5.5 Mid‐upper arm circumference (MUAC) (mean, SD)

Fenn 2015 (+)

cRCT (1208 HHs/mothers)

24.4 (3.4)

NR

607

24.3 (3.2)

NR

601

Beta‐coefficient 0.09, 95% CI –0.13 to 0.30, P = 0.41 (6 months)

cRCT (1683 children)

13.5 (1.3)

NR

874

13.5 (1.2)

NR

809

beta‐coefficient 0.06, 95% CI –0.02 to 0.15, P = 0.15 (6 months)

1.6Change in biochemical indicators

1.6.1 Outcome measure: haemoglobin concentration (g/dL) (mean, SD)

Fenn 2015 (+)

cRCT (1208 HHs/mothers)

mean 103 (SD 18)

NR

607 mothers

mean 100 (SD 19)

NR

601 mothers

MD –0.42, 95% CI –0.63 to –0.20, P < 0.001 (6 months)

cRCT (1683 children)

mean 89 (17)

NR

874 children

mean 88 (16)

NR

809 children

MD –0.12, 95% CI –0.31 to 0.08, P = 0.24 (6 months)

Yes

Fernald 2011 (?)

cRCT (922 children)

9.7 (1.3)

10.4 (1.5)

9.5 (1.3)

10.3 (1.3)

MD 0.04, 95% CI –0.21 to 0.29, P > 0.1

1.7 Cognitive function and development

1.7.1 Outcome measure: cognitive and development scales/indices (mean, SD)

Baird 2013 (+)

cRCT (RCPM; 2057 adolescents)

MD 0.136, SE 0.119, 95% CI –0.097 to 0.369, P > 0.1 (2 years)

No (no n to calculate SMD)

Daidone 2014 (+)

cRCT (ECD Index; 5670 children)

5.174

4.926

MD 0.311, 95% CI –0.065 to 0.687, P > 0.1 (2 years)

1.7.2 Outcome measure: Individual cognitive function measures scores (mean, SD)

Fernald 2011 (?)

cRCT (Language: TVIP; 1894 children 36 months and older)

MD 0.013, 95% CI –0.076 to 0.102, P > 0.1 (2 years)

N/A

Language: IDHC‐B 1192 children aged 12–35 months)

45.0 (35.1)

42.3 (32.2)

MD 2.43, 95% CI –1.01 to 5.86, P > 0.1 (2 years)

1.8 Change in proportion of anxiety and depression

1.8.1 Outcome measure: depression score (CES‐D scale) (mean change in score, SD)

Fernald 2011 (?)

cRCT (1430 mothers)

19.6 (11.1)

18.9 (10.6)

MD 0.71, 95% CI –0.84 to 2.25, P > 0.1 (2 years)

Yes

Haushofer 2013 (?)

RCT (2140 adults)

471 HHs

26.48 (9.31)

469 HHs

MD –0.99, 95% CI –1.54 to –0.44, P < 0.1 (3 years)

Hjelm 2017 (?)

cRCT (1765 HHs with adolescents)

Effect estimate 0.00, robust t‐statistic 0.00, P not significant (2 years)

cRCT (2217 HHs with adolescents)

19.24

Effect estimate –0.54, 95% CI –1.80028 to 0.72028 (3 years)

1.8.2 Outcome measure: Perceived Stress Scale (mean, SD)

Fernald 2011 (?)

cRCT (n = 1430)

Top 3 income quartiles: MD 0.045, 95% CI –0.112 to 0.202, P > 0.1.

Bottom income quartile: MD 0.177, 95% CI –0.017 to 0.371, P < 0.1

(2 years)

Yes

Haushofer 2013 (?)

RCT (2140 adults)

0.00 (1.00)

MD –0.14, 95% CI –0.258 to –0.022, P < 0.05 (3 years)

Hjelm 2017 (?)

cRCT (2490 HHs)

9.58 (4.64)

9.92 (4.73)

Effect estimate –0.42, 95% CI –1.12364 to 0.28364 (3 years)

1.8.3 Outcome measure: proportion with psychological distress (psychological distress, anxiety and depression, social dysfunction, loss of confidence)

Baird 2013 (+)

cRCT (2089 adults)

0.374

pp –14.3, 95% CI –21.0 to –7.6, P < 0.001 (1 year)

N/A

0.308

pp –3.8, 95% CI –13.14 to 5.8 P > 0.1 (2 years)

1.8.4 Outcome measure: Psychological Well‐being Score (mean, SD)

Haushofer 2013 (?)

RCT (2140 adults)

–0.00 (1.00)

Coefficient 0.20 SD, 95% CI 0.082 to 0.318, P < 0.1 (2 years)

N/A

1.9 Morbidity

1.9.1 Outcome measure: incidence of respiratory infections (reference period: 1 and 2 weeks)

Daidone 2014 (+)

cRCT

Proportion children aged 0–60 months with ARI in previous 2 weeks (n = 7232)

0.0511

0.0832

pp –3.6, 95% CI –8.6 to 14.0, P > 0.05 (2 years)

No. 2 different measures of effect that could not be compared (IRR vs OR/pp).

Fenn 2015 (+)

cRCT (1683 children)

n (%): 310 (34.3)

NR

874 children

n (%): 273 (32.2)

NR

809 children

OR, 0.73, 95% CI 0.51 to 1.03, P = 0.07 (6 months)

Tonguet Papucci 2015 (+)

cRCT

Episodes/child‐month (1250 children aged 0–15 months)

N 0.87, 95% CI 0.84 to 0.89

N 0.95, 95% CI 0.92 to 0.97

IRR 0.79, 95% CI 0.78 to 0.81, P < 0.001 (2 years)

Asfaw 2014 (‐)

cRCT 957 children aged 0–7 years)

613 children

344 children

IRR 0.556, t‐statistics –2.40, P < 0.05 (2 years)

1.9.2 Outcome measure: incidence diarrhoeal disease

Fenn 2015 (+)

cRCT (1683 children)

n (%): 228 (25.2)

NR

874 children

n (%): 298 (35.0)

NR

809 children

OR 1.05, 95% CI 0.67 to 1.63, P = 0.84 (6 months)

No. Different measure of effect for one study (IRR vs OR/pp)

Daidone 2014 (+)

cRCT

Proportion children aged 0–60 months with diarrhoea in previous 2 weeks (n = 7232)

0.0684

0.0925

pp –4.9, 95% CI –8.9 to –0.9, P < 0.05 (2 years)

Tonguet Papucci 2015 (+)

cRCT

Episodes/child/month (1250 children aged 0–15 months)

n 0.85, 95% CI 0.82 to 0.88

n 0.83, 95% CI 0.80 to 0.85

IRR 1.00, 95% CI 0.97 to 1.03, P = 0.89 (2 years)

Ahmed 2019a (?)

cRCT (n NR)

Coefficient –0.003, pp –0.3, SE 0.02, 95% CI –0.0422 to 0.0362, P > 0.05 (2 years)

Ahmed 2019b (?)

cRCT (n NR)

Coefficient –0.009, pp –0.9, SE 0.02, 95% CI –0.0482 to 0.0302, P > 0.05

1.9.3 Outcome measure: proportion with any illness in previous reference period (1 month/3 months)

Pellerano 2014 (+)

cRCT (1996 children aged 0–5 years)

38.9

31.4

36.7

45.3

pp –15.38, P < 0.1 (2 years)

No. Variance estimates not available for all studies.

Merttens 2013 (‐)

cRCT (n = 14,342) (includes injury)

22.5

12.1

23.1

11.7

pp 1.0, P > 0.05 (2 years)

1.9.4 Proportion with anaemia (any)

Fenn 2015 (+)

cRCT (1683 children)

874 children

809 children

OR 1.13, 95% CI 0.68 to 1.86, P = 0.64 (6 months)

N/A

cRCT (1208 mothers)

607 mothers

601 mothers

OR 1.34, 95% CI 0.82 to 2.18, P = 0.24 (6 months)

1.10 Adverse events: proportion who were overweight (according to International standards and Bukana Health Card)

Pellerano 2014 (+)

cRCT (total n: 6 months old: 474; 12 months old: 293)

6 months old: 4.5; 12 months old: 6.0

6 months old: 2.2; 12 months old: 0.0

6 months old: 0.8; 12 months old: 0.0

6 months old: 2.0; 12 months old: 0.0

6 months old: pp –5.082, P > 0.05; 12 months old: pp –6.461, P > 0.05 (2 years)

N/A

aEach triangle represents one study; bValues are derived from graphs

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0; □: Effect measure is the null; (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias. FDCS: Food diversity consumption score; FCS: Food consumption score.

ARI: acute respiratory infection; CES‐D: Center for Epidemiologic Studies Depression Scale; CI: confidence interval; cRCT: cluster randomised controlled trial; DD: Diet diversity; DDS: Dietary Diversity Score; ECD: Early Childhood Development; FCS: Food Consumption Score; FDCS: Food Diversity Composite Score; FSI: Food Security Index; HAZ: height‐for‐age z‐score; HDDS: Household Dietary Diversity Score; HFIAS: Household Food Insecurity Access Scale; HH: household; IDHC‐B: Inventario do Desenvolvemento de Habilidades Comunicativas‐B; IQR: interquartile range; IRR: incidence rate ratio; MD: mean difference; MDD: minimum dietary diversity; n: number; NR: not reported; OR: odds ratio; pp: percentage point; RCPM: Ravens Coloured Progressive Matrices ; RCT: randomised controlled trial; SD: standard deviation; SE: standard error; SMD: standardised mean difference; TVIP: Test de Vocabulario en Imagenes Peabody; WHZ: weight‐for‐height z‐score.

Figures and Tables -
Table 9. Unconditional cash transfers – results of included trials
Table 10. Unconditional cash transfers – results of included prospective controlled studies

Study ID

(risk of bias)

Study design (n)

Unconditional cash transfers

No intervention

Effect measure (time point)

Effect directiona

Meta‐analysis (yes/no)

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

1.3.2 Dietary diversity

1.3.2.1 Outcome measure: Dietary diversity scores, including composite Food Consumption Score (FCS) (weighted) (mean, SD) (scores refer to number food groups consumed; reference periods and scales vary)

Breisinger 2018 (‐)

PCS (6003 HHs) – HDDS

NR

9.58 (1.38)

2190?

NR

9.48 (1.55)

3813?

MD (SE) 0.16 (0.117), 95% CI –0.06932 to 0.38932, P > 0.1 (1 year??)

N/A

Breisinger 2018 (‐)

PCS (5799 HHs) – mother DDS)

NR

4.21 (1.28)

2190?

NR

4.04 (1.26)

3813?

MD 0.011 (SE 0.100), 95% CI –0.185 to 0.207, P > 0.1 (1 year?),

Breisinger 2018 (‐)

PCS (1684 HHs) DDS children aged 6–23 months

NR

3.35 (1.73)

2190?

NR

3.39 (1.61)

3813?

MD –0.342 (SE 0.209) 95% CI –0.752 to 0.068, P > 0.1 (1 year)

Breisinger 2018 (‐)

PCS (3202 HHs) DDS children aged 24–59 months

NR

5.09 (1.37)

2190?

NR

4.89 (1.40)

3813?

MD –0.057 (SE 0.144) 95% CI –0.33924 to 0.22524, P > 0.1 (1 year)

1.5Change in anthropometric indicators

1.5.1Height‐for‐age z‐scores; chronic undernutrition)

1.5.1.1 Outcome measure: proportion stunted (HAZ < –2SD)

Renzaho 2017 (‐)

Prospective controlled study (n = 1491)

66.7

59.8

748

63

52.9

743

Adjusted DID (pp): –5.16, 95% CI –9.55 to –0.77 (5 years), SE 2.2

1.5.1.3 Outcome measure: HAZ (mean, SD)

Aguero 2006 (‐)

PCS

–0.84

–1.08

NR (MD 0.15 at 45%, and 0.25 at 80% of nutritional window; data derived from graph (6 years))

No. SE not available for all studies.

Renzaho 2017 (‐)

PCS (1491 children)

–2.6 (1.4)

–2.2 (1.4)

748

–2.3 (1.3)

–2.1 (1.3)

743

Adjusted DID: 0.18, 95% CI 0.09 to 0.27 (5 years)

1.5.2WHZ; acute undernutrition/wasting

1.5.2.1 Outcome measure: proportion wasted (WHZ < –2SD) (proportion)

Renzaho 2017 (‐)

PCS (1491 children)

12.7

5.7

748

5.8

6.4

743

Adjusted DID: pp –2.84, 95% CI –5.58 to –0.1 (5 years)

1.5.2.3 Outcome measure: WHZ (mean, SD)

Renzaho 2017 (‐)

PCS (1491 children)

–0.8 (1.1)

–0.4 (1.0)

748

–0.5 (0.9)

–0.4 (1.1)

743

Adjusted DID: MD 0.19, 95% CI 0.09 to 0.3 (5 years)

1.5.3 Weight‐for‐age z‐score (WAZ; underweight)

1.5.3.1 Outcome measure: proportion underweight (WAZ <2SD)

Renzaho 2017 (‐)

PCS (1491 children)

50.7

34.8

748

37.3

28.9

743

Adjusted DID: pp –7.35, 95% CI –11.62 to –3.08 (5 years)

N/A

1.5.3.3 Outcome measure: mean WAZ

Renzaho 2017 (‐)

PCS (1491 children)

–2.1 (1.1)

–1.6 (1.1)

748

–1.7 (1.0)

–1.4 (1.1)

743

Adjusted DID:

0.22, 95% CI 0.15 to 0.29 (5 years)

N/A

aEach triangle represents one study.

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

DDS: Dietary Diversity Score; DID: difference in differences; HAZ: height‐for‐age z‐score; HH: household; MD: mean difference; n: number; N/A: not applicable/available; NR: not reported; PCS: prospective controlled study; SD: standard deviation; SE: standard error; WAZ: weight‐for‐age z‐score; WHZ: weight‐for‐height z‐score.

Figures and Tables -
Table 10. Unconditional cash transfers – results of included prospective controlled studies
Table 11. Conditional cash transfers – overview of included studies

Study name (year) country of conduct

Study design

Overall risk of biasa

Other key detail of intervention

Population (sample size at baseline: intervention/ control)

Outcome domains and measures with available data

Timepoint of measurement

Baird 2013

(Malawi)

cRCT

Low

Programme name: Schooling, Income, and Health Risks study (SIHR). Includes unconditional and conditional groups.

Type, amount and frequency of payments: payments were split between guardian and girl in each HH. HH amount varied randomly from USD 4, USD 6, USD 8, to USD 10 per month. Amount paid to girl beneficiaries varied randomly from USD 1, USD 2, USD 3, USD 4, to USD 5 per month.

Conditionalities: school attendance for 80% of the days during the previous month.

Provider: 2 NGOs

Delivery: payments to girl beneficiaries at local distribution points

Co‐interventions: NR

Adolescent girls who were never married from urban and rural HHs (1211/1495 girls)

Cognitive function and development:

  • Cognitive test score (Raven's Coloured matrices and other)

Anxiety/depression:

  • Psychological distress test score (GHQ‐12)

1 and 2 years

Macours 2012

(Nicaragua)

cRCT

Low

Programme name: Atención a Crisis

Amount and frequency of payments: Standard payment of USD 145 per HH every 2 months. 3 intervention groups:

1. Standard transfer + education: additional USD 145 per HH and USD 25 per child for HHs with children aged 7–15 years; 2. Standard transfer + scholarship for vocational training; and 3. Standard transfer + lump sum to start non‐agricultural activity.

Conditionalities: 1. Regular health check‐ups for children aged 0–5 years, school enrolment; 2. regular attendance, however not monitored in practice; and 3. developing a business plan.

Provider: government

Delivery: payments to child's primary carer.

Co‐interventions: NR

Poor rural HHs with 2377 children aged < 6 years (3002/1019 HHs)

HH expenditure on food:

  • Percentage of total expenditure

Anthropometric indicators:

  • WAZ

  • HAZ

Anxiety/depression:

  • Depression score (CES‐D)

Cognitive function and development:

  • Language test score (TVIP score)

Morbidity – child

  • Number of days ill in bed in the past month

9 months

(12 months for CES‐D)

Maluccio 2005

(Nicaragua)

cRCT

Low

Programme name: Red de Protección Social

Amount and frequency of payments: amount NR; payments every 2 months.

Conditionalities: school attendance; preventive health care visits for children for growth and development monitoring, vaccination, and provision of antiparasites, vitamins, and iron supplements.

Provider: government. Preventive health services provided by private healthcare providers.

Delivery: NR

Co‐interventions: NR

Poor, rural HHs (1396 HHs)

HH expenditure on food:

  • Percentage of total expenditure

Anthropometric indicators:

  • HAZ

  • WAZ

  • WHZ

1 and 2 years

Kusuma 2017a

(Indonesia)

cRCT

Unclear

Programme name: Program Keluarga Harapan (PKH)

Amount and frequency of payments: USD 60–220 per HH per year, depending on the number and age of children in the HH.

Conditionalities: health: pre‐ and postnatal visits, iron supplementation and assisted deliveries for pregnant women, growth monitoring, immunisation and vitamin A supplementation of children aged < 5 years. Education: primary and junior secondary school enrolment and attendance rates of 85%.

Provider: government

Delivery: payment to mothers through local post offices

Co‐interventions: NR

Very poor urban HHs with children aged 24–36 months (1395 HHs)

Anthropometry:

  • Underweight (WAZ < –2SD)

  • Severe underweight (WAZ < –3SD)

  • Wasting (WHZ < –2SD)

  • Severe wasting (WHZ < –3SD)

  • Stunting (HAZ < –2SD)

  • Severe stunting (HAZ < –3SD)

2 years

Gertler 2000 (PROGRESA)

(Mexico)

cRCT

Unclear

Programme name: Oportunidades (previously known as PROGRESA)

Type, amount and frequency of payments: scholarships of up to MXN 490 (January–June 98) and MXN 625 per HH (July–December 1999), every 2 months; payments for school supplies; and monthly payments for food.

Conditionalities: health: attendance of preventive health services by every family member; growth monitoring and immunisation of children aged 0–5 years; nutrition supplements (for lactating women, children aged 6–23 months or low‐weight children), antenatal care for pregnant women. Education: school enrolment and school attendance > 85%.

Provider: government

Delivery: lump sum payment to mothers once completed forms were submitted by HHs to verify school attendance.

Co‐interventions: NR

Poor rural HHs

(506 villages; 320/186)

Anthropometric indicators:

  • HAZ

  • Stunting (HAZ < –2SD)

  • BMIZ

Biochemical indicators:

  • Anaemia

Cognitive function and development:

  • Cognitive test scores (verbal, cognitive, behavioural)

Morbidity – Child

  • Illness during past 4 weeks

8, 12, 15, 20 months, 10 years

Evans 2014

(Tanzania)

cRCT

High

Programme name: N/A

Amount and frequency of payments: USD 12–36, depending on the number of people in the HH, every 2 months.

Conditionalities: education: primary school enrolment and attendance for children aged 7–15 years; health: health facility visits for growth monitoring 6 times a year for children aged 0–5 years; vaccination and growth monitoring for children 0–2 years; yearly visit to health facility for elderly people (aged ≥ 60 years).

Provider: Tanzania Social Action Fund (TASAF), World bank

Delivery: payments disbursed by TASAF to bank accounts managed by local government authorities. Funds disbursed directly to community‐managed accounts who made payments to mothers.

Co‐interventions: transfers from government/TASAF or from NGOs/religious organisation

Poor HHs with vulnerable children or elderly people, or both

(80 villages; 40/40)

Anthropometric indicators: NR

30 and 42 month

Hidrobo 2014

(Colombia)

cRCT

High

Programme name: N/A

Amount and frequency of payments: USD 40 per month per HH.

Conditionalities: attendance of monthly nutrition sensitisation training sessions by HH members.

Provider: World Food Programme (NPO)

Delivery: money transferred on to pre‐programmed debit cards.

Co‐interventions: NR

Poor urban HHs (2357 HHs)

HH expenditure on food:

  • Proportion of total expenditure per month

Dietary diversity:

  • DDI

  • HDDS

  • FCS

7 months

Kandpal 2016

(Philippines)

cRCT

High

Programme name: Pantawid Pamilyang Pilipino Programme

Type, amount and frequency of payments: health grant of PHP 500 (USD 11) per HH per month; education grant of PHP 300 (USD 6.50) per child per month for ≤ 10 months/year, and for ≤ 3 children in the HH. Payments every 2 months.

Conditionalities: health: clinic visits for immunisation and vaccination, growth monitoring, and management of childhood disease in children aged < 5 years; antenatal care for pregnant women, starting from the first trimester; school‐aged children (6–14 years) to receive deworming tablets 2 times/year; and HHs with children 0–14 years, the HH grantee (mother) or spouse (or both) had to attend family development sessions monthly. Education: enrolment of children aged 6–14 years in primary or secondary school and 85% school attendance every month.

Provider: government

Delivery: NR

Co‐interventions: NR

Poor HHs with children aged 0–14 years or pregnant women (714/ 704 HHs)

Anthropometric indicators:

  • WAZ

  • Underweight (WAZ < –2SD)

  • Severely underweight (WAZ < –3SD)

  • HAZ

  • Stunted (HAZ < –2SD)

  • Severely stunted (HAZ < –3SD)

Morbidity – child:

  • Seeking treatment for child for fever, cough or diarrhoeal disease in past 2 weeks

36 months

Kurdi 2019

(Yemen)

cRCT

High

Programme name: Cash for Nutrition programme

Amount and frequency of payments: payments every 3 months (YER 30,000 per month for 9 months in 2015; YER 10,000 (USD 30) per month for 12 months in 2016/2017) to mothers of children aged 2 years of age and pregnant women.

Conditionalities: attending monthly nutrition‐focused trainings, complying with child monitoring and treatment of malnutrition. Attendance tracked but conditionality not strictly enforced.

Provider: government, Yemen Emergency Crisis Response Project (funded by the World Bank)

Delivery: nutrition sessions delivered by trained local women. Details of cash transfer not reported.

Co‐interventions: unspecified other food distribution programmes.

Women from poor and vulnerable (1001/999 women)

Diet diversity:

  • HDDS

Anthropometric indicators:

  • HAZ

  • WHZ

2.5 years

Andersen 2015

(Peru)

Prospective controlled study

High

Programme name: Juntos

Amount and frequency of payments: PEN 100 (30 US dollars) each month regardless of HH composition.

Conditionalities: regular health visits for children aged < 5 years, or pregnant and lactating women. Children aged 6–14 years with primary school attendance ≥ 85%.

Provider: Peruvian government

Delivery: NR

Co‐interventions: NR

Poor HHs with children aged 6–18 months (374/586 children)

Anthropometric indicators:

  • HAZ

  • Stunting (HAZ < –2SD)

  • BMIZ

Cognitive function and development:

  • Language (TVIP) score

  • Grade attainment

Adverse effects:

  • Overweight (BMIZ > 2SD)

< 2 years and ≥ 2 years

Ferre 2014

(Bangladesh)

Prospective controlled study

High

Programme name: Shombhob project

Amount and frequency of payments: BDT 400 per months for HHs with children 0–36 months and BDT 400 per month for HHs with primary school children (6–15 years).

Conditionalities: Health: Attending growth monitoring of children aged 0 – 36 months, and nutrition session for mother/carer. Education: school attendance of at least 80% every month.

Provider: Government

Delivery: Cash cards provided to beneficiary mothers. Electronic transfer to their accounts with the Bangladesh Post Office (BPO). Withdrawal from mobile machines on a designated day during each payment cycle in each village, or from Upazila BPO branch office at any time point.

Rural HHs (700/1587)

HH expenditure on food:

  • Proportion of total expenditure

Dietary diversity:

  • MDD

Anthropometric indicators:

  • Stunting (HAZ < –2SD)

  • Wasting (WHZ < –2SD)

  • Underweight (WAZ < –2SD)

13 months

Huerta 2006 (PROGRESA) (Mexico)

Prospective controlled study

High

Programme name: Oportunidades (previously known as PROGRESA)

Type, amount and frequency of payments: SeeGertler 2000 (PROGRESA)

Conditionalities: seeGertler 2000 (PROGRESA)

Provider: Mexican government

Delivery: seeGertler 2000 (PROGRESA)

Co‐interventions: NR

Poor rural HHs with ≥ 1 child aged < 5 years (205/142 communities)

Anthropometric indicators:

  • LAZ or HAZ

  • WAZ

  • WLZ or WHZ

Biochemical indicators:

  • Anaemia

  • Hb

Morbidity – child:

  • Respiratory infection during the past 2 weeks

  • Diarrhoeal disease during the past 2 weeks

14 and 26 months

Leroy 2008 (PROGRESA)

(Mexico)

Prospective controlled study

High

Programme name: Oportunidades (previously known as PROGRESA)

Type, amount and frequency of payments: USD 32.5–41.3 per month (see Gertler 2000 (PROGRESA))

Conditionalities: seeGertler 2000 (PROGRESA)

Provider: government of Mexico

Delivery: see Gertler 2000 (PROGRESA)

Co‐interventions: NR

Poor and vulnerable urban HHs

(733 children aged 0–24 months)

Anthropometric indicators:

  • HAZ

  • WHZ

2 years

Lopez Arana 2016

(Colombia

Prospective controlled study

High

Programme name: Familias en Acción

Type, amount and frequency of payments:

COP 40,000 for children aged < 7 years; COP 14,000 per primary school and COP 28,000 per secondary school child. Periodic payments.

Conditionalities: children aged < 7 years to attend vaccination programmes and growth and development check‐ups regularly; children aged 7–17 years to attend ≥ 80% of school lessons.

Provider: government, World Bank and Inter‐American Development Bank

Delivery: transfer of cash to mothers into the HH bank account.

Co‐interventions: some children participated in a childcare supplementary nutrition and psychosocial stimulation programme (Hogares Comunitarios programme).

Poor HHs with children aged 0–17 years (9293/4424)

Anthropometric indicators:

  • HAZ

  • Stunting (HAZ < –2SD)

  • BMIZ

  • Thinness (BMIZ < –2SD)

Adverse events:

  • Overweight (BMIZ > 1)

  • Obesity (BMIZ > 2)

About 4 years

aOverall Risk of Bias based on risk of selection and attrition bias.

BMIZ: body mass index‐for‐age z‐score; CES‐D: Center for Epidemiologic Studies Depression Scale; COP: Colombian peso; cRCT: cluster randomised controlled trial; DDI: Dietary Diversity Index; FCS: Food Consumption Score; GHQ‐12: 12‐item General Health Questionnaire; HAZ: height‐for‐age z‐score; Hb: haemoglobin; HDDS: Household Dietary Diversity Score; HH: household; LAZ: length‐for‐age z‐score; MXN: Mexican peso; N/A: not applicable/available; non‐governmental organisation; NPO: non‐profit organisation; NR: not reported; PEN: Yemeni rial; PHP: Philippine peso; TVIP: Test de Vocabulario en Imagenes Peabody; WAZ: weight‐for‐age z‐score; WHZ: weight‐for‐height z‐score; WLZ: weight‐for‐length z‐score; YER: Yemeni rial.

Figures and Tables -
Table 11. Conditional cash transfers – overview of included studies
Table 12. Conditional cash transfers – results of included trials

Study ID (risk of bias)

Study design (n)

Conditional cash transfers

No intervention

Effect measure (time point)

Effect directiona

Meta‐analysis (yes/no)

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

2.2: Proportion of HH expenditure on food

2.2.1 Outcome measure: proportion of HH expenditure on food (weekly/monthly)

Maluccio 2005 (+)

cRCT (1490 HHs)

69.8

70

766

70.2

66.5

724

pp 3.9, SE 1.7, 95% CI 0.568 to 7.232, P < 0.01 (1 year)

N/A

cRCT (1434 HHs)

722

712

pp 4.1, SE 1.3, 95% CI 1.552 to 6.648, P < 0.01 (2 years)

2.2.2 Outcome measure: proportion of food in total expenditures (SDs)

Macours 2012 (+)

cRCT (3326 HHs)

70%

70.7%

Effect 0.005, SD, SE 0.009, 95% CI –0.013 to 0.023, P > 0.1 (9 months)

2.3: Proportion of HHs who were food secure

2.3.2 Dietary diversity

2.3.2.2 Outcome measure: HDDS (012) (mean)

Hidrobo 2014 (‐)

cRCT (2087 HHs)

9.23

9.11

MD 0.46, SE 0.11, 95% CI 0.244 to 0.676, P < 0.01 (7 months)

Yes

Kurdi 2019 (‐)

cRCT (1850 HHs)

935 HHs

915 HHs

MD 0.374, SE 0.262, 95% CI –0.13952 to 0.88752 (2.5 years)

Secondary outcomes

2.5Change in anthropometric indicators

2.5.1 Stunting (chronic undernutrition)

2.5.1.1 Outcome measure: proportion stunted (HAZ < –2SD)

Maluccio 2005 (+)

cRCT (722 children aged < 5 years)

41.9

37.1

40.9

41.5

pp –5.3, 95% CI –11.376 to 0.776, P < 0.1 (2 years)

Yes; this subset.

This was entered as MD: difference in percentage stunted

Gertler 2000 (PROGRESA) (?)

cRCT (n at follow‐up 1062)

0.396

0.410

OR 0.914, P = 0.495 (20 months)

Kusuma 2017a (?)

cRCT (1394 children aged 24–36 months)

mean 0.55

DID 0.035, SE 0.046, 95% CI –0.05516 to 0.12516

pp 3.5, 95% CI –5.5 to 12.5, P > 0.05 (2 years)

Yes; this subset.

This was entered as MD: difference in percentage stunted

Kandpal 2016 (‐)

cRCT (351 children aged < 36 months)

49.701

pp –3.768, 95% CI –13.830 to 6.294, P > 0.1 (36 months)

2.5.1.2 Outcome measure: proportion with severe stunting (HAZ < –3SD)

Kusuma 2017a (?)

cRCT (1394 children aged 24–36 months)

mean 0.29

DID 0.047, SE 0.053, 95% CI –0.05688 to 0.15088.

pp 4.7, 95% CI –5.7 to 15.1, P > 0.05 (2 years)

Yes

Kandpal 2016 (‐)

cRCT (351 children aged < 36 months)

–10.189

23.952

pp –10.189, 95% CI –18.769 to –1.607 (3 years)

2.5.1.3 Outcome measure: HAZ (mean, SD)

Maluccio 2005 (+)

cRCT (1036 children aged < 5 years)

–1.79 (1.14)

–1.65 (1.15)

479

–1.76 (1.15)

–1.80 (1.18)

557

MD 0.17, 95% CI 0.0132 to 0.327, P < 0.05 (2 years)

Yes

Macours 2012 (+)

cRCT (3082 children aged < 6 years)

–1.27b

–1.08b

MD 0.072, 95% CI 0.005 to 0.139, P < 0.05 (9 months)

Evans 2014 (‐)

cRCT (102 children aged 0–4 years)

MD 0.86, 95% CI –2.358 to 3.718, P > 0.1 (1.5 years)

Kandpal 2016 (‐)

cRCT (351 children)

0.284

–1.903

MD 0.284, 95% CI –0.034 to 0.600, P < 0.1 (3 years)

Kurdi 2019 (‐)

cRCT (1048 children)

MD 0.109, SE 0.146, 95% CI –0.18 to 0.395 (2.5 years)

2.5.2Wasting (acute undernutrition)

2.5.2.1 Outcome measure: proportion wasted (WHZ < –2SD)

Maluccio 2005 (+)

cRCT (722 children aged < 5 years)

1.0%

0.4%

479

0.3

0.2

557

pp –0.4, SE 0.5, 95% CI –1.38 to 0.58, P > 0.1 (2 years)

Yes

Kusuma 2017a (?)

cRCT (1394 children aged 24–36 months)

Mean 0.19

DID –0.063, SE 0.032, 95% CI –0.12572 to –0.00028, P < 0.05

2.5.2.2 Outcome measure: proportion severely wasted (WHZ < –3SD

Kusuma 2017a (?)

cRCT (1394 children aged 24–36 months)

Mean 0.09

Beta –0.037, SE 0.022, 95% CI –0.08012 to 0.00612, P < 0.1

2.5.2.3 Outcome measure: WHZ (mean, SD)

Evans 2014 (‐)

cRCT (63 children aged 0–4 years)

MD –0.03, SE 0.45, 95% CI –0.9120 to 0.852, P > 0.1 (1.5 years)

Yes

Kurdi 2019 (‐)

cRCT (1048 children)

MD 0.190, SE 0.148, 95% CI –0.10008 to 0.48008 (2.5 years)

2.5.3 Underweight

2.5.3.1 Outcome measure: proportion underweight (WAZ < –2SD)

Maluccio 2005 (+)

cRCT (722 children aged < 5 years)

15.3

10.4

14.7

15.8

pp –6, SE 2.6, P < 0.05 (2 years)

Yes

Kusuma 2017a (?)

cRCT (1394 children aged 24–36 months)

Mean 0.38

DID –0.040, SE 0.036, 95% CI –0.11056 to 0.03056, P > 0.05

Kandpal 2016 (‐)

cRCT (390 children aged < 36 months)

28.72

pp –2.57, 95% CI –11.980 to 6.839 (3 years)

2.5.3.2 Outcome measure: proportion severely underweight (WAZ < –3SD)

Kusuma 2017a (?)

cRCT (1394 children aged 24–36 months)

Mean 0.10

DID –0.025, SE 0.024, 95% CI –0.07204 to 0.02204

Kandpal 2016 (‐)

cRCT (390 children aged < 36 months)

8.51

pp 1.075, 95% CI –4.72 to 6.87, P > 0.1 (3 years)

Yes

2.5.3.3 Outcome measure: weight‐for‐age z‐score (WAZ) (mean standard deviation)

Macours 2012 (+)

cRCT (3082 children aged < 6 years)

–1.06

–0.88

MD 0.036, SE 0.037, 95% CI –0.037 to 0.109, P > 0.1 (9 months)

Yes

Evans 2014 (‐)

cRCT (76 children 0–4 years)

MD –0.29, SE 1.25, 95% CI –2.74 to 2.16, P > 0.1 (1.5 years)

Kandpal 2016 (‐)

cRCT (390 children < 36 months)

0.14

MD 0.140, 95% CI –0.161 to 0.438, P > 0.1 (3 years)

2.5.3.4 Outcome measure: BMI‐for‐age z‐score

Evans 2014 (‐)

cRCT (64 children aged 0–4 years)

MD –1.55, 95% CI –4.43 to 1.33, P > 0.1 (1.5 years)

2.7 Cognitive function and development

2.7.1 Outcome measure: cognitive test scores/cognitive and socioemotional outcomes (mean, SD)

Macours 2012 (+)

cRCT (3326 children)

MD 0.1211, SE 0.028, 95% CI 0.066 to 0.176 P < 0.01 (9 months)

Yes

Baird 2013 (+)

cRCT (2057 schoolgirls)

MD 0.174, 95% CI 0.0799 to 0.268, SE 0.048, P < 0.01 (2 years)

2.8 Change in proportion of anxiety and depression

2.8.1 Outcome measure: proportion with psychological distress

Baird 2013 (+)

cRCT (2089 schoolgirls)

Mean 0.374, SE 0.02, P < 0.01

pp –0.063, SE 0.03, P < 0.05 (1 year)

N/A

Mean 0.308, SE 0.017, P < 0.01

pp –0.039, SE 0.047, P > 0.1 (2 years)

2.9 Morbidity

2.9.1 Outcome measure: proportion reporting being ill in past 4 weeks/parents seeking care for illness in past 2 weeks

Gertler 2000 (PROGRESA) (?)

cRCT (7703 children aged 0–35 months)

OR 0.777, P = 0.000 (20 months)

Yes. Gertler subgroup 3–5 years selected as converting OR to SMD not possible due to missing group sizes.

cRCT (19,939 children aged 3–5 years at baseline)

0.280

0.097

0.263

0.127

Estimate –0.021, 95% CI –0.045 to 0.003 (20 months)

Evans 2014 (‐)

cRCT (18,192 participants)

Estimate –0.04, 95% CI –0.099 to 0.019, P > 0.1 (32 months)

Kandpal 2016 (‐)

cRCT (456 children aged 6–36 months)

229

41.85

227

pp 9.830, 95% CI 0.179 to 19.481, P < 0.05 (36 months)

2.9.2 Outcome measure: number of days ill in bed (SD)

Macours 2012 (+)

cRCT (3326 children)

0.669

MD –0.357 SD, SE 0.133, 95% CI –0.6178 to –0.096, P < 0.01 (9 months)

2.9.3 Outcome measure: proportion with anaemia

Gertler 2000 (PROGRESA) (?)

cRCT (2010 children)

0.410

0.483

OR 0.745, P = 0.012 (20 months)

aEach triangle represents one study.
bValues derived from graphs

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

CI: confidence interval; cRCT: cluster randomised controlled trial; DID: difference in differences; HAZ: height‐for‐age z‐score; HDDS: Household Dietary Diversity Score; HH: household; MD: mean difference; n: number; N/A: not applicable/available; OR: odds ratio; pp: percentage point; SD: standard deviation; SE: standard error; SMD: standardised mean difference; WAZ: weight‐for‐age z‐score; WHZ: weight‐for‐height z‐score.

Figures and Tables -
Table 12. Conditional cash transfers – results of included trials
Table 13. Conditional cash transfers – results of included prospective controlled studies

Study ID (risk of bias)

Study design (n)

Conditional cash transfers

No intervention

Effect measure (time point)

Effect directiona

Meta‐analysis (yes/no)

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

2.2 Proportion of HH expenditure on food

2.2.1 Outcome measure: proportion of HH expenditure on food (weekly/monthly)

Ferre 2014 (‐)

PCS (n NR)

3168/5548 = 0.57

3153/5780 = 0.55

Proportion after study period is 337.0/378.8 = 0.89 (not impact) (13 months)

N/A

2.3: Proportion of HHs who were food secure

2.3.2 Dietary diversity

2.3.2.1 Proportion with MDD

Ferre 2014 (‐)

Prospective controlled study (n = 1318 children)

12.1

12.5

MD 0.031, SE 0.05, 95% CI –0.067 to 0.129 (13 months)

Secondary outcomes

2.5Change in anthropometric indicators

2.5.1 Stunting (chronic undernutrition)

2.5.1.1 Outcome measure: proportion stunted (HAZ < –2SD)

Ferre 2014 (‐)

Prospective controlled study (1580 children)

47.2

43.3

MD 0.034, SE 0.05, 95% CI –0.064 to 0.132 (13 months)

Yes. Subset. (except Lopez‐Arana as OR could not be converted to SMD due to missing group sizes)

Andersen 2015 (‐)

Prospective controlled study (n = 188 children)

91 (48.4%)

72 (38.3%)

80 (42.6%)

76 (40.4%)

Treatment effect: –7.98, 95% CI –22.3 to 6.34, P = 0.27 (< 2 years)

Prospective controlled study (n = 169 children)

101 (59.8%)

67 (39.6%)

84 (49.7%)

81 (47.9%)

Treatment effect –18.3, 95% CI –38.3 to 1.59, P = 0.07 (≥ 2 years)

Lopez Arana 2016 (‐)

Prospective controlled study (2874 children)

391 (30.3%)

442 (27.9%)

OR 0.92, 95% CI 0.82 to 1.05, P > 0.05 (4 years)

2.5.1.2 Outcome measure: height‐for‐age z‐score (HAZ) (mean, SD)

Leroy 2008 (PROGRESA) (‐)

Prospective controlled study (432 children)

–1.29 (1.36)

–1.4 (1.16)

MD 0.1, 95% CI –0.086 to 0.306, P = 0.13 (2 years)

Yes

Andersen 2015 (‐)

Prospective controlled study (n = 188 children)

–1.97 (1.1)

–1.76 (0.864)

–1.80 (1.02)

–1.71 (0.757)

MD 0.12, 95% CI –0.10 to 0.33, P = 0.28 (< 2 years)

Prospective controlled study (n = 169 children)

–2.11 (1.24)

–1.85 (0.829)

–2.08 (1.12)

–1.95 (0.813)

MD 0.14, 95% CI –0.20 to 0.49, P = 0.41 (≥ 2 years)

Lopez Arana 2016 (‐)

Prospective controlled study (2874 children)

–1.47 (1.21)

–1.42 (1.13)

MD 0.00, 95% CI –0.10 to 0.11, P > 0.05 (4 years)

2.5.2:Wasting (acute undernutrition)

2.5.2.1 Outcome measure: proportion wasted (WHZ < –2SD)

Ferre 2014 (‐)

Prospective controlled study (2244 children)

27.8

22.9

MD/DID –0.036, SE 0.04, 95% CI –0.1144 to 0.0424 (ages 22–46 months when enrolled)

MD –0.125, SE 0.07, 95% CI –0.2622 to 0.0122 (aged 10–22 months when enrolled) pp –12.5

(13 months)

No. Lopez‐Arana/Ferre 2014 could not be converted to SMD due to missing group sizes.

Lopez Arana 2016 (‐)

Prospective controlled study (2874 children)

25 (1.9%)

14 (0.9%)

OR 0.25, 95% CI 0.09 to 0.74, P < 0.05 (4 years)

2.5.2.2 Outcome measure: WHZ (mean, SD)

Leroy 2008 (PROGRESA) (‐)

Prospective controlled study (432 children)

0.30 (1.07)

0.33 (1.00)

MD 0.085, 95% CI –0.113 to 0.283, P = 0.2 (2 years)

2.5.3 Underweight

2.5.3.1 Outcome measure: proportion underweight (WAZ <2SD)

Ferre 2014 (‐)

Prospective controlled study (1638 children)

47.1

42.9

MD/DID 0.046, SE 0.05, 95% CI –0.052 to 0.144

pp 4.6 (13 months)

N/A

2.5.3.2 Outcome measure: BMIZ (mean, SD)

Andersen 2015 (‐)

Prospective controlled study (n = 188 children)

0.527 (1.15)

0.145 (0.833)

0.790 (0.986)

0.436 (0.739)

MD –0.028, 95% CI –0.31 to 0.25, P = 0.84 (< 2 years)

Yes

Prospective controlled study (n = 169 children)

0.613 (1.23)

0.248 (0.788)

0.622 (1.3)

0.622 (0.773)

MD –0.36, 95% CI –0.79 to 0.06, P = 0.09 (≥ 2 years)

Lopez Arana 2016 (‐)

Prospective controlled study (2874 children)

MD 0.14, 95% CI 0.00 to 0.27, P < 0.05 (4 years)

2.7 Cognitive function and development

2.7.1 Outcome measure: language score (TVIP) (mean, SD)

Andersen 2015 (‐)

Prospective controlled study (n = 243 children)

–0.538 (0.782)

–0.718 (0.959)

–0.531 (0.761)

–0.552 (1.03)

Coefficient –0.15, 95% CI –0.37 to 0.066, P = 0.17 (≥ 2 years)

N/A

2.10: Adverse outcomes: overweight/obesity

2.10.1 Outcome measure: overweight (BMI z‐score >2SD)

Andersen 2015 (‐)

Prospective controlled study (n = 188 children)

n = 65, 34.6%

n = 24, 12.8%

n = 81, 43.1%

n = 34, 18.1%

pp 3.19, 95% CI –9.93 to 16.3, P = 0.63 (< 2 years)

Yes

Prospective controlled study (n = 169 children)

n = 65, 37.9%

n = 28, 16.6%

n = 64, 37.9%

n = 42, 24.9%

pp –8.89, 95% CI –24.7 to 7.0, P = 0.27 (≥ 2 years); log OR –0.2784

Lopez Arana 2016 (‐)

Prospective controlled study (2874 children)

OR 1.30, 95% CI 0.83 to 2.03, P > 0.05 (4 years)

2.10.2 Obesity

Lopez Arana 2016 (‐)

Prospective controlled study (2874 children)

41 (3.2%)

37 (2.3%)

OR 0.56, 95% CI 0.20 to 1.53, P > 0.05

aEach triangle represents one study.

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

BMIZ: body mass index‐for‐age z‐score; CI: confidence interval; DID: difference in differences; HAZ: height‐for‐age z‐score; HH: household; MD: mean difference; n: number; N/A: not applicable/available; NR: not reported; OR: odds ratio; PCS: prospective controlled study; SD: standard deviation; SE: standard error; TVIP: Test de Vocabulario en Imagenes Peabody; WAZ: weight‐for‐age z‐score; WHZ: weight‐for‐height z‐score.

Figures and Tables -
Table 13. Conditional cash transfers – results of included prospective controlled studies
Table 14. Income‐generation interventions – results of included trials

Study ID (risk of bias)

Study design (n)

Income‐generation interventions

No intervention

Effect measure (time point)

Effect direction

Meta‐analysis (yes/no)

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

3.3 Proportion of HHs who were food secure

3.3.1 Food security

3.3.1.1 Outcome measure: proportion experiencing food security (based on HFIAS)

Osei 2017 (?)

cRCT (2614 HHs)

79.7, 95% CI 77.2 to 82.0

53.6, 95% CI 51.0 to 56.1

87.4, 95% CI 85.3 to 89.3

78.3, 95% CI 76.0 to 80.4

— (2.5 years)

No n for individual groups to calculate MD.

3.3.1.2 Outcome measure: HH food security score (mean, SD)

Beegle 2017 (‐)

RCT (2193 HHs)

1083 HHs

–3.12 (1.29)

1110 HHs

MD –0.060, SE 0.080, 95% CI –0.2168 to 0.0968 (3/4 months)

3.3.1.3 Outcome measure: Resilience Index (mean, SD)

Beegle 2017 (‐)

RCT (2195 participants)

–9.32 (9.84)

MD –0.224, SE 0.630 (3/4 months)

3.3.1.4 Outcome measure: Principal Components Analysis index (mean, SD)

Beegle 2017 (‐)

RCT (2123 participants)

0.15 (2.08)

MD –0.029, SE 0.135 (3/4 months)

3.3.2 Dietary diversity

3.3.2.1 Outcome measure: odds of consuming an additional food group based on the DDS

Darrouzet Nardi 2016 (?)

(DDS 0–7)

cRCT (2584 children)

OR 1.524, 95% CI 1.45 to 4.38, P = 0.001 (2 years)

3.3.2.2 Outcome measure: HDDS (mean, SD)/Food Consumption Score

Olney 2016 (?)

(HDDS 0–11)

cRCT (1476 HHs)

5.6 (1.93)

5.6 (2.07)

880 HHs

5.8 (1.70)

5.2 (2.11)

596 HHs

MD 0.7, SE 0.44, 95% CI –0.1624 to 1.5624, P = 0.17 (2 years)

Yes. SMD.

Beegle 2017 (‐)

(FCS 0–126)

RCT (2201 HHs)

1191 HHs

38.82 (16.01)

1110 HHs

MD –0.708, SE 1.072, 95% CI –2.80912 to 1.39312 (3/4 months)

3.3.2.3 Outcome measure: MDD (n, %)

Marquis 2018 (+)

cRCT (428 children)

30.9

80.2

247

33.8

69.5

181

OR 1.65, SE 0.41, 95% CI 0.8464 to 2.4536, P < 0.05 (12 months)

Yes. Olney groups combined.

Darrouzet Nardi 2016 (?)

cRCT (2604 children)

OR 1.146, 95% CI 1.02 to 1.29, P = 0.021 (2 years)

Olney 2016 (?)

cRCT (758 children)

OWL: 7 (3.0)

HC: 4 (1.7)

OWL: 35 (15.0)

HC: 43 (18.2)

OWL: 220

HC: 231

8 (2.6)

20 (6.3)

307

OWL villages vs control: pp 8.3, P = 0.17

HC villages vs control: pp 12.6, P = 0.08

(2 years)

Combined effect: MD pp 10.08, 95% CI 1.02 to 19.14

Secondary outcomes

3.5Change in anthropometric indicators

3.5.1 Stunting

3.5.1.1 Outcome measure: Height‐for‐Age z‐score (HAZ) (mean, SD or SE)

Marquis 2018 (+)

cRCT (428 children)

–0.88 (1.27)

247

–0.78 (1.30)

181

MD 0.22, SE 0.06, P < 0.01, 95% CI 0.10 to 0.34 (12 months)

No. Effect sizes calculated for Darrouzet (2 years) and Osei from group estimates.

Darrouzet Nardi 2016 (?)

cRCT (303 children)

–1.47 (0.07)

–1.38 (0.06)

–1.48 (0.06)

–1.41 (0.06)

MD 0.109, 95% CI 0.000 to 0.218, P = 0.048 (12 months)

609 children

–1.47 (0.07)

–1.30 (0.06)

305

–1.48 (0.06)

–1.33 (0.06)

304

MD 0.03, SE 0.0049, 95% CI 0.020 to 0.040 (2 years)

Osei 2017 (?)

cRCT (2569 children)

–2.23 (0.03)

–2.1 (0.03)

1299

–2.4 (0.04)

–2.32 (0.03)

1297

MD 0.22, SE 0.0012, 95% CI 0.218 to 0.222 (2.5 years)

3.5.1.2 Outcome measure: proportion stunted (HAZ < –2SD) (CI)

Osei 2017 (?)

cRCT (2569 children)

57.7

55.1

1299

65.8

63.5

1297

OR 0.94, 95% CI 0.74 to 1.19 (2.5 years)

Yes. Verbowski groups combined

Verbowski 2018 and aquaculture (?)

cRCT (597 children)

27.9

29.9

299

29.3

32.0

298

MD pp –0.62, P = 0.927 (1.8 years)

MD pp 2.2, 95% CI –5.64 to 10.05

Verbowski 2018 (?)

cRCT (598 children)

22.7

28.9

300

29.3

32.0

298

MD pp 3.73, P = 0.453 (1.8 years)

3.5.2Wasting

3.5.2.1 Outcome measure: WHZ (mean, SD or SE)

Marquis 2018 (+)

cRCT (428 children)

–0.37 (1.08)

247

–0.31 (1.24)

181

MD 0.07, SE 0.08, 95% CI –0.087 to 0.227, P > 0.10 (12 months)

No. Effect for Osei calculated from group estimates.

Osei 2017 (?)

cRCT (2603 children)

–0.91 (0.03)

–0.85 (0.03)

1300

–0.93 (0.03)

–0.71 (0.03)

1303

MD –0.14, SE 0.0012, 95% CI –0.142 to –0.138 (2.5 years)

3.5.2.2 Outcome measure: proportion wasted (WHZ < –2SD)

Osei 2017 (?)

cRCT (2603 children)

10.6

10.5

1300

10.1

9.7

1303

OR 1.03, 95% CI 0.70 to 1.52 (2.5 years)

Yes. Verbowski groups combined.

Verbowski 2018 and aquaculture (?)

cRCT (597 children)

6.7

10.2

299

8.3

8.9

298

MD pp 2.75, P = 0.424 (22 months)

MD pp 3.19, 95% CI –1.95 to 8.33

Verbowski 2018 (?)

cRCT (598 children)

8.4

13.0

300

8.3

8.9

298

MD pp 3.80, P = 0.348 (22 months)

3.5.3 Underweight

3.5.3.1 Outcome measure: Weight‐for‐age z‐score (WAZ) (mean, SD or SE)

Marquis 2018 (+)

cRCT (428 children)

–0.78 (1.12)

247

–0.68 (1.27)

181

MD 0.15, SE 0.07, P < 0.05 (12 months)

Yes. Effect estimates calculated using group estimates.

Darrouzet Nardi 2016 (?)

cRCT (634 children)

–2.04 (0.07)

–1.97 (0.06)

301

–1.94 (0.06)

–1.89 (0.06)

333

NR (1 year)

–2.04 (0.07)

–1.97 (0.06)

–1.94 (0.06)

–2.07 (0.06)

MD 0.10, 95% CI 0.09 to 0.11 (2 years)

Osei 2017 (?)

cRCT (2613 children)

–1.87 (0.03)

–1.77 (0.03)

1306

–1.97 (0.03)

–1.77 (0.03)

1307

MD 0.00, 95% CI –0.00 to 0.00 (2.5 years)

3.5.3.2 Outcome measure: percentage underweight (WAZ < 80% standard/ < –2SD) (includes severe underweight)

Osei 2017 (?)

cRCT (2613 children)

43.4

41.0

1306

48.0

40.6

1307

OR 1.15, 95% CI 0.91 to 1.46 (2.5 years)

Yes. Verbowski groups combined.

Verbowski 2018 and aquaculture (?)

cRCT (597 children)

23.5

32.0

299

23.0

28.8

298

MD pp 2.75, P = 0.670 (22 months)

MD pp –1.16, 95% CI –9.02 to 6.70

Verbowski 2018 (?)

cRCT (598 children)

26.1

28.8

300

23.0

28.8

298

MD pp –3.63, P = 0.479 (22 months)

3.5.3.3 Outcome measure: BMI (kg/m2) (mean, SD or SE)

Olney 2016 (?)

cRCT (1297 women)

20.2 (2.22)

20.7 (2.34)

787

20.6 (2.27)

21.1 (2.70)

510

MD 0.2, 95% CI –0.192 to 0.592, SE 0.20, P = 0.26 (2 years)

Yes. Effect estimate for Osei calculated from group estimates.

Osei 2017 (?)

cRCT (2614 mothers)

19.6 (0.07)

19.8 (0.05)

1182

20.1 (0.06)

19.9 (0.05)

1303

MD –0.10, 95% CI –0.10 to –0.10 (2.5 years)

3.5.3.4 Proportion of women who were underweight (BMI < 18.5 kg/m2)

Olney 2016 (?)

cRCT (1297 women)

23

15

787

15

16

510

pp –8.7, P = 0.01 (2 years)

No. Verbowski groups combined.

Osei 2017 (?)

cRCT (2614 mothers)

28.2

28.6

1182

17.5

19.9 (0.05)

1303

OR 0.61, 95% CI 0.46 to 0.82 (2.5 years)

Verbowski 2018 and aquaculture (?)

cRCT (541 women)

14.2

9.0

270

16.6

9.4

271

MD pp 1.19, P = 0.920 (22 months)

MD 3.88, 95% CI –4.36 to 12.12

Verbowski 2018 (?)

cRCT (541 women)

13.4

13.5

270

16.6

9.4

271

MD pp 4.27, P = 0.347 (22 months)

3.6 Change in biochemical indicators

3.6.1 Mean haemoglobin concentration (children) (mean, SE)

Osei 2017 (?)

cRCT (2614 children)

115.3 (0.1)

114.3 (0.1)

1307

113.6 (0.1)

110.8 (0.1)

1307

MD 3.5, SE 0.0039, 95% CI 3.492 to 3.507 (2.5 years)

Yes. Verbowski groups combined.

Verbowski 2018 and aquaculture (?)

cRCT (597 children)

104.5 (13.7)

108.4 (13.1)

298

105.7 (13.6)

107.1 (12.9)

299

MD 2.54, SE 1.43, P = 0.076 (22 months)

MD 2.48, 95% CI 0.51 to 4.46

Verbowski 2018 (?)

cRCT (597 children)

104.1 (13.8)

108.0 (12.3)

298

105.7 (13.6)

107.1 (12.9)

299

MD 2.43, SE 1.42, P = 0.088 (22 months)

3.6.2 Mean haemoglobin concentration (women) (mean, SD or SE)

Osei 2017 (?)

cRCT (2614 mothers)

129.3 (0.1)

126.5 (0.1)

1307

129.6 (0.1)

121.9 (0.1)

1307

MD 4.6, SE 0.0039, 95% CI 4.592 to 4.608 (2.5 years)

Yes. Verbowski groups combined.

Verbowski 2018 and aquaculture (?)

cRCT (541 women)

122.4 (12.1)

122.9 (12.9)

270

121.5 (12.5)

121.1 (12.1)

271

MD 0.49, SE 1.33, P = 0.714 (22 months)

MD –0.07, 95% CI –1.92 to 1.78

Verbowski 2018 (?)

cRCT (541 women)

121.7 (13.7)

121.0 (11.9)

270

121.5 (12.5)

121.1 (12.1)

271

MD –0.63, SE 1.34, P = 0.637 (22 months)

3.9 Morbidity

3.9.1 Prevalence of anaemia (children)

Osei 2017 (?)

cRCT (2614 children)

28.2

30.8

1307

31.6

42.5

1307

OR 0.76, 95% CI 0.59 to 0.98 (2.5 years)

Yes. Verbowski groups combined.

Verbowski 2018 and aquaculture (?)

cRCT (597 children)

63.1

54.3

298

59.2

59.5

299

MD pp –9.74, P = 0.119 (22 months)

MD pp –11.90, 95% CI –20.47 to –3.33

Verbowski 2018 (?)

cRCT (597 children)

65.4

52.6

298

59.2

59.5

299

MD pp –14.0, P = 0.023 (22 months)

3.9.2 Prevalence of anaemia (women)

Osei 2017 (?)

cRCT (2614 mothers)

19.6

24.6

1307

21.1

35.8

1307

OR 0.62, 95% CI 0.48 to 0.82 (2.5 years)

Yes. Verbowski groups combined.

Verbowski 2018 and aquaculture (?)

cRCT (541 women)

38.9

35.8

270

40.4

38.7

271

MD pp –1.10, P = 0.865 (22 months)

MD pp 1.34, 95% CI –7.94 to 10.61

Verbowski 2018 (?)

cRCT (541 women)

41.9

43.5

270

40.4

38.7

271

MD pp 4.14, P = 0.551 (22 months)

aEach triangle represents one study.

(+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias. = Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0.

BMI: body mass index; cRCT: cluster randomised controlled trial; CI: confidence interval; DDS: Dietary Diversity Score; EHFP: enhanced homestead food production; FCS: Food Consumption Score; HAZ: height‐for‐age z‐score; HC: health committee; HDDS: Household Dietary Diversity Score; HFIAS: Household Food Insecurity Score; HH: household; MD: mean difference; MDD: Minimum Dietary Diversity; n: number; NR: not reported; OR: odds ratio; OWL: older women leaders; pp: percentage point; SD: standard deviation; SE: standard error; SMD: standardised mean difference; WAZ: weight‐for‐age z‐score; WHZ: weight‐for‐height z‐score.

Figures and Tables -
Table 14. Income‐generation interventions – results of included trials
Table 15. Income‐generation interventions – results of included prospective controlled studies

Study ID (risk of bias)

Study design (n)

Income‐generation interventions

No intervention

Effect measure (time point)

Effect of combined groups/calculated effect

Effect directiona

Meta‐analysis (yes/no)

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

3.2: Proportion of HH expenditure on food

3.2.1 Outcome measure: proportion of HH expenditure on food

Kennedy 1989 (?)

Prospective controlled study (378 HHs)

(2 years)

N/A

Alaofe 2016 (?)

Prospective controlled study (56 HHs)

(1 year)

3.3: Proportion of HHs who were food secure

3.3.1 Food security

3.3.1.1 Outcome measure: proportion experiencing food security (0 months with insufficient food in past 12 months)/ Doocy: based on HFIAS

Weinhardt 2017 (?)

Prospective controlled study (827 participants)

165/564 (29.3%)

309/564 (54.8%)

564

71/262 (27.1%)

117/263 (44.5%)

263

OR 1.36, 95% CI 0.93 to 1.97, P = 0.108 (1.5 years)

N/A no effect measure for Doocy

165/564 (29.3%)

36 months: 308/531 (58.0%)

71/262 (27.1%)

129/245 (52.7%)

OR 1.12, 95% CI 0.75 to 1.67, P = 0.585 (3 years)

Doocy 2017 – FFS (‐)

Prospective controlled study (571 HHs)

1.90%

27.80%

317 HHs

0.40%

14.60%

254 HHs

— (3.5 years)

Doocy 2017– WEG (‐)

Prospective controlled study (548 HHs)

0,3%

29.9%

0.4%

14.6%

— (3.5 years)

3.3.1.2 Proportion experiencing food deficit always

Asadullah 2015 (‐)

Prospective controlled study (4038 HHs)

60.1

15.3

2098

41.91

28.87

1940

pp –28.85, P < 0.01 (3 years)

60.1

21.02

41.91

28.45

pp –17.15, P < 0.01 (6 years)

60.1

42.9

41.91

44.38

pp –13.91, P < 0.01 (9 years)

3.3.1.3 Outcome measure: HFIAS (mean, SD or SE)

Doocy 2017 – FFS (‐)

Prospective controlled study (571 HHs)

14.4 (4.6)

5.7 (5.1)

317

14.8 (5.3)

10.1 (6.1)

254

MD –4.6, 95% CI –5.0 to –4.2, P < 0.001 (3.5 years)

MD –4.23, 95% CI –4.96 to –3.49

No

Doocy 2017 – WEG (‐)

Prospective controlled study (548 HHs)

15.3 (5.3)

6.3 (5.5)

294

14.8 (5.3)

10.1 (6.1)

254

MD –3.85, 95% CI –4.26 to –3.43, P < 0.01 (3.5 years)

Kangmennaang 2017 (‐)

Prospective controlled study (1000 HHs)

1.255 (0.029)

1.173 (0.033)

571

1.136 (0.044)

1.359 (0.071)

429

MD –0.304, SE 0.095, P < 0.01 (about 2 years)

3.3.1.4 Outcome measure: proportion of HHs improving a HFIAS category (95% CI)

Doocy 2017 – FFS (‐)

Prospective controlled study (571 HHs)

55.3 (48.8 to 61.9)

317

32.4 (24.6 to 40.3)

254

MD 22.9, 95% CI 12.7 to 33.1, P < 0.001 (3.5 years)

MD pp 24.21, 95% CI 16.67 to 31.76

N/A

Doocy 2017 – WEG (‐)

Prospective controlled study (548 HHs)

59.5

294

31.5

254

MD 25.8, 95% CI 14.6 to 36.9, P < 0.001 (3.5 years)

3.3.2 Dietary diversity

3.3.2.1 Outcome measure: probability weighted DDS (mean, SD)

Jodlowski 2016 (+)

Prospective controlled study (283 HHs)

105 HHs

178 HHs

MD –0.123, 95% CI –0.43 to 0.18, P > 0.1 (18 months)

3.3.2.2 Outcome measure: HDDS (mean, SD)

Jodlowski 2016 (+)

Prospective controlled study (283 HHs)

5.86 (1.848)

105 HHs

5.747 (1.774)

178 HHs

MD 0.267, 95% CI –0.13 to 0.66, P > 0.1 (18 months)

Yes. (Doocy groups combined)

Alaofe 2019b (?)

Prospective controlled study (423 HHs)

6.07 (1.26)

6.50 (1.23)

282

6.05 (1.26)

6.24 (1.24)

214

MD 0.94, SE 0.24, 95% CI 0.4696 to 1.4104, P < 0.01 (1 year)

Doocy 2017 – FFS (‐)

Prospective controlled study (571 HHs)

3.4 (1.4)

3.4 (1.5)

317

3.4 (1.5)

4.8 (2.1)

254

MD 0.9, 95% CI 0.5 to 1.3, P < 0.001 (3.5 year)

MD 0.80, 95% CI 0.51 to 1.09

Doocy 2017 – WEG (‐)

Prospective controlled study (548 HHs)

3.4 (1.7)

5.5 (2.2)

294

3.4 (1.5)

4.8 (2.1)

254

MD 0.69, 95% CI 0.27 to 1.10, P = 0.001 (3.5 year)

3.3.2.3 Outcome measure: Women's Household Dietary Diversity Score (WDDS‐10) (mean, SD)

Alaofe 2019b (?)

Prospective controlled study (430 women)

4.58 (1.04)

4.91 (0.97)

286

4.83 (0.97)

4.01 (1.12)

220

MD 0.83, SE 0.19, P < 0.01, 95% CI 0.46 to 1.20 (1 year)

3.3.2.4 Outcome measure: proportion achieving target dietary diversity at endline according to HDDS

Doocy 2017 – FFS (‐)

Prospective controlled study (571 HHs)

21.3

69.7

317

18.1

67.6

254

MD 21.7, 95% CI 12.3 to 31.1, P < 0.001 (3.5 year)

MD 17.03, 95% CI 7.81 to 26.24

N/A

Doocy 2017 – WEG (‐)

Prospective controlled study (548 HHs)

18.7

62.2

294

18.1

67.6

254

MD 12.3, 95% CI 2.8 to 21.8, P = 0.011 (3.5 years)

3.4 Change in adequacy of dietary intake

3.4.1 Outcome measure: percentage of calorie‐deficient HHs (< 80% of caloric requirement/adult equivalent)

Kennedy 1989 (?)

Prospective controlled study (374 HHs)

30.7

28.1

30

28.7

(2 years)

3.4.2 Outcome measure: percentage of preschool‐aged children meeting caloric requirements

Kennedy 1989 (?)

Prospective controlled study (1297 children)

69

66

58

62

(2 years)

Secondary outcomes

3.5Change in anthropometric indicators

3.5.1 Stunting

3.5.1.1 Outcome measure: HAZ (mean, SD or SE)

Kennedy 1989 (?)

Prospective controlled study (746 children)

–1.34

–1.67

–1.50

–1.76

NR

(2 years)

3.5.1.2 Outcome measure: proportion stunted (HAZ <2SD) (CI)

Kennedy 1989 (?)

Prospective controlled study (222 children)

25.3

94

25.7

128

NR (2 years)

N/A

Doocy 2017 – FFS (‐)

Prospective controlled study (471 children)

60.2 (50.8 to 69.6)

265

58.8 (50.1 to 67.5)

206

(adjusted) MD 1.4, 95% CI –10.7 to 13.6, P = 0.81 (3.5 year)

3.5.2:Wasting

3.5.2.1 Outcome measure: WHZ (mean, SD or SE)

Kennedy 1989 (?)

Prospective controlled study (651 children)

–0.22

–0.15

–0.31

–0.04

NR (2 years)

3.5.2.2 Outcome measure: proportion wasted (WHZ < –2SD)

Kennedy 1989 (?)

Prospective controlled study (118 children)

13.0

48

14.1

70

NR (2 years)

3.5.3 Underweight

3.5.3.1 Outcome measure: WAZ (mean, SD or SE)

Kennedy 1989 (?)

Prospective controlled study (198 children)

–1.03

–1.14

–1.17

–1.10

NR (2 years)

3.5.3.2 Outcome measure: percentage underweight (WAZ < 80% standard/ < –2SD) (includes severe underweight)

Kennedy 1989 (?)

Prospective controlled study (198 children)

19.7

74

24.1

124

NR (2 years)

No. Subset. Except Kennedy – effect could not be calculated.

Weinhardt 2017 (?)

Prospective controlled study (509 children)

14.8%

16.8%

322

22.5%

19.8%

187

OR 1.52, 95% CI 0.80 to 2.90, P = 0.205 (1.5 years)

Prospective controlled study (538 children)

14.8%

18.6%

344

22.5%

24.2%

194

OR 1.27, 95% CI 0.54 to 3.01, P = 0.585 (3 years)

Doocy 2017 – FFS (‐)

Prospective controlled study (471 children)

22.3 (14.8 to 29.8)

265

29.8 (22.0 to 37.7)

206

(adjusted)

MD –7.6, CI –17.7 to 2.5, P = 0.13 (3.5 year)

3.5.3.3 Outcome measure: BMI (kg/m2) (mean, SD or SE)

Kennedy 1989 (?)

Prospective controlled study (753 women)

22.3

22.2

NR (2 years)

No. No effect estimate for Kennedy and variance estimate cannot be calculated for Asadullah (missing group sizes)

Alaofe 2019b (?)

Prospective controlled study (359 women)

23.01 (2.94)

22.95 (3.73)

256

22.03 (3.14)

21.69 (3.24)

167

MD 0.43, SE 0.24, 95% CI –0.0504 to 0.8904, P < 0.1 (1 year)

Asadullah 2015 (‐)

Prospective controlled study (3547 women)

19.0

18.95

19.17

18.98

MD 0.14, P = 0.29

3.5.3.4 Proportion of women who were underweight (BMI < 18.5 kg/m2)

Alaofe 2019b (?)

Prospective controlled study (359 women)

4.88

3.10

256

6.57

14.08

167

MD –0.22, SE 0.27, 95% CI –0.749 to 0.309, P > 0.1 (1 year)

3.5.3.5 Outcome measure: mid‐upper arm circumference (mean, SD)

Katz 2001 (‐)

Prospective controlled study (718 women)

22.8 (2.0)

335

23.0 (2.2)

383

MD in intervention group –0.20 cm

MD in control group –0.25 cm,

P = 0.67 (2 years)

3.6 Change in biochemical indicators

3.6.1 Proportion with iron deficiency

Alaofe 2019b (?)

Prospective controlled study (68 women)

15.3%

13.5%

17.9%

12.8%

DID –0.11, SE 0.83, 95% CI –0.94 to 0.72, P > 0.05 (1 year)

3.6.2 Proportion with vitamin A deficiency

Alaofe 2019b (?)

Prospective controlled study (60 women)

14.3%

5.8%

20.2%

10.8%

DID 0.54, SE 0.95, 95% CI –0.41 to 1.49, P > 0.05 (1 year)

3.9 Morbidity

3.9.1 Outcome measure: proportion seriously ill in past year

Asadullah 2015 (‐)

Prospective controlled study (4038 HHs)

23.38%

15.89

24.24

17.17

pp –1.72, P > 0.1 (3 years)

23.38

12.93

24.24

12.53

pp –0.78, P > 0.1 (6 years)

23.38

22.16

24.24

22.37

pp –0.70, P > 0.1 (9 years)

3.9.2 Outcome measure: % time ill

Kennedy 1989 (?)

Prospective controlled study (1055 children)

29.8

31.2

NR (2 years)

Prospective controlled study (420 women)

23.8

24.3

NR (2 years)

3.9.3 Outcome measure: % time ill with diarrhoea

Kennedy 1989 (?)

Prospective controlled study (1055 children)

4.6

4.0

NR (2 years)

3.9.4 Prevalence of anaemia (women)

Alaofe 2019b (?)

Prospective controlled study (126 women)

49.3%

36.9%

49%

53.2%

MD –1.25, SE 0.58, 95% CI –1.83 to –0.67, P < 0.05 (1 year)

3.9.5 Prevalence of iron‐deficiency anaemia (women)

Alaofe 2019b (?)

Prospective controlled study (564 women)

6.6%

4.2%

13.8%

8.4%

MD –0.99, SE 1.40, 95% CI –2.39 to 0.41, P > 0.05 (1 year)

aEach triangle represents one study.
bThis study also has a component comparing the intervention plus a working group versus a comparison group with a working group. Results are not presented here.

(+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias; = Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0.

CI: confidence interval; DDS: Dietary Diversity Score; DID: difference in differences; FFS: Farmer Field School; HAZ: height‐for‐age z‐score; HDDS: Household Dietary Diversity Score; HFIAS: Household Food Insecurity Scale; HH: household; MD: mean difference; N/A: not applicable/available; NR: not reported; OR: odds ratio; PCS: prospective controlled study; SD: standard deviation; SE: standard error; WAZ: weight‐for‐age z‐score; WEG: Women Empowerment Group; WHZ: weight‐for‐height z‐score.

Figures and Tables -
Table 15. Income‐generation interventions – results of included prospective controlled studies
Table 16. Food vouchers, subsidies, social support: overview of included studies

Study ID (country)

Study design

Overall risk of biasa

Other key details of intervention

Population (sample size at baseline: intervention/ control)

Outcome domains and measures with available data

Timepoint of measurement

Comparison 4: food vouchers

Fenn 2015

(Pakistan)

cRCT

Low

Programme name: REFANI Pakistan

Intervention description and frequency: 3 intervention groups all disbursed at the same time every month for 6 consecutive months:

  • Unconditional transfer (see OSIS Table comparison 1);

  • Unconditional transfer (see OSIS table comparison 2) and

  • Fresh food vouchers with a cash value of PKR 1500 (approximately USD 14), which could be exchanged for specified fresh foods (fruits, vegetables, milk and meat) in nominated shops.

Provider: Action Against Hunger field staff

Delivery: food vouchers disbursed monthly at distribution points. Verbal messaging from Action Against Hunger field staff at distribution that children should benefit from the transfers.

Co‐interventions: WINS programme in all villages provided outpatient treatment for children aged 6 (SD 59) months with SAM, micronutrient supplementation (children, pregnant and lactating women), and behaviour change communication.

Poor and very poor HHs in agrarian district

(food voucher intervention/control: 632/632 HHs)

Anthropometric indicators:

  • Wasting (WHZ < –2SD)

  • Severe wasting (WHZ < –3SD)

  • WHZ

  • Stunting (HAZ < –2SD)

  • Severe stunting (HAZ < –3SD)

  • HAZ

  • MUAC

  • BMI

Biochemical indicators:

  • Hb

Morbidity:

  • ARI

  • Diarrhoea

  • Anaemia

6 and 12 months

Jensen 2011

(China)

RCT

Unclear

Programme name: N/A

Intervention description and frequency: 1‐month supply of vouchers entitling HHs to a price reduction of CNY 0.10, CNY 0.20 or CNY 0.30 (Rmb; 1 Rmb = USD 0.13) off the price of 1 jin (1 jin = 500 g) of the local staple (rice or wheat flour) to the value of 750 g per person per day.

Provider: employees of the provincial‐level agencies of the Chinese National Bureau of Statistics.

Delivery: printed vouchers redeemed by HHs at local grain shops. Shop owners reimbursed for the cost of the vouchers and given a fixed payment for complying with implementation guidelines. Re‐sale of vouchers or goods purchased with vouchers not permitted.

Co‐interventions: NR

Poor urban HHs (969/324)

Adequacy of dietary intake

  • Mineral Sufficiency index

  • Vitamin Sufficiency index

6–7 months

Hidrobo 2014

(Ecuador)

cRCT

High

Programme name: N/A

Intervention description and frequency: included a CCT group (see OSIS table comparison 2) and a food voucher group. Value of USD 40 per month per HH, given in denominations of USD 20. Participants were required to attend monthly nutrition sensitisation training sessions by HH members.

Provider: World Food Programme (NPO)

Delivery: printed serialised vouchers redeemed at central supermarkets in urban centres for a list of nutritionally approved foods, within 30 days of receipt.

Co‐interventions: NR

Poor urban HHs (2087 HHs)

Dietary diversity:

  • DDI;

  • HDDS;

  • FCS

7 months

Ponce 2017

(Ecuador)

cRCT

High

Programme name: N/A

Intervention description and frequency: 2 intervention groups:

  • HHs received a food voucher of USD 40 monthly;

  • HHs received a food voucher of USD 40 monthly + monthly training sessions on topics that included malnutrition, food preparation, children's health, mother's health, women's rights and women's empowerment.

Provider: NR

Delivery: NR

Co‐interventions: NR

HHs based in 3 provinces in Ecuador (food voucher only group/food voucher + training on health and nutrition/control: 171/401/201 HHs)

Dietary diversity:

  • FCS

12 months

Comparison 5: food and nutrition subsidies

Chen 2019

(China)

cRCT

High

Programme name: N/A

Intervention description and frequency: Schools in 2 intervention groups received a one‐off nutrition subsidy with a monetary equivalent of CNY 225 (USD 33) per enrolled student. Schools could use these for nutrition‐related expenses, e.g. buying food. Schoolmasters received information about the proportion of enrolled students who were anaemic, elective methods for reducing iron‐deficient anaemia, and details about anaemia's relation with school attendance, educational performance, and cognitive development. Schoolmasters in treatment group 1 were given a general policy target of 'malnutrition reduction' and in treatment group 2 a specific policy target of 'anaemia reduction', with a potential monetary bonus tied to a reduction in anaemia prevalence (CNY 150/USD 22 per student whose anaemia status changed).

Provider: project team and local government

Delivery: CNY 225 (equivalent to USD 33) per student was transferred into the school's bank account. Incentive payment for treatment group 2 was only calculated and transferred after the intervention period.

Co‐interventions: NR

Primary schools in rural areas (nutritional subsidy only/nutritional subsidy + monetary incentive/control: 15/15/29 schools)

Dietary diversity:

  • Dietary Diversity Score

Anthropometric indicators:

  • BMIZ

  • Underweight

Biochemical indicators:

  • Hb

Morbidity:

  • Anaemia

6 months

Andaleeb 2016

(India)

Prospective controlled study

High

Programme name: PDS

Intervention description and frequency: universal access to the PDS. All HHs that possess a ration card were eligible for 25 kg of subsidised rice, whether they are the poorest of the poor, below the poverty line or above the poverty line.

Provider: state government

Delivery: a ration card was a document issued by the government which entitled an individual/family to purchase from the PDS. Ration cards classified HHs based upon their poverty status and were also used as an identity card to avail many of the other government schemes.

Co‐interventions: other government schemes (not specified)

Rural HHs (3819 HHs)

Adequacy of dietary intake

  • Ratio of nutrient intake to RDA

7 years

Chakrabarti 2018

(India)

Prospective controlled study

High

Programme name: PDS

Intervention description and frequency: subsidising a variety of pulses in different districts as part of the PDS, in addition to the usual subsidising of rice, wheat, sugar and kerosene oil.

Provider: state governments (subsiding of pulses) and central Indian government (subsiding of rice, wheat, sugar and kerosene).

Delivery: government‐issued ration cards are given to poor HHs enabling them to purchase from the PDS.

Co‐interventions: NR

Rural and urban HHs in selected states (23,558/101,086 HHs)

No relevant outcome measures reported

5 years

Sturm 2013

(South Africa)

Prospective controlled study

High

Programme name: HealthyFood Program

Intervention description and frequency: provided a rebate of up to 25% on healthy food purchases in > 400 designated supermarkets across South Africa, for members of the private Discovery Health Insurance and their Vitality programme.

Provider: Discovery Health Insurance company in collaboration with Pick n Pay (brand) supermarkets.

Delivery: members had specific Discovery credit cards that they use for shopping. Scanner data from pay points available every time the card was swiped when purchasing certain healthy food items at Pick n Pay supermarket. These data were collated monthly.

Co‐interventions: NR

169,485 Discovery Vitality members who shopped at Pick n Pay supermarkets with linkable purchasing data (100,344 activated participants and 69,141 non‐participants, i.e. who were not actively using their benefits.)

Proportion of HH expenditure on food

  • Ratio of healthy to total food expenditure: for 10%/25% rebate group compared to control

Maximum 28 months (period November 2009 to March 2012)

Comparison 6: Social support interventions

Kusuma 2017b

(Indonesia)

cRCT

Unclear

Programme name: Generasi

Intervention description and frequency: block payments to villages of USD 8500 (2007) and USD 18200 (2009) per village.

Provider: government

Delivery: trained facilitators advised village management team on allocation of funds (41% villages implemented financial incentives for health worker outreach, 79% villages implemented SFP, and 96% villages implemented financial assistance for mothers)

Co‐interventions: NR

Rural HHs 1481 children aged 24–36 months

Anthropometric indicators:

  • Stunting (HAZ < –2SD)

  • Severe stunting (HAZ < –3SD)

  • Wasting (WHZ < –2SD)

  • Severe wasting (WHZ < –3SD)

  • Underweight (WAZ < –2SD)

  • Severe underweight (WAZ < –3SD)

1 year

Brunie 2014

(Mozambique)

Prospective controlled study

High

Programme name: VSL or a combination of VSL and Ajuda Mútua.

Intervention description and frequency: VSLs are self‐managed and capitalised microfinance programmes where members pool savings and can borrow from the pool and repay with interest. Programmes work in cycles which terminate in paying out the accumulated savings and interest to members proportional to their initial deposit. The Ajuda Mútua rotating labour scheme operates with groups of HHs working together on each family's land or enterprise on a rotational basis.

Provider: Save the Children (NGO)

Delivery: NR

Co‐interventions: SANA (Segurança Alimentar de Nutrição e Agricultura) – food security through nutrition and agriculture multiyear assistance programme targeting aspects of food utilisation. Communities are mobilised to adopt good nutrition practices, and pregnant women and carers are taught to prevent malnutrition in young children.

Interested HHs in randomised district (VSL: 395; VSL+Ajuda Mútua: 401; control: 480)

Food security:

  • Self‐reported months of food sufficiency in previous year

Dietary diversity:

  • HDDS

  • IDDS

Anthropometric indicators:

  • WAZ

3 years

aOverall risk of bias based on key domains: selection and attrition bias. If any of these were high, overall risk of bias was considered high.

ARI: acute respiratory infection; BMI: body mass index; BMIZ: body mass index‐for‐age z‐score; CCT: conditional cash transfer; CNY: Chinese yuan; cRCT: cluster randomised controlled trial; DDI: Dietary Diversity Index; FCS: Food Consumption Score; HAZ: height‐for‐age z‐score; Hb: haemoglobin; HDDS: Household Dietary Diversity Score; HH: household; IDDS: Individual Dietary Diversity Score; MUAC: mid‐upper arm circumference; N/A: not applicable/available; NPO: non‐profit organisation; NR: not reported; PDS: Public Distribution System; PKR: Pakistani rupee; RCT: randomised controlled trial; RDA: recommended daily allowance; SAM: severe acute malnutrition; SD: standard deviation; SFP: Supplementary Feeding Programme; VSL: village savings and loan; WAZ: weight‐for‐age z‐score; WINS: Women and Children/Infants Improved Nutrition in Sindh; WHZ: weight‐for‐height z‐score.

Figures and Tables -
Table 16. Food vouchers, subsidies, social support: overview of included studies
Table 17. Food vouchers – results of included trials

Study ID (risk of bias)

Study design (n)

Food vouchers

No intervention

Effect measure (time point)

Effect direction a

Meta‐analysis

Notes

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

4.3 Proportion of HHs who were food secure

4.3.1 Dietary diversity

4.3.1.1 Outcome measure: Food Consumption Score (mean): different scales (out of 112 and 8)

Hidrobo 2014 (‐)

cRCT (2087 HHs)

59.75

59.05

Coefficient 9.40, 95% CI 6.6 to 12.2, P < 0.01 (7 months)

No. SMD needed as scales are different. SMD could not be calculated due to missing group sizes for Hidrobo – MV to email authors.

SE calculated from CI

Ponce 2017 food voucher alone (‐)

cRCT (372 HHs)

5.96

NR

171 HHs

5.89

NR

201 HHs

Coefficient 0.394, SE 0.05, 95% CI 0.296 to 0.492, P < 0.01 (1 year)

SE available

Ponce 2017 food voucher + education (‐)

cRCT (602 HHs)

5.83

NR

401 HHs

5.89

NR

201 HHs

Coefficient 0.291, SE 0.081, P < 0.01 (1 year)

SE available

Secondary outcomes

4.4 Change in adequacy of dietary intake

4.4.1 Outcome measure: Mineral Sufficiency Index (mean, SD)

Jensen 2011 (?)

RCT (1265 HHs)

1.02 (0.36)

969

1.00 (0.34)

% change –0.061, 95% CI –0.219 to 0.098 (5 months)

4.4.2 Outcome measure: Vitamin Sufficiency Index (mean, SD)

Jensen 2011 (?)

RCT (1265 HHs)

1.2 (0.44)

1.17 (0.38)

% change –0.051, 95% CI –0.218 to 0.116 (5 months)

4.5 Change in anthropometric indicators

4.5.1 Stunting

4.5.1.1 Outcome measure: % stunted (HAZ < –2SD), n (%)

Fenn 2015 (+)

cRCT (1643 children)

473 (54.9)

NR

834 children

437 (51.7)

NR

809 children

OR 0.41, 95% CI 0.25 to 0.67, P < 0.001 (6 months)

Fenn 2015 (+)

cRCT (1633 children)

473 (54.9)

NR

818 children

437 (51.7)

NR

815 children

OR 0.48, 95% CI 0.31 to 0.73, P = 0.001 (12 months)

4.5.1.2 Outcome measure: % severely stunted (HAZ < –3SD)

Fenn 2015 (+)

cRCT (1643 children)

NR

NR

834 children

NR

NR

809 children

OR 0.38, 95% CI 0.23 to 0.63, P < 0.001 (6 months)

Fenn 2015 (+)

cRCT (1633 children)

NR

NR

818 children

NR

NR

815 children

OR 0.51, 95% CI 0.33 to 0.79, P = 0.003 (12 months)

4.5.1.3 Outcome measure: HAZ, mean (SD)

Fenn 2015 (+)

cRCT (1643 children)

–2.12 (1.69)

NR

834 children

–1.97 (1.75)

NR

809 children

Beta‐coefficient 0.27, 95% CI 0.19 to 0.34, P < 0.001 (6 months)

Fenn 2015 (+)

cRCT (1633 children)

–2.12 (1.69)

NR

818 children

–1.97 (1.75)

NR

815 children

Beta‐coefficient 0.29, 95% CI 0.19 to 0.40, P < 0.001 (12 months)

4.5.2 Wasting

4.5.2.1 Outcome measure: % wasted (WHZ <2SD), n (%)

Fenn 2015 (+)

cRCT (1643 children)

165 (19.3)

NR

834 children

184 (21.9)

NR

809 children

OR 1.16, 95% CI 0.67 to 2.01, P = 0.6 (6 months)

Fenn 2015 (+)

cRCT (1633 children)

165 (19.3)

NR

818 children

184 (21.9)

NR

815 children

OR 1.17, 95% CI 0.75 to 1.82, P = 0.5 (12 months)

4.5.2.2 Outcome measure: % severely wasted (WHZ) < –3SD

Fenn 2015 (+)

cRCT (1643 children)

46 (5.4)

NR

834 children

62 (7.4)

NR

809 children

OR 1.27, 95% CI 0.45 to 3.55, P = 0.66 (6 months)

4.5.2.3 Outcome measure: WHZ, mean (SD)

Fenn 2015 (+)

cRCT (1643 children)

–1.08 (1.14)

NR

834 children

–1.15 (1.30)

NR

809 children

Beta‐coefficient 0.16, 95% CI 0.05 to 0.26, P = 0.004 (6 months)

Fenn 2015 (+)

cRCT (1633 children)

–1.08 (1.14)

NR

818 children

–1.15 (1.30)

NR

815 children

Beta‐coefficient 0.02, 95% CI –0.10 to 0.14, P = 0.79 (12 months)

4.5.3 Underweight

4.5.3.1 Outcome measure: MUAC, mean (SD)

Fenn 2015 (+)

cRCT (1643 children)

13.8 (1.2)

NR

834 children

13.5 (1.2)

NR

809 children

Beta‐coefficient –0.05, 95% CI –0.14 to 0.04, P = 0.27 (6 months)

Fenn 2015 (+)

cRCT (1204 women)

25.2 (3.2)

NR

603 mothers

24.3 (3.2)

NR

601 mothers

Beta‐coefficient –0.16, 95% CI –0.38 to 0.05, P = 0.14 (6 months)

4.5.3.2 Outcome measure: BMI, mean (SD)

Fenn 2015 (+)

cRCT (1204 women)

20.8 (18.5 ± 24.0)

NR

603 mothers

20.0 (18.1 ± 22.7)

NR

601 mothers

Beta‐coefficient 0.29, 95% CI 0.03 to 0.54, P = 0.03 (6 months)

aEach triangle represents one study.

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

CI: confidence interval; cRCT: cluster randomised controlled trial; FCS: Food Consumption Score; HAZ: height‐for‐age z‐score; HH: household; MUAC: mid‐upper arm circumference; n: number; NR: not reported; OR: odds ratio; RCT: randomised controlled trial; SD: standard deviation; SE: standard error; SMD: standardised mean difference.

Figures and Tables -
Table 17. Food vouchers – results of included trials
Table 18. Food and nutrition subsidies – results of included trials

Study ID (risk of bias)

Study design (n)

Food rebate/subsidy

No intervention

Effect measure (timepoint)

Effect direction

Meta‐analysis

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

5.3 Proportion of HHs who were food secure

5.3.1 Dietary diversity

5.3.1.1 Outcome measure: DDS for nutrition subsidy only (general target: malnutrition reduction) group vs control (mean, SD)

Chen 2019 – nutrition subsidy (‐)

DDS 0–10

cRCT (656 students)

4.75 (2.17)

5.21 (2.18)

219 students

5.33 (2.32)

4.82 (2.36)

437 students

MD 0.956, robust SE 0.255, 95% CI 0.4562 to 1.4558, P < 0.01 (6 months)

N/A

Chen 2019 – nutrition subsidy+monetary incentive (‐)

cRCT

4.65 (2.20)

5.32 (2.09)

210 students

5.33 (2.32)

4.82 (2.36)

437 students

Mean score 1.263, robust SE 0.224, P < 0.01 (6 months)

Secondary outcomes

5.5 Change in anthropometric indicators

5.5.1 Outcome measure: BMI‐for‐age z‐score (mean, SD)

Chen 2019 – nutrition subsidy (‐)

cRCT

–0.70 (0.91)

–0.71 (0.95)

219 students

–0.68 (0.94)

–0.76 (0.97)

437 students

Mean score 0.080, robust SE 0.058

No significant difference from control (6 months)

N/A

Chen 2019 – nutrition subsidy+monetary incentive (‐)

cRCT

–0.63 (0.91)

–0.60 (0.89)

210 students

–0.68 (0.94)

–0.76 (0.97)

437 students

Mean score 0.123, robust SE 0.047, P < 0.01 (6 months)

5.5.2 Outcome measure: proportion underweight (mean, SD)

Chen 2019 – nutrition subsidy (‐)

cRCT

0.07 (0.25)

0.07 (0.26)

219 students

0.08 (0.26)

0.11 (0.32)

437 students

Mean proportion –0.032, robust SE 0.024, 95% CI –0.079 to 0.015 (6 months)

N/A

Chen 2019 – nutrition subsidy+monetary incentive (‐)

cRCT

0.06 (0.24)

0.06 (0.23)

210 students

0.08 (0.26)

0.11 (0.32)

437 students

Mean proportion –0.041, robust SE 0.022, 95% CI –0.084 to 0.002 (6 months)

5.6 Change in biochemical indicators

5.6.1 Outcome measure: haemoglobin concentration in children in nutrition subsidy only (general target: malnutrition reduction) group vs control (mean, SD)

Chen 2019 – nutrition subsidy (‐)

cRCT

128.51 (12.63)

128.11 (15.86)

219 students

128.03 (12.95)

127.93 (14.86)

437 students

Mean concentration 0.512, robust SE 1.348, 95% CI –2.130 to 3.154 (6 months)

N/A

Chen 2019 – nutrition subsidy+monetary incentive (‐)

cRCT

127.84 (12.80)

130.95 (15.66)

210 students

128.03 (12.95)

127.93 (14.86)

437 students

Mean concentration 4.490, robust SE 1.241, 95% CI 2.058 to 6.922, P < 0.01 (6 months)

5.9 Morbidity

5.9.1 Outcome measure: proportion of anaemic children in nutrition subsidy only (general target: malnutrition reduction) group vs control (mean, SD)

Chen 2019 – nutrition subsidy (‐)

cRCT

0.18 (0.38)

0.22 (0.42)

219 students

22 (0.42)

0.23 (0.42)

437 students

Mean proportion –0.005, robust SE 0.048, 95% CI –0.099 to 0.089, P > 0.01 (6 months)

N/A

Chen 2019 – nutrition subsidy+monetary incentive (‐)

cRCT

0.23 (0.42)

0.16 (0.36)

210 students

0.22 (0.42)

0.23 (0.42)

437 students

Mean proportion –0.120, robust SE 0.046, 95% CI –0.210 to –0.029, P < 0.01 (6 months)

cRCT: cluster randomised controlled trial; DDS: Dietary Diversity Score; MD: mean difference; n: number; N/A: not applicable/available; SD: standard deviation; SE: standard error.

Figures and Tables -
Table 18. Food and nutrition subsidies – results of included trials
Table 19. Food and nutrition subsidies – results of included prospective controlled studies

Study ID (risk of bias)

Study design (n)

Food rebate

No intervention

Effect measure (time point)

Effect directiona

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Primary outcomes

5.2 Proportion of HH expenditure on food

5.2.1 Outcome measure: ratio of healthy to total food expenditure (mean, SD)

Sturm 2013 – 10% rebate (‐)

Prospective controlled study (169,485 HHs)

0.21 (0.11)

67,343 HHs

0.17 (0.13)

69,141 HHs

Increase by 6.0%, 95% CI 5.3% to 6.8% (3 years)

Sturm 2013 – 25% rebate (‐)

Prospective controlled study (136,484 HHs)

0.21 (0.12)

0.17 (0.13)

Increase by 9.3%, 95% CI 8.5% to 10.0% (2 years and 4 months)

5.4 Change in adequacy of dietary intake

5.4.1 Outcome measure: ratio of current caloric intake to the RDA (multiplied by 100)

Andaleeb 2016 (‐)

Controlled before‐after study

NR

NR

1134 HHs

NR

NR

NR

DID estimate 2.55, SE 1.31, 95% CI –0.018 to 5.118, P < 0.1 (7 years)

5.4.2 Outcome measure: ratio of current protein intake to the RDA (multiplied by 100)

Andaleeb 2016 (‐)

Controlled before‐after study

NR

NR

1134 HHs

NR

NR

NR

DID estimate 3.75, SE 1.65, 95% CI 0.516 to 6.984, P < 0.05 (7 years)

5.4.3 Outcome measure: ratio of current fat intake to the RDA (multiplied by 100)

Andaleeb 2016 (‐)

Controlled before‐after study

NR

NR

1134 HHs

NR

NR

NR

DID estimate –0.1, SE 0.00, P > 0.1 (7 years)

aEach triangle represents one study.

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

CI: confidence interval; DID: difference in differences; HH: household; n: number; NR: not reported; RDA: recommended daily allowance; SD: standard deviation; SE: standard error.

Figures and Tables -
Table 19. Food and nutrition subsidies – results of included prospective controlled studies
Table 20. Social support interventions – results of included trials

Study ID (risk of bias)

Study design (n)

Village savings/grants

No intervention

Effect measure (time point)a

Effect direction

Results at baseline

Results at follow‐up

n

Results at baseline

Results at follow‐up

n

Secondary outcomes

6.5 Change in anthropometric indicators

6.5.1 Stunting

6.5.1.1 Outcome measure: proportion stunted (HAZ < –2SD)

Kusuma 2017b (?)

cRCT (1481 children aged 24–36 months)

Mean 0.48

DID 0.034, SE 0.055, 95% CI –0.074 to 0.142, P > 0.05 (2 years)

1.1.2 Outcome measure: proportion severely stunted (HAZ < –3SD)

Kusuma 2017b (?)

cRCT (1481 children aged 24–36 months)

Mean 0.29

DID –0.06, SE 0.053, 95% CI –0.164 to 0.044, P > 0.05 (2 years)

6.5.2 Wasting

6.5.2.1 Outcome measure: proportion wasted (WHZ < –2SD)

Kusuma 2017b (?)

cRCT (1481 children aged 24–36 months)

Mean 0.19

DID –0.010, SE 0.035, 95% CI –0.079 to 0.059

pp –1.0, 95% CI –7.86 to 5.86, P > 0.05 (2 years)

6.5.2.2 Outcome measure: proportion severely wasted (WHZ < –3SD)

Kusuma 2017b (?)

cRCT (1481 children aged 24–36 months)

Mean 0.10

DID –0.021, SE 0.025, 95% CI –0.07 to 0.028, P > 0.05 (2 years)

6.5.3 Underweight

6.5.3.1 Outcome measure: proportion underweight (WAZ < –2SD)

Kusuma 2017b (?)

cRCT (1481 children aged 24–36 months)

Mean 0.34

Beta –0.020, SE 0.051, 95% CI –0.120 to 0.080, P > 0.05 (2 years)

6.5.3.2 Outcome measure: proportion severely underweight (WAZ < –3SD)

Kusuma 2017b (?)

cRCT (1481 children aged 24–36 months)

Mean 0.12

Beta –0.056, SE 0.034, 95% CI –0.123 to 0.011, P < 0.1 (2 years)

aEach triangle represents one study.

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

DID: difference in differences; HAZ: height‐for‐age z‐score; n: number; pp: percentage point; SD: standard deviation; SE: standard error; WAZ: weight‐for‐age z‐score; WHZ: weight‐for‐height z‐score.

Figures and Tables -
Table 20. Social support interventions – results of included trials
Table 21. Social support interventions – results of included prospective controlled studies

Study ID (risk of bias)

Study design (n)

Village savings/grants

No intervention

Effect measure (time point)a

Combined group effect

Effect direction

Meta‐analysis

Results

at baseline

Results

at follow‐up

n

Results

at baseline

Results

at follow‐up

n

Primary outcomes

6.3 Proportion of HH who were food secure

6.3.1 Food security

6.3.1.1 Outcome measure: self‐reported months of food sufficiency in previous year (mean, SD)

Brunie 2014 – VSL (‐)

Prospective controlled study (851 HHs)

10.41

10.52

10.58

10.21

DID estimate 0.47, 95% CI –0.04 to 0.98, P < 0.1 (3 years)

MD 1.25, 95% CI –0.28 to 2.79

N/A

Brunie 2014 – VSL+AM (‐)

836 HHs

9.27

11.18

10.47

10.35

DID estimate 2.04, 95% CI 1.53 to 2.55, P < 0.1 (3 years)

6.3.2 Dietary diversity

6.3.2.1 Outcome measure: HDDS (mean, SD)

Brunie 2014 – VSL (‐)

Prospective controlled study (802 HHs)

4.06

5.44

3.73

4.84

DID estimate 0.27, 95% CI –0.16 to 0.70, P > 0.1 (3 years)

MD –0.30, 95% CI –1.46 to 0.87

N/A

Brunie 2014 – VSL+AM (‐)

813 HHs

4.2

4.56

3.82

5.11

DID estimate −0.92, 95% CI –1.567 to –0.273, P < 0.001

6.3.2.2 Outcome measure: IDDS (mean, SD)

Brunie 2014 – VSL (‐)

Prospective controlled study (542 children)

2.51

3.43

2.87

2.97

DID estimate 0.81, 95% CI 0.36 to 1.26, P < 0.01 (3 years)

MD 0.52, 95% CI –0.18 to 1.23

N/A

Brunie 2014 – VSL+AM (‐)

(579 children)

2.99

3.46

2.82

3.22

DID estimate 0.07, 95% CI –0.7532 to 0.8932, P > 0.01 (3 years)

Secondary outcomes

6.5 Change in anthropometric indicators

6.5.1 Outcome measure: weight‐for‐age z‐scores (WAZ)

Brunie 2014 – VSL (‐)

Prospective controlled study (503 children)

–1.21

–0.91

–1.25

–0.83

DID estimate –0.11, 95% CI –0.561 to 0.341, P > 0.1 (3 years)

MD 0.05, 95% CI –0.37 to 0.48

N/A

Brunie 2014 – VSL+AM (‐)

(550 children)

–0.96

–0.93

–1.15

–0.78

DID estimate 0.34, 95% CI –0.31 to 0.99, P > 0.01

aEach triangle represents one study.

= Favours the intervention, 95% CI excludes 0; △ = Unclear effect potentially favouring the intervention, 95% CI includes zero; ▼ = Favours the control, 95% CI excludes 0; ▽ = Unclear effect potentially favouring the control, 95% CI includes 0. (+): low overall risk of bias; (?): unclear overall risk of bias; (‐): high overall risk of bias.

AM: Ajuda Mútua; CI: confidence interval; DID: difference in differences; HDDS: Household Dietary Diversity Score; HH: household; IDDS: Individual Dietary Diversity Score; MD: mean difference; n: number; N/A: not applicable/available; SD: standard deviation; VSL: village savings and loan; WAZ: weight‐for‐age z‐score.

Figures and Tables -
Table 21. Social support interventions – results of included prospective controlled studies
Comparison 1. Unconditional cash transfers (UCT) versus no intervention

Outcome or subgroup title

No. of studies

No. of participants

Statistical method

Effect size

1.1 Proportion of household expenditure on food Show forest plot

3

Mean Difference (IV, Random, 95% CI)

4.24 [‐2.88, 11.36]

1.2 Proportion consuming > 1 meal/day Show forest plot

2

Mean Difference (IV, Random, 95% CI)

Subtotals only

1.3 Food security scores Show forest plot

3

Std. Mean Difference (IV, Random, 95% CI)

0.18 [0.13, 0.23]

1.4 Dietary Diversity Score including composite food consumption score (FCS) (weighted) Show forest plot

3

Std. Mean Difference (IV, Random, 95% CI)

Subtotals only

1.5 Proportion with minimum dietary diversity Show forest plot

2

Std. Mean Difference (IV, Random, 95% CI)

Subtotals only

1.6 Proportion of food poverty (per capita daily caloric intake < 2122 calories) Show forest plot

2

Mean Difference (IV, Random, 95% CI)

‐4.64 [‐9.34, 0.06]

1.7 Proportion stunted (height‐for‐age z‐score (HAZ) < ‐2SD) Show forest plot

2

Odds Ratio (IV, Random, 95% CI)

0.62 [0.46, 0.84]

1.8 HAZ Show forest plot

7

Mean Difference (IV, Random, 95% CI)

Subtotals only

1.8.1 Change in z‐scores

6

Mean Difference (IV, Random, 95% CI)

0.07 [‐0.04, 0.18]

1.8.2 Change in z‐score/month

1

Mean Difference (IV, Random, 95% CI)

‐0.00 [‐0.00, 0.00]

1.9 Weight‐for‐height z‐score (WHZ) Show forest plot

6

Mean Difference (IV, Random, 95% CI)

Subtotals only

1.9.1 Change in z‐scores

5

Mean Difference (IV, Random, 95% CI)

‐0.02 [‐0.10, 0.06]

1.9.2 Change in z‐scores/month

1

Mean Difference (IV, Random, 95% CI)

‐0.00 [‐0.01, 0.00]

1.10 Weight‐for‐age z‐score (WAZ) Show forest plot

2

Mean Difference (IV, Random, 95% CI)

‐0.04 [‐0.43, 0.35]

1.11 Haemoglobin concentration (g/dL) Show forest plot

2

Mean Difference (IV, Random, 95% CI)

‐0.06 [‐0.21, 0.09]

1.12 Depression score (CES‐D scale) Show forest plot

3

Mean Difference (IV, Random, 95% CI)

‐0.41 [‐1.31, 0.49]

1.13 Perceived Stress Scale (PSS) Show forest plot

2

Mean Difference (IV, Random, 95% CI)

‐0.15 [‐0.26, ‐0.03]

Figures and Tables -
Comparison 1. Unconditional cash transfers (UCT) versus no intervention
Comparison 2. Conditional cash transfers (CCT) versus no intervention

Outcome or subgroup title

No. of studies

No. of participants

Statistical method

Effect size

2.1 Household Dietary Diversity Score (HDDS) Show forest plot

2

Mean Difference (IV, Random, 95% CI)

0.45 [0.25, 0.65]

2.2 Proportion stunted (height‐for‐age z‐score (HAZ) < ‐2SD) – RCTs Show forest plot

3

Mean Difference (IV, Random, 95% CI)

‐2.51 [‐7.78, 2.75]

2.3 Proportion with severe stunting (HAZ < ‐3 SD) – RCTs Show forest plot

2

Mean Difference (IV, Random, 95% CI)

‐3.05 [‐17.63, 11.53]

2.4 HAZ – RCTs Show forest plot

5

Mean Difference (IV, Random, 95% CI)

0.09 [0.04, 0.15]

2.5 Proportion stunted (HAZ < ‐2 SD) – PCS Show forest plot

2

Mean Difference (IV, Random, 95% CI)

‐5.63 [‐26.59, 15.34]

2.6 HAZ – PCS Show forest plot

3

Mean Difference (IV, Random, 95% CI)

0.03 [‐0.06, 0.12]

2.7 Proportion wasted (weight‐for‐height z‐score (WHZ) < ‐2 SD) – RCTs Show forest plot

2

Mean Difference (IV, Random, 95% CI)

‐2.50 [‐8.04, 3.04]

2.8 WHZ – RCTs Show forest plot

2

Mean Difference (IV, Random, 95% CI)

0.17 [‐0.11, 0.44]

2.9 Proportion underweight (weight‐for‐age z‐score (WAZ) < ‐2SD) – RCTs Show forest plot

3

Mean Difference (IV, Random, 95% CI)

‐4.87 [‐8.65, ‐1.09]

2.10 Proportion severely underweight (WAZ < ‐3 SD) – RCTs Show forest plot

2

Mean Difference (IV, Random, 95% CI)

‐1.08 [‐4.73, 2.57]

2.11 WAZ – RCTs Show forest plot

3

Mean Difference (IV, Random, 95% CI)

0.04 [‐0.03, 0.11]

2.12 BMI‐for‐age z‐score – PCS Show forest plot

2

Mean Difference (IV, Random, 95% CI)

Subtotals only

2.13 Cognitive test scores – RCTs Show forest plot

2

Mean Difference (IV, Random, 95% CI)

0.13 [0.09, 0.18]

2.14 Proportion reporting being ill in past 4 weeks/parents seeking care for illness past 2 weeks – RCTs Show forest plot

3

Mean Difference (IV, Random, 95% CI)

‐0.28 [‐5.92, 5.35]

2.15 Overweight (BMI z‐score > 2 SD)_PCS Show forest plot

2

Odds Ratio (IV, Random, 95% CI)

1.00 [0.59, 1.71]

Figures and Tables -
Comparison 2. Conditional cash transfers (CCT) versus no intervention
Comparison 3. Income generation (IG) versus no intervention

Outcome or subgroup title

No. of studies

No. of participants

Statistical method

Effect size

3.1 HFIAS – PCS Show forest plot

2

Mean Difference (IV, Random, 95% CI)

Subtotals only

3.2 HDDS – RCTs Show forest plot

2

Std. Mean Difference (IV, Random, 95% CI)

0.02 [‐0.09, 0.13]

3.3 Minimum dietary diversity (MDD) – RCTs Show forest plot

3

Odds Ratio (IV, Random, 95% CI)

1.28 [1.11, 1.47]

3.4 HDDS – PCS Show forest plot

3

Mean Difference (IV, Random, 95% CI)

0.67 [0.29, 1.05]

3.5 Proportion stunted (HAZ < ‐2 SD) – RCTs Show forest plot

2

Odds Ratio (IV, Random, 95% CI)

1.00 [0.84, 1.19]

3.6 HAZ – RCTs Show forest plot

3

Mean Difference (IV, Random, 95% CI)

Subtotals only

3.7 Proportion wasted (WHZ < ‐2 SD) – RCTs Show forest plot

2

Odds Ratio (IV, Random, 95% CI)

1.13 [0.92, 1.40]

3.8 WHZ – RCTs Show forest plot

2

Mean Difference (IV, Random, 95% CI)

‐0.05 [‐0.25, 0.15]

3.9 Percentage underweight – RCTs Show forest plot

2

Odds Ratio (IV, Random, 95% CI)

1.06 [0.89, 1.26]

3.10 WAZ – RCTs Show forest plot

3

Mean Difference (IV, Random, 95% CI)

Subtotals only

3.11 Percentage underweight – PCS Show forest plot

2

Odds Ratio (IV, Random, 95% CI)

0.83 [0.61, 1.12]

3.12 Proportion of women underweight – RCTs Show forest plot

3

Std. Mean Difference (IV, Random, 95% CI)

Subtotals only

3.13 BMI – RCTs Show forest plot

2

Mean Difference (IV, Random, 95% CI)

‐0.02 [‐0.28, 0.25]

3.14 Haemoglobin concentration (children) – RCTs Show forest plot

2

Mean Difference (IV, Random, 95% CI)

3.49 [3.25, 3.72]

3.15 Haemoglobin concentration (women) – RCTs Show forest plot

2

Mean Difference (IV, Random, 95% CI)

Subtotals only

3.16 Prevalence of anaemia (children) – RCTs Show forest plot

2

Odds Ratio (IV, Random, 95% CI)

0.73 [0.61, 0.88]

3.17 Prevalence of anaemia (women) – RCTs Show forest plot

2

Odds Ratio (IV, Random, 95% CI)

1.06 [0.82, 1.38]

Figures and Tables -
Comparison 3. Income generation (IG) versus no intervention
Comparison 4. Food vouchers vs no intervention

Outcome or subgroup title

No. of studies

No. of participants

Statistical method

Effect size

4.1 Food consumption score Show forest plot

2

Std. Mean Difference (IV, Random, 95% CI)

Subtotals only

Figures and Tables -
Comparison 4. Food vouchers vs no intervention